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PRESENTED BY –
SHILPA C
BAITAEO71
III B.Sc II Sem B Section
DISEASES OF TOMATO
TOMATO
DISEASE CAUSAL ORGANISM
EARLY BLIGHT Alternaria solani
LATE BLIGHT Phytophthora infestens
WILT Fusarium oxysporium
f.sp.lycopersici
Verticillium wilt
BUCK EYE /FRUIT
ROT
Phytopthora nicotianae var.
parasitica
SEPTORIA LEAF
SPOT
Septoria lycopersici
POWDERY MILDEW Levillula taurica
DAMPING OFF Pythium aphanidermatum
BACTERIAL DISEASES OF
TOMATO
DISEASE CAUSAL ORGANISM
BACTERIAL WILT Ralstonia solanacearum
BACTERIAL LEAF
SPOT
Xanthomonas vesicatoria
VIRAL DISEASES OF
TOMATO
DISEASE CAUSAL ORGANISM
LEAF CURL Tomato leaf curl virus
Vector – white fly
TOMATO MOSAIC Tobacco mosaic virus
Vector – aphid
TOMATO SPOTTED WILT Tomato spotted wilt virus
Vector - thrips
CONTENT :
Early blight
Late blight
Early blight - Alternaria
solani
• Early blight is the most destructive disease of
tomatoes in the tropical and subtropical regions.
Each 1% increase in intensity can reduce yield by
1.36%, and complete crop failure can occur when the
disease is most severe.Yield losses of up to 79%
have been reported in the U.S., of which 20-40% is
due to seedling losses (i.e., collar rot) in the field.
• It can affect almost all parts of the tomato plants,
including the leaves, stems, and fruits.
• The plants may not die, but they will be weakened
and will set fewer tomatoes than normal.
Leaves
 Initially small dark spots form on older foliage near the
ground
 Leaf spots are round, brown and can grow up to half inch
in diameter.
 Larger spots have target board or bull eye symptom
like concentric rings and tissue around spots often turns
yellow.
 Severely infected leaves turn brown and fall off, or dead,
dried leaves may cling to the stem
Stem
 Seedling stems are infected at or just above the soil line.
The stem turns brown, sunken and dry (collar rot). If the
infection girdles the stem, the seedling wilts and dies.
 Stem infections on older plants are oval to irregular, dry
brown areas with dark brown concentric rings.
Fruit
 Fruit can be infected at any stage of maturity
 Fruit spots are leathery, black, with raised concentric
FAVOURABLE CONDITION :
 Disease develops at moderate to warm 15 to 27
degree celsius;
 Rainy weather or heavy dew, 90% humidity ,
 June-July sowing - weak and old plants prone to
infection high soil moisture.
DISEASE CYCLE :
 PSI : Mycelium or conidia in infected plant debris.
 SSI : Conidia by wind, water or rain splash .
Mode of spread:
Seed borne and air borne conidia
spore
 Light brown conidia with muriform
shape, but the isolates varied in the size
(length, width and beak length) of the
conidia, the number of cells per
conidium and sporulation times.
 Among the nitrogen sources, potassium
nitrate followed by sodium nitrate and
ammonium molybdate increased the
growth of mycelium.
 Low pH (4-4.5) was found as ideal for
the growth of A. alternata, while the
minimum growth was observed in pH
9.0, irrespective of isolates
 The conidia of A. alternata isolates
germinated at 46°C.
DISEASE CYCLE
MANAGAMENT
CULTURAL CONTROL
 Use pathogen-free seed, or collect seed only from
disease-free plants.
 Remove infected debris from field to reduce inoculum
for the next year.
 Water plants in the morning so plants are wet for the
shortest amount of time.
 Apply plastic or organic mulch to reduce humidity and
provide a barrier between contaminated soil and leaves.
 Use a drip irrigation than over head system to minimize
leaf wetness which provides optimal conditions for
fungal growth.
 Rotate to a non-Solanaceous crop for at least three
years.
 If possible control wild population of Solanaceae. This
 IIHR 2101 (Solanum habrochaires LA-1777) showed highly
resistant reaction, IIHR- 2758 showed moderately resistant
reaction
 Fertilize properly to maintain vigorous plant growth.
Particularly, do not over-fertilize with potassium and maintain
adequate levels of both nitrogen and phosphorus.
 For greenhouse production, early blight has been reduced by
as much as 50% by covering houses with UV-absorbing vinyl
film.
 Increase air circulation in rows. Damp conditions allow for
optimal growth of A. Solani and the disease spreads more
rapidly. This can be achieved by planting farther apart or by
trimming leaves.
CHEMICAL CONTROL
 Some of the fungicides are chlorothalonil, copper
product,mancozeb, potassium bicarbonate, and ziram.
 Bacillus subtilis
FORECAST MODEL EPIDEM
LATE BLIGHT- Phytophthora infestens
 Late blight is a potentially devastating disease of
tomato and potato, infecting leaves, stems, and fruits
of tomato plants. The disease spreads quickly in
fields and can result in total crop failure if untreated.
Late blight of potato was responsible for the Irish
potato famine of the late 1840s.
 phytophthora in Latin means "plant destroyer."
Infected plant tissue dies.
Signs and symptoms :
Leaves :
 Leaves have large, dark brown blotches with a
green gray edge; not confined by major leaf veins
 Infections progress through leaflets and petioles,
resulting in large sections of dry brown foliage.
Stem :
 Stem infections are firm and dark brown with a
rounded edge.
Fruit :
 Firm, dark brown, circular spots grow to cover large
parts of fruits. Spots may become mushy as
secondary bacteria invade.
 In high humidity, thin powdery white fungal growth
appears on infected leaves, fruit, and stems
 In cool, wet weather, entire fields turn brown and
FAVOURABLE CONDITIONS:
 High relative humidity >90 %
 Low temperature 10 – 25 degree celsius
 Cloudy weather and rainfall with splashing rains.
DISEASE CYCLE :
 PSI : Oospores in the infected debris and soil.
 SSI : Sporangia or zoospores dispersed by wind
or
rainwater
Mode of spread:
Soil borne-spread through sporangia or zoospores
dispersed by
wind or rainwater.
MANGEMENT :
CULTURAL METHOD
 Destroy potato piles before the growing season
begins,
 Control volunteer potato plants, as infected plants
can grow from infected tubers.
 Seed infection is unlikely on commercially prepared
tomato seed or on saved seed that has been
thoroughly dried.
 If infection is found in only a few plants within a field,
infected plants should be removed, disced-under,
killed with herbicide to avoid spreading through the
entire field
 Movement of water from infected to healthy field
should be avoided.
 Crop rotation with cereals
CHEMICAL CONTROL
 Prophylactic sprays with copper fungicides or
dithiocarbomates 0.25% .
 Metalxyl / amiston 0.2%.
FORECAST MODEL : TOMCAST
Diseases of Tomato: Early Blight, Late Blight and Management
Diseases of Tomato: Early Blight, Late Blight and Management

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Diseases of Tomato: Early Blight, Late Blight and Management

  • 1. PRESENTED BY – SHILPA C BAITAEO71 III B.Sc II Sem B Section DISEASES OF TOMATO
  • 2. TOMATO DISEASE CAUSAL ORGANISM EARLY BLIGHT Alternaria solani LATE BLIGHT Phytophthora infestens WILT Fusarium oxysporium f.sp.lycopersici Verticillium wilt BUCK EYE /FRUIT ROT Phytopthora nicotianae var. parasitica SEPTORIA LEAF SPOT Septoria lycopersici POWDERY MILDEW Levillula taurica DAMPING OFF Pythium aphanidermatum
  • 3. BACTERIAL DISEASES OF TOMATO DISEASE CAUSAL ORGANISM BACTERIAL WILT Ralstonia solanacearum BACTERIAL LEAF SPOT Xanthomonas vesicatoria
  • 4. VIRAL DISEASES OF TOMATO DISEASE CAUSAL ORGANISM LEAF CURL Tomato leaf curl virus Vector – white fly TOMATO MOSAIC Tobacco mosaic virus Vector – aphid TOMATO SPOTTED WILT Tomato spotted wilt virus Vector - thrips
  • 6. Early blight - Alternaria solani • Early blight is the most destructive disease of tomatoes in the tropical and subtropical regions. Each 1% increase in intensity can reduce yield by 1.36%, and complete crop failure can occur when the disease is most severe.Yield losses of up to 79% have been reported in the U.S., of which 20-40% is due to seedling losses (i.e., collar rot) in the field. • It can affect almost all parts of the tomato plants, including the leaves, stems, and fruits. • The plants may not die, but they will be weakened and will set fewer tomatoes than normal.
  • 7. Leaves  Initially small dark spots form on older foliage near the ground  Leaf spots are round, brown and can grow up to half inch in diameter.  Larger spots have target board or bull eye symptom like concentric rings and tissue around spots often turns yellow.  Severely infected leaves turn brown and fall off, or dead, dried leaves may cling to the stem Stem  Seedling stems are infected at or just above the soil line. The stem turns brown, sunken and dry (collar rot). If the infection girdles the stem, the seedling wilts and dies.  Stem infections on older plants are oval to irregular, dry brown areas with dark brown concentric rings. Fruit  Fruit can be infected at any stage of maturity  Fruit spots are leathery, black, with raised concentric
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  • 15. FAVOURABLE CONDITION :  Disease develops at moderate to warm 15 to 27 degree celsius;  Rainy weather or heavy dew, 90% humidity ,  June-July sowing - weak and old plants prone to infection high soil moisture. DISEASE CYCLE :  PSI : Mycelium or conidia in infected plant debris.  SSI : Conidia by wind, water or rain splash . Mode of spread: Seed borne and air borne conidia
  • 16. spore  Light brown conidia with muriform shape, but the isolates varied in the size (length, width and beak length) of the conidia, the number of cells per conidium and sporulation times.  Among the nitrogen sources, potassium nitrate followed by sodium nitrate and ammonium molybdate increased the growth of mycelium.  Low pH (4-4.5) was found as ideal for the growth of A. alternata, while the minimum growth was observed in pH 9.0, irrespective of isolates  The conidia of A. alternata isolates germinated at 46°C.
  • 18. MANAGAMENT CULTURAL CONTROL  Use pathogen-free seed, or collect seed only from disease-free plants.  Remove infected debris from field to reduce inoculum for the next year.  Water plants in the morning so plants are wet for the shortest amount of time.  Apply plastic or organic mulch to reduce humidity and provide a barrier between contaminated soil and leaves.  Use a drip irrigation than over head system to minimize leaf wetness which provides optimal conditions for fungal growth.  Rotate to a non-Solanaceous crop for at least three years.  If possible control wild population of Solanaceae. This
  • 19.  IIHR 2101 (Solanum habrochaires LA-1777) showed highly resistant reaction, IIHR- 2758 showed moderately resistant reaction  Fertilize properly to maintain vigorous plant growth. Particularly, do not over-fertilize with potassium and maintain adequate levels of both nitrogen and phosphorus.  For greenhouse production, early blight has been reduced by as much as 50% by covering houses with UV-absorbing vinyl film.  Increase air circulation in rows. Damp conditions allow for optimal growth of A. Solani and the disease spreads more rapidly. This can be achieved by planting farther apart or by trimming leaves. CHEMICAL CONTROL  Some of the fungicides are chlorothalonil, copper product,mancozeb, potassium bicarbonate, and ziram.  Bacillus subtilis FORECAST MODEL EPIDEM
  • 20. LATE BLIGHT- Phytophthora infestens  Late blight is a potentially devastating disease of tomato and potato, infecting leaves, stems, and fruits of tomato plants. The disease spreads quickly in fields and can result in total crop failure if untreated. Late blight of potato was responsible for the Irish potato famine of the late 1840s.  phytophthora in Latin means "plant destroyer." Infected plant tissue dies.
  • 21. Signs and symptoms : Leaves :  Leaves have large, dark brown blotches with a green gray edge; not confined by major leaf veins  Infections progress through leaflets and petioles, resulting in large sections of dry brown foliage. Stem :  Stem infections are firm and dark brown with a rounded edge. Fruit :  Firm, dark brown, circular spots grow to cover large parts of fruits. Spots may become mushy as secondary bacteria invade.  In high humidity, thin powdery white fungal growth appears on infected leaves, fruit, and stems  In cool, wet weather, entire fields turn brown and
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  • 31. FAVOURABLE CONDITIONS:  High relative humidity >90 %  Low temperature 10 – 25 degree celsius  Cloudy weather and rainfall with splashing rains. DISEASE CYCLE :  PSI : Oospores in the infected debris and soil.  SSI : Sporangia or zoospores dispersed by wind or rainwater Mode of spread: Soil borne-spread through sporangia or zoospores dispersed by wind or rainwater.
  • 32. MANGEMENT : CULTURAL METHOD  Destroy potato piles before the growing season begins,  Control volunteer potato plants, as infected plants can grow from infected tubers.  Seed infection is unlikely on commercially prepared tomato seed or on saved seed that has been thoroughly dried.  If infection is found in only a few plants within a field, infected plants should be removed, disced-under, killed with herbicide to avoid spreading through the entire field  Movement of water from infected to healthy field should be avoided.  Crop rotation with cereals
  • 33. CHEMICAL CONTROL  Prophylactic sprays with copper fungicides or dithiocarbomates 0.25% .  Metalxyl / amiston 0.2%. FORECAST MODEL : TOMCAST