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NCM 102- Group Activity PowerPoint Presentation February 23- 27, 2009 ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Urinary System ,[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Kidney ,[object Object],[object Object],[object Object]
 
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
 
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Ureters ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
  Urinary Bladder ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Urethra ,[object Object],[object Object],[object Object],[object Object]
Acid- Base Regulation ,[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Regulation System ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object]
Acid Base Imbalances ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],D. Causes -DKA or Diabetic Ketoacidosis with starvation -Salicylate overdose -Lactic Acidosis 2 o  hypoperfusion -Methanol and ethylene Glycol toxicity -uremia
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],E. Manifestations A. Acute - headache - drowsiness - nausea and vomiting - confusion - increased RR and depth - shock - peripheral vasodilation - dysrhythmia - cold and clammy skin - decreased BP B. Chronic -asymptomatic
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],C. Laboratory Findings (ABG) - high plasma pH (above 7.45) - normal or high PCO 2  (above 45 mmHg) as a compensatory elevation - high plasma bicarbonate: - above 28 mEq/L in adults - above 25 mEq/L in children - high urine pH (above 7) - excessive diarrhea -  hyperaldosteronism - Cushing’s  syndrome - villous adenoma - cystic fibrosis - hypokalemia
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object]
HYDRONEPHROSIS ,[object Object]
Etiology ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
PRESENCE OF CALCULUS, TUMORS, SCAR TISSUE, CONGENITAL DEFECTS, KINK IN THE URETER URINE FLOW OBSTRUCTION URINE ACCUMULATION & STASIS PRESSURE IN THE KIDNEY WALLS DISTENTION OF THE KIDNEYS SUSTAINED/INTERMITTENT INCREASE PRESSURE IRREVERSIBLE NEPHRON DESTRUCTION Pathophysiology
Assessment ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Diagnostics: ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Complications ,[object Object],[object Object]
Nursing Diagnosis ,[object Object],[object Object],[object Object],[object Object],[object Object]
INTERVENTION ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],Medical
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],Surgical
NEPHROTIC SYNDROME ,[object Object]
ETIOLOGY ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Pathophysiology   Membranous glomerulonephritis Glomerular basement membrane damage Glomerular permeability to plasma protein Albumin depletion in the blood Alteration in osmotic pressure in the vessels Fluid moves to interstitial spaces Increase synthesis of LDL, HDL in the liver with decrease lipid catabolism Decreased plasma volume Stimulates aldosterone secretion Sodium & water retention Decreased glomerular filtration rate edema edema Hyperlipidemia lipiduria proteinuria hypoalbuminemia
Assessment ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Diagnostics: ,[object Object],[object Object],[object Object],[object Object]
Complications ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Nursing Diagnosis ,[object Object],[object Object],[object Object],[object Object],[object Object]
URINARY TRACT INFECTION ,[object Object]
Etiology ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Assessment ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Diagnostics ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Complications ,[object Object],[object Object],[object Object],[object Object]
Nursing Diagnosis ,[object Object],[object Object],[object Object],[object Object],[object Object]
Nursing Interventions ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
ACUTE GLOMERULONEPHRITIS ,[object Object]
Etiology ,[object Object],[object Object]
A ntigen (group a beta-hemolytic streptococcus Antigen- antibody product Deposition of antigen-antibody complex in glomerulus Increased production of epithelial cells lining the glomerulus Leukocytes infiltrate the glomerulus  Thickening of the glomerular filtration membrane Pathophysiology Scarring and loss of glomerular filtration membrane Decreased glomerular filtration rate
Assessment ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Diagnostics: ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Complications ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Nursing Diagnosis ,[object Object],[object Object],[object Object],[object Object],[object Object]
Nursing Interventions ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
CHRONIC GLOMERULONEPHRITIS ,[object Object]
Etiology ,[object Object],[object Object]
Assessment ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Diagnostics: ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Complications ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Nursing Diagnosis ,[object Object],[object Object],[object Object],[object Object],[object Object]
Nursing Interventions ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
REFERENCES: ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object]

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Group 1

  • 1.
  • 2.
  • 3.
  • 4.
  • 5.  
  • 6.
  • 7.
  • 8.
  • 9.  
  • 10.
  • 11.
  • 12.
  • 13.
  • 14.
  • 15.
  • 16.
  • 17.
  • 18.
  • 19.
  • 20.
  • 21.
  • 22.
  • 23.
  • 24.
  • 25.
  • 26.
  • 27.
  • 28.
  • 29.
  • 30.
  • 31.
  • 32.
  • 33.
  • 34.
  • 35.
  • 36. PRESENCE OF CALCULUS, TUMORS, SCAR TISSUE, CONGENITAL DEFECTS, KINK IN THE URETER URINE FLOW OBSTRUCTION URINE ACCUMULATION & STASIS PRESSURE IN THE KIDNEY WALLS DISTENTION OF THE KIDNEYS SUSTAINED/INTERMITTENT INCREASE PRESSURE IRREVERSIBLE NEPHRON DESTRUCTION Pathophysiology
  • 37.
  • 38.
  • 39.
  • 40.
  • 41.
  • 42.
  • 43.
  • 44.
  • 45.
  • 46.
  • 47. Pathophysiology Membranous glomerulonephritis Glomerular basement membrane damage Glomerular permeability to plasma protein Albumin depletion in the blood Alteration in osmotic pressure in the vessels Fluid moves to interstitial spaces Increase synthesis of LDL, HDL in the liver with decrease lipid catabolism Decreased plasma volume Stimulates aldosterone secretion Sodium & water retention Decreased glomerular filtration rate edema edema Hyperlipidemia lipiduria proteinuria hypoalbuminemia
  • 48.
  • 49.
  • 50.
  • 51.
  • 52.
  • 53.
  • 54.
  • 55.
  • 56.
  • 57.
  • 58.
  • 59.
  • 60.
  • 61. A ntigen (group a beta-hemolytic streptococcus Antigen- antibody product Deposition of antigen-antibody complex in glomerulus Increased production of epithelial cells lining the glomerulus Leukocytes infiltrate the glomerulus Thickening of the glomerular filtration membrane Pathophysiology Scarring and loss of glomerular filtration membrane Decreased glomerular filtration rate
  • 62.
  • 63.
  • 64.
  • 65.
  • 66.
  • 67.
  • 68.
  • 69.
  • 70.
  • 71.
  • 72.
  • 73.
  • 74.
  • 75.