2. ⢠GOO is the clinical and pathophysiological
consequence of any symptom complex that
produces a mechanical impediment to gastric
emptying.
HISTORY
⢠AGE :20-45 years with peak 30-35 years
⢠Abdominal pain
site:epigastric and left hypochondrial pain
relationship to food: food - pain -
relief=du
food â pain =gu
3. relieved by alkali,milk
association with time of the day
h/o radiation to the back(? Pancreas
penetration)
generalised pain(perforation)
⢠Anorexia,nausea
⢠Early satiety
⢠Vomiting- characteristic unpleasant
-copious
-projectile
-non bilous,food taken several hours
to days ago
4. ⢠Feeling of unwell
⢠Weight loss
⢠Abdominal swelling
EXAMINATION
⢠Chronically ill looking
⢠Wasted,dehydrated
⢠Pale
⢠May be in shock
⢠Epigastric /left or right hypochondrial
tenderness
6. INVESTIGATIONS
⢠To stabilise patient
-complete haemogram
-serum electrolytes,
-arterial blood gases
-urinalysis
⢠To confirm diagnosis
-plain x-ray abdomen erect
-gastric function tests:>400ml resting juice
aspirated shows presumptive diagnosis of GOO
-endoscopy and biopsy
7. -barium meal:findings
markedly dialated stomach with a lot
of residue
gastritis,stasis
chronic cicatrised ulcer,diverticula
trifoliate deformity of duodenal cap
pyloric opening narrowed or total
obstruction
⢠Detection of H.pylori
-Non invasive
serology
8. carbon labelled urea breath test
-invasive
rapid ureasetest
histology and culture
15. ⢠POST OP COMPLICATIONS
immediate:primary haemorrhage
injury to contiguous strictures
aneasthetic complications
early: postgastrecrtomy syn
i)early dumping: 20-30 mins after
ingestion ofmeal
both GI and cardiovascular
symptoms
Mgt-pt.informed preop
dietary modification,long
acting somatostatin analogue,jejunal 20cm
isoperistaltic loop interposition,jejunal 10cm
antiperistaltic loop interposition
16. 2) Late dumping: due to hypoglycaemia
Mgt:small meals,less
carbohydrates,antiperistaltic loop
⢠duodenal blow out:4-5th post op day,life
threatening, mgt;fluid and electrolyte
correction,enteroentostomy