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JOINT PAIN
Group 14 English Class
dr. Ogirahma
MEMBER OF GROUP 14
110 2016 0004 ANDI M. SHOFWATUL ISLAM HAFID
110 2016 0018 MUHAMMAD SOFHYAN FAJRIN
110 2016 0046 NURUL FITRIAH JUNAID
110 2016 0060 MUSTIKA
110 2016 0076 MUTMAINNA
110 2016 0110 NUR FITRIANY LIHAWA
110 2016 0118 NINADIYAH NURUL AZIZAH
110 2016 0125 S. AHMAD GUFRAN IDRUS
110 2016 0131 ANDI SESARINA TENRI OLA SAPADA
110 2016 0160 NUR AKHSAN DIANA A.R.
110 2016 0171 FIRMAWATI. AR
SCENARIO
A woman, 60 years old, come to community
health center with chief complain pain on
left flank after slipped on the floor 2 hours
earlier. The pain also spread to left hip joint
so that she can’t walk because of the pain.
KEYWORDS
• Woman
• 60 years old
• Complain pain
• On the left flank
• After slipped
• Pain spread
• To the left hip joint
• Can’t walk
• Because of the pain
DIFFICULT WORDS
Pain
The sensation of discomfort that is more or less localized due to
stimulation on the classify nerve endings. It serves as a
protective mechanism because it allows us to withdraw or away
from its source
Dorland, W.A Newman. 2002. Kamus Kedokteran Dorland. Jakarta : EGC. Edisi 29
QUESTIONS
1. Explain about the anatomy and physiology of
joint include the physiology formation of bone!
2. Explain the mechanism of pain and the types of
mechanical and inflammation pain!
3. Explain About Rotating Motion!
4. The Differential Diagnosis based on the scenario!
ANATOMY OF HIP JOINT
Syarifuddin. (2006). Anatomi Fisiologi untuk Mahasiswa Kedokteran, Edisi 3. Jakarta: EGC.
HISTOLOGY OF THE BONE
Vincent J. Vigorita. Orthophaedic Phathology 2nd Edition
PHYSIOLOGY OF THE BONE
Smeltzer, C.S., Bare, G.B., (2001). Buku Ajar Keperawatan Medical Bedah Brunner& Suddarth, Edisi 8, Volume 3, Penerbit EGC,
Jakarta.
MECHANISM OF PAIN
•
Setiyohadi, Bambang, Dkk. 2014. Buku Ajar Ilmu Penyakit Dalam. Jakarta: Interna Publishing.
MECHANISM OF INFLAMMATION
PAIN
MECHANISM OF MECHANICAL
PAIN
ROTATING MOTION
Hip Joint
• Flexion : 130’
• Extension : 30’
• Abduction : 45’
• Adduction : 20’
• Internal Rotation : 40’
• External Rotation : 40’
Nelson Arnold, Kokkonen Jouko,Stretching Anatomy,Human Kinetics,Edition 3,2012, Page 21
4. The Differential Diagnosis based on the
scenario!
OSTEOARTHRITIS TROCHANTERIC
BURSITIS
FRAKTUR CAPUT
FEMORIS
GOUT RA
Definition OA is a
degenerative
joint disease
associated with
joint cartilage
damage.
Irritation or
inflammation of
the trochanteric
bursa
a disconnected
state of the
relationship of
the head of the
femur or the
neck of the
femur caused
by trauma.
is a metabolic
disorder
characterized
by increased
uric acid
concentration
in the
extracellular
fluid
Chronic systemic
inflammatory
disease of
unknown cause
Epidemiology • Men >
woman
• Prevalence
man 15,5 %
and woman
12,7%
• About
people who
aged > 40 th
predominantly
male (80%) with
a mean age of
52 years.
Anserine bursitis
is thought to be
more common in
middle-aged
women, but
there is no
evidence to
substantiate this.
most of these
fractures
occur in
elderly
women
whose bones
are already
osteoporosis
Gout is a
dominant
disease in adult
men and in
women who
have
monopouse
• The
prevalence
rate is
approximatel
y 1%
• between the
ages of 35
and 50
years.
OSTEOARTHRITIS TROCHANTERIC
BURSITIS
FRAKTUR
CAPUT
FEMORIS
GOUT RA
Etiology Age,
(genetic,
nutricional),
obesity,
malaligment,
joint injury,
excessive
joint use, and
muscle
weakness,
degradation of
articular
cartilage
The
trochanteric
bursa may be
inflamed by a
group of
muscles or
tendons
rubbing over
the bursa and
causing friction
against the
thigh bone.
Soft tissue
trauma
(injury
bleeding,
bruising,
partial tear
(sprain),
rupture,
blood vessel
and nerve
disorder
Abnormalities of
uric acid levels
in serum with
accumulation of
crystalline
deposits of
monosodium
urate.
An external
trigger or trauma
that triggers an
autoimmune
reaction, leading
to synovial
hypertrophy and
chronic joint
inflammation
along with the
potential for
extra-articular
manifestations,
OSTEOARTHRITI
S
TROCHANTERIC
BURSITIS
FRAKTUR
CAPUT
FEMORIS
GOUT RA
Mechanism
of injury
Degradation
(failure) of
cartilage,
inflammation
of joint fluid
Inflammation
of the bursa
increases
thinning in the
synovia
Fractures,
factors where
one side of the
bone is
cracked /
swollen / italic
when
monosodium
crystals have
been formed
on the joints.
OSTEOARTHRITI
S
TROCHANTERIC
BURSITIS
FRAKTUR CAPUT
FEMORIS
GOUT RA
hyperplasia of
joint fluid cells
and endothelial
cell activation
are the
incidence of
early
pathological
processes that
develop into
uncontrolled
inflammation
and result in
bone and
cartilage
destruction.
Mansjoer, Arif. 2000. Kapita Selekta Kedokteran. Edisi Ketiga. Jakarta: Media Aesculapius. Page 355-356
Signs of
symptoms
Joint pain, bone
enlargement,
crepitation,
morning
stiffness lasting
< 30 minutes
Pain, swelling of
the bones, heat
and red,
tenderness over
lateral pelvic
region and the
buffer radiating
downward to the
legs / knees.
Pain at night and
more pain if bent
or pressed.
There is a
history of
trauma, pain,
swelling in the
fractured
bone,
deformity,
musculoskelet
al dysfunction
due to pain,
bone
centraction
and
neurovascular
disorders.
Sweeling,
severe pain
Fever,
malaise,
arthalgias,
joint
inflammation
and
sweeling
OSTEOARTHRITIS TROCHANTERIC
BURSITIS
FRAKTUR CAPUT
FEMORIS
GOUT RA
Therapy NSAID
therapy,
health edu,
compress,
surgical
intervention
Local
injections of
corticosteroid
s, NSAIDs,
weight loss,
strengthening
and flexing
gluteus
medius and
iliotibial
muscles.
Bone
grafting
surgery.
NSAID Corticosteroid,
NSAID, surgical
treatment.
OSTEOARTHRITI
S
TROCHANTERIC
BURSITIS
FRAKTUR CAPUT
FEMORIS
GOUT RA
Noor, Zairin. Musculoskeletal Disorders Teachers. Jakarta: Salemba Medika. Page 506-507
Supporting
Examination
Complete
blood tests
include
hemoglobin
and
leukocyte
levels,
synovial test
and X- Ray
Erythrocyte
Sedimentation
Rate test, C-
reaktive
protein test
and X- Ray
X- Ray Synovial
test,
serum
uric acid
test,
complete
blood
tests
and X-
Ray
Erythrocyte
Sedimentation
Rate test, C-
reaktive protein
test and X- Ray,
MRI, Bone
Scanning
OSTEOARTHRITI
S
TROCHANTERIC
BURSITIS
FRAKTUR CAPUT
FEMORIS
GOUT RA
Noor, Zairin. Musculoskeletal Disorders Teachers. Ed. 2. 2015. Jakarta: Salemba Medika.
OSTEOARTHRITIS TROCHANTERIC
BURSITIS
FRAKTUR CAPUT
FEMORIS
GOUT RA
COMPLICATION Pharmacology:
1. NSAIDs
2. Analogues
such as
tramadol.
3. Muscle
relaxants
(muscle
relaxants).
4. Intraarticular
glucocorticoid
injection.
 Nonpharmacolo
gy:
1. Surgical
intervention
2. Atroscopy
3. Osteotomy
4. Fusion
(arthrodesis)
5. Joint
replacement
(arthroplasty)
Pharmacology:
NSAIDs,
acetaminophen,
and corticosteroid
injections
Non
pharmacology:
1. Protection by
debate or by
brace
2. Rest to avoid
the activity of
the joints to
reduce pain
3. Compress with
ice packs
4. Compression
with elastic
bandage
5. Elevation
6. Surgical
therapy
 Pharmacology
1. Antibiotics
2. Analgetic
3. Drugs that
contain lots
of calcium 
triocol
 Nonpharmaco
logy
1. Surgery
internal fixation 
in spongiosum
mounting pen,
wire, or nail
externa fixation 
on the skin
2. Non surgery
Installation of a
cast
Skin tractionn
skelet traction
Pharmacology:
1. NSAID's :
2. Colsichine.
3. Corticosteroi
d.
4. Probenecid
5. Allopurinol.
6. Uricosuric.
 Nonpharmaco
logy:
Surgical
intervention
 Pharmacologic
therapy
1. DMARD's
2. Glucocorticoids
3. NSAIDs
4. Analgesic
 Nonpharmacolo
gic therapy
1. Fasting therapy
2. Spa and
exercise
3.Supplementation
of essential fatty
acids
4. Provision of fish
oil supplements
5. Provide
education and
multidisciplinary
approach in patient
care
6. Surgery
• X- Ray
• Computed Tomography (Ct-scan)
• Magnetic Resonance Imaging (MRI)
• Angiography
• Digital Subtraction Angiography (DSA)
• Venogram
• Mielography
• Discography
• Artography
Sukman, Ningsih Nurna. 2009. Asuhan Keperawatan Pada Klien Dengan Gangguan Sistem Muskuloskeletal. Jakarta : Salemba Medika.
RADIOGRAPH IMAGE
Fraktur Collum Femoris Gout
KOREKSIAN!
• KATA KUNCI, BUKAN KALIMAT KUNCI
• EX : - WOMAN
• - 60 YEARS OLD
• - etc
• CANTUMKAN SEMUA PERTANYAAN DLM 1 SLIDE, KMDIAN JWBN
DIBELAKANG
• LENGKAPI EPIDEMIOLOGI
• PEMERIKSAAN PENUNJANG MASUKKAN DI TABEL MASING-MASING DD
• PISAHKAN TERAPI NONFARMAKOLOGI&FARMAKOLOGI
• MASUKKAN KOMPLIKASI PENYAKIT & PREVENTIVE

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Pbl english class power point klp 14 joint pain 2

  • 1. JOINT PAIN Group 14 English Class dr. Ogirahma
  • 2. MEMBER OF GROUP 14 110 2016 0004 ANDI M. SHOFWATUL ISLAM HAFID 110 2016 0018 MUHAMMAD SOFHYAN FAJRIN 110 2016 0046 NURUL FITRIAH JUNAID 110 2016 0060 MUSTIKA 110 2016 0076 MUTMAINNA 110 2016 0110 NUR FITRIANY LIHAWA 110 2016 0118 NINADIYAH NURUL AZIZAH 110 2016 0125 S. AHMAD GUFRAN IDRUS 110 2016 0131 ANDI SESARINA TENRI OLA SAPADA 110 2016 0160 NUR AKHSAN DIANA A.R. 110 2016 0171 FIRMAWATI. AR
  • 3. SCENARIO A woman, 60 years old, come to community health center with chief complain pain on left flank after slipped on the floor 2 hours earlier. The pain also spread to left hip joint so that she can’t walk because of the pain.
  • 4. KEYWORDS • Woman • 60 years old • Complain pain • On the left flank • After slipped • Pain spread • To the left hip joint • Can’t walk • Because of the pain
  • 5. DIFFICULT WORDS Pain The sensation of discomfort that is more or less localized due to stimulation on the classify nerve endings. It serves as a protective mechanism because it allows us to withdraw or away from its source Dorland, W.A Newman. 2002. Kamus Kedokteran Dorland. Jakarta : EGC. Edisi 29
  • 6. QUESTIONS 1. Explain about the anatomy and physiology of joint include the physiology formation of bone! 2. Explain the mechanism of pain and the types of mechanical and inflammation pain! 3. Explain About Rotating Motion! 4. The Differential Diagnosis based on the scenario!
  • 7. ANATOMY OF HIP JOINT Syarifuddin. (2006). Anatomi Fisiologi untuk Mahasiswa Kedokteran, Edisi 3. Jakarta: EGC.
  • 8. HISTOLOGY OF THE BONE Vincent J. Vigorita. Orthophaedic Phathology 2nd Edition
  • 9. PHYSIOLOGY OF THE BONE Smeltzer, C.S., Bare, G.B., (2001). Buku Ajar Keperawatan Medical Bedah Brunner& Suddarth, Edisi 8, Volume 3, Penerbit EGC, Jakarta.
  • 10. MECHANISM OF PAIN • Setiyohadi, Bambang, Dkk. 2014. Buku Ajar Ilmu Penyakit Dalam. Jakarta: Interna Publishing.
  • 13. ROTATING MOTION Hip Joint • Flexion : 130’ • Extension : 30’ • Abduction : 45’ • Adduction : 20’ • Internal Rotation : 40’ • External Rotation : 40’ Nelson Arnold, Kokkonen Jouko,Stretching Anatomy,Human Kinetics,Edition 3,2012, Page 21
  • 14. 4. The Differential Diagnosis based on the scenario! OSTEOARTHRITIS TROCHANTERIC BURSITIS FRAKTUR CAPUT FEMORIS GOUT RA Definition OA is a degenerative joint disease associated with joint cartilage damage. Irritation or inflammation of the trochanteric bursa a disconnected state of the relationship of the head of the femur or the neck of the femur caused by trauma. is a metabolic disorder characterized by increased uric acid concentration in the extracellular fluid Chronic systemic inflammatory disease of unknown cause
  • 15. Epidemiology • Men > woman • Prevalence man 15,5 % and woman 12,7% • About people who aged > 40 th predominantly male (80%) with a mean age of 52 years. Anserine bursitis is thought to be more common in middle-aged women, but there is no evidence to substantiate this. most of these fractures occur in elderly women whose bones are already osteoporosis Gout is a dominant disease in adult men and in women who have monopouse • The prevalence rate is approximatel y 1% • between the ages of 35 and 50 years. OSTEOARTHRITIS TROCHANTERIC BURSITIS FRAKTUR CAPUT FEMORIS GOUT RA
  • 16. Etiology Age, (genetic, nutricional), obesity, malaligment, joint injury, excessive joint use, and muscle weakness, degradation of articular cartilage The trochanteric bursa may be inflamed by a group of muscles or tendons rubbing over the bursa and causing friction against the thigh bone. Soft tissue trauma (injury bleeding, bruising, partial tear (sprain), rupture, blood vessel and nerve disorder Abnormalities of uric acid levels in serum with accumulation of crystalline deposits of monosodium urate. An external trigger or trauma that triggers an autoimmune reaction, leading to synovial hypertrophy and chronic joint inflammation along with the potential for extra-articular manifestations, OSTEOARTHRITI S TROCHANTERIC BURSITIS FRAKTUR CAPUT FEMORIS GOUT RA
  • 17. Mechanism of injury Degradation (failure) of cartilage, inflammation of joint fluid Inflammation of the bursa increases thinning in the synovia Fractures, factors where one side of the bone is cracked / swollen / italic when monosodium crystals have been formed on the joints. OSTEOARTHRITI S TROCHANTERIC BURSITIS FRAKTUR CAPUT FEMORIS GOUT RA hyperplasia of joint fluid cells and endothelial cell activation are the incidence of early pathological processes that develop into uncontrolled inflammation and result in bone and cartilage destruction. Mansjoer, Arif. 2000. Kapita Selekta Kedokteran. Edisi Ketiga. Jakarta: Media Aesculapius. Page 355-356
  • 18. Signs of symptoms Joint pain, bone enlargement, crepitation, morning stiffness lasting < 30 minutes Pain, swelling of the bones, heat and red, tenderness over lateral pelvic region and the buffer radiating downward to the legs / knees. Pain at night and more pain if bent or pressed. There is a history of trauma, pain, swelling in the fractured bone, deformity, musculoskelet al dysfunction due to pain, bone centraction and neurovascular disorders. Sweeling, severe pain Fever, malaise, arthalgias, joint inflammation and sweeling OSTEOARTHRITIS TROCHANTERIC BURSITIS FRAKTUR CAPUT FEMORIS GOUT RA
  • 19. Therapy NSAID therapy, health edu, compress, surgical intervention Local injections of corticosteroid s, NSAIDs, weight loss, strengthening and flexing gluteus medius and iliotibial muscles. Bone grafting surgery. NSAID Corticosteroid, NSAID, surgical treatment. OSTEOARTHRITI S TROCHANTERIC BURSITIS FRAKTUR CAPUT FEMORIS GOUT RA Noor, Zairin. Musculoskeletal Disorders Teachers. Jakarta: Salemba Medika. Page 506-507
  • 20. Supporting Examination Complete blood tests include hemoglobin and leukocyte levels, synovial test and X- Ray Erythrocyte Sedimentation Rate test, C- reaktive protein test and X- Ray X- Ray Synovial test, serum uric acid test, complete blood tests and X- Ray Erythrocyte Sedimentation Rate test, C- reaktive protein test and X- Ray, MRI, Bone Scanning OSTEOARTHRITI S TROCHANTERIC BURSITIS FRAKTUR CAPUT FEMORIS GOUT RA Noor, Zairin. Musculoskeletal Disorders Teachers. Ed. 2. 2015. Jakarta: Salemba Medika.
  • 21. OSTEOARTHRITIS TROCHANTERIC BURSITIS FRAKTUR CAPUT FEMORIS GOUT RA COMPLICATION Pharmacology: 1. NSAIDs 2. Analogues such as tramadol. 3. Muscle relaxants (muscle relaxants). 4. Intraarticular glucocorticoid injection.  Nonpharmacolo gy: 1. Surgical intervention 2. Atroscopy 3. Osteotomy 4. Fusion (arthrodesis) 5. Joint replacement (arthroplasty) Pharmacology: NSAIDs, acetaminophen, and corticosteroid injections Non pharmacology: 1. Protection by debate or by brace 2. Rest to avoid the activity of the joints to reduce pain 3. Compress with ice packs 4. Compression with elastic bandage 5. Elevation 6. Surgical therapy  Pharmacology 1. Antibiotics 2. Analgetic 3. Drugs that contain lots of calcium  triocol  Nonpharmaco logy 1. Surgery internal fixation  in spongiosum mounting pen, wire, or nail externa fixation  on the skin 2. Non surgery Installation of a cast Skin tractionn skelet traction Pharmacology: 1. NSAID's : 2. Colsichine. 3. Corticosteroi d. 4. Probenecid 5. Allopurinol. 6. Uricosuric.  Nonpharmaco logy: Surgical intervention  Pharmacologic therapy 1. DMARD's 2. Glucocorticoids 3. NSAIDs 4. Analgesic  Nonpharmacolo gic therapy 1. Fasting therapy 2. Spa and exercise 3.Supplementation of essential fatty acids 4. Provision of fish oil supplements 5. Provide education and multidisciplinary approach in patient care 6. Surgery
  • 22. • X- Ray • Computed Tomography (Ct-scan) • Magnetic Resonance Imaging (MRI) • Angiography • Digital Subtraction Angiography (DSA) • Venogram • Mielography • Discography • Artography Sukman, Ningsih Nurna. 2009. Asuhan Keperawatan Pada Klien Dengan Gangguan Sistem Muskuloskeletal. Jakarta : Salemba Medika.
  • 24. KOREKSIAN! • KATA KUNCI, BUKAN KALIMAT KUNCI • EX : - WOMAN • - 60 YEARS OLD • - etc • CANTUMKAN SEMUA PERTANYAAN DLM 1 SLIDE, KMDIAN JWBN DIBELAKANG • LENGKAPI EPIDEMIOLOGI • PEMERIKSAAN PENUNJANG MASUKKAN DI TABEL MASING-MASING DD • PISAHKAN TERAPI NONFARMAKOLOGI&FARMAKOLOGI • MASUKKAN KOMPLIKASI PENYAKIT & PREVENTIVE