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GASTRIC OUTLET OBSTRUCTION
DR.EDWINA VASANTHA,M.S.,D.G.O
• GOO is the clinical and pathophysiological
consequence of any symptom complex that
produces a mechanical impediment to gastric
emptying.
HISTORY
• AGE :20-45 years with peak 30-35 years
• Abdominal pain
site:epigastric and left hypochondrial pain
relationship to food: food - pain -
relief=du
food – pain =gu
relieved by alkali,milk
association with time of the day
h/o radiation to the back(? Pancreas
penetration)
generalised pain(perforation)
• Anorexia,nausea
• Early satiety
• Vomiting- characteristic unpleasant
-copious
-projectile
-non bilous,food taken several hours
to days ago
• Feeling of unwell
• Weight loss
• Abdominal swelling
EXAMINATION
• Chronically ill looking
• Wasted,dehydrated
• Pale
• May be in shock
• Epigastric /left or right hypochondrial
tenderness
• Distended abdomen
• Visible gastric peristalsis
• hepatosplenomegaly
• Succussion splash
• Auscultopercussion test-to look for stomach
dialatation
• Internal pelvic,per rectal examination
• Vitals
• Lymph nodal enlargement- left supraclavicular
INVESTIGATIONS
• To stabilise patient
-complete haemogram
-serum electrolytes,
-arterial blood gases
-urinalysis
• To confirm diagnosis
-plain x-ray abdomen erect
-gastric function tests:>400ml resting juice
aspirated shows presumptive diagnosis of GOO
-endoscopy and biopsy
-barium meal:findings
markedly dialated stomach with a lot
of residue
gastritis,stasis
chronic cicatrised ulcer,diverticula
trifoliate deformity of duodenal cap
pyloric opening narrowed or total
obstruction
• Detection of H.pylori
-Non invasive
serology
carbon labelled urea breath test
-invasive
rapid ureasetest
histology and culture
• Differential diagnosis
 PUD
 Gastric polyps
 Ingestion of caustics
 Pyloric stenosis; mostly fisrt borne male child
 Congenital duodenal webs
 Gallstone obstruction (Bouveret syndrome)
 Pancreatic pseudocysts
 bezoars
 Cast syndrome(superior mesentric artery
 Malignancy
pancreatic cancer
ampullary cancer
duodenal cancer
cholangiocarcinoma
gastric cancer
metastases to gastric outlet from other
primary
• TREATMENT
General measures
resuscitation : IVF
urethral catheter
nasogastric tube
correction of electrolyte
imbalance ideally under ECG monitoring
anaemia correction
Antisecretory therapy
Non operative : warm saline lavage
H.pylori eradication
Invasive :endoscopic balloon dialatation
Operative measures
highly selective
vagotomy+GJ+H.pylori eradication
truncal/selective vagotomy
+Billroth II +kocherisation +HP Eradication
TV/SV+
Antrectomy+GJ/GD+Kocherisation+HP
eradication
OBSTRUCTING TYPES-distal
gastrectomy+TV+GJ+HPE
• POST OP COMPLICATIONS
immediate:primary haemorrhage
injury to contiguous strictures
aneasthetic complications
early: postgastrecrtomy syn
i)early dumping: 20-30 mins after
ingestion ofmeal
both GI and cardiovascular
symptoms
Mgt-pt.informed preop
dietary modification,long
acting somatostatin analogue,jejunal 20cm
isoperistaltic loop interposition,jejunal 10cm
antiperistaltic loop interposition
2) Late dumping: due to hypoglycaemia
Mgt:small meals,less
carbohydrates,antiperistaltic loop
• duodenal blow out:4-5th post op day,life
threatening, mgt;fluid and electrolyte
correction,enteroentostomy
Thank you

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GASTRIC OUTLET OBSTRUCTION TREATMENT

  • 2. • GOO is the clinical and pathophysiological consequence of any symptom complex that produces a mechanical impediment to gastric emptying. HISTORY • AGE :20-45 years with peak 30-35 years • Abdominal pain site:epigastric and left hypochondrial pain relationship to food: food - pain - relief=du food – pain =gu
  • 3. relieved by alkali,milk association with time of the day h/o radiation to the back(? Pancreas penetration) generalised pain(perforation) • Anorexia,nausea • Early satiety • Vomiting- characteristic unpleasant -copious -projectile -non bilous,food taken several hours to days ago
  • 4. • Feeling of unwell • Weight loss • Abdominal swelling EXAMINATION • Chronically ill looking • Wasted,dehydrated • Pale • May be in shock • Epigastric /left or right hypochondrial tenderness
  • 5. • Distended abdomen • Visible gastric peristalsis • hepatosplenomegaly • Succussion splash • Auscultopercussion test-to look for stomach dialatation • Internal pelvic,per rectal examination • Vitals • Lymph nodal enlargement- left supraclavicular
  • 6. INVESTIGATIONS • To stabilise patient -complete haemogram -serum electrolytes, -arterial blood gases -urinalysis • To confirm diagnosis -plain x-ray abdomen erect -gastric function tests:>400ml resting juice aspirated shows presumptive diagnosis of GOO -endoscopy and biopsy
  • 7. -barium meal:findings markedly dialated stomach with a lot of residue gastritis,stasis chronic cicatrised ulcer,diverticula trifoliate deformity of duodenal cap pyloric opening narrowed or total obstruction • Detection of H.pylori -Non invasive serology
  • 8. carbon labelled urea breath test -invasive rapid ureasetest histology and culture
  • 9. • Differential diagnosis  PUD  Gastric polyps  Ingestion of caustics  Pyloric stenosis; mostly fisrt borne male child  Congenital duodenal webs  Gallstone obstruction (Bouveret syndrome)  Pancreatic pseudocysts  bezoars  Cast syndrome(superior mesentric artery
  • 10.  Malignancy pancreatic cancer ampullary cancer duodenal cancer cholangiocarcinoma gastric cancer metastases to gastric outlet from other primary
  • 11. • TREATMENT General measures resuscitation : IVF urethral catheter nasogastric tube correction of electrolyte imbalance ideally under ECG monitoring anaemia correction Antisecretory therapy Non operative : warm saline lavage H.pylori eradication
  • 12. Invasive :endoscopic balloon dialatation Operative measures highly selective vagotomy+GJ+H.pylori eradication truncal/selective vagotomy +Billroth II +kocherisation +HP Eradication TV/SV+ Antrectomy+GJ/GD+Kocherisation+HP eradication OBSTRUCTING TYPES-distal gastrectomy+TV+GJ+HPE
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  • 15. • POST OP COMPLICATIONS immediate:primary haemorrhage injury to contiguous strictures aneasthetic complications early: postgastrecrtomy syn i)early dumping: 20-30 mins after ingestion ofmeal both GI and cardiovascular symptoms Mgt-pt.informed preop dietary modification,long acting somatostatin analogue,jejunal 20cm isoperistaltic loop interposition,jejunal 10cm antiperistaltic loop interposition
  • 16. 2) Late dumping: due to hypoglycaemia Mgt:small meals,less carbohydrates,antiperistaltic loop • duodenal blow out:4-5th post op day,life threatening, mgt;fluid and electrolyte correction,enteroentostomy
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