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Management of Nontraumatic,
Endodontic Emergencies
• Patient
• Staff
• Dentist
Emergency Impacts
• Pain
• Pain and swelling
• Trauma (later
lecture)
Patient Presentation
• Diagnosis
• Definitive dental treatment
• Drugs
3 D’s of
Successful Management
Diagnosis
• Determine the CC
• Take an accurate
medical history
• Complete a
thorough exam,
with all necessary
tests
• Perform a
radiographic exam
• Analyze and
synthesize results
• Establish a
treatment plan
Treatment Plan
to
REMOVE
the
ETIOLOGY
When do patients present for
emergency endodontic care?
• No prior RCT / initial infection
• After RCT initiated
• After obturation
Initial Presentation
• PAIN!
• Primary
infection
After Initiation of
Endodontic Therapy
FLARE-UP!
After Initiation
of
Endodontic Treatment
Before obturation
After Obturation
• Recent
obturation
• Non-healing
endodontic
therapy
Determine a
Pulpal
and
Periradicular
Diagnosis
• Normal pulp
• Reversible pulpitis
• Irreversible pulpitis
• Necrotic pulp
• Pulpless/
previously treated
Pulpal DiagnosisPulpal Diagnosis
• Normal periradicular
tissues
• Acute periradicular
periodontitis
• Acute periradicular
abscess
Periradicular DiagnosisPeriradicular Diagnosis
• Chronic periradicular
periodontitis
• Symptomatic
• Asymptomatic
• Chronic periradicular
abscess (suppurative
periradicular
periodontitis)
Periradicular DiagnosisPeriradicular Diagnosis
• Focal sclerosing
osteomyelitis
(condensing osteitis):
LEO
Periradicular DiagnosisPeriradicular Diagnosis
Etiology
• After listening to the patient,
begin to determine the
etiology of the chief
complaint:
• Contents of the root canal?
• Dentist controlled factors?
• Host factors?
Contents of the
Root Canal
• Pulp tissue
• Bacteria
• Bacterial by-products
• Endodontic therapy materials
Dentist
Controlled Factors
• Over-instrumentation
• Inadequate debridement
• Missed canal
• Hyper-occlusion*
• Debris extrusion
• Procedural complications*
Hyperocclusion
• Rosenberg PA, Babick PJ,
Schertzer L, Leung A. The effect
of occlusal reduction on pain after
endodontic instrumentation. J
Endodon 1998;24:492.
Hyperocclusion
• Researchers have
found that patients
most likely to benefit
from occlusal
reduction are those
whose teeth initially
present with
symptoms.
• Indiscriminant
reduction of the
occlusal surface is
not indicated
• PRE-OP PAIN
• PULP VITALITY
• PERCUSSION
SENSITIVITY
• ABSENCE OF A
PERIRADICULAR
RADIOLUCENCY
• COMBINATION OF
THESE SYMPTOMS
Procedural
Complications
• Perforation
• Separated instrument
• Zip
• Strip
• NaOCl accident
• Air emphysema
• Wrong tooth
Dentist
Controlled Factors
Dentist’s personalityDentist’s personality
Host Factors
• Allergies
• Age
• Sex
• Emotional state
Host Factors
• Complex
etiology
• Microbiologic
• Immunologic
• Inflammatory
Bacteria!
• Bacterial by-
products/
endotoxin
Host Defense is Multi-
factorial
C E L L S
n e u tro p h ils , lym p h o c yte s ,
p la s m a c e lls , m a c ro p h a g e s ,
o s te o c la s ts , e p ith e lia l c e lls , d e n d ritic c e lls
M O L E C U L A R M E D IA T O R S
c yto k in e s (IL , IF N , C S F , T G F )
e ic o s a n o id s (P G , L T )
e n z ym a tic e ffe c to r m o le c u le s
A N T IB O D IE S
im m u n o g lo b u lin s (Ig G , e tc .)
p ro d u c e d b y p la s m a c e lls
M ixe d M ic ro flo ra
• Diagnosis
• Definitive dental treatment
• Drugs
Three D’s
of
Successful Management
Emergency
Treatment
• Non-surgical
• Surgical
• Combined
• Pulpotomy
• Partial pulpectomy
• Complete pulpectomy
• Debridement of the
root canal system*
Non-surgical
Emergency Treatment
Surgical
Emergency Treatment
Incision for drainage
Trephination/apical fenestration
• Decreases number of bacteria
• Reduces tissue pressure
• Alleviates pain/trismus
• Improves circulation
• Prevents spread of infection
• Alters oxidation-reduction potential
• Accelerates healing
Rationale for I & D
Management
• Inadequate
debridement
• Debris extrusion
• Over-instrumentation
• Missed canal
• Fluctuant swelling
• Severe pain, no
swelling
Treatment
• For severe pain without visible
swelling…
• Trephination!
QUESTIONS
“Should I leave the tooth
OPEN or CLOSED?”
“Should I place an
Interappointment
Medicament?”
Ca(OH)2
“Should I prescribe
ANTIBIOTICS?”
• Diagnosis
• Definitive Dental Treatment
• Drugs
Three D’s
of
Successful Management
Remember, there is a
Complex Etiology
• Microbiologic
• Immunologic
• Inflammatory
And, not all can be
easily treated...
• Debris extrusion
• Over-instrumentation
• Over-filling
• Over-extension
Breaking
the
Use a Flexible
Analgesic
Strategy
• Pre - op / loading dose
• Long acting anesthesia
• Prescription
Drugs
Codeine
• Prototype opioid for orally available
combination drugs
• Studies found that 60 mg of codeine
(2 T-3) produces significantly more
analgesia than placebo but less
analgesia than 650 mg aspirin, or
600 mg acetaminophen
Codeine
Patients taking 30 mg of codeine
report only as much analgesia as
placebo
• 57 patients
• Local anesthesia,
pulpectomy, post- op
analgesic
• Placebo
• 600 mg ibuprofen
• 600 mg ibuprofen & 1000 mg
acetaminophen
Ibuprofen and
Acetaminophen
• Visual analogue scale &
baseline 4-point category pain
scale
• 1 hr, 4 hr, 6 hr, 8 hr
• General linear model analyses
• Significant differences
• Placebo and combination
• Ibuprofen and combination
• No significant difference
• Placebo and ibuprofen
Ibuprofen and
Acetaminophen*
“The results demonstrate
that the combination of
ibuprofen and
acetaminophen may be
more effective than
ibuprofen alone for the
management of
postoperative endodontic
pain.”
Ibuprofen and
Acetaminophen*
Analgesic Doses
Codeine 60 mg
Oxycodone 5-6
Hydrocodone 10
Dihydrocodone 60
Propoxyphene HCl
(Darvon)
102
Meperidine (Demerol) 90
Tramadol (Ultram) 50
Flexible Analgesic Plan
M I L D
2 0 0 - 4 0 0 m g ib u p r o fe n
o r 6 5 0 m g a s p ir in
M O D E R A T E
6 0 0 - 8 0 0 m g ib u p r o fe n
p lu s c o m b o a n a lg e s ic =
6 0 m g c o d e in e
S E V E R E
6 0 0 - 8 0 0 m g ib u p r o fe n
p lu s c o m b o a n a lg e s ic =
1 0 m g o x y c o d o n e
A s p ir in - lik e D r u g s a r e I n d ic a t e d
Flexible Analgesic Plan
M IL D
6 0 0 -1 0 0 0 a c e ta m in o p h e n
M O D E R A T E
6 0 0 -1 0 0 0 m g a c e ta m in o p h e n
a n d o p ia te =
6 0 m g c o d e in e
S E V E R E
1 0 0 0 m g a c e ta m in o p h e n
a n d o p ia te =
1 0 m g o x y c o d o n e
A s p irin -lik e D ru g s a re C o n tra in d ic a te d
Selected NSAID Drug
Interactions
Anticoagulants Increased prothrombin time or bleeding time
ACE Inhibitors Reduced antihypertensive effectiveness
Beta Blockers Reduced antihypertensive effects
Cyclosporine Increased risk of nephrotoxicity
Lithium Increased serum levels of lithium
Sympathomimetics Increased blood pressure
Thiazide Reduced antihypertensive effectiveness
• Systemic involvement
• Compromised host
resistance
• Fascial space involvement
• Inadequate surgical
drainage
Indications for
Antibiotic Therapy
Select antibiotic with
anaerobic spectrum
Use a larger dose for a
shorter period of time (“hard
and fast” rule)
Guidelines for
Antibiotic Therapy
• Gram stain results available:
antibiotic-sensitivity charts
• C & S results available:
antibiotic-sensitivity charts
• No gram stain or C & S results:
PCN is antibiotic of choice
Selecting the
Appropriate Antibiotic
Penicillin V
• Still, the drug of choice for
infections of endodontic origin
• Loading dose: 1-2 g then 500 mg qid
x 7-10 days
Metronidozole
(Flagyl)
• Used in conjunction with Penicillin
V
• 500 mg of Penicillin V with 250 mg
Metronidozole, qid x 7-10 days
Clindamycin
• Loading dose: 300 mg
• 150-300 mg qid x 10 days
Closely Follow All Infected Patients
Components of a
Successful Management
• Appropriate attitude of
dentist
• Proper patient management
• Accurate diagnosis
• Profound anesthesia
• Prompt and effective
treatment
Patient Instructions
• By the Clock
• NOT
• PRN
Questions ?

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Yaatraa maryaathakal
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Urakkam sharddikkenda sunnathukal
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Pravachakanmaariloodae
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Namaskarikkunnavar santhoshikkuka
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Namaskaara sheshamulla dikrukal
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Hope..
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