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Endodontic Emergency 
PROF.DR. 
MEHMET OMER GORDUYSUS 
DDS, PHD
Endodontic Emergency 
IT IS A SITUATION ASSOCIATED WITH PAIN 
AND/OR SWELLING THAT REQUIRES 
IMMEDIATE DIAGNOSIS AND TREATMENT. 
IT MAY INVOLVE RESCHEDULING OF THE 
NORMAL APPOINTMENTS.
DIFFERENCE BETWEEN 
URGENCY AND EMERGENCY 
ď‚—An urgency represent a less 
severe problem. 
ď‚—An emergency is more severe and 
requires immediate attention Now. 
ď‚—A Rule of the true emergency 
:One tooth is the offender.
Key questions to differentiate between 
emergency and urgency 
ď‚—Is the problem such that it disturbs your sleeping, 
eating, working, concentration? → An emergency 
condition affects these activities. 
How long has it been bothering you? →Short 
duration emergencies, with pain of long duration are 
urgencies. 
ď‚—Have you taken any pain medication, Did it help? 
→Medications are usually ineffective during an 
emergency condition.
Etiologies: 
ď‚—Microbial 
ď‚—Mechanical 
ď‚—Chemical
Factors causing pain are: 
ď‚—1-Chemical mediators 
ď‚—2-Pressure
Chemical mediators 
1-Direct: 
By activating nociceptors causing spontaneous pain 
Or by lowering their pain threshold 
2-Indirectly: 
By increasing vascular permeability & producing 
edema
Pressure: 
ď‚—Edema results in increased fluid pressure, which 
mechanically stimulates pain reseptors.
Emergency Impacts 
ď‚—Patient 
ď‚—Staff 
ď‚—Dentist
Patient Presentation
3D’s of Successful Management 
ď‚—Diagnosis 
ď‚—Definitive Dental Treatment 
ď‚—Drugs
Diagnosis 
ď‚—Determine the CC 
ď‚—An accurate medical 
history 
ď‚—Complete a thorough 
exam, with all necessary 
tests 
ď‚—Perform a radiographic 
exam 
ď‚—Analyze the results 
ď‚—Establish the treatment 
plan
Treatment Plan 
to 
REMOVE 
the 
Etiology
When do patients present for emergency 
endodontic care? 
ď‚—No prior RCT/ initial infection 
ď‚—After RCT initiated 
ď‚—After obturation
Initial Presentation 
ď‚—Pain 
ď‚—Primary Infection
After Initiation of Endodontic Therapy 
ď‚—FLARE -UP
After Initiation of Endodontic Therapy 
ď‚—Before 
obturation
After Obturation 
ď‚—Recent obturation 
ď‚—Non-healing endodontic therapy
Determine a 
PULPAL 
And 
PERIAPICAL 
Diagnosis
Pulpal Diagnosis
Periradicular Diagnosis 
ď‚—Normal periradicular tisbsecesssues 
ď‚—Symtomatic periradicular periodontitis 
ď‚—Acute periradicular abscess
Etiology 
After listening to the patient, begin to determine 
the etiology of the chief complaint: 
ď‚—Contents of the root canal 
ď‚—Dentist controlled factors 
ď‚—Host factors
Contents of the root canal 
ď‚—Pulp tissue 
ď‚—Bacteria 
ď‚—Bacterial by-products 
ď‚—Endodontic therapy materials
Dentist controlled factors 
ď‚—Over-instrumentation 
ď‚—Inadequate debridement 
ď‚—Missed canal 
ď‚—Hyper-occlusion 
ď‚—Debris extrusion 
ď‚—Procedural complications
Hyperocclusion 
ď‚—Research have found 
that patients most 
likely to benefit from 
occlusal reduction are 
those teeth whose 
initially present with 
symptoms. 
ď‚—Indiscriminant 
reduction of occlusal 
surface is not indicated 
ď‚—Pre-Op Pain 
ď‚— Pulp vitality 
ď‚—Percussion sensitivity 
ď‚—Absence of a 
periradicular 
radiolucency 
ď‚—Combination of these 
symptoms
Procedure complications 
ď‚—Perforation 
ď‚—Separated instrument 
ď‚—Zip 
ď‚—Strip 
ď‚—NaOCl accident 
ď‚—Air emphysema 
ď‚—Wrong tooth
Dentist Controlled Factors 
Dentist’s personality
Host Factors 
ď‚—Allergies 
ď‚—Age 
ď‚—Sex 
ď‚—Emotional state
Host Factors 
ď‚—Complex etiology 
Microbiologic 
Immunologic 
Inflammatory
Emergency Treatment 
ď‚—Non surgical 
ď‚—Surgical 
ď‚—Combined
Non surgical Emergency Treatment 
ď‚—Pulpotomy 
ď‚—Partial pulpoctomy 
ď‚—Complete pulpectomy 
ď‚—Debridement of the root canal system
Surgical Emergency Treatment 
ď‚—Incision for drainage 
ď‚—Trephination/ Apical fenestration
Rationale for Incision for drainage 
ď‚—Decreases number of bacteria 
ď‚—Reduce tissue pressure 
Alleviates pain/trismus 
Improves circulation 
ď‚—Prevents spread of infection 
ď‚—Alters oxidation-reduction potential 
ď‚—Accelerates healing
Management of Acute Pulpitis: 
Diagnosis: 
ď‚—Pain: +ve 
ď‚—Vitality: +ve 
ď‚—Tenderness to percussion : 
ď‚—Radiographic changes: 
No change from normal 
ď‚—Deep caries, 
extensive restoration, 
trauma, pulp capping may be seen.
Management: 
ď‚—Limited time: 
Anteriors/Premolar 
ď‚—Profound anesthesia. 
ď‚—Complete pulp extirpation. 
ď‚—Temporary dressing. 
Molar 
ď‚—Pulpotomy
Lots of time: 
Anteriors/ Premolars/Molars 
ď‚—Complete 
pulp extirpation 
ď‚—Temporary dressing
Management of Acute pulpitis with 
apical periodontitis: 
ď‚—Diagnosis: 
ď‚—Vitality: +ve 
ď‚—Tenderness to percussion: +ve 
ď‚—The tooth feels high and/or loose 
and that the teeth will not close 
together. 
ď‚—X-ray: Normal to slight widening of 
periodontal ligament space to small 
radiolucence.
Management 
Minimal Time: Molar 
ď‚—Profound Anesthesia: May need an additional 
carpule. 
ď‚—Pulpectomy of the largest canal ( distal of lowers 
and palatal of upper). 
ď‚—Temporary dressing. 
ď‚—May need to call the next day to remove pulp from 
the other canal, pain will not subside if the other 
canals are the cause of pain.
Anterior teeth/ Premolar 
ď‚—Complete pulp extirpation followed 
by temporary dressing.
Lots of time: 
ď‚—Complete pulp extirpation of all 
the canals must be done followed 
by a temporary dressing.
Management of Pulp Necrosis: 
Rarely seen as an emergency 
Diagnosis: 
ď‚—Non vital tooth( may be one or more of its root 
canal). 
ď‚—No tenderness to percussion. 
ď‚—Periapical radiolucency seen on the radiograph.
Management: 
1- Canal debridement followed by a 
temporary dressing. 
2- Extraction of non restorable tooth. 
(Analgesics and antibiotics may be 
required).
Acute apical Abscess: 
The position of the swelling will 
depend on: 
ď‚—1- Orientation of the tooth apex. 
ď‚—2-Relationship of the site of perforation 
to muscle attachment on the maxilla 
and mandible.
Spreading submandibular swelling 
due to an acute apical abscess
Facial swelling should be detected
Palatal swelling associated with 
upper lateral incisor
Facial Swelling Associated with 
maxillary canine
Acute apical abscess producing a facial swelling 
Tooth drainge of an apical abscess
To resolve swelling: 
1)Establish drainage through the root canal. 
2)Establish drainage by incising a fluctuant 
swelling. 
3)Prescribe antibiotics.
Management of a localized soft tissue 
swelling: 
* If it is fluctuance, it indicate that pus is present, 
soft tissue infiltration of anesthesia around the 
periphery of the infected area. 
* Incise at the site of greatest fluctuance down to 
the level of apical bone.
• A vertical incision offers improved post 
operative healing compared with a 
horizontal incision. 
• Place the incision in a position to 
encourage drainage by gravity.
•Dissect gently through the deeper 
tissues and explore all parts of 
abscess cavity. 
•The wound should be kept clean 
with hot salt-water mouth rinses 
to promote drainage.
Diffuse swelling: 
• From endodontic point of view, the 
tooth is opened, and the canal is 
thoroughly instrumented and irrigated, 
if no drainage is achieved, the apical 
foramen is instrumented through to 
encourage drainage from the periapical 
tissues.
• In the absence of drainage through 
tooth, soft tissue drainage might be 
established through incision. The drain 
is sutured into incision wound to 
ensure tissue drainage.
ď‚—The patient who show sign of 
toxicity, CNS changes, or airway 
compromise should be considered 
for immediate hospitalization.
Guidelines for Antibiotic Therapy 
ď‚—Select antibiotic with anaerobic spectrum 
ď‚—Use a larger dose for a short period of time
Antibiotic therapy: 
• For localized swellings the antibiotic 
therapy is usually unnecessary (except 
with patient with depressed host 
defense). 
• For diffuse swelling antibiotic are 
indicated.
ď‚—1st choice: penicillin VK 
Initial dose 1-2 g then 500mg every 
6 hours for 7-10 days 
ď‚—The combination of penicillin and 
metranidazole (250mg) is 
recommended 7-10 days.
ď‚—Clindamycin are suitable alternatives for 
patients who are allergic to amoxicillin. 
ď‚—The dose 300mg followed by 150 to 300mg 
every 6 hours for 7-10 days. 
ď‚—(some times signs of colitis)
As a general rule: 
ď‚—Antibiotic therapy should be 
considered for patients who 
have signs and symptoms of 
infection, such as cellulites, 
fever, or lymphadenitis.
FFlleexxiibbllee aannaallggeessiicc ssttrraatteeggyy 
Aspirin like drug 
indicated 
Ibuprofen 
200-400mg 
Aspirin like drug 
contraindicated 
Aspirin like drug 
contraindicated 
Ibuprofen 
400-600mg 
Ibuprofen 
400-600mg 
Acetaminophen 
650-1000mg 
Acetaminophen 
650-1000mg 
Acetaminophen 650-1000mg 
Plus 
equivalent of Codeine 60 mg 
Acetaminophen 1000mg 
Plus 
equivalent of Oxycodone 10 mg 
Ibuprofen 400-600mg 
Ibuprofen 400-600mg 
Plus 
Plus 
Acetaminophen 650-1000mg 
Acetaminophen 650-1000mg 
Ibuprofen 600-800mg 
Plus 
Acetaminophen 1000mg 
Sever Moderate Mild
Crown-Root Fractures
Untreated undiagnosed fractres
Root fracture induced during 
obturation
Endo emergency

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Endo emergency

  • 1. Endodontic Emergency PROF.DR. MEHMET OMER GORDUYSUS DDS, PHD
  • 2. Endodontic Emergency IT IS A SITUATION ASSOCIATED WITH PAIN AND/OR SWELLING THAT REQUIRES IMMEDIATE DIAGNOSIS AND TREATMENT. IT MAY INVOLVE RESCHEDULING OF THE NORMAL APPOINTMENTS.
  • 3. DIFFERENCE BETWEEN URGENCY AND EMERGENCY ď‚—An urgency represent a less severe problem. ď‚—An emergency is more severe and requires immediate attention Now. ď‚—A Rule of the true emergency :One tooth is the offender.
  • 4. Key questions to differentiate between emergency and urgency ď‚—Is the problem such that it disturbs your sleeping, eating, working, concentration? → An emergency condition affects these activities. ď‚—How long has it been bothering you? →Short duration emergencies, with pain of long duration are urgencies. ď‚—Have you taken any pain medication, Did it help? →Medications are usually ineffective during an emergency condition.
  • 6. Factors causing pain are: ď‚—1-Chemical mediators ď‚—2-Pressure
  • 7. Chemical mediators 1-Direct: By activating nociceptors causing spontaneous pain Or by lowering their pain threshold 2-Indirectly: By increasing vascular permeability & producing edema
  • 8. Pressure: ď‚—Edema results in increased fluid pressure, which mechanically stimulates pain reseptors.
  • 9. Emergency Impacts ď‚—Patient ď‚—Staff ď‚—Dentist
  • 11. 3D’s of Successful Management ď‚—Diagnosis ď‚—Definitive Dental Treatment ď‚—Drugs
  • 12. Diagnosis ď‚—Determine the CC ď‚—An accurate medical history ď‚—Complete a thorough exam, with all necessary tests ď‚—Perform a radiographic exam ď‚—Analyze the results ď‚—Establish the treatment plan
  • 13. Treatment Plan to REMOVE the Etiology
  • 14. When do patients present for emergency endodontic care? ď‚—No prior RCT/ initial infection ď‚—After RCT initiated ď‚—After obturation
  • 15. Initial Presentation ď‚—Pain ď‚—Primary Infection
  • 16. After Initiation of Endodontic Therapy ď‚—FLARE -UP
  • 17. After Initiation of Endodontic Therapy ď‚—Before obturation
  • 18. After Obturation ď‚—Recent obturation ď‚—Non-healing endodontic therapy
  • 19. Determine a PULPAL And PERIAPICAL Diagnosis
  • 21. Periradicular Diagnosis ď‚—Normal periradicular tisbsecesssues ď‚—Symtomatic periradicular periodontitis ď‚—Acute periradicular abscess
  • 22. Etiology After listening to the patient, begin to determine the etiology of the chief complaint: ď‚—Contents of the root canal ď‚—Dentist controlled factors ď‚—Host factors
  • 23. Contents of the root canal ď‚—Pulp tissue ď‚—Bacteria ď‚—Bacterial by-products ď‚—Endodontic therapy materials
  • 24. Dentist controlled factors ď‚—Over-instrumentation ď‚—Inadequate debridement ď‚—Missed canal ď‚—Hyper-occlusion ď‚—Debris extrusion ď‚—Procedural complications
  • 25. Hyperocclusion ď‚—Research have found that patients most likely to benefit from occlusal reduction are those teeth whose initially present with symptoms. ď‚—Indiscriminant reduction of occlusal surface is not indicated ď‚—Pre-Op Pain ď‚— Pulp vitality ď‚—Percussion sensitivity ď‚—Absence of a periradicular radiolucency ď‚—Combination of these symptoms
  • 26. Procedure complications ď‚—Perforation ď‚—Separated instrument ď‚—Zip ď‚—Strip ď‚—NaOCl accident ď‚—Air emphysema ď‚—Wrong tooth
  • 27. Dentist Controlled Factors ď‚—Dentist’s personality
  • 28. Host Factors ď‚—Allergies ď‚—Age ď‚—Sex ď‚—Emotional state
  • 29. Host Factors ď‚—Complex etiology Microbiologic Immunologic Inflammatory
  • 30. Emergency Treatment ď‚—Non surgical ď‚—Surgical ď‚—Combined
  • 31. Non surgical Emergency Treatment ď‚—Pulpotomy ď‚—Partial pulpoctomy ď‚—Complete pulpectomy ď‚—Debridement of the root canal system
  • 32. Surgical Emergency Treatment ď‚—Incision for drainage ď‚—Trephination/ Apical fenestration
  • 33. Rationale for Incision for drainage ď‚—Decreases number of bacteria ď‚—Reduce tissue pressure Alleviates pain/trismus Improves circulation ď‚—Prevents spread of infection ď‚—Alters oxidation-reduction potential ď‚—Accelerates healing
  • 34. Management of Acute Pulpitis: Diagnosis: ď‚—Pain: +ve ď‚—Vitality: +ve ď‚—Tenderness to percussion : ď‚—Radiographic changes: No change from normal ď‚—Deep caries, extensive restoration, trauma, pulp capping may be seen.
  • 35. Management: ď‚—Limited time: Anteriors/Premolar ď‚—Profound anesthesia. ď‚—Complete pulp extirpation. ď‚—Temporary dressing. Molar ď‚—Pulpotomy
  • 36. Lots of time: Anteriors/ Premolars/Molars ď‚—Complete pulp extirpation ď‚—Temporary dressing
  • 37. Management of Acute pulpitis with apical periodontitis: ď‚—Diagnosis: ď‚—Vitality: +ve ď‚—Tenderness to percussion: +ve ď‚—The tooth feels high and/or loose and that the teeth will not close together. ď‚—X-ray: Normal to slight widening of periodontal ligament space to small radiolucence.
  • 38. Management Minimal Time: Molar ď‚—Profound Anesthesia: May need an additional carpule. ď‚—Pulpectomy of the largest canal ( distal of lowers and palatal of upper). ď‚—Temporary dressing. ď‚—May need to call the next day to remove pulp from the other canal, pain will not subside if the other canals are the cause of pain.
  • 39. Anterior teeth/ Premolar ď‚—Complete pulp extirpation followed by temporary dressing.
  • 40. Lots of time: ď‚—Complete pulp extirpation of all the canals must be done followed by a temporary dressing.
  • 41. Management of Pulp Necrosis: Rarely seen as an emergency Diagnosis: ď‚—Non vital tooth( may be one or more of its root canal). ď‚—No tenderness to percussion. ď‚—Periapical radiolucency seen on the radiograph.
  • 42. Management: 1- Canal debridement followed by a temporary dressing. 2- Extraction of non restorable tooth. (Analgesics and antibiotics may be required).
  • 43. Acute apical Abscess: The position of the swelling will depend on: ď‚—1- Orientation of the tooth apex. ď‚—2-Relationship of the site of perforation to muscle attachment on the maxilla and mandible.
  • 44. Spreading submandibular swelling due to an acute apical abscess
  • 45. Facial swelling should be detected
  • 46. Palatal swelling associated with upper lateral incisor
  • 47. Facial Swelling Associated with maxillary canine
  • 48. Acute apical abscess producing a facial swelling Tooth drainge of an apical abscess
  • 49. To resolve swelling: 1)Establish drainage through the root canal. 2)Establish drainage by incising a fluctuant swelling. 3)Prescribe antibiotics.
  • 50. Management of a localized soft tissue swelling: * If it is fluctuance, it indicate that pus is present, soft tissue infiltration of anesthesia around the periphery of the infected area. * Incise at the site of greatest fluctuance down to the level of apical bone.
  • 51. • A vertical incision offers improved post operative healing compared with a horizontal incision. • Place the incision in a position to encourage drainage by gravity.
  • 52. •Dissect gently through the deeper tissues and explore all parts of abscess cavity. •The wound should be kept clean with hot salt-water mouth rinses to promote drainage.
  • 53. Diffuse swelling: • From endodontic point of view, the tooth is opened, and the canal is thoroughly instrumented and irrigated, if no drainage is achieved, the apical foramen is instrumented through to encourage drainage from the periapical tissues.
  • 54. • In the absence of drainage through tooth, soft tissue drainage might be established through incision. The drain is sutured into incision wound to ensure tissue drainage.
  • 55. ď‚—The patient who show sign of toxicity, CNS changes, or airway compromise should be considered for immediate hospitalization.
  • 56. Guidelines for Antibiotic Therapy ď‚—Select antibiotic with anaerobic spectrum ď‚—Use a larger dose for a short period of time
  • 57. Antibiotic therapy: • For localized swellings the antibiotic therapy is usually unnecessary (except with patient with depressed host defense). • For diffuse swelling antibiotic are indicated.
  • 58. ď‚—1st choice: penicillin VK Initial dose 1-2 g then 500mg every 6 hours for 7-10 days ď‚—The combination of penicillin and metranidazole (250mg) is recommended 7-10 days.
  • 59. ď‚—Clindamycin are suitable alternatives for patients who are allergic to amoxicillin. ď‚—The dose 300mg followed by 150 to 300mg every 6 hours for 7-10 days. ď‚—(some times signs of colitis)
  • 60. As a general rule: ď‚—Antibiotic therapy should be considered for patients who have signs and symptoms of infection, such as cellulites, fever, or lymphadenitis.
  • 61. FFlleexxiibbllee aannaallggeessiicc ssttrraatteeggyy Aspirin like drug indicated Ibuprofen 200-400mg Aspirin like drug contraindicated Aspirin like drug contraindicated Ibuprofen 400-600mg Ibuprofen 400-600mg Acetaminophen 650-1000mg Acetaminophen 650-1000mg Acetaminophen 650-1000mg Plus equivalent of Codeine 60 mg Acetaminophen 1000mg Plus equivalent of Oxycodone 10 mg Ibuprofen 400-600mg Ibuprofen 400-600mg Plus Plus Acetaminophen 650-1000mg Acetaminophen 650-1000mg Ibuprofen 600-800mg Plus Acetaminophen 1000mg Sever Moderate Mild
  • 64. Root fracture induced during obturation