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Head Injury: Common Issues
Dr Amit Agrawal, MCh
 To describe which are the common pathophysiological
 features ofhead injury
 To define the mechanisms of head injuries
 Characteristic clinical and imaging findings
 To define the management and outcome
Objectives
Basic Anatomy
 Scalp
 Skull
 Meninges
 Dura Mater
 Arachnoid
 Pia Mater
 Brain Tissue
 CSF and Blood
Head Injury
 Any trauma to the scalp, skull, or brain
 Head trauma includes an alteration in consciousness
Head Injury: Causes
 Motor vehicle accidents
 Firearm-related injuries
 Falls
 Assaults
 Sports-related injuries
 Recreational accidents
Epidemiology
 Mild TBI: 82%
 Moderate to severe TBI: 14%
 Fatal TBI: 5% (7000 deaths per year)
Pathophysiology of Brain Injury
Brain Injury
Primary Secondary
1.Delayed cell death2.Intracranial hypertension
and mass lesion
3.Ischemia systemic
hypoxia,hypercarbia and
hypotension
Primary Brain Injury
Primary
Focal
@SITE
Contra-
coup
Diffuse DAI
Type of injuries
 Scalp Injuries
 Skull Fractures
 Depressed Skull Fractures
 Basilar Skull Fractures
 Vascular Injuries
 Penetrating Head Injury
 Intracranial Hemorrhage
 Epidural Hematoma
 Subdural Hematoma
 Subarachnoid Hemorrhage
 Intracerebral Hemorrhage
Clinical features
 Diminishing level of consciousness
 Headache
 Vomiting
 Seizures
 Ear, nasal or oral bleed
 Focal weakness
 Pupillary changes
 Papilledema
Epidural haematoma
 Classic: arterial origin, blood
collects between skull and dura
 Middle Meningeal Artery
 Dural Venous Sinuses
 Brief loss of consciousness
 Headache
 Drowsiness
 Nausea
 Vomitting
 Rapid clinical deterioration
 Talk & die
Subdural Hematoma
 Due to tearing of bridging
veins, blood collects
between dura and cortex
Subarachnoid hemorrhage
 Disruption of small vessels on
the cortex, occur along the
falx, tentorium, or outer
cortical surface
Intraventricular Hemorrhage
Diffuse Axonal Injury
Skull fractures
 Vault
 Skull base
 Linear
 Stellate
 Depressed
 Non-depressed
 Open/closed
Intra Cerebral Heamatoma
 Formed within brain tissue &
caused by shearing or tensile
forces that mechanically
stretch and tear deep small
caliber arterioles
 Most common in temporal
and frontal regions
 C/F depend on site involved
Concussion
 Temporary & brief interruption of neurological function after
minor head injury
 Due to shearing / stretching of white matter fibers at the time
of impact or temporary neuronal dysfunction
 Headache
 Confusion
 Amnesia
 CT/MRI cannot detect
Spine injury
Neurological Assessment
 Rapid Trauma Neurological Examination
1. Level Of Consciousness
2. Pupils
3. EOM
4. Fundi
5. Extremity Movement
6. Response To Pain
7. Deep Tendon Reflexes
8. Plantar Responses
9. Brainstem Reflexes
Level of Consciousness
 Glasgow Coma Scale
Eye Opening Best Verbal Best Motor
Spontaneous 4 Oriented 5 Obeys Command
6
To Voice 3 Confused 4 Localizes 5
To Pain 2 Inappropriate 3 Withdraws 4
None 1 Incomprehensible 2 Flexion 3
None 1 Extension 2
None 1
Diagnosis
 CT is diagnostic
 MRI
 Cerebral angiography
Indications for CT scan
 Skull fracture
 Deteriorating GCS
 Neurologic deficit
 Amnesia, headache
 Seizure
Management
 History
 General Examination
 Limited Neurologic Examination
 C-spine and other X-rays as indicated
 CT scan
Management
 Primary Survey and Resuscitation
 A- Airway
 B- Breathing
 C- Circulation
 D- Disability assessment
 E- Exposure
 Secondary Survey and ‘AMPLE’ history
 Conservative management
 Surgical management
Initial Stabilization
 Cervical spine injury
 Hypotension
 Hypothermia
 Neurogenic Hypertension
 CT scan only after hemodynamic stabilization
 Medical therapy for raised ICP
 Immediate neurosurgeon opinion
 If needed surgical management
Criteria for admission
 No CT scanner available
 Abnormal CT scan findings
 All penetrating head injuries
 Skull fractures
 CSF leak
 Deteriorating level of consciousness
 Moderate to severe headache
 Significant alcohol / drug intoxication
 Significant associated injuries
 Head end elevation – 30 degree
 Intravenous fluids
 Maintain normovolemia
 Anti-epileptics
 Hypotonic/glucose containing fluids should not be used
 Serum sodium levels monitored daily
Conservative management
Surgical management
 Scalp wounds cleaning & debridemant
 Elevation of depressed Fractures
 Craniotomy & evacuation of Haematoma
 Cranial decompression for reduction of ICT
Prevention
Health Promotion
 Prevent car and motorcycle accidents
 Wear safety helmets
Head Injury: Common Issues

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Head Injury: Common Issues

  • 1. Head Injury: Common Issues Dr Amit Agrawal, MCh
  • 2.  To describe which are the common pathophysiological  features ofhead injury  To define the mechanisms of head injuries  Characteristic clinical and imaging findings  To define the management and outcome Objectives
  • 3. Basic Anatomy  Scalp  Skull  Meninges  Dura Mater  Arachnoid  Pia Mater  Brain Tissue  CSF and Blood
  • 4. Head Injury  Any trauma to the scalp, skull, or brain  Head trauma includes an alteration in consciousness
  • 5. Head Injury: Causes  Motor vehicle accidents  Firearm-related injuries  Falls  Assaults  Sports-related injuries  Recreational accidents
  • 6. Epidemiology  Mild TBI: 82%  Moderate to severe TBI: 14%  Fatal TBI: 5% (7000 deaths per year)
  • 7. Pathophysiology of Brain Injury Brain Injury Primary Secondary 1.Delayed cell death2.Intracranial hypertension and mass lesion 3.Ischemia systemic hypoxia,hypercarbia and hypotension
  • 9. Type of injuries  Scalp Injuries  Skull Fractures  Depressed Skull Fractures  Basilar Skull Fractures  Vascular Injuries  Penetrating Head Injury  Intracranial Hemorrhage  Epidural Hematoma  Subdural Hematoma  Subarachnoid Hemorrhage  Intracerebral Hemorrhage
  • 10. Clinical features  Diminishing level of consciousness  Headache  Vomiting  Seizures  Ear, nasal or oral bleed  Focal weakness  Pupillary changes  Papilledema
  • 11. Epidural haematoma  Classic: arterial origin, blood collects between skull and dura  Middle Meningeal Artery  Dural Venous Sinuses  Brief loss of consciousness  Headache  Drowsiness  Nausea  Vomitting  Rapid clinical deterioration  Talk & die
  • 12. Subdural Hematoma  Due to tearing of bridging veins, blood collects between dura and cortex
  • 13. Subarachnoid hemorrhage  Disruption of small vessels on the cortex, occur along the falx, tentorium, or outer cortical surface
  • 16. Skull fractures  Vault  Skull base  Linear  Stellate  Depressed  Non-depressed  Open/closed
  • 17. Intra Cerebral Heamatoma  Formed within brain tissue & caused by shearing or tensile forces that mechanically stretch and tear deep small caliber arterioles  Most common in temporal and frontal regions  C/F depend on site involved
  • 18. Concussion  Temporary & brief interruption of neurological function after minor head injury  Due to shearing / stretching of white matter fibers at the time of impact or temporary neuronal dysfunction  Headache  Confusion  Amnesia  CT/MRI cannot detect
  • 20. Neurological Assessment  Rapid Trauma Neurological Examination 1. Level Of Consciousness 2. Pupils 3. EOM 4. Fundi 5. Extremity Movement 6. Response To Pain 7. Deep Tendon Reflexes 8. Plantar Responses 9. Brainstem Reflexes
  • 21. Level of Consciousness  Glasgow Coma Scale Eye Opening Best Verbal Best Motor Spontaneous 4 Oriented 5 Obeys Command 6 To Voice 3 Confused 4 Localizes 5 To Pain 2 Inappropriate 3 Withdraws 4 None 1 Incomprehensible 2 Flexion 3 None 1 Extension 2 None 1
  • 22. Diagnosis  CT is diagnostic  MRI  Cerebral angiography
  • 23. Indications for CT scan  Skull fracture  Deteriorating GCS  Neurologic deficit  Amnesia, headache  Seizure
  • 24. Management  History  General Examination  Limited Neurologic Examination  C-spine and other X-rays as indicated  CT scan
  • 25. Management  Primary Survey and Resuscitation  A- Airway  B- Breathing  C- Circulation  D- Disability assessment  E- Exposure  Secondary Survey and ‘AMPLE’ history  Conservative management  Surgical management
  • 26. Initial Stabilization  Cervical spine injury  Hypotension  Hypothermia  Neurogenic Hypertension  CT scan only after hemodynamic stabilization  Medical therapy for raised ICP  Immediate neurosurgeon opinion  If needed surgical management
  • 27. Criteria for admission  No CT scanner available  Abnormal CT scan findings  All penetrating head injuries  Skull fractures  CSF leak  Deteriorating level of consciousness  Moderate to severe headache  Significant alcohol / drug intoxication  Significant associated injuries
  • 28.  Head end elevation – 30 degree  Intravenous fluids  Maintain normovolemia  Anti-epileptics  Hypotonic/glucose containing fluids should not be used  Serum sodium levels monitored daily Conservative management
  • 29. Surgical management  Scalp wounds cleaning & debridemant  Elevation of depressed Fractures  Craniotomy & evacuation of Haematoma  Cranial decompression for reduction of ICT
  • 30. Prevention Health Promotion  Prevent car and motorcycle accidents  Wear safety helmets