2. INTRODUCTION
The treatment of pulpally involved teeth in primary dentition poses an unique
challenge
Young dental pulp in primary teeth , has a high potential for repair
High degree of cellularity and vascularity is seen in these tissues in stages prior to
advanced physiological resorption of roots
The young pulp lends itself more readily to procedures concerned with preservation
of pulp vitality
3. PULP is defined as soft tissue forming
inner structure of tooth and
containing nerve and blood vessel , also
called as tooth pulp.
The dental pulp occupies the center of each
tooth and shapes itself to a maturation of
the tooth .
7. Defined as a procedure where a small amount of carious dentin is retained
in deep areas of cavity to avoid exposure of pulp, followed by placement
of a suitable medicament and restorative material that seals off the carious
dentin and encourages pulp recovery – Ingle
A procedure in which only the gross caries is removed from the lesion and
the cavity is sealed for a time with a biocompatible material – McDonald
DEFINITION
8. OBJECTIVES:
Arresting the carious process
Promoting dentin sclerosis
Stimulating formation of tertiary dentin
Remineralization of carious dentin
9. INDICATIONS:
CLINICAL EXAMINATION:
Large carious lesion
Absence of lymphadenopathy
Normal appearance of adjacent
gingiva
Normal colour of tooth
RADIOGRAPHIC
EXAMINATION
Large carious lesion in close
proximity to the pulp
Normal lamina dura
Normal periodontal ligament
space
No interradicular or periapical
radiolucency
When pulp inflammation is judged to be minimal and complete removal of caries would cause pulp exposure
HISTORY:
o Mild discomfort from chemical and thermal stimuli
o Absence of spontaneous pain
10. CONTRAINDICATIONS
CLINICAL EXAMINATION:
Excessive tooth mobility
Soft leathery dentin covering a
large area of the cavity, in a non-
restorable tooth
Parulis approximating the affected
tooth
Tooth discoloration
Non-responsiveness to pulp testing
RADIOGRAPHIC
EXAMINATION:
Pulp exposure
Interrupted or broken lamina
dura
Widened PDL space
Radiolucencies in the root apices
or furcation areas
Any signs of pulpal or periapical pathology & Soft leathery dentin covering a very large area of the cavity
HISTORY:
o Sharp, penetrating pain persisting after withdrawal of stimulus
o Prolonged spontaneous pain(nocturnal)
11. Treatment procedure
Administer LA and isolate with rubber dam
Prepare outline cavity and remove the infected
dentin(caries detector dye may be used)
Stop excavation after firm resistance of sound
dentin is felt
Cavity flushed with saline and dried with
cotton pellet
Site covered with Ca(OH)2 and remainder with
ZOE
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12. 6-8 WEEKS LATER
Final restoration is then placed
Cover the floor with Ca(OH)2 and base is built with reinforced ZOE or GIC
Cavity is washed out and dried gently
The area around the potential exposure will appear whitish, may be soft, which is predentin, don’t
disturb this area
Previous remaining carious dentin will have become dried out, flaky and easily removed
But if caries remain on re-entry, remove the caries
If a reparative dentin is formed, permanent restn followed by full coverage restoration is chosen
Between the appointment, history must be negative and temp restn should be intact
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14. OUTCOME
3 new types of dentin are formed
Cellular fibrillar dentin – first 2 months
Globular dentin – 3 months
Tubular dentin – (uniform mineralized dentin)
1/5th of reparative dentin formation occurs in less than 30 days
After 3 months, 0.1mm is formed
16. DEFINITION:
It is the placement of the calcium hydroxide preparation on a small
(pinpoint) pulpal exposure – Mathewson
It consists merely of placing a protective material over the site of the
exposed pulp prior to restoring the tooth - Finn
17. OBJECTIVES
Preservation of vitality of the radicular pulp.
Relief of pain in patients with acute pulpagia.
Ensuring the continuity of normal apexogenesis in immature
permanent teeth
18. INDICATIONS:
Accidental pin point exposure of pulp when excavating deep caries,
less than 1 sq. mm. surrounded by clean dentin for (<24hours)
Traumatic fracture of tooth(<24 hours) with pin point exposure
Iatrogenic exposure during cavity preparation & crown preparation
Bleed if touched but not excessively and controlled easily with cotton
pellet
Normal vitality tests without tender to percussion
No radiographic evidence of periradicular pathology
Young patient
19. CONTRAINDICATIONS
History of severe spontaneous tooth aches at night
Excessive tooth mobility
Periodontal ligament thickening
Intraradicular radiolucency
Excessive bleeding at exposure site
Purulent , serous exudate from exposure
External or internal resorption
Swelling and fistula with associated tooth
20. Treatment procedure
Final restoration is done under determining the success by determination of dentin bridge,
maintenance of pulp vitality, lack of pain and minimal inflammatory response
Place temporary restoration
Place the pulp capping material on exposed pulp with application of minimal pressure so as to avoid
forcing the material into pulp chamber
Hemorrhage is arrested with light pressure from sterile cotton pellet
Cavity irrigated with saline / chloramine T / distilled water
Once exposure is encountered, further manipulation of pulp is avoided
21. DPC CONTRAINDICATED IN PRIMARY TEETH
Localization of infection & inflammation in primary teeth is poorer than in permanent teeth. [Mc
Donalds,1956]
Incidence of reparative dentin formation in primary teeth is more extensive than permanent Teeth.
[Sayegh , 1968]
Primary pulp contain high cellular content which might be responsible for failures. Primary pulp
responds more rapidly to the effects of dentinal caries then the perm. Teeth. [Rayner & Southam,
1979]
Undifferentiated mesenchymal cells may differentiate into osteoclasts in response to caries or pulp
capping material which could lead to internal resorption. [Kennedy,1985]