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CHRONIC LARYNGITIS
By
Lt Col Saeed Ullah, FCPS
Classified ENT, Head and neck surgeon
CMH Quetta
Anatomy
Calssification
 Chronic specific laryngitis
 Chronic non specific laryngitis
Chronic non specific
laryngitis
1. Chronic hyperemic
2. Chronic Hypertrophic
3. Atrophic laryngitis
4. Pachydermia (contact granuloma / contact
ulcer
Chronic hypreamic laryngitis
 Diffuse inflammatory condition involving the
whole larynx particularly true & false vocal
cords
Causes
 Recurrent acute laryngitis
 Incompletely resolved acute laryngitis
 Chronic infection in the vicinity
 Sinusitis, tonsillitis, bad orodental hygiene ,
 Ch Bronchitis
 Occupational factors
 dust & Fumes
 Smoking & Alcohol
 Voice abuse
 GERD
Clinical features
 Males affected more than females
 Hoarseness of voice
 Fatigue of voice
 Hawking & irritation larynx
 Dry irritating cough
Clinical features
 Laryngoscopy
 Hyperemia ,Vocal cords appear dull & edges are
rounded,Viscid secretions onVC / interarytnoid
region
Treatment
 EliminateThe cause
 infection / irritating factors
 Complete voice rest / Observe proper vocal
hygiene
 Medicated steam inhalation
Chronic hypertrophic
laryngitis
 Advanced stage of hyperemic laryngitis
 Cellular infiltrate in the submucosa
 Epithelium may undergo hyperplasia /
metaplasia
 May be generalized involvement
 FalseVC (ventricular bands)
 “Dysphonia plica ventricularis”
 True vocal cords
 Rinke’s Edema
Causes & features
Clinical findings
 Laryngoscopy
 Laryngeal mucosa is thickened & dusky red in
colour
 Vocal cords red & swollen, Edges loose sharp
demarcation
Treatment
 Conservative
 Surgical
 Stripping of edematous mucosa with micro-
scissor
 Ablation with LASER
 One side done at a time to preventWeb
Chronic atrophic laryngitis
 Common in women
 Associated with Atrophic Rhinitis
 Laryngitis Sicca
 Exact cause not known
 Hormonal disturbance, Dietary deficiency,
Autoimmune disorder
 Bacillus ozaenae secondary infection
Clinical features
 Hoarseness
 Dry irritating cough
 Dyspnoea due to crusts
 Laryngoscopy
 Laryngeal mucosa is dry & atrophic
 Covered with foul smelling crusts
Treatment
 Primary condition
 Atrophic rhinitis
 Steam inhalation
 25% glucose in glycerin sprays
 Expectorants containing Iodides to loosen the
crusts
Chronic specific laryngitis
Tubercolous laryngitis
 Secondary to Pulmonary tuberculosis
 Common in adult males
 Brochogenic / hematogenous route
 Bronchogenic route
 affects posterior larynx
 Interarytnoid region
 submucosal tubercles & granuloma
 Hematogenous
 Multiple painful ulcers in larynx & pharynx
Clinical features
 Hoarseness
 Painful ulcers / referred otalgia
 Odynophagia
 Laryngoscopy
 Pale granulations in the interarytnoid region
 Ulcers of vocal cords
 Mouse eaten appearance
 Swelling of falseVCs & Aryepiglottic folds
Investigations
 Xray chest PA view
 Sputum for AFB
 Biopsy
Treatment
Vocal nodules
 Singer‘ nodules
 Common in voice misusers
 Teachers, singers, preachers &Vendors
 Vocal trauma
 submucosal hemorrhage
 fibrosis & hyalinization
 At the junction of anterior 1/3 7 posterior 2/3 (
subject to maximum trauma )
Clinical features
 H/o misuse of voice
 Hoarseness
 Vocal fatigue
 Laryngoscopy
 Symmetrical nodular pinkish or grey masses at
junction of ant 1/3 & post 2/3 of true vocal cords
Treatment
 Conservative
1. Avoid misuse of voice
2. Speech therapy / proper use of voice
1. No shouting / No whisper
3. ? May change the profession
 Surgical
 Microlaryngoscopy & LASER
Thanks

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Chronic laryngeal infections

  • 1.
  • 2.
  • 3. CHRONIC LARYNGITIS By Lt Col Saeed Ullah, FCPS Classified ENT, Head and neck surgeon CMH Quetta
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  • 9. Calssification  Chronic specific laryngitis  Chronic non specific laryngitis
  • 10. Chronic non specific laryngitis 1. Chronic hyperemic 2. Chronic Hypertrophic 3. Atrophic laryngitis 4. Pachydermia (contact granuloma / contact ulcer
  • 11. Chronic hypreamic laryngitis  Diffuse inflammatory condition involving the whole larynx particularly true & false vocal cords
  • 12. Causes  Recurrent acute laryngitis  Incompletely resolved acute laryngitis  Chronic infection in the vicinity  Sinusitis, tonsillitis, bad orodental hygiene ,  Ch Bronchitis  Occupational factors  dust & Fumes  Smoking & Alcohol  Voice abuse  GERD
  • 13. Clinical features  Males affected more than females  Hoarseness of voice  Fatigue of voice  Hawking & irritation larynx  Dry irritating cough
  • 14. Clinical features  Laryngoscopy  Hyperemia ,Vocal cords appear dull & edges are rounded,Viscid secretions onVC / interarytnoid region
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  • 17. Treatment  EliminateThe cause  infection / irritating factors  Complete voice rest / Observe proper vocal hygiene  Medicated steam inhalation
  • 18.
  • 19. Chronic hypertrophic laryngitis  Advanced stage of hyperemic laryngitis  Cellular infiltrate in the submucosa  Epithelium may undergo hyperplasia / metaplasia  May be generalized involvement  FalseVC (ventricular bands)  “Dysphonia plica ventricularis”  True vocal cords  Rinke’s Edema
  • 21. Clinical findings  Laryngoscopy  Laryngeal mucosa is thickened & dusky red in colour  Vocal cords red & swollen, Edges loose sharp demarcation
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  • 23. Treatment  Conservative  Surgical  Stripping of edematous mucosa with micro- scissor  Ablation with LASER  One side done at a time to preventWeb
  • 24. Chronic atrophic laryngitis  Common in women  Associated with Atrophic Rhinitis  Laryngitis Sicca  Exact cause not known  Hormonal disturbance, Dietary deficiency, Autoimmune disorder  Bacillus ozaenae secondary infection
  • 25. Clinical features  Hoarseness  Dry irritating cough  Dyspnoea due to crusts  Laryngoscopy  Laryngeal mucosa is dry & atrophic  Covered with foul smelling crusts
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  • 28. Treatment  Primary condition  Atrophic rhinitis  Steam inhalation  25% glucose in glycerin sprays  Expectorants containing Iodides to loosen the crusts
  • 29.
  • 31. Tubercolous laryngitis  Secondary to Pulmonary tuberculosis  Common in adult males  Brochogenic / hematogenous route  Bronchogenic route  affects posterior larynx  Interarytnoid region  submucosal tubercles & granuloma  Hematogenous  Multiple painful ulcers in larynx & pharynx
  • 32. Clinical features  Hoarseness  Painful ulcers / referred otalgia  Odynophagia  Laryngoscopy  Pale granulations in the interarytnoid region  Ulcers of vocal cords  Mouse eaten appearance  Swelling of falseVCs & Aryepiglottic folds
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  • 34. Investigations  Xray chest PA view  Sputum for AFB  Biopsy
  • 36.
  • 37. Vocal nodules  Singer‘ nodules  Common in voice misusers  Teachers, singers, preachers &Vendors  Vocal trauma  submucosal hemorrhage  fibrosis & hyalinization  At the junction of anterior 1/3 7 posterior 2/3 ( subject to maximum trauma )
  • 38. Clinical features  H/o misuse of voice  Hoarseness  Vocal fatigue  Laryngoscopy  Symmetrical nodular pinkish or grey masses at junction of ant 1/3 & post 2/3 of true vocal cords
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  • 40.
  • 41. Treatment  Conservative 1. Avoid misuse of voice 2. Speech therapy / proper use of voice 1. No shouting / No whisper 3. ? May change the profession  Surgical  Microlaryngoscopy & LASER