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PROMPT Course (Proper Recognition and Management of Acutely Ill Patients)
Orientation for the New R1 Residents - Academic year 2019-2020
Wednesday, 25 September 2019
Approach to Chest Pain
2019
Dr Ihab Suliman MBBS ECFMG MRCP (UK) MRCP Spec( DM&Endoc)
CBNC ABcv FESC
Consultant
Advanced Cardiac imaging
King Abdul-Aziz Cardiac Center
0505244473
Ihab.suliman@ngha.med.sa
Twitter@IhabFathiSulima
https://www.slideshare.net/isuliman
Differential
 Cardiac
 MI
 Pericarditis
 Myocarditis
 Aortic Stenosis
 Pulmonary
 PE
 PNA
 Asthma/COPD
 Acute Chest Syndrome
 Pleura
 Pleuritis
 Pneumothorax
 Aorta
 Dissection
 Perforated ulcer
 Chest wall
 Costocondiritis/musculos
keletal
 Esophagus
 Esophageal Spasm
 Eosinophilic Esophagitis
 Esophageal
Rupture/Perforation
 GERD
 Mediastinitis
 RUQ pathology
 Panic attack
Lethal Causes of Chest Pain
▣ 1- Acute Coronary
Syndrome(STEMI/NSTEMI/UA)
▣ 2- Acute Aortic dissection Type A.
▣ 3-Massive Pulmonary Embolism.
▣ 4-Pericardial Tamponade.
▣ 5-Esophageal Rupture.
▣ 6-Tension Pneumothorax.
▣ * Fulminant Myocarditis
▣ x
Pearl: ALWAYS have
the patient point to
the pain!
Typical vs. Atypical Chest Pain
Typical (Ischemic)
Characterized as
discomfort/pressure rather
than pain, Include Upper
Abdomen.
Time duration >2 mins.
Provoked by activity/exercise.
Radiation (i.e. arms, jaw).
Does not change with
respiration/position.
Associated with
diaphoresis/nausea.
Relieved by rest/nitroglycerin.
▣ Atypical
▣ Pain that can be
localized with one finger.
▣ Constant pain lasting for
days.
▣ Fleeting pains lasting for
a few seconds.
▣ Pain reproduced by
movement/palpation.
Typical vs. Atypical Chest Pain
UpToDate
Evaluation of Chest Pain
Case 1:
▣ Ask nurse for most current set of vital
signs
▣ Ask nurse to get an EKG
▣ See the patient!
Evaluation of Chest Pain
▣ Once at bedside, determine if patient is stable
or unstable
▣ Perform focused history and physical exam
▣ Read and interpret the EKG. Compare EKG to
old EKG if available
▣ If patient looks unstable or has concerning EKG
findings, call ER/Cardio On call for help
▣ Write a clinical event note!
Evaluation of Chest Pain
Important Physical signs
▣ focused physical exam for chest pain
� Vital Signs: tachycardia, hypertension/hypotension or
hypoxia
� General: Sick appearing, actively having chest pain
� HEENT: JVD, carotid bruits
� Chest: Rales, wheezes or decreased breath sounds
� CVS: New murmurs, reproducible chest pain, s3 gallop
� Abd: Abdominal tenderness, pulsatile mass
� Ext: Edema, peripheral pulses
� Skin: Rash on chest wall
CASE 1 49 years old male with severe CP
Criteria for type 1 MI
▣ Detection of a rise and/or fall of cTn values with at
least one value above the 99th percentile URL and
with at least one of the following:
▣ Symptoms of acute myocardial ischaemia;
▣ New ischaemic ECG changes;
▣ Development of pathological Q waves;
▣ Imaging evidence of new loss of viable
myocardium or new regional wall motion
abnormality in a pattern consistent with an
ischaemic aetiology;
▣ Identification of a coronary thrombus by
angiography including intracoronary imaging or by
autopsy.a
Case 2
▣ 25 years old lady suddenly developed chest pain that is L-sided, 8/10 and
ECG
CT Angiogram
Incidence
▣ Ranges from 2-10 per 100,000 person-years
▣ Evidence of dissection is found in 1-3% of all
autopsies
Who’s affected?
▣ International Registry of Acute Aortic
Dissection (IRAD)
◼ 65% men
◼ mean age 63yrs
◼ Women tend to present older (67 vs. 60yrs)
▣ Highest incidence in patients 50 to 70 years old.
▣ Male-to-female ratio 2:1
▣ Half of dissections in females before age 40
occur during pregnancy
Clinical Features
▣ Abrupt onset of severe, sharp or "tearing"
posterior chest or back pain (70-90%)
▣ Pulse deficit
◼ weak/absent carotid, brachial, or femoral pulse
resulting from intimal flap or compression by
hematoma
▣ HTN at initial presentation is more common in
those with a type B dissection .%)
Management
▣ Untreated aortic dissection or intramural
hematoma
◼ 25% die within 24hrs
◼ 50% by 48hrs
▣ Basic management
◼ Type A dissection  surgery
◼ Type B dissection  medical management/BP
management
▣ Surgery -- prevents medial extension reaching
the pericardium and producing fatal
tamponade or worsening other complications
Case 3
25 years old female 7th day PP with severe localized CP
Case 3
Case 4
55 years old lady stopped Warfarin for uterine surgery came with
CP Severe SOB
MECHANICAL MVR
What TO DO if No Click
▣ What should be done if No Click
▣ Fluoroscopy or CT.
▣ Valve Thrombosis
Case 5
▣ 70 years old lady with malignant ascites.
▣ Sent for surgery.
MV
calcification
more
posteriorly
Gastric thickening
liver lesion ascites
PRE OP ECG
Normal
POST OPE ECG
Troponin
Nef HM, et al. Tako-tsubo
Cardiomyopathy (Apical
Ballooning). Heart. 2007; 93:1309-
1315.
Stress Induced Cardiomyopathy
Sharkey SW, Lesser JR, Zenovich AG, et al. Acute and reversible cardiomyopathy provoked by stress in
women from the United States. Circulation 2005;111:472–9.
▣ Women 95% cases
▣ Mean age 68
▣ Not just emotional stress
◼ Intense physical stress
◼ Acute medical illness (ICU)
◼ No stress in 10%
▣ Probably 2% of ACS cases
Etiology ???
◼ Wall motion abnormality doesn’t correlate to single coronary
distribution
◼ Catecholamine induced vascular spasm?
◼ Catecholamine induced reversible myocyte injury?
� Is LV apex more sensitive to injury?
END
▣ Thanks a lot

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Prompt course cp 2019 ihab suliman

  • 1. PROMPT Course (Proper Recognition and Management of Acutely Ill Patients) Orientation for the New R1 Residents - Academic year 2019-2020 Wednesday, 25 September 2019 Approach to Chest Pain 2019 Dr Ihab Suliman MBBS ECFMG MRCP (UK) MRCP Spec( DM&Endoc) CBNC ABcv FESC Consultant Advanced Cardiac imaging King Abdul-Aziz Cardiac Center 0505244473 Ihab.suliman@ngha.med.sa Twitter@IhabFathiSulima https://www.slideshare.net/isuliman
  • 2. Differential  Cardiac  MI  Pericarditis  Myocarditis  Aortic Stenosis  Pulmonary  PE  PNA  Asthma/COPD  Acute Chest Syndrome  Pleura  Pleuritis  Pneumothorax  Aorta  Dissection  Perforated ulcer  Chest wall  Costocondiritis/musculos keletal  Esophagus  Esophageal Spasm  Eosinophilic Esophagitis  Esophageal Rupture/Perforation  GERD  Mediastinitis  RUQ pathology  Panic attack
  • 3.
  • 4. Lethal Causes of Chest Pain ▣ 1- Acute Coronary Syndrome(STEMI/NSTEMI/UA) ▣ 2- Acute Aortic dissection Type A. ▣ 3-Massive Pulmonary Embolism. ▣ 4-Pericardial Tamponade. ▣ 5-Esophageal Rupture. ▣ 6-Tension Pneumothorax. ▣ * Fulminant Myocarditis ▣ x
  • 5. Pearl: ALWAYS have the patient point to the pain!
  • 6. Typical vs. Atypical Chest Pain Typical (Ischemic) Characterized as discomfort/pressure rather than pain, Include Upper Abdomen. Time duration >2 mins. Provoked by activity/exercise. Radiation (i.e. arms, jaw). Does not change with respiration/position. Associated with diaphoresis/nausea. Relieved by rest/nitroglycerin. ▣ Atypical ▣ Pain that can be localized with one finger. ▣ Constant pain lasting for days. ▣ Fleeting pains lasting for a few seconds. ▣ Pain reproduced by movement/palpation.
  • 7. Typical vs. Atypical Chest Pain UpToDate
  • 8. Evaluation of Chest Pain Case 1: ▣ Ask nurse for most current set of vital signs ▣ Ask nurse to get an EKG ▣ See the patient!
  • 9. Evaluation of Chest Pain ▣ Once at bedside, determine if patient is stable or unstable ▣ Perform focused history and physical exam ▣ Read and interpret the EKG. Compare EKG to old EKG if available ▣ If patient looks unstable or has concerning EKG findings, call ER/Cardio On call for help ▣ Write a clinical event note!
  • 10. Evaluation of Chest Pain Important Physical signs ▣ focused physical exam for chest pain � Vital Signs: tachycardia, hypertension/hypotension or hypoxia � General: Sick appearing, actively having chest pain � HEENT: JVD, carotid bruits � Chest: Rales, wheezes or decreased breath sounds � CVS: New murmurs, reproducible chest pain, s3 gallop � Abd: Abdominal tenderness, pulsatile mass � Ext: Edema, peripheral pulses � Skin: Rash on chest wall
  • 11. CASE 1 49 years old male with severe CP
  • 12.
  • 13.
  • 14.
  • 15.
  • 16. Criteria for type 1 MI ▣ Detection of a rise and/or fall of cTn values with at least one value above the 99th percentile URL and with at least one of the following: ▣ Symptoms of acute myocardial ischaemia; ▣ New ischaemic ECG changes; ▣ Development of pathological Q waves; ▣ Imaging evidence of new loss of viable myocardium or new regional wall motion abnormality in a pattern consistent with an ischaemic aetiology; ▣ Identification of a coronary thrombus by angiography including intracoronary imaging or by autopsy.a
  • 17.
  • 18.
  • 19.
  • 20. Case 2 ▣ 25 years old lady suddenly developed chest pain that is L-sided, 8/10 and
  • 21. ECG
  • 23. Incidence ▣ Ranges from 2-10 per 100,000 person-years ▣ Evidence of dissection is found in 1-3% of all autopsies
  • 24. Who’s affected? ▣ International Registry of Acute Aortic Dissection (IRAD) ◼ 65% men ◼ mean age 63yrs ◼ Women tend to present older (67 vs. 60yrs) ▣ Highest incidence in patients 50 to 70 years old. ▣ Male-to-female ratio 2:1 ▣ Half of dissections in females before age 40 occur during pregnancy
  • 25. Clinical Features ▣ Abrupt onset of severe, sharp or "tearing" posterior chest or back pain (70-90%) ▣ Pulse deficit ◼ weak/absent carotid, brachial, or femoral pulse resulting from intimal flap or compression by hematoma ▣ HTN at initial presentation is more common in those with a type B dissection .%)
  • 26. Management ▣ Untreated aortic dissection or intramural hematoma ◼ 25% die within 24hrs ◼ 50% by 48hrs ▣ Basic management ◼ Type A dissection  surgery ◼ Type B dissection  medical management/BP management ▣ Surgery -- prevents medial extension reaching the pericardium and producing fatal tamponade or worsening other complications
  • 27. Case 3 25 years old female 7th day PP with severe localized CP
  • 29. Case 4 55 years old lady stopped Warfarin for uterine surgery came with CP Severe SOB MECHANICAL MVR
  • 30. What TO DO if No Click ▣ What should be done if No Click ▣ Fluoroscopy or CT. ▣ Valve Thrombosis
  • 31. Case 5 ▣ 70 years old lady with malignant ascites. ▣ Sent for surgery.
  • 37. Nef HM, et al. Tako-tsubo Cardiomyopathy (Apical Ballooning). Heart. 2007; 93:1309- 1315.
  • 38.
  • 39. Stress Induced Cardiomyopathy Sharkey SW, Lesser JR, Zenovich AG, et al. Acute and reversible cardiomyopathy provoked by stress in women from the United States. Circulation 2005;111:472–9. ▣ Women 95% cases ▣ Mean age 68 ▣ Not just emotional stress ◼ Intense physical stress ◼ Acute medical illness (ICU) ◼ No stress in 10% ▣ Probably 2% of ACS cases Etiology ??? ◼ Wall motion abnormality doesn’t correlate to single coronary distribution ◼ Catecholamine induced vascular spasm? ◼ Catecholamine induced reversible myocyte injury? � Is LV apex more sensitive to injury?