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Peripheral Nerve Injuries
Part-II
Dr. Anshu Sharma
Assistant Prof.
Dept. of Orthopaediscs, GMC&H.
Ulnar nerve
• This nerve arises from the medial cord of the
brachial plexus.
• Root value: C7,C8 and T1.
Motor branches of the ulnar
nerve
Muscles supplied by ulnar nerve
In the arm: Nil
In the forearm:
1. proximal 1/3
2.distal 1/3
Flexor carpi ulnaris, medial half of flexor
digitorum profundus
Nil
In the hand:
Superficial branch
Deep branch
Hypothenar m/s
Adductor pollicis,
All interossei and
Medial two lumbricals
Low Ulnar Nerve lesions:-
• Injury in distal third of the forearm.
• Sparing of forearm muscles but muscles of hand are
affected.
• Complain of numbness of ulnar one and a half
fingers.
• Claw hand derformity with hyperextension of MCP
joints of the ring and the little fingers.
• Hypothenar and interosseous wasting.
• Froment’s sign positive.
High Ulnar Nerve lesions:-
• Injury proximal to elbow.
• Motor and sensory loss are the same as in low lesions.
• Hand is not markedly deformed because the ulnar
half of flexor digitorum profundus is paralysed and
the fingers are therefore less clawed. (High Ulnar
Paradox)
Tests for individual muscles in Ulnar n. palsy:-
1. Flexor carpi ulnaris:- Asked to palmar flex the wrist against
gravity and the hand deviates towards radial side.
2.Abductor digiti minimi:-
-Asked to abduct the little finger against resistance while
keeping the hand flat on the table.
3. Interossei:
-fn: Palmar interossei do AD-duction(PAD)
Dorsal interossei do AB-duction(DAB) of the fingers at MCP
joints
EGAWA’S TEST
- For dorsal interossei(abductors) of the middle finger.
- With the hand kept on a flat table palmar surface down, pt
is asked to move his middle finger sideways.
- First dorsal interossei muscle can be separately
examinated by asking the pt to abduct the index finger
against resistance.
Egawa’s test
CARD TEST
- For palmar interossei
(adductors) of the fingers.
- Examined by inserting a card
between two extended
fingers and asked to hold
tightly while examiners try to
pull the card out.
- In case of weak palmar
interossei, it is easy to pull out
the card.
Claw hand:-
 Hyperextensiom at MCP joint and flexion at PIP and DIP
joint (paralysis of lumbricals).
 Ulnar paradox= Clawing is more marked in low ulnar
nerve palsy than high ulnar nerve palsy (flexors of fingers
are also paralysed).
 In ulnar nerve palsy, only medial 2 fingers develop
clawing while all 4 fingers develop clawing in combined
median and ulnar nerve palsies.
Claw hand (In Ulnar n. palsy)
3.Adductor pollicis:
- "book test” or froment’s sign.
(use of adductor pollicis and 1st dorsal interosseous)
- In case of paralysis, pt will hold a book by using flexor pollicis
longus(supplied by median n.) in place of the adductor.
- This produces flexion at the IP joint of the thumb.
Book test
Sciatic
Nerve:-
• Most commonly injured in traumatic hip dislocation
(posteriorly) and pelvic fractures.
• Clinical Features include:-
Paralysis of hamstrings and all muscles below the knee.
 Absent ankle jerk.
Loss of sensation below knee except on medial side of the
leg(saphenous branch of the femoral nerve).
Patient walks with Foot Drop and a high stepping gait.
Electro-physiological Nerve Studies:
-Helps in diagnosing nerve injury and
predecting nerve recovery even before it is
apparent clinically.
(A) Electromyography
(B) Nerve conduction study
(C) S-D Curve.
Electromyography (EMG):-
• A graphic recording of the electrical activity of a muscle
at rest and during activity.
• A concentric needle electrode is inserted into the muscle
and connected to an oscilloscope screen and a loudspeaker.
• Useful in deciding:
– Whethere or not a nerve injury is present
– Whether it is a complete or incomplete nerve injury
– Whether any regeneration occurring
– Level of nerve injury
In normal muscles
• A normal muscle at rest shows no electrical activity.
• As the patient slowly contracts the muscle there is
recruitment of one, then more and then multiple
motor units.
• A motor unit defined as the anterior horn cell in the
spinal cord, with its motor axon and the variable
number of muscle fibres it innervates in the muscle.
• In strong contraction, impulses of a number of motor
units firing simultaneously are superimposed, giving
rise to an interference pattern.
In denervated muscles
• The denervated muscle has spontaneous electrical activity
at rest(denervation potentials)
• These potentials are normally suppressed by stronger nerve
action potentials.
• Appears around 15-20 days after the muscle denervation.
• As denervation progresses, more and more denervation
potentials appear.
• If these potentials have not appeared by the end of the 2nd
week of after nerve injury, it is a good prognostic sign.
Nerve Conduction Studies
(NCV):-
• It is a measure of the velocity of conduction of impulse
in a nerve.
• A stimulating electrode is applied over a point on the nerve
trunk and the response is picked up by an electrode at a
distance or directly over the muscle.
• The velocity of the conduction of the impulse b/w any two
points of the nerve can be calculated.
• The normal nerve conduction velocity of motor nerve is
70 Meter/sec.
• Helps to determine
Whether a nerve injury is present
Whether it is a complete or partial nerve injury
Compressive lesions
Treatment of Nerve
Injury
(A) Conservative management:-
 Splintage of the paralysed limb
 Preserve mobility of the joint
 Care of skin and nails
 Physiotherapy
 Relief of pain: analgesics
(B) Operative management :-
 1.Neurolysis
 2.Nerve repair
 3.Nerve grafting
 4.Nerve transfer
 5.Tendon Transfer
Neurolysis:-
• Application of physical or chemical agents to a nerve
in order to cause a temporary degeneration of
targeted nerve fibres (fibrosis).
• Operation where nerve is freed from enveloping scar
(perineural fibrosis) ; called external neurolysis.
• The nerve sheath may be dissected longitudinally to
relieve the pressure from the fibrous tissue within
the nerve(intraneural fibrosis ; internal neurolysis.
Nerve Repair:-
• May be performed within a few days of injury or later.
• Types:
Primary repair: Indicated in clean sharp nerve injuries; done in
the first 6 to 8 hours of injury.
Delayed primary repair: Done in the first 7 to 18 days of injury
when the wound is clean and there are no other major
complicating injuries.
Secondary repair: Done in crushed, avulsed injuries; done at a
delay of 3-6 weeks.
Techniques of Nerve Repair:-
1. Nerve suture
• Indicated when the nerve ends can be brought close to
each other.
• Techniques:
Epineural suture
Epi-perineural suture
Perineural suture
Group fascicular repair
2. Nerve grafting
• Indicated when the gap is more than 10 cm or end to end
suture is likely to result in tension at the suture line.
• Most common nerve used is sural nerve.
• Other source:
Medial antebrachial cutaneous nerve
Third webspace branch of median n
Lateral antebrachial cutaneous nerve
Palmar cutaneous and dorsal cutaneous branch of ulnar n
Methods of closing Nerve
Gaps:-
• Mobilization of the nerve on both sides of the lesion.
• Alteration of the course of the nerve.
• Sacrificing some unimportant branch if it is hampering
nerve mobilization.
• Ralaxation of the nerve by temporarily positioning the
joints in a favourable position.
• Bone Resection.
• Nerve grafting
• Nerve crossing ( pedicle grafting )
Signs of Nerve
Regeneration:-1. Tinel’s sign: On gently tapping over the nerve along its
course,from distal to proximal, a pin and needle sensation
is felt in the area of the skin supplied by the nerve. A
distal progression of the level at which it occurs, suggests
regeneration(1 mm/day).
2. Motor examination: The muscle supplied nearest to the
site of injury is the first to recover. Muscles in the more
distal area begin to contract as they are reinnervated one
after another (motor march: absent in neuropraxia).
3. Electordiagnostic test: Helps in predecting nerve
recovery even before it is apparent clinically.
Electromyography
Nerve conduction study
S-D Curve.
Prognostic Factors for Nerve repair:-
Factors outside our influence
• Nerve injured (motor, sensory, mixed)
• Level of lesion (proximal – distal)
• Accompanying lesion (fractures etc.)
• Age of patient
Factors which we can influence
• Delay between injury and surgery
• Surgical technique
Peripheral Nerve Injuries: Ulnar Nerve Palsy Tests

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Peripheral Nerve Injuries: Ulnar Nerve Palsy Tests

  • 1. Peripheral Nerve Injuries Part-II Dr. Anshu Sharma Assistant Prof. Dept. of Orthopaediscs, GMC&H.
  • 2. Ulnar nerve • This nerve arises from the medial cord of the brachial plexus. • Root value: C7,C8 and T1.
  • 3.
  • 4. Motor branches of the ulnar nerve Muscles supplied by ulnar nerve In the arm: Nil In the forearm: 1. proximal 1/3 2.distal 1/3 Flexor carpi ulnaris, medial half of flexor digitorum profundus Nil In the hand: Superficial branch Deep branch Hypothenar m/s Adductor pollicis, All interossei and Medial two lumbricals
  • 5. Low Ulnar Nerve lesions:- • Injury in distal third of the forearm. • Sparing of forearm muscles but muscles of hand are affected. • Complain of numbness of ulnar one and a half fingers. • Claw hand derformity with hyperextension of MCP joints of the ring and the little fingers. • Hypothenar and interosseous wasting. • Froment’s sign positive.
  • 6. High Ulnar Nerve lesions:- • Injury proximal to elbow. • Motor and sensory loss are the same as in low lesions. • Hand is not markedly deformed because the ulnar half of flexor digitorum profundus is paralysed and the fingers are therefore less clawed. (High Ulnar Paradox)
  • 7. Tests for individual muscles in Ulnar n. palsy:- 1. Flexor carpi ulnaris:- Asked to palmar flex the wrist against gravity and the hand deviates towards radial side. 2.Abductor digiti minimi:- -Asked to abduct the little finger against resistance while keeping the hand flat on the table.
  • 8. 3. Interossei: -fn: Palmar interossei do AD-duction(PAD) Dorsal interossei do AB-duction(DAB) of the fingers at MCP joints EGAWA’S TEST - For dorsal interossei(abductors) of the middle finger. - With the hand kept on a flat table palmar surface down, pt is asked to move his middle finger sideways. - First dorsal interossei muscle can be separately examinated by asking the pt to abduct the index finger against resistance.
  • 10. CARD TEST - For palmar interossei (adductors) of the fingers. - Examined by inserting a card between two extended fingers and asked to hold tightly while examiners try to pull the card out. - In case of weak palmar interossei, it is easy to pull out the card.
  • 11. Claw hand:-  Hyperextensiom at MCP joint and flexion at PIP and DIP joint (paralysis of lumbricals).  Ulnar paradox= Clawing is more marked in low ulnar nerve palsy than high ulnar nerve palsy (flexors of fingers are also paralysed).  In ulnar nerve palsy, only medial 2 fingers develop clawing while all 4 fingers develop clawing in combined median and ulnar nerve palsies.
  • 12. Claw hand (In Ulnar n. palsy)
  • 13. 3.Adductor pollicis: - "book test” or froment’s sign. (use of adductor pollicis and 1st dorsal interosseous) - In case of paralysis, pt will hold a book by using flexor pollicis longus(supplied by median n.) in place of the adductor. - This produces flexion at the IP joint of the thumb.
  • 16. • Most commonly injured in traumatic hip dislocation (posteriorly) and pelvic fractures. • Clinical Features include:- Paralysis of hamstrings and all muscles below the knee.  Absent ankle jerk. Loss of sensation below knee except on medial side of the leg(saphenous branch of the femoral nerve). Patient walks with Foot Drop and a high stepping gait.
  • 17.
  • 18. Electro-physiological Nerve Studies: -Helps in diagnosing nerve injury and predecting nerve recovery even before it is apparent clinically. (A) Electromyography (B) Nerve conduction study (C) S-D Curve.
  • 19. Electromyography (EMG):- • A graphic recording of the electrical activity of a muscle at rest and during activity. • A concentric needle electrode is inserted into the muscle and connected to an oscilloscope screen and a loudspeaker. • Useful in deciding: – Whethere or not a nerve injury is present – Whether it is a complete or incomplete nerve injury – Whether any regeneration occurring – Level of nerve injury
  • 20. In normal muscles • A normal muscle at rest shows no electrical activity. • As the patient slowly contracts the muscle there is recruitment of one, then more and then multiple motor units. • A motor unit defined as the anterior horn cell in the spinal cord, with its motor axon and the variable number of muscle fibres it innervates in the muscle. • In strong contraction, impulses of a number of motor units firing simultaneously are superimposed, giving rise to an interference pattern.
  • 21. In denervated muscles • The denervated muscle has spontaneous electrical activity at rest(denervation potentials) • These potentials are normally suppressed by stronger nerve action potentials. • Appears around 15-20 days after the muscle denervation. • As denervation progresses, more and more denervation potentials appear. • If these potentials have not appeared by the end of the 2nd week of after nerve injury, it is a good prognostic sign.
  • 22. Nerve Conduction Studies (NCV):- • It is a measure of the velocity of conduction of impulse in a nerve. • A stimulating electrode is applied over a point on the nerve trunk and the response is picked up by an electrode at a distance or directly over the muscle. • The velocity of the conduction of the impulse b/w any two points of the nerve can be calculated. • The normal nerve conduction velocity of motor nerve is 70 Meter/sec. • Helps to determine Whether a nerve injury is present Whether it is a complete or partial nerve injury Compressive lesions
  • 23. Treatment of Nerve Injury (A) Conservative management:-  Splintage of the paralysed limb  Preserve mobility of the joint  Care of skin and nails  Physiotherapy  Relief of pain: analgesics (B) Operative management :-  1.Neurolysis  2.Nerve repair  3.Nerve grafting  4.Nerve transfer  5.Tendon Transfer
  • 24. Neurolysis:- • Application of physical or chemical agents to a nerve in order to cause a temporary degeneration of targeted nerve fibres (fibrosis). • Operation where nerve is freed from enveloping scar (perineural fibrosis) ; called external neurolysis. • The nerve sheath may be dissected longitudinally to relieve the pressure from the fibrous tissue within the nerve(intraneural fibrosis ; internal neurolysis.
  • 25. Nerve Repair:- • May be performed within a few days of injury or later. • Types: Primary repair: Indicated in clean sharp nerve injuries; done in the first 6 to 8 hours of injury. Delayed primary repair: Done in the first 7 to 18 days of injury when the wound is clean and there are no other major complicating injuries. Secondary repair: Done in crushed, avulsed injuries; done at a delay of 3-6 weeks.
  • 26. Techniques of Nerve Repair:- 1. Nerve suture • Indicated when the nerve ends can be brought close to each other. • Techniques: Epineural suture Epi-perineural suture Perineural suture Group fascicular repair
  • 27.
  • 28.
  • 29.
  • 30. 2. Nerve grafting • Indicated when the gap is more than 10 cm or end to end suture is likely to result in tension at the suture line. • Most common nerve used is sural nerve. • Other source: Medial antebrachial cutaneous nerve Third webspace branch of median n Lateral antebrachial cutaneous nerve Palmar cutaneous and dorsal cutaneous branch of ulnar n
  • 31.
  • 32. Methods of closing Nerve Gaps:- • Mobilization of the nerve on both sides of the lesion. • Alteration of the course of the nerve. • Sacrificing some unimportant branch if it is hampering nerve mobilization. • Ralaxation of the nerve by temporarily positioning the joints in a favourable position. • Bone Resection. • Nerve grafting • Nerve crossing ( pedicle grafting )
  • 33. Signs of Nerve Regeneration:-1. Tinel’s sign: On gently tapping over the nerve along its course,from distal to proximal, a pin and needle sensation is felt in the area of the skin supplied by the nerve. A distal progression of the level at which it occurs, suggests regeneration(1 mm/day). 2. Motor examination: The muscle supplied nearest to the site of injury is the first to recover. Muscles in the more distal area begin to contract as they are reinnervated one after another (motor march: absent in neuropraxia). 3. Electordiagnostic test: Helps in predecting nerve recovery even before it is apparent clinically. Electromyography Nerve conduction study S-D Curve.
  • 34. Prognostic Factors for Nerve repair:- Factors outside our influence • Nerve injured (motor, sensory, mixed) • Level of lesion (proximal – distal) • Accompanying lesion (fractures etc.) • Age of patient Factors which we can influence • Delay between injury and surgery • Surgical technique