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Essential
Clinical skill
“ จากพี่...สู่น้อง สู่มวลชน ”
The last pre-clinic course
อ.นพ.ปฏิญญา ยุทธชาวิทย์
ตรวจทาน : อ.พญ.ศุภวดีวงศ์นิจศีล
ขอขอบคุณเพื่อนๆพี่ๆแพทย์ขอนแก่น รุ่นที่ 37 และ 38 ที่รักทุกคน
คณะแพทยศาสตร์ มหาวิทยาลัยมหาสารคาม
SPEEDY ECG!
MIXED! (BASICS AND USES)
สอนและปรับปรุง โดย
อ.นพ.ปฏิญญา ยุทธชาวิทย์
คณะแพทยศาสตร์ มหาวิทยาลัยมหาสารคาม
สาหรับนักศึกษาแพทย์ชั้นปี 3
True education is what remains
after one has forgotten
what one has learned
in school.
-Albert Einstein
STEPS !
• Part I :To know ECG
• What is ECG ?
• Anatomy of heart and (electro)physiology
• Anatomy of ECG
• Part II :To use ECG
• Leads placement positions
• Read ECG like a pro (?) : Step by step
• Ischemic pattern
• Need-to-know ECGs
TO KNOW ECG…
What is ECG ?
Anatomy of heart and (electro)physiology
Anatomy of ECG
WHAT IS ECG ?
• An electrocardiogram (ECG / EKG) is an electrical
recording of the heart and is used in the investigation of
heart disease.
• 1895 ;Willem Einthoven invented ECG.
• A representation of electrical events
in cardiac cycle.
• Each events has a distinctive waveform.
• Study ECG >> study shadow of heart.
Look from different aspects.
ANATOMY OF HEART
ANATOMY OF HEART
CONDUCTION PATHWAY
Depolarized at SA node
Internodal pathway to AV node
interatrial tract
to Lt. atrium
Bundle of His
Left and right
bundle branches
Purkinje fiber
to contractile cell
(~0.05 m/sec)
(~ 4 m/sec)
CONDUCTION PATHWAY
Depolarized at SA node
Internodal pathway to AV node
interatrial tract
to Lt. atrium
Bundle of His
right
bundle branches
Purkinje fiber
to contractile cell
Left
bundle branches
Purkinje f
to contra
Purkinje fiber
to contractile cell
Purkinje fiber
to contractile cell
Purkinje fiber
to contractile ce
Purkinje fiber
to contractile cell
Purkinje fibe
to contractil
Purkinje fibe
to contracti
Purkinje fiber
to contractile cell
Purkinje fiber
to contractile cell
Purkinje fiber
to contractile cell
ELECTROPHYSIOLOGY
2Types of cardiac cells
• Myocardial cell
• Mechanical
• Cab be electrical stimulated
• Cannot generate electrical itself
• Pacemaker cell : SA node,AV node
• Generate electrical impulse spontaneously
ELECTROPHYSIOLOGY
ELECTROPHYSIOLOGY
ELECTROPHYSIOLOGY
ELECTROPHYSIOLOGY
https://www.osmosis.org/
ELECTROPHYSIOLOGY
ELECTROPHYSIOLOGY
ELECTROPHYSIOLOGY
ELECTROPHYSIOLOGY
ELECTROPHYSIOLOGY
ELECTROPHYSIOLOGY
ELECTROPHYSIOLOGY
ELECTROPHYSIOLOGY
Depolarization, Repolarization
+-
Depolarization wave
+ -
Depolarization wave
Repolarization wave
-+
ELECTROPHYSIOLOGY
ELECTROPHYSIOLOGY
ANATOMY OF ECG AND
CORRELATION
https://www.youtube.com/user/ECGTeacher
ANATOMY OF ECG
…THE PAPER
Basics
• ECG paper has many squares
• Big square = 5 small square
• Each small square = 1 mm in width and
height (1mm x 1 mm)
• Rate of paper
• 25 mm/sec
• So 0.04 sec/1 mm (small square)
• Or 0.2 sec/ 5 mm (large square)
• Electrode and machine record
• 1mV = 10 mm
• So 0.1 mV/1 mm (small square)
ANATOMY OF ECG
…ABOUT 12 LEADS
Electrodes ≠ leads ; basic 10 electrodes make 12 leads
3 types of leads
• Bipolar limb leads (frontal plane):
• Lead I: RA (-) to LA (+) (Right Left, or lateral)
• Lead II: RA (-) to LL (+) (Superior Inferior)
• Lead III: LA (-) to LL (+) (Superior Inferior)
• Augmented unipolar limb leads (frontal plane):
• Lead aVR: RA (+) to [LA & LL] (-) (Rightward)
• Lead aVL: LA (+) to [RA & LL] (-) (Leftward)
• Lead aVF: LL (+) to [RA & LA] (-) (Inferior)
• Unipolar (+) chest leads (horizontal plane):
• Leads V1, V2, V3: (Posterior Anterior)
• Leads V4, V5, V6:(Right Left, or lateral)
ANATOMY OF ECG
…ABOUT 12 LEADS
ANATOMY OF ECG
…ABOUT 12 LEADS
ANATOMY OF ECG
…ABOUT 12 LEADS
ANATOMY OF ECG
‘PQRSTU’
NORMAL ECG COMPLEX
 PWave
- depolarization of the atria
- normal sinus rhythm: the P wave is upright in leads I, II, aVF,V4,V5, andV6 and
inverted in aVR
 QRS Complex
- ventricular depolarization
V1,V2; Rt.Ventricle
V3,V4; septum (R wave = S wave ; transition point”)
If Rt.Ventricle enlargement;transition pointV4/V5 orV5/V6
V5,V6; Lt. ventricle
THE ‘PQRSTU’
 QRS complex
 QWave. The first negative deflection of the QRS complex
(not always present)
pathologic:the Q wave should be > 25% of the QRS complex
 RWave: The first positive deflection
 S Wave :The negative deflection following the R wave
THE ‘PQRSTU’
• TWave
- repolarization of the ventricles and follows the QRS complex
- normal : upright in leads I, II,V3,V4,V5, andV6 and inverted in
aVR
… AndWhere’s
atrial depolarization ???
THE ‘PQRSTU’
Intervals (PR, QRS, and QT intervals)
measured in the limb leads (ดู lead II ง่าย)
1. PR interval = 0.12–0.20 s (3-5 ช่องเล็ก)
2.QRS duration < 0.12 s (เล็กกว่า 3 ช่องเล็ก) ***
3. QT interval increases with decreasing heart rate,
usually < 0.44 s
* QT interval (not exceed one half of the RR
interval)
THE ‘PQRSTU’
TO USE ECG…
Electrodes placement positions
Read ECG like a pro (?) : Step by step
Ischemic pattern
Need-to-know ECGs
Price of ECG depends on how you read it.
The more you know, the more you see.
The more you see, the more you know!
ELECTRODES PLACEMENT
POSITIONS
ซ้ายกาขี้
ขวากาเลือด
ซ้ายเหยียบหญ้า
ขวาเหยียบดิน
ELECTRODES PLACEMENT
POSITIONS
Midclavicular line
Midaxillary line
V1: 4 intercostal space to
right sternum,
V2: 4 intercostal space to
left sternum
V3: halfway betweenV2-V4,
V4: 5 intercostal space at
the midclavicle Line
V6: 5 intercostal space at
the madaxillary Line,
V5: halfway betweenV4-V6
READ ECG LIKE A PRO (?)
• Rhythm
• Rate
• Axis
• Chamber enlargement
• Myocardial ischemia and infarction
• Miscellaneous
• Drug effect
• Electrolyte imbalance
NORMAL SINUS RHYTHM
Rhythm
• ทุก QRS ตามหลัง P wave โดย QRS เป็น positive ใน II และเป็น negative ใน aVR
• regular PR and RR interval
• rate between 60 and 100 beats/min
ABNORMAL RHYTHM
• Varying rhythm
• Extra beats
and skip
• Heart block
• Total irregularity
• Basically regularity
• Regular irregularity
(Grouping beat)
Rhythm
VARYING RHYTHM
Normal Sinus Rhythm
Atrial Fibrillation
Rhythm
A Fib
Totally irregular
Can’t identify/
fibrillated p wave
Sinus bradycardia
Sinus tachycardia
Rhythm
Sinus tach
Sinus rhythm
Rate > 100
Other normal
Sinus brad
Sinus rhythm
Rate < 60
Other normal
Sinus pause
Atrial flutter
Rhythm
AF
Saw tooth wave
Atrial rate near 300
Sinus pause
Sinus rhythm
Sometime sinus
fails to pace
Ventricular fibrillation
Ventricular tachycardia
Rhythm
VT
Fast ventricular rate
100-150
Wide QRS
Advanced life support!
(tachy)
VF
Severely
unorganized
CPR!!
Wide QRS ; > 3 ช่องเล็ก
(0.12 s)
Torsades de Pointes
Supraventricular tachycardia
Rhythm
SVT
Fast atrial and
ventricular rate
100-250
narrow QRS
Advanced life support!
(tachy, adenosine)
Torsades
Twist around point
PolymorphicVT
Advanced life support!
(tachy, MgSO4)
Premature ventricular beat
Asystole
Rhythm
Asystole
No electrical activity
Check leads!
CPR!!
PVCs
Ventricle >> extra beat
Wide QRS
Occasional irregular
Premature atrial contraction
Atrial tachycardia
Rhythm
PACs
Supraventricular origin
Narrow QRS
Occasional irregular
Compensatory/ non-
compensatory pause
AF
A Fib
MAT ; multifocal atrial tach
Pre-excitation syndrome
+- accessory pathway (WPW)
AVRT
AVNRT
ไว้รอเรียน
ตอนโตกว่านี้
ก็ได้ครับ
Pulseless electrical activity
Rhythm
PEA
Any ECGs
That produce no pulse
ExceptVT,VFNo pulse !!
HEART BLOCK
First degree
Second degree
Third degree
Rhythm
PR interval ยาวกว่า 5 ช่องเล็ก (0.2s)
SECOND DEGREE
Mobitz I
Mobitz II
Rhythm
RATE
Rate
• Rule of 300
• จงนับช่องระหว่าง RR ว่ามีกี่ช่องใหญ่
• แล้วนามาหาร 300
• Rate = 300/N
= 1500/n
N=จานวนช่องใหญ่ , n = จานวนช่องเล็ก
RATE
Rate
• ทาไมหล่ะ ?
• กระดาษไหล 25 mm/sec
• เท่ากับ 25x60=1500 mm/60 sec
• 1500 ช่องเล็ก/นาที
• 300 ช่องใหญ่/นาที
• ดังนั้นถ้าความถี่ QRS 300 ครั้งต่อนาที
(HR=300 bpm) จะเกิด QRS ทุก
ตาแหน่ง 1 ช่องใหญ่พอดี ก็เทียบ
บัญญัติไตรยางค์
RATE
Rate
RATE
Sinus Tachycardia
Sinus Bradycardia
Rate
RATE
Rate
• กรณี QRS มาไม่สม่าเสมอ
• ให้นับจานวน QRS ให้หมด
ใน 10 วินาที (50 ช่องใหญ่)
• นับใน lead II/lead ยาวสุดจะสะดวก
• นับแล้ว x 6 = rate
• ปกติ ECG record 10 sec อยู่แล้ว
33 x 6 = 198 bpm
AXIS
Axis
ELECTRICAL AXIS
Axis
AXIS MADE IT EASY!
Axis
Axis
ELECTRICAL AXIS
Axis
ELECTRICAL AXIS
Axis
ELECTRICAL AXIS
Axis
CHAMBER ENLARGEMENT
AND HYPERTROPHY
• Right atrium enlargement
• Peak of P wave > 2.5 mm
• Left atrium enlargement
• Width of P wave > 0.12 seconds
• Right ventricular hypertrophy
• R/S wave ratio in leadV1 > 1 + RAD
• Left ventricular hypertrophy
• S in leadV1 + R wave in leadV5 orV6 > 35 mm
Chamber
enlargement
CHAMBER ENLARGEMENT
AND HYPERTROPHY
• Check P wave for atrial enlargement
• Check R wave in leadV1 for RV hypertrophy
(RAD)
• Check S wave in leadV1 and R wave in lead
V5, 6 for LV hypertrophy
Chamber
enlargement
THE P WAVE
Chamber
enlargement
ABNORMAL P WAVE
RA enlargement
(P pulmonale)
LA enlargement
(P mitralae)
Biatrial
enlargement
Chamber
enlargement
RIGHT ATRIUM ENLARGEMENT
Chamber
enlargement
RIGHT ATRIAL ENLARGEMENT
Chamber
enlargement
LEFT ATRIAL ENLARGEMENT
Chamber
enlargement
RIGHT VENTRICULAR
HYPERTROPHY
Chamber
enlargement
LEFT VENTRICULAR
HYPERTROPHY
Chamber
enlargement
MYOCARDIAL ISCHEMIA, INJURY,
AND INFARCTION
• ST-T segments depression andT wave
inversion
• myocardial ischemia
• ST -T elevation
• myocardial injury
• Q wave
• myocardial infarction
Ischemic pattern
CORONARY SUPPLY MYOCARDIUM
Ischemic pattern
CORONARY DISEASE
TRANSMURAL
• ISCHEMIA : invert T
• INJURY : ST elevate
• INFARCTION : Q wave
• RECIPROCAL
CHANGE
SUBENDOCARDIAL
• ISCHEMIA: QT prolong , upright
tallT
• INJURY : ST depress
• INFARCTION : symmetrical
deep inverted T, ST depress
• RECIPROCAL
CHANGE
Ischemic pattern
CORONARY DISEASE
Ischemic pattern
Q-WAVE
Ischemic pattern
ST-SEGMENT
Ischemic pattern
• Flattening
• ST depress
– Plain ST depress (Horizontal)
– Non plain ST depress
• Downslope pattern : dig intox
• Reverse correct check mark : strain pattern
ABNORMAL ST SEGMENT
(ST DEPRESSION)
Normal Flattening
ST segment
Horizontal
ST depress
Downsloping
ST segment
Ischemic pattern
ST DEPRESS IN ISCHEMIA
Ischemic pattern
ST DEPRESS IN UNSTABLE ANGINA
Ischemic pattern
NON-ISCHAEMIC ST DEPRESS
Digoxin effect
Strain from
Hypertrophy
Ischemic pattern
T WAVE IN ISCHEMIA
Ischemic pattern
INVERTED T WAVE IN UA
Ischemic pattern
BIPHASIC T WAVE IN UA
Ischemic pattern
ABNORMAL ST SEGMENT
(ST ELEVATION)
• Elevate
• convex upward
• Injury
• Aneurysm
• concave upward
• early repolarization
• Pericarditis
• Reciprocal strain in hypertrophy
• nonspecific
Ischemic pattern
ST ELEVATE IN LV ANEURYSM
• QS pattern
• ST elevated
• T wave inversion
Ischemic pattern
ST ELEVATE IN EARLY REPOLARIZATION
• Common in M>FM
• Common in black
• Begins elevated J point and
usually concave form,
commonly associated with
notching of the
downstroke of the QRS
complex and can reach 500
microvolte in amplitude
Ischemic pattern
ST ELEVATION IN PERICARDITIS
Ischemic pattern
ST ELEVATION HYPERTROPHY
Ischemic pattern
ANATOMY OF ECG
…ABOUT 12 LEADS
Ischemic pattern
MYOCARDIAL ISCHEMIA
Ischemic pattern
SUBENDOCARDIAL ISCHEMIA
Ischemic pattern
MYOCARDIAL INJURY
Ischemic pattern
MYOCARDIAL INFARCTION
Ischemic pattern
HEART BLOCK
First degree
Second degree
Third degree
Other
Other
BUNDLE BRUNCH BLOCK
COMPREHENSION
RBBB CRITERIA
• Widening QRS > 0.12
sec
• S in I
• R’ inV1
• Repolarization
abnormality
Other
RBBB
Right bundle branch block manifest by prominent S wave in leads I, aVl , and
the left ventricular precordial leads and an rsR pattern in lead V1 . The left
anterior fascicular block is indicated by the marked left-axis deviation in the
frontal leads.
Other
LBBB CRITERIA
• Widening QRS > 0.12 sec
• Notch R (M shape)
inV5-6
• Negative QRS inV1-2
• Repolarization
abnormality
Other
Other
LBBB
A FLUTTER
Other
AF
Other
VENTRICULAR TACHYCARDIA
• Wide complex tachycardia.Atrioventricular dissociation
confirms ventricular origin.
Other
HYPERKALEMIA
Other
HYPOKALEMIA
Other
DIGITALIS EFFECT
Other
Salvador Dali sagging
CARDIAC TEMPONADE
• Low voltage , electrical alternan
Other
PERICARDITIS
Other
• ST elevate all lead (Concave elevation)
COR PULMONALE (E.G. PE)
Other
• Abnormal wave - S in lead I - Q กว้างกว่า 1.5ช่อง lead III
- invertedT in lead III
BRUGADA SIGN
Other
• Coved ST segment elevation >2mm in >1 ofV1-V3 followed by a negativeT wave
• Abnormal cardiac Na+ channel
• Sudden death สัมพันธ์กับโรคไหลตาย
WOLFF-PARKINSON-WHITE
Other
• Delta wave ; slurring slow rise of
initial portion of the QRS
• Syndrome is a combination of
the presence of a congenital
accessory pathway and
episodes of tachyarrhythmia.
• Bundle of Kent
QUIZ TIME?!
Let’s see what remains…
ดูในเอกสาร Speedy ECG (exercise)
Thank you!
A little bit hard today…
>w<!!

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