Hipsters Unite! Explore the hip's role in patients with low back pain
1. Hipsters Unite!
Examining the hip’s influence for patients with back pain.
Eric K. Robertson, PT, DPT, OCS, FAAOMPT
Assistant Professor
Texas State University
2. Hipster Defined
What is a hipster?
You‘d be surprised. It could very well be YOU.
A hipster is an individual—one that usually fits within a
certain subculture. Which subculture? It doesn‘t
matter. Because the definition for hipster is so very
vague
In its most simplest terms, a hipster is an individual
who wants to know things.
Source: quotes from a Hipster named Adam
(http://travelsofadam.com/what-is-a-hipster/)
3.
4.
5. Fact #52
―If you were to lay every hipster in the world
end to end, we don‘t know the length of the
line they would form, but there would be an
Apple store at the end of the line.‖
6. Austin: Epic Hipster Population
*Ranked #2 behind Seattle, WA for hipsterism by designtrends.com
7.
8. Actual Austin Hipsters
A confirmation that a) hipsters do exist in a nearby geographic
locale, and that b) sometimes they gather, and that c)
whatever, I‘m going to just write what I want in the true spirit of
hipsterism.
9. Actually What This Talk is REALLY About:
An examination into the age-old concept that a) yes, the hip bone is
connected to the back bone, that b) when one hurts, the other most
often suffers, and that c) by paying attention to trends in movement
patterns we can flesh out how to direct treatments at the hip that may
assist us in treating patients with low back pain.
10. Actually What This Talk is REALLY About:
An examination into the age-old concept that a) yes, the hip bone is
connected to the back bone, that b) when one hurts, the other most
often suffers, and that c) by paying attention to trends in movement
patterns we can flesh out how to direct treatments at the hip that may
assist us in treating patients with low back pain.
15. Which of the following are predictors of LBP?
• Bulging disc without
herniation or root • History of depression
contact • History of
• Bulging disc without occupational-related
herniation but with LBP
nerve root contact • Fearful beliefs about
• Herniated/Prolapsed work as reported in a
discs survey
• End plate changes /
Shmorl‘s nodes
• Foraminal or canal Psychosocial
stenosis findings
Physical findings
/ Imaging
16. How did you do?
Physically:
Only disc contact with nerve root has been shown
to be a WEAK predictor of LBP
Psychosocial:
Depression, occupational injuries, and fear-
avoidance are all STRONG predictors of LBP
Implications in terms of pain?
18. The Hip – Spine Relationship
It‘s hip to have a high yield!
19. The Hip Bone‘s Connected to the…
Hip – Spine Syndrome (Offierski and MacNab, 1983)
Proposed a formal relationship between hip and spine pain
Three subgroups
Simple – Primary Dysfunction of one area, but symptoms in
another
Complex – Dysfunction in both hip and spine
Secondary – Hip and Spine pain are dependent and inter-related
This tight hip flexors and increased lumbar lordosis, as example
22. Hip – Spine Syndrome
Though frequently cited, few have expanded upon the work of
Offierski and MacNab.
Ben-Galim et al., 2007 – Effects of THA on LBP
25 individuals with severe hip OA, received THA
No radiographic spine changes, but baseline LBP was 5.8 on VAS
Subjects had PT for ambulation, hip motion, but not for lumbar spine
23. Ben-Galim et al., 2007
Pain - LBP Disability - LBP
6 40
5
30
4
3 20
2
10
1
0 0
VAS ODI
24. More recently…
10 Saito et al, Spine, May 2012
Case series:
4 patients with leg
pain and confirmed
spinal stenosis and Hip
OA.
1
Fusion Helped by THA Helped by
Fusion TKA
―I‘m sorry we fused your spine…‖
―Conclusions. It is difficult to determine the origin of lower
leg pain by spinal nerve block and hip joint block in
patients with lumbar spinal stenosis and hip osteoarthritis.
We take this into consideration before surgery.‖
25. So let‘s think about this connection…
How can we evolve as hipsters?
33. Normal Lumbopelvic Rhythm
Lumbar spine and hips contribute to flexion in equal
magnitudes.
Early flexion from the lumbar spine
Later flexion from the hip contribution
Extension is the reverse, with muscle activation
occurring from a caudad to cephalad direction
Twisting motions are primarily from hip contribution
Lee & Wang
34. How about with LBP?
We move slower, and with less magnitude.
Hip:Spine Ratio (Shum et al, 4 studies)
Normal ratio: 0.50
LBP: 0.38-0.40 (reduced lumbar spine contribution)
Perhaps even less lumbar motion in those with +SLR
Peak angular velocity also reduced for individuals with LBP
35. Shum et al. Contribution
Examined patients with and without LBP
Looked at:
Simple sit-stand
Sock donning
Twisting
Picking up and object from a seated position
Altered movement strategies may be a strategy to protect
injured lumbar tissues
These changes are larger in those with neural tension signs
36. Lumbopelvic Motion
Many authors agree that more lumbar motion, earlier in the
range, especially with rotation is apparent in patients with low
back pain. (Burnett et al., 2004; Esola et al., 1996; Luomajoki et al., 2008;
McClure et al., 1997; Roussel et al., 2009)
Inconclusive overall.
Heterogeneous populations?
Can sub-grouping help us?
37. Low Back Pain Classification
Systems:
McKenzie-based
Treatment-based classification
Movement System Impairment
Commonalities?
Movement
Symptoms reproduced matters
Hip motion matters
38. Low Back Pain Classification
Systems:
McKenzie-based
Treatment-based classification
Movement System Impairment
Commonalities?
Movement
Symptoms reproduced matters
Hip motion matters
39. Treatment-based Classification
Hip Influence:
Spinal Manipulation and Exercise Group
CPR:
< 16 days
No pain beyond knee
Hypomobile lumbar spine accessory motion
Low Fear-Avoidance Beliefs
Hip Motion (>35 degrees)
Also, we see something interesting in the Stability subgroup…
Important factors: Age, recurrent pain, SLR ROM, etc
40. Arthrokinematics of a Subgroup
Teyhen et al, 2007 in PTJ
Examined subgroup
indentified to have suspected
spinal instability per CPR
Fluroscopic computer analysis
Found segmental HYPO-mobility in
this subgroup!
41. Hip Hypomobility with LBP
Tafazzoli and Lamontagne, 1996
With LBP:
Increased passive elastic moment on the hamstrings
Increased hip stiffness to oscillatory motions
A rationale to treat the hips for patients presenting with primary
LBP
42. More Hip Trouble from LBP
Reduced Hip Flexion in patients with LBP
Wong and Lee, 2004
3-dimensional analysis of functional motion in patients with LBP
Increased ER compared to IR in patients with LBP
Cibulka et al, 1998
Examined over 100 patients with LBP
43. Low Back Pain Classification
Systems:
McKenzie-based
Treatment-based classification
Movement System Impairment
Commonalities?
Movement
Symptoms reproduced matters
Hip motion matters
44. Movement System Impairment System
People with LBP demonstrate earlier and greater lumbopelvic
rotation during hip lateral rotation compared to people
without LBP
5 groups (based on history, symptoms with movement and alignment)
lumbar extension rotation syndrome
lumbar extension syndrome
lumbar flexion rotation syndrome
lumbar flexion syndrome
lumbar rotation syndrome
Important exam items related to the hip:
Hip Ext, Rot ROM, Hip abduction with lateral rotation in hook-
lying, relative hip flexibility, hip motion in supine
Van Dillen, Sahrmann, et al. 2003
45. Hip Musculature
and LBP
If we‘re moving differently, we‘re using our
muscles differently, right?
46. Neuromuscular control of walking
with chronic low-back pain
Vogt et al., 2003, Manual Therapy
17 male subjects, hip ROM, surface EMG during gait
Subjects with moderate low back pain had:
Decreased hip motion and stride time
Decreased hip extensor and lumbar spine extensor muscle activity
Persistent firing of the above noted muscle groups compared to
controls
Leinonen et al., 2000 – observed similar decreased
gluteus maximus activation in patients with LBP
47. Lumbopelvic Stabilization
Hungerford et
al., 2003, Spine.
EMG analysis of 14 individuals
with SIJ pain compared to
age-matched controls
Reduced activation of
Internal oblique
Multifidus
Gluteus maximus
48. Where We Stand As Hipsters
Diagnostic Dilemma
Patterns of Known Impairments of the hip in patients with low
back pain
Presence of hip-based factors in classification systems for
clinicians to use when treating patients with low back pain.
Practically Speaking: Education about the role of the hip in low
back pain is something that people tend to teach in weekend
courses and at conferences like this. It‘s not something much
research has supported.
49. Evidence for
Interventions
What guidance do we have concerning the
hip-spine connection?
Image by mpascoe via Flickr
50. Regional Interdependence Defined
―Seemingly unrelated impairments in a remote anatomical
region may contribute to, or be associated with, the patient‘s
primary complaint.‖
Wainner, Whitman, Cleland & Flynn. Regional Interdependence: A Musculoskeletal
Examination Model Whose Time Has Come. JOSPT, 2007, (37)11.
51. Spine & Extremity Regions
Typical referred & radiating pain patterns
Impairments - often seemingly unrelated
53. Examples in the Literature:
Intervention Studies
Primary LBP
• Cibulka, JOSPT, 1999 Primary Knee Pain
• Boyle & Demske, Physiotherapy Deyle et al, Ann Int Med, ‗00
Theory & Practice, ‗09 Deyle et al, PT, ‘05
• Whitman et al, PM&R Clinics of Cliborne et al, JOSPT ‘04
North America 2003 Currier et al, Phys Ther, ‘07
• Whitman et al, Spine 2006 Lowry et al, JOSPT, ‘08
Iverson et al, Phys Ther, ‘08
Mascal et al, JOSPT, ‘03
Cibulka & Threlkeld-
Watkins, JOSPT, ‘05
Primary Hip Pain Vaughn, JOSPT, ‗08
• Cibulka &
Delitto, JOSPT, 1993
Primary Foot Pain
• Konczak & Ames, JMPT, 2004
• Cleland et al, JOSPT, 2009
• Wisdo, JMPT, 2004
54. Treat the Lower
Quarter for
Primary Low Back Pain
Classic Examples in the Literature
55. Lower Quarter Treatment for Primary LBP
Low Back Pain
• Cibulka, JOSPT, ’99
• Boyle & Demske, Physiotherapy Theory &
Practice, ‗09
Lumbar Spinal Stenosis
• Whitman et al, Spine. ‘06
• Whitman et al, PM&R Clinics of North
America, ‘03
56. Differential Dx for Hip vs. Spine Disease
Brown, et al. Clin Orthop. Feb 2004:280-284
97 pts with LE pain referred to spine specialty clinic
Age - mean 67.5 ± 11.6 years
History, physical exam, & diagnostic testing completed
Imaging studies gold standard for diagnosis
Findings:
19% - spinal disorders only
36% - hip & spine in conjunction
45% - hip only disorders
81% - had some hip involvement present
Limitations
Diagnostic & test review with spectrum bias
57. • Limp, groin pain, limited IR at hip
– More likely to be present in a patient with a hip disorder
• Positive femoral stretch test
– More likely to have a spine disorder
58. Hamstring Muscle Strain Treated by
Mobilizing the Sacroiliac Joint
Cibulka et al, Phys Ther, 1986
59. Hamstring Muscle Strain Treated by
Mobilizing the Sacroiliac Joint
20 patients with a HS Dependent Measures
strain Hamstring flexibility
Hamstring muscle torque
Quadriceps muscle torque
Control- MH and
stretching
Experimental-
MH, stretching and SI
manip
Significant difference in hamstring torque for
experimental group.
60.
61. N = 60 patients with LSS
Outcomes:
• GRC, Disability (OSW)
• Baseline, 6-weeks, 1-year, long-term (mean 27-29 mo)
Interventions:
• All subjects: 2x/wk for 6 wks in-clinic, walk at home 3x/week
Flexion Exercise & Manual PT, Exercise, and
Walking Group (FExWG) Walking Group
(MPTExWG)
• Sub-therapeutic US, SKC/DKC, •BWS TM walking & SKC/DKC
TM walking program
• Manual physical therapy -
Impairment-Based,
Comprehensive Lower Quarter
67. Other RI Facts:
Hip-Spine Syndrome / Relationships
Hip ROM & LBP/SIJ
Ellison et al, Phys Ther, ‘90; Chesworth et al, Physiother Canada, ‘94;
Cibulka et al, Spine, ‘98; Sjolie , Scand J Med Sci, sports, ‘04;
Vad et al, Am J Sports Med, ‘04; Coplan, JOSPT, ‘02; Mellin, Spine, ‘88
Porter & Wilkinson; Spine ‘97; Mellin. Spine ‘88
Hip Region Muscle Performance & LBP
Nadler et al, Clin J Sport Med, ‘00; Nadler et al, Am J Phys Med Rehabil, ‘01;
Nadler et al, Med Sci Sports Exerc, ‘02; Kandaanpaa et al, Arch Phys Med Rehabil, ‘98;
Nourbakhsh & Arab, JOSPT, ‗02
Hip Region Muscle Performance & LBP
Offierski & McNab, Spine ‘83; Ben-Galim et al, Spine, ‘07; Murata et al, Clin Orthop Surg, ‘02;
Nakamura et al, Acta Orthop Scand, ‘03; Yoshimoto et al, Spine, ‘05; Takemitsu et al, Spine, ‘88;
Sato et al, J Musculoskelet Syst, ‘89; Itoi, Spine, ‘91; Watanabe et al, Orthopedics, ‗02
Reiman, Weisbach, and Glynn. The Hip’s Influence on Low Back Pain: A Distal Link to a Proximal
Problem. Journal of Sport Rehab, ‘09
68. Other RI Facts
Associations between:
Hip ROM & response to spinal manipulation
Flynn et al. Spine ’02.Childs et al, Ann Int Med ’04
Hip Abd, ER, and Ext Weakness and PFPS
Robinson, JOSPT, ‘07; Ireland, JOSPT, ‗03
SIJ manipulation and quadriceps facilitation
Suter et al. JMPT ‘00
SIJ manipulation and HS peak torque changes (pts with HS strain)
Cibulka et al, Phys Ther, ‗85
LSS source of pain identified after THA
Bohl et al, Spine, ‘79, Saito et el, Spine 2012
Concomitant spine and hip disease extremely common
Brown et al, CORR, ‘04
69. Primary Hip Interventions for Patients
with Low Back Pain and Hip Impairments:
A Prospective Case Series (Preliminary Results)
Eric K. Robertson1, Cheryl Sparks2, Derek Clewley3
1Faculty,Department of Physical Therapy, Texas State University, San Marcos, TX, USA, 2Faculty, Department of
Physical Therapy, Bradley University, Peoria, IL, USA, 3Benchmark Physical Therapy, Atlanta, GA, USA
*Acknowledgement: Dr. Julie M. Whitman for her review of the study design and report.
Research performed as part of the Evidence in Motion Fellowship Program
70. Although many clinicians who
are perceived as experts often
describe examination and
interventions at the hip for
patients with low back
pain, very little evidence
describing this exists.
Lack of Research
71. Management of a Female with Chronic
Sciatica and LBP: A Case Report
History
6 weeks of lumbar stabilization and flexibility intervention did not
eliminate symptoms
Intervention
Treatment plan revised to include impairment-based left hip
capsule flexibility
Also included hip strengthening to glut med
Outcome
Complete resolution of pain, disability (0 NPRS, 0% on ODI (reduced
from 41% at baseline))
Boyle and Demske, 2009 Physiotherapy Theory and Practice
72. Management of a Female with Chronic Sciatica and LBP:
A Case Report
• History 60
• 6-weeks of lumbar
stabilization and flexibility 50
intervention did not
eliminate symptoms 40
• Intervention 30 41%
Reduction.
• Treatment plan revised to
20
include impairment-based
left hip capsule flexibility
10
• Also included hip
Spine Interventions
strengthening to glut med
0
Hip Interventions
NPRS ODI
Boyle and Demske, 2009 Physiotherapy Theory and Practice
73. Purpose
The purpose of this prospective case series was two-fold.
1. Describe the clinical decision making process involved in the
management of patients with primary complaints of low back
and hip pain.
2. Provide an evidence-based rationale for directing treatment at
the hip in a sub-group of patients with low back pain.
74. Number of Subjects
Six subjects referred to physical therapy with low back pain who
also demonstrated impairments at the hip were recruited to
participate. (Note: Data collection is still in progress, currently
have 11 enrolled.)
75. About the Subjects
Inclusion Criteria:
• 1. Adults referred to PT with a primary complaint of low back pain (LBP)
• 2. Positive hip impairments as identified in the patient’s initial physical
examination*
Definition of positive hip impairment:
• Positive special tests, decreased ROM, decreased muscle
strength, reversed lumbo-pelvic rhythm, decreased mobility or pain with
accessory motions, or findings consistent with the clinical presentation
of hip osteoarthritis per the criteria established by Altman et al.
76. About the Subjects
Exclusion Criteria:
• Any medical red flags
• Spinal or femoral fractures, except for degenerative spondylolisthesis or
spondylolisis
• Upper motor neural compromise
• Pregnancy
• Lower motor neuron changes suggestive of nerve root irritation and/or
compression (positive straight leg raise at <45º or diminished lower extremity
strength, sensation, or reflexes
• High fear-avoidance beliefs (>35 on the Fear Avoidance Belief Questionnaire
Work [FABQW] subscale)
• Previous history of spine or hip surgery
• Inability to read and understand English
77. Methods
• Primary outcomes: • Secondary outcomes:
• Oswestry Disability • Global Rating of Change
Index, (ODI) (GROC)
• Harris Hip Score (HHS) • Assessment of hip impairments.
• Numerical Pain Rating Scale
(NPRS)
Initial Examination
Week 4
Visit #2 (2-3 days after Initial Examination)
Institutional Review Board approval was obtained from Bradley University for this study.
78. Important Exam Items
Hip Examination
ROM, all planes
• +/- Hip Impairment Special Tests: Scour, FABER, other
• Positive special tests, decreased Flexibility: Hip flexor, extensor length
ROM, decreased muscle Strength: All planes
strength, reversed lumbo-pelvic
rhythm, decreased mobility or pain
with accessory motions, or findings Lumbar Spine Examination
consistent with the clinical
presentation of hip osteoarthritis per ROM Screen
the criteria established by Altman et al. Accessory Mobility
Core musculature assessment
Special Tests: SLR, PIT, etc
79. Hip Examination Item Results
Hip ROM Impairments 6/6
Hip Weakness 4/6
+ Hip Special Test 5/6
Median age was 50 years (range, 27-61 years)
Examination Results
80. Interventions
Day 1: Examination and Initial Treatment
If hip impairment was noted, then treatment progressed in the following manner:
• Manual Interventions focused on hip. (thrust/non-thrust)
• Hip-specific therapeutic exercise
• Reinforcing Home Exercise Program
• Re-assess Day 2.
81. Interventions
Exercises provided to focus on
• gluteus maximus,
• hip abduction and external rotation
• core stabilization progression.
Neuromuscular re-education:
• Movement re-education
consisting of patient
instruction, practice if
abnormalities noted, i.e. sit to
stand
• Squatting and proper lifting
mechanics
• Proprioceptive core stability
exercises
82. Interventions
Day 2 – Week 4 Treatment
• Treat per impairment-based approach or a pragmatic application
of the Treatment-based classification approach.
• HEP and TherEx continued to stress hip-focused interventions
83. Results
Results: At 4 weeks, the average improvement in ODI scores was 39%, and
24% for the HHS. 66% of the patients experienced a significant decrease in the
NPRS. 100% of patients for which data was collected experienced at least a
change of 4 on the GROC (4-7), indicating at least moderate improvement.
Avg. visits = 7.3 (3-10).
Numeric Pain Rating
Global Rating of Change*
Scale*
5.3 2.8
Median 5.25, (range, 4-7) Median 3, (range, 0-5)
“Quite a bit better” MCID: 1.8
*Note: Data on GROC collected for 4/6 patients.
84. ODI Avg. Change
Day 2 4
Week 4 20
Oswestry Disability Index
90
80
70
60
50 Baseline
40 Day 2
Week 4
30
20
10
0
Patient 1 Patient 2 Patient 3 Patient 4 Patient 5 Patient 6
86. • Small Case Series = No Cause and Effect
• Not consecutive subjects
• Control over interventions limited, especially the
neuromuscular reeducation to the core
Limitations
87. Short-term response of hip mobilizations and
exercise in individuals with chronic low back
pain: a case series
• Subjects:
• 8 Subjects with CLBP, Avg age 49, with ROM impairments
• Intervention
• Impairment-based manual therapy to Bilateral Hips
• 3 Sessions over 1 week
• Outcome
• 5/8 (65%) reports >4 “Moderately Better” on the GROC
• 24% reduction in ODI scores
“This case series suggests that an impairment-based approach directed at the hip joints may lead to
improvements in pain, function, and disability in patients with CLBP. A neurophysiologic mechanism may be a
plausible explanation regarding the clinical outcomes of this study. A larger, well-controlled trial is needed to
determine the potential effectiveness of this approach with patients with CLBP.”
Burns, Mintken, Austin, Cleland. 2010 JMMT
88. Conclusions
• Significant improvements were observed in all primary outcome measures
at 4 weeks.
• Secondary outcome measures trended towards significant improvements.
• This case series provides preliminary evidence that some patients with low
back pain may receive a benefit from interventions directed at the hip.
• Future studies should work to determine the factors that can predict patients
that may realize this benefit.
90. Posterior Pelvic Tilts
Williams Flexion Exercises
Hot Packs
Massage
Ultrasound
That‘s so yesterday!
Once we acknowledge the role of the LE in
back pain, it frees us from the constraints of
patients with low back pain. It allows us to
become creative and challenge our patients in
new ways.
93. • Progression of:
• Limb support
• Surface
• Visual input
• Perturbations
• Task demands
Proprioceptive/Balance
Training
DPTMWG
94. Testing For Strength
In CKC Positions
• Sit to Stand
• Squat
• Leg Press
• Lunge
95. • Used most often with older individuals
• Performance is based on given period of
time (10-30 seconds) and reps recorded or
given reps and time recorded in seconds
• Sit to stand performance has direct
correlation to knee extension force and leg
press force.
Sit To Stand Test
Jones et al
DPTMWG
97. • Sit to Stand Test (Csuka et al, Am J Med 1985)
• Regression equation for predicting normal performance for
10 stand ups. Results are in seconds.
• Women: 7.6 + .17 x age
• Men: 4.9 +.19 x age
Sit to Stand Test
DPTMWG
98. • Utilized to compare
uninvolved to involved
and also to compare to
norms
• Associated with
thigh, hip and buttock
strength
• Takes balance and core
factors out of strength
assessment.
• Greater than 90% of
contra-lateral
considered acceptable
Leg Press Test for Strength
DPTMWG
99. Plisky et al. N Am J Sports Phys Ther. 2009 May; 4(2): 92–99.
DPTMWG
100. • Significant differences associated with chronic ankle
instability and ACL insufficiency
• Can be predictive of LE injury
• What about LBP?!
Population Reach Distance Implication P value
All Players R:L Difference of 2.5x more likely to p<.05
>4 cm sustain LE injury
Females <94% of LE 6x more likely to p<.05
length sustain LE injury
Y Balance Test Utility
DPTMWG
101. Mascal et al. Management of patellofemoral pain targeting hip, pelvis,
and trunk muscle function: 2 case reports. JOSPT 2003
N=2
20, 37 y/o females
102. Mascal et al. Management of patellofemoral pain targeting hip, pelvis,
and trunk muscle function: 2 case reports. JOSPT 2003
• 14 week treatment period focused on recruitment and endurance
training of the hip, pelvis and trunk (including TrA)
• Patients attended PT 1-2 times per week
• Hip muscles (particularly glut max, med and external rotators)
were progressively strengthened, starting in NWB followed by WB
functional tasks
103. Hip Abductors and ERs
• Mini-squats, step-ups/downs and leg presses for
concentric/eccentric control, BAPS board/lunges for
proprioceptive training
Core muscle training
Restore Neuromuscular Control
107. Pain…
It might not be as much of a
physical thing as we think!
We need to consider the
cognitive components!
108. L E V E L
P A I N
Injury!
Adapted from Butler & Mosely, 2008, ―Explain Pain‖
H E A L I N G R E S P ON SE
109. ―Nociception is neither sufficient
for, or necessary to experience pain.‖
Adapted from Butler & Mosely, 2008, ―Explain Pain‖
110. Identifying Patients at Risk for
Chronic Pain
George & Zepperi, JOSPT, July- 2009
Fear-avoidance model of musculoskeletal pain
(FAM) (Measured by FABQ)
Factors influencing pain perception
Anxiety
Fear of re-injury
Catastrophizing
Confrontation Avoidance
Anxiety Anxiety
Fear of re-injury Fear of re-injury
Catastrophizing Catastrophizing
111. FDAQ – A Measurement
George & Zepperi, JOSPT, July- 2009
George et al., PTJ, July- 2009
112. Establishing a Baseline
Therapist: ‗How long can you walk before you flare-up?‘
Patient: ‗I can walk for 30 min but I pay for it the next day‘
Therapist: ‗Can you walk for 20 min without flaring up?‘
Patient: ‗No, but I have‘
Therapist: ‗Can you walk for 10 min without flaring up?‘
Patient: ‗Probably not — definitely not up hills‘
Therapist: ‗5 min on a flat surface?‘
Patient: ‗Probably‘
Therapist: ‗3 min on a flat surface?‘
Patient: ‗Definitely‘
113. Continuous Progression
‗Every day you do more than you did
yesterday, but not much more‘ …at least initially.
Setting clear measurable goals and objectives!
114. KEY POINTS
Pain is not nociception
The representation of the body in the human brain
The brain changes as the pain persists
Body-brain is a 2 way street
Training the brain for people in Pain
This is VERY Hip.
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Hinweis der Redaktion
https://www.biodigitalhuman.com/#/pubkid=29Y
Low Back Pain: $86 billion/year (2005)• All arthritis: $80 billion (2003)• Cancer: $89 billion (2007)• Diabetes: $98 billion (2002)• Heart dis & stroke: $258 billion (2005)• Costs for LBP rose 65% (inflationadjusted),1997-2005Martin BI, Deyo RA, et al. JAMA
Low Back Pain: $86 billion/year (2005)• All arthritis: $80 billion (2003)• Cancer: $89 billion (2007)• Diabetes: $98 billion (2002)• Heart dis & stroke: $258 billion (2005)• Costs for LBP rose 65% (inflationadjusted),1997-2005Martin BI, Deyo RA, et al. JAMA
Flexion-relaxation response – refers to the electromyographic silence of the lumbar spine extensors in end ranges of flexion, thought to be why hip extensors activate first to rise from a flexed position.
Given the findings of Lee and Shum4,8,10-13, these findings do not seem to be in so much contrast to expectations, but perhaps representative of the arthrokinematics of many patients with low back pain.
Image is of control, note early activity of OI, multifidus during gait and early quieting of biceps femoris.
Hip–Spine SyndromeThe concept of a biomechanical link between the hip joint and the lumbar spine has been described as hip–spine syndrome (HSS).40 (offierski and mcnap – hip spine syndrome – spine – ’83) HSS specifically depicts the influence of a pathological hip joint on the alignment of the spine and subsequent muscle length and joint forces.40The most recent documentation of this relationship has been that of severehip osteoarthritis (OA) potentially causing abnormal spinal sagittal alignment andensuing LBP.41 Ben-Galim et al41 evaluated the effects of surgical treatment of hipOA on low back disability in patients preoperative and postoperative total hipreplacement and found significant (P < .01) improvements in both visual analogscores for LBP and Oswestry Disability Index scores after surgery that remainedat the 2-year follow-up.Other specific related interactions of the hip and spine in HSS can include ahip-flexion contracture resulting in compensatory hyperlordosis of the lumbarspine or a posteriorly inclined pelvis with increased kyphotic posture and primaryor rapidly destructive hip OA.40,42–48 In each of these examples, although there isa relationship between the hip and spine, the evidence demonstrating the significanceof its effect on LBP is deficient.Although the biomechanical influences of the hip on LBP are not fully evidentat this time, the current level of evidence does support a regional relationshipbetween the 2 areas. From the preliminary work of Ben-Galim et al,41 one canbegin to appreciate the importance of further investigating hip ROM, as well asregional soft-tissue characteristics, in patients with LBP.Offierski and McNabb published a paper describing a “hip-spine syndrome” in which patients with pain in the lumbo-pelvic-hip region can have concurrent pathologies in both the lumbar spine and hip joints. 1 They stated that the inability to properly determine the primary source of a patient’s pain may lead to inappropriate or incomplete treatments. Additional studies have been published that support the existence of hip-spine syndrome in patients with the primary complaint of LBP.2-5 . Offierski CM, MacNabb MB. Hip-Spine Syndrome. Spine. 1983;8(3):316-321.2. Fogel GR, Esses SI. Hip spine syndrome: management of coexisting radiculopathy and arthritis of the lower extremity. Spine J. 2003;3(3):238-241.3. Vad VB, Bhat AL, Basrai D, Gebeh A, Aspergren DD, Andrews JR. Low back pain in professional golfers: the role of associated hip and low back range-of-motion deficits. Am J Sports Med. 2004;32(2):494-497.4. Brown MD, Gomez-Marin O, Brookfield KF, Li PS. Differential diagnosis of hip disease versus spine disease. ClinOrthopRelat Res. 2004;419:280-284. 5. Lee RYW, Wong TKT. Relationship between the movements of the lumbar spine and hip. Hum Mov Sci. 2002;21(4):481-494.6. Ben-Galim P, Ben-Galim T, Rand N, et al. Hip-spine syndrome: the effect of total hip replacement surgery on low back pain in severe osteoarthritis of the hip. Spine. 2007;32(19):2099-2102.
Treatment with hip long axis distraction thrust manipulation if not contraindicated.Hip and low back pain will be re-assessed following treatment.Non-thrust and thrust hip joint mobilization including those to improve flexion,extension, and rotation as indicated by impairments and stretches to theanterior hip musculature to maximize hip joint mobility. Hip and low back painwill be re-assessed following treatment.Perform therapeutic exercises for hip. Prescribe home exercise program (HEP) for hip.
Treatment with hip long axis distraction thrust manipulation if not contraindicated.Hip and low back pain will be re-assessed following treatment.Non-thrust and thrust hip joint mobilization including those to improve flexion,extension, and rotation as indicated by impairments and stretches to theanterior hip musculature to maximize hip joint mobility. Hip and low back painwill be re-assessed following treatment.Perform therapeutic exercises for hip. Prescribe home exercise program (HEP) for hip.
Stabilization progression exercises described by Hicks et alHip extension in prone with knee flexion.Specific exercise to gluteus medius.
Treatment with hip long axis distraction thrust manipulation if not contraindicated.Hip and low back pain will be re-assessed following treatment.Non-thrust and thrust hip joint mobilization including those to improve flexion,extension, and rotation as indicated by impairments and stretches to theanterior hip musculature to maximize hip joint mobility. Hip and low back painwill be re-assessed following treatment.Perform therapeutic exercises for hip. Prescribe home exercise program (HEP) for hip.
NPRS (Childs, Piva 2005, Spine, reported for 1 week)ODI:39%HHS: 24%66% reduced NPRS (1 didn’t report pain)100% at least 4 on GROC= moderate improvement
The intervention for these two patients consisted of an impairment-based approach … Here you can see some of the exercises performed. On the left, basic strength training at 0-6 weeks, In the middle more dynamic training at 6-10weeks, and on the right, functional plyometric exercises at 10-14 weeks. Note that the exercises were steeped in neuromuscular recruitment of the proper muscles and that this trial focused somewhat extensively on hip strengthening. Both patients experienced significant decreases in pain, increased function, muscle strength and endurance.
I do want to talk specifically about the concept of VMO strengthening. Many PT’s hold onto what they were taught back in the 80’s and 90’s about things. The VMO is one example. It’s not possible for us to selective activate any one part of the quad over another, since they all come from the same nerve supply. No evidence exists that consistently supports this concept. However, it’s not uncommon to see this in the clinic, or read articles that includes statements like the one you see here, which are perhaps logical from an embryologic standpoint, but not from a practical application standpoint. The key is to focus on the nm control of the entire lower extremity kinetic chain and leave behind the focus on the VMO.
Low Back Pain: $86 billion/year (2005)• All arthritis: $80 billion (2003)• Cancer: $89 billion (2007)• Diabetes: $98 billion (2002)• Heart dis & stroke: $258 billion (2005)• Costs for LBP rose 65% (inflationadjusted),1997-2005Martin BI, Deyo RA, et al. JAMA