Personal Information
Unternehmen/Arbeitsplatz
Nepal Nepal
Beruf
Student at ucms college of dental surgery
Tags
release after replication
protect dna from nuclease degradation
keep two strand separate
activity of helicase
3. single strand binding protein (ssbp)
unwind the supercoil
2. gyrase (topoisomerases)
to separate the strand
1. dna helicases
at the 3’ carbon of deoxyribose
a free hydroxyl group (-oh)
the phosphate group attached to the 5’ carbon of d
attached to the 1’ carbon of deoxyribose
consists of the nitrogenous base
the nucleotide structure
vit d3
also stresses & lack of exercise
high alkalinity
excessive fat
dietary fiber
phosphorous
oxalic acid
calcium-phosphorus imbalance
absorption decreased by: vitamin d deficiency
nerve transmission
secretory process
release of hormones
activation of enzymes
blood coagulation
muscle contraction
calcium
normal buffer base level
phosphate buffer systems
and
plasma protein
hemoglobin
comprising the bicarbonate
blood’s total buffer capacity
represents the
actual base excess is the concentration of titrata
aseptic meningitis.
bacterial meningitis
is also significantly higher in
lactate ld activity
lactate dehydrogenase:
brudunski’s sign
kernig’s sign
photophobia
meningism
headache
parasites pyrexia
fungi
virus
it may be caused by bacteria
it is meningoencephalitis
infection spread to brain parenchyma
leptomeninges (aracnoid-piamater) if
inflammation of
involving mainly the
infectious disease
* local atopic allergies
anaphylactic shock
* systemic life threatening
* soluble molecules cause the manifestation of dis
soluble molecules (mediators)
an antigen (allergen)
leading to release of soluble molecules
(ig e)
an antigen reacts with cell fixed antibody
* type iv is cell mediated
ii and iii are antibody mediated
* types i
type iv
type iii
type ii
type i
* there are four types of hypersensitivity reactio
* an immune response results in exaggerated reacti
or allergy
hypersensitivity
16 cell stage – app 96 hrs after fertilization
12 cell stage – app 72 hrs after fertilization
4 cell stage – app 40-50 hrs after fertilization
2 cell stage – app 30 hrs after fertilization.
appearance of bilaminar & trilaminar germ disc
blastocyst & its implantation. differentiation of
formation of morula
germinal period – 1st – 3rd week cleavage
major cause of thrombosis
and 3.hemorrhage. . vasculitis
2.embolism
(3) vascular rupture. cerebrovascular diseases in
(2) embolic occlusion of vessels
blood vessels. the three basic processes are thr
involving
pathologic process
denotes any abnormality of the brain caused by a
cerebrovascular disease
mosquito
colchicine
plasmodium
benign tertian malaria 48 hrs
benign quartan malaria : 72 hours p.ovale
malignant tertiary malaria:36-48hrs p.malariae
benign tertiary malaria : 48hrs p.falciparum
p.vivax
toxins
hyperthermia
by ionizing radiation
sublethal damage to the cells
hepatitis)
natural killer cells or cytotoxic t lymphocytes).
immune system action (e.g.
tumor necrosis factor)
direct action of cytokines (e.g.
loss of growth factors.
cell injury that damage dna
cell injury
description
apoptosis
detection
uses
epitopes
adjuvent
haptens
chemical nature of antigen
super antigen
antigenicity
immunogen
antibody
1. definition.
bioavailability
renal clearance
mechanism of drug
route
netabolism
biotransformation
adsorption
drugs
pharmacology
thyroid
pituitary gland
biological rhythm
hormone
corticosteroids
thyroid hormone
growth hormone
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Personal Information
Unternehmen/Arbeitsplatz
Nepal Nepal
Beruf
Student at ucms college of dental surgery
Tags
release after replication
protect dna from nuclease degradation
keep two strand separate
activity of helicase
3. single strand binding protein (ssbp)
unwind the supercoil
2. gyrase (topoisomerases)
to separate the strand
1. dna helicases
at the 3’ carbon of deoxyribose
a free hydroxyl group (-oh)
the phosphate group attached to the 5’ carbon of d
attached to the 1’ carbon of deoxyribose
consists of the nitrogenous base
the nucleotide structure
vit d3
also stresses & lack of exercise
high alkalinity
excessive fat
dietary fiber
phosphorous
oxalic acid
calcium-phosphorus imbalance
absorption decreased by: vitamin d deficiency
nerve transmission
secretory process
release of hormones
activation of enzymes
blood coagulation
muscle contraction
calcium
normal buffer base level
phosphate buffer systems
and
plasma protein
hemoglobin
comprising the bicarbonate
blood’s total buffer capacity
represents the
actual base excess is the concentration of titrata
aseptic meningitis.
bacterial meningitis
is also significantly higher in
lactate ld activity
lactate dehydrogenase:
brudunski’s sign
kernig’s sign
photophobia
meningism
headache
parasites pyrexia
fungi
virus
it may be caused by bacteria
it is meningoencephalitis
infection spread to brain parenchyma
leptomeninges (aracnoid-piamater) if
inflammation of
involving mainly the
infectious disease
* local atopic allergies
anaphylactic shock
* systemic life threatening
* soluble molecules cause the manifestation of dis
soluble molecules (mediators)
an antigen (allergen)
leading to release of soluble molecules
(ig e)
an antigen reacts with cell fixed antibody
* type iv is cell mediated
ii and iii are antibody mediated
* types i
type iv
type iii
type ii
type i
* there are four types of hypersensitivity reactio
* an immune response results in exaggerated reacti
or allergy
hypersensitivity
16 cell stage – app 96 hrs after fertilization
12 cell stage – app 72 hrs after fertilization
4 cell stage – app 40-50 hrs after fertilization
2 cell stage – app 30 hrs after fertilization.
appearance of bilaminar & trilaminar germ disc
blastocyst & its implantation. differentiation of
formation of morula
germinal period – 1st – 3rd week cleavage
major cause of thrombosis
and 3.hemorrhage. . vasculitis
2.embolism
(3) vascular rupture. cerebrovascular diseases in
(2) embolic occlusion of vessels
blood vessels. the three basic processes are thr
involving
pathologic process
denotes any abnormality of the brain caused by a
cerebrovascular disease
mosquito
colchicine
plasmodium
benign tertian malaria 48 hrs
benign quartan malaria : 72 hours p.ovale
malignant tertiary malaria:36-48hrs p.malariae
benign tertiary malaria : 48hrs p.falciparum
p.vivax
toxins
hyperthermia
by ionizing radiation
sublethal damage to the cells
hepatitis)
natural killer cells or cytotoxic t lymphocytes).
immune system action (e.g.
tumor necrosis factor)
direct action of cytokines (e.g.
loss of growth factors.
cell injury that damage dna
cell injury
description
apoptosis
detection
uses
epitopes
adjuvent
haptens
chemical nature of antigen
super antigen
antigenicity
immunogen
antibody
1. definition.
bioavailability
renal clearance
mechanism of drug
route
netabolism
biotransformation
adsorption
drugs
pharmacology
thyroid
pituitary gland
biological rhythm
hormone
corticosteroids
thyroid hormone
growth hormone
Mehr anzeigen