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There are opportunities to use PL information for optimizing ruminant feeding efficiency and carcass quality I NTRODUCTION M ATERIALS  & M ETHODS ,[object Object],[object Object],[object Object],[object Object],[object Object],C ONCLUSION R ESULTS * For each graph, each point corresponds to an experimental treatment ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],R ECOMMENDED  R EFERENCES ,[object Object],[object Object],[object Object],[NEFA (  )] CNS appetite  (  ) IGF1(  ),  insulin (  ),  PERIPHERAL ORGANS   energy intake other functions: i.e.  gonads gravid uterus lipogenesis lipids storage lipolysis ADIPOCYTE ADIPOSE TISSUE mammary gland &  milk synthesis growth & tissue  composition energy    expenditure physical  activity energy partition leptin leptin  synthesis [glucose (  )] [ β -OH (  )] [leptin] n  = 6 exp PL, ng/mL PL, ng/mL PL, ng/mL PL, ng/mL + + + + + + Figures . Examples of correlations found between physiological, metabolic and carcass performance variables, and plasmatic leptin (PL) - - + Leptin is the hormone product the obese gene that is synthesized and predominantly expressed by adipocytes. We aimed to update knowledge on relationship between plasma leptin (PL), feed intake (FI) and body fat reserves (BF) in ruminants

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