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Lbp assessement
1. LOW BACK PAIN ASSESSMENT
DR. VARUN SINGLA
MD (PGI Chandigarh)
PDCC-Pain Management (SGPGI Lucknow)
Fellow FPCI (Mumbai)
Member, Indian Society for Study of Pain
Peer Reviewer, Cochrane PaPaS, UK
3. EPIDEMIOLOGY
Among top 10 diseases
and injuries that
account for highest
number of DALYs .
Lifetime prevalence -
60-70%
Leading cause of
activity limitation and
work absence with
significant social and
economic impact.
Global number of
individuals with LBA is
likely to increase in
future
4. ANATOMY
LBA : Pain in posterior aspect of body from
lower margin of 12th ribs to lower gluteal folds
± referred pain to one or both lower limbs
that lasts for atleast one day
10. CATEGORIZATION
Non- specific low back pain
Back pain with radiculopathy or spinal
stenosis
Back pain with other spinal cause
11. APPROACH
Rule out serious pathology “red flags”
Confirm that pain is :
In lower back
Mechanical
Not inflammatory
Rule out specific causes
14. HISTORY
Search should be directed to answer two
questions: “Where is the disease causing
the pain—in the brain, spinal cord, plexus,
muscle, tendon, or bone?” and “What is
the nature of the disease?”
“One physician willing to
sit and actually listen to
patients can be of more
practical benefit than 100
magnets (of any Tesla
strength).”
15. HOW TO GO ABOUT IT!!
Build rapport
A routine social history
Establish chief complaint. Why is the patient
here?
Use pain litany. Where is the pain? What is its
nature?
Do not jump to conclusions
Determine impact of pain on patient's life
Explore past medical and family history
Drug history
16. PAIN LITANY
1. Mode of onset
2. Location
3. Chronicity
4. Tempo (duration and frequency)
5. Character and severity
6. Associated factors: Premonitory symptoms and aura,
Precipitating factors, Environmental factors (occupation),
Family history, Age at onset, Pregnancy and menstruation,
Gender, Past medical and surgical history, Socioeconomic
considerations, Psychiatric history, Medications and drug
and alcohol use
17. MODE OF ONSET
Sudden or insiduous
Distinguishes sick from well patients
18. LOCATION
SPECIFIC POINT VS ACROSS
BACK
SUPERFICIAL VS DEEP
INVOLVE ANY OTHER
REGION (LOWER
EXTREMITY)
SITE OF MAXIMUM PAIN
19. CHRONICITY
first or worst syndrome - accentuated pain
presentation, deserve serious concern, view
with medical urgency
Equating chronicity with benign disease is
dangerous.
Identify ominous changes in a long-standing,
stable pain syndrome
20. DURATION AND FREQUENCY
Gives an idea about nature of disease.
Severity of disease
Make Differential diagnosis
24. MEDICATION HISTORY
Duration, frequency and amount
Red flag drugs : morphine, codeine, fentanyl,
cocaine, hydromorphone, methadone,
barbiturates, BZDs,
Other important drugs : Oral contraceptives,
Anticoagulants (heparin, warfarin,
clopidogrel), Antiplatelet agents (aspirin,
ticlopidine), Antianginals (nitrates)
25. PSYCHOLOGY
Patient’s expectations – cure?
Physician pursuing it!!!
Psychosocial issues often contribute to, and
may be the main cause of disability too.
Need to address psychological aspect.
26. PSYCHOSOCIAL FACTORS – PREDICTORS OF
POOR OUTCOME
Depression, anxiety
Job dissatisfaction, stress (time of work)
Stress – marital/other
Secondary gain
Maladaptive thinking and coping styles
Multiple somatic complaints
28. INSPECTION/ GENERAL SURVEY
Skin and Subcutaneous tissue - lumbar
lipoma, an abnormal hair patch, or a port
wine stain, cafe au lait spots and nodular
skin swellings
30. GAIT
Sciatica - walk with the hip more
extended and the knee more flexed
Antalgic gait - putting as little weight
as possible on the affected side and
then quickly transferring the weight
to the unaffected side.
Heel walking – ankle dorsiflexors
strength. L4 innervated tibialis
anterior, L3-L4 disc herniation.
Toe walking – S1 innervated
gastrosoleus muscle, L5-S1 disc
herniation
31. RANGE OF MOTION
Flexion : angle between the final
position of the trunk and a vertical
plane (80° to 90°) OR distance from
the patient's fingertips to the floor (10
cm).
Extension : angle between trunk and
vertical line; 20° to 30°
Lateral bending :angle between
imaginary line of vertebra prominens
and sacrum; 20 -30°
Rotation : angle between new plane of
rotated shoulders and coronal plane;
30° to 40°
32. PALPATION
Spinous process
Tuffier’s line : L4-L5 interspace
Step off : spondylolisthesis, vertebral
compression fracture
Paraspinal muscle spasm : lateral bending
Muscular tenderness, trigger points, tender
nodules
Facet joint tenderness, transverse process
fracture
40. SENSORY
Pain and temperature : spinothalamic tract
Light touch, position sense, vibration :
posterior column
41. NERVE ROOT TENSION TESTS
SLR
Cross leg SLR
Lasegue’s test
Slump test
Bowstring sign
42. SLR, CROSSED SLR AND LASEGUE TEST
stretches the L5 and SI nerve roots 2 mm to 6 mm
hamstring tightness
SLR - extremely sensitive (0.9) but less specific
(0.26)
Crossed SLR - less sensitive (.29) but more
specific (.88)
Lasegue test – Sensitivity (0.7).
45. FEMORAL NERVE STRETCH TEST
L2, L3, or L4
nerve root
compression
VALSALVA MANOUVRE
•Increases
intrathecal pressure
– thus pain due to
pressure on spinal
cord or its nerve
roots
47. WADDELL SIGNS FOR NON-ORGANIC PAIN
Superficial non-anatomic tenderness
Pain from maneuvers that should not
ellicit pain
Distraction maneuvers that should ellicit
pain BUT don’t
Disturbances not consistent with known
patterns of pain
Over-reacting during the exam
Not definitive to rule out organic disease
48. SACROILIAC JOINT TESTS
Yeomen test
Patrick’s test(FABER)
Gaenslen’s test
Sacroiliac shear test
Distraction test
Thigh thrust test
49. CLINICAL TESTS FOR SACROILIAC JOINT
DYSFUNCTION
Faber Patrik
Test
Yeoman’s Test
What are we dealing with
Global burden of disease study 2010
Out of 291 diseases LBA ranked at 6th in health burden
Future – as population ages
Global burden of disease study 2010
Spitzer, W.O an dLeblanc , F.E, 1987
Most people recover from an acute episode within 8 weeks
NASS (north american spine society, 2000) guidelines define “initila phase of care” = 8 weeks
Symptomatic after 6 months = poor prognosis for significant improvement
Based on history and examination
remains vigilant for signals of an urgent situation. Pain of uncertain origin should always be regarded as a potential emergency.
Rule out hip pathology
Aggravated or relieved by certain movemnets or postures
Inflammatory – after waking / second half of night
- morning stiffness >30 minutes
- relieved by activity
- lab test / inflammatory markers
Saddle anaesthesia Bladder/ bowel involvement Motor weakness
Sudden onset severe central pain (relieved by lying down) H/O trauma : major / minor spinal deformity
>50 yr or <20 yr H/O cancer Constitutional symptoms Pain when supine, causing sleep disturbance, thoracic
Major truma - RTA or FFH
Minor trauma – strenous lifting in osteoporotic patient
introduce self
occupation, place of employment, marital status, and number of children
Assess patient's mood, anxiety level, and capability of giving a history on his or her own.
Is it pain?, prescription renewal, morbid fear of cancer. Open ended questions allow the patient to tell his or her own story
ask about other doctors whom the patient has seen and their treatments.
psychological fears, family issues (marriage), compensation, and work record.
current pain should be placed in context with other major medical events: previous surgery, hospitalizations, cancer
Pain litany - formulaic exploration of the patient’s pain history
office should be both professional and comfortable
patients have a private place where they undress and are examined
.
distinguishing sick from well patients. For example, the sudden, explosive presentation of a subarachnoid hemorrhage secondary to a ruptured intracranial aneurysm, manifested by severe headache, neck pain, and a sense of impending doom, contrasts sharply with
the chronic diffuse headache and vague neck tightness of tension-type cephalalgia
The pain in trigeminal neuralgia is Usually limited to one or more branches of cranial nerve (CN) V and does not spread beyond the distribution of the nerve.4 The V2 and V3 divisions of this nerve are much more frequently involved than is V1 (Fig. 5.1). The pain is rarely bilateral except in certain cases of multiple sclerosis, brainstem neoplasms and skull base tumors, and infections.
burning, prickling dysesthesias of meralgia paresthetica. The unilateral involvement of the lateral femoral cutaneous nerve produces painful dysesthesias in the anterior thigh, more
Commonly in men, who notice the disturbance when they put a hand in a trouser pocket.
“How long have you had this pain?”
try to date the pain in relation to other medical events, such as trauma, surgery, and other illnesses.
back pain that has been present for 30 years and is not associated with any progression is strong evidence of a self-limited pain syndrome
severe low back pain of sudden onset or pain that suddenly changes in character must be assigned to the category of “sick until proved otherwise.”
Identify ominous changes in a long-standing, stable pain syndrome (e.g., when a patient with chronic low
back pain suddenly becomes incontinent).
n Recognize new symptoms superimposed on chronic complaints
trigeminal neuralgia
(tic douloureux) is described as brief electric shocks or stabbing pain. Onset and termination of attacks are abrupt,
and affected patients are usually pain free between episodes. Attacks last only a few seconds. It is not unusual for
a series of attacks to occur in rapid succession over several hours. In contrast, the pain of temporal (giant cell) arteritis is usually described as a dull, persistent, gnawing pain that is exacerbated by chewing.3
The patient with acute lumbar disk herniation often writhes before the physician and is essentially unable to
sit in a chair. The body language and facial expressions
Vascular headaches tend to be throbbing
and pulsatile, and the pain intensity is often described as
severe.3 Cluster headaches may have a deeper, boring, burning,
wrenching quality.
Trigeminal neuralgia is typically described as paroxysmal,
jabbing, or shocklike, in contrast to non-neuralgic pain such as
experienced in temporomandibular joint (TMJ) dysfunction,
which is often described as a unilateral, dull, aching pain
Standing, walking, sitting, froward flexion, extension, weight bearing, lying down
Until drug dependency issues are addressed, effective inroads into the management of chronic pain will be thwarted.
anticoagulants (warfarin, heparin) or antiplatelet agents (aspirin, clopidogrel [Plavix], and ticlopidine
Inadvertent overdosing of an older, confused patient can cause intracerebral bleeding (headache) or back and radicular pain (secondary to retroperitoneal hemorrhage
LBA is recurrent in 33-70% of patients. Expectations fail to reflect it (von korf , spine 1996. haestback, european spine journal 2003)
Treat these and if refractory to treatment anticipate poor outcome
Skin and Subcutaneous Tissue :
lumbal lipoma, an abnormal hair patch, or a port wine stain may be associated with spina bifida or even myelomeningocele.
cafe au lait spots and nodular skin swellings may indicate neurofibromatosis, a condition that may cause secondary deformity of the spine.
Longitudinal furrow in midline. Spinous processes run in this and prominent in flexion.
Paraspinous muscles – superficial column – erector spinae or sacrospinalis muscle – Multifidus, longissimus, iliocostalis
Paraspinous muscle spasm – prominence on one side (due to any painful lesion of lumbar spine or paraspinal muscle strain)
A, spinous processes; B, erector spinae; C, iliac crests; D, posterior facet joints; E, transverse processes.
Symmetry – space between upper limb and trunk (detect a subtle coronal deformity of the spine )
Pelvic obliquity – imaginary line between PSIS or iliac crest parallel to floor. (scoliosis, anamolous vertebra, leg length discrepancy, list)
Spondylolisthesis or vertebral compression fracture : Step-Off Deformity. body of involved vertebra slide forward . M/C L5-S1
normal lumbar lordosis (60%) should exactly complement the thoracic kyphosis and cervical lordosis, so that the base of the occiput rests directly above the sacrum
Hyperlordosis -swayback – buttocks prominent
lumbar flatback syndrome - Compression fractures that result in anterior wedging of the lumbar vertebral bodies, Advanced degeneration of the lumbar intervertebral disks,
Decreased lumbar lordosis is often a temporary, reversible deformity related to pain and associated muscle spasm
spondylolysis, reflexive decrease in lumbar lordosis.
Ankylosing spondylitis more rigid decrease
Gibbus - sharp, angular kyphotic deformity, protruding spinous process,
tuberculosis of the spine, localized collapse of anterior portion of vertebral column
Spinal stenosis, spndylolisthesis – pain increase in extension , relieved with flexion
Discogenic , vertebral body source – increase with flexion, axial loading
Pain in extension – LCS, SOL, ds of posterior element of vertebrae (spondylosis, facet arthropathy)
Herniated disc - avoid lateral bending towards side of impingement.
Paraspinal muscle spasm – muscles on side of bend do not relax unusually.
Lumbar discogenic pain is often restricted to the axial spine and is associated with intolerance of the sitting position and pain provoked by coughing, sneezing, and Valsalva
maneuvers
Localized tenderness at a particular level - sprains or disruptions of posterior ligaments of the spine, fractures and tumors of the posterior elements
The amount of slippage usually must be at least 50% of the diameter of the lumbar vertebral bodies before the step-off can be detected by physical examination.
Trigger points may also indicate the presence of fibromyalgia
facet joint arthritis or a painful facet joint syndrome.
localized unilateral tenderness deep to the paraspinous muscles following trauma should suggest the possibility of a transverse process fracture
crunch or sit-up.
Extension : pain associated with disorders of the posterior elements of the lumbar spine, such as spondylolysis or facet joint arthritis, or of spinal stenosis may be exacerbated by this test.
gluteus medius - hip abduction
Grades 1 to 3 are relatively objective and less prone to interobserver variation.
Monosynaptic arc. The afferent limb is provided by sensory fibers, which innervate muscle spindles. These fibers project centrally toward the spinal cord and synapse with alpha motor neurons in the ventral horn. The alpha motor neurons comprise the efferent limb of the reflex arc.
patellar tendon L4 , some L3
Reinforcement - by jendrassik manouvre (interlock fingers) and jaw clenching
Tibialis Posterior
examiner holds the patient's foot in a small amount of eversion and dorsiflexion and strikes the posterior tibial tendon just below the medial malleolus
plantar flexion inversion response
Medial Hamstring
strikes the thumb, which is pressing on the semitendinosus tendon
Achilles' Tendon
dorsiflexes the foot to place the Achilles tendon under tension, and then strikes the Achilles about 3 cm above the calcaneus
unilateral decrease
herniated L5-S1 disk impinging the ipsilateral S1 nerve root.
Bilateral hyperreflexia –
upper motor neuron lesion
undue briskness of the Achilles or the patellar tendon
reflexes - upper motor neuron lesion, carry out provocative tests for ankle clonus and the Babinski sign
Performing the straight-leg raising test on the side opposite that of the sciatica
but it puts little tension on the more proximal nerve roots suggests a lesion of either the L5 or the S1 nerve root.
tightness in the posterior thigh rather than sciatica,
associated with a wide variety of conditions, including spondylolysis.
for confirming the presence of a compressed or irritated lumbar nerve root
SLUMP TEST
back straight, looking straight ahead (Fig. 9-28A). The patient is then encouraged to slump, allowing the thoracic and lumbar spines to collapse into flexion while still looking straight ahead (Fig. 9-28B). The next step is to fully flex the cervical spine (Fig. 9-28C). The patient is then instructed to extend one knee, thus performing a straight-leg raise (Fig. 9-28D). The patient then dorsiflexes the foot on the same side, thus duplicating the Lasegue test
prone on the examination table with the knee flexed to at least 90°. The patient's hip is then extended passively by lifting the thigh off the examination table
When one of the nerve roots that contribute to the femoral nerve is compressed, this maneuver reproduces the patient's radicular pain in the anterior thigh.
stand in the straddle position with one lower limb extended behind the other. The patient is then instructed to lean back as far as possible
procedure is then repeated with the position of the lower limbs reversed
unilateral spondylolysis, hyperextension tends to exacerbate the patient's pain and the pain tends to be more severe when the lower limb on the affected side is extended posteriorly
Weakness involving multiple muscle groups in a nonmyotomal distribution with “give-way” effort
Sensory loss in a segmental pattern (e.g., glove-stocking rather than a dermatomal distribution) observed in a patient in whom polyneuropathy is not an appropriate Diagnosis
Limitations
Widespread superficial tenderness is commonly found in patients with fibromyalgia, and tenderness on deep palpation is part of myofascial pain syndrome
Sensory loss in a glove-stocking distribution is present with peripheral polyneuropathy.
Recent data - Waddell’s signs cannot accurately distinguish between organic and nonorganic
causes of pain
review of prior imaging Studies in light of a newly derived specific historical or physical finding can be particularly helpful
To assess the structure of spine and alignment of vertebra
Spondylolisthesis
Grade 1: 1% to 25% slippage. B, Grade 2: 26% to 50% slippage.
C, Grade 3: 51% to 75% slippage. D, Grade 4: 76% to 100% slippage.
Visulalise spinal cord and nerves in relation to surrounding spine structures (bone, joint, disc, etc)