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By
MS. Sonam Patode
 At the end of the seminar the group will
be able to get the knowledge about the
oxygen insufficiency, develop a positive
attitude towards it and practice this
knowledge in teaching and clinical areas.
At the end of the class the student will be able
to :
 Define oxygen insufficiency.
 Enlist the etiology of oxygen insufficiency.
 Enumerate factors affecting oxygenation
 Know the types of oxygen insufficiency.
 Discuss the pathophysiology of oxygen
insufficiency.
 Enlist sign and symptoms of oxygen
insufficiency.
 Explain different diagnostic evaluation of
oxygen insufficiency.
 Describe the management of oxygen
insufficiency.
 Identify the nurses role in the management of
oxygen insufficiency.
 Explain about oxygen therapy.
 Discuss about nurses responsibility while
administering oxygen.
 All living things use oxygen or depend on
organisms that use oxygen in some way.
 We use oxygen to break down simple sugar
and release energy.
 This can be done through respiration or
fermentation.
 Animals mainly use respiration.
 The process that breaks apart simple food
molecules to release energy.
 It occurs inside cells.
 What YOU do with the oxygen you take in.
The molecule most living things use for
energy — including us!
 We break down food into smaller
molecules during digestion. One of the
small molecules is glucose.
 Glucose leaves your intestines, goes into
your blood and is taken to every cell in
your body.
 In your cells, oxygen is used to split glucose
apart — releasing energy, water and carbon
dioxide.
 Plants take in carbon dioxide and water and
use them to make food. Their food is simple
sugar — glucose.
 Plants pull the carbon off Co2 and use the
carbon in glucose. (They do not need the
oxygen for this. They get that from water, H2O.)
 Plants release the oxygen (O2) back into
the atmosphere.
 Other organisms use the free oxygen for
respiration.
 We keep destroying natural areas, especially
forested areas with many plants and replacing
them with buildings, parking lots, lawns, etc.
 Fewer plants mean less oxygen and more carbon
dioxide.
 This disturbs the balance of the natural cycle.
 Every time something burns (combustion),
more carbon dioxide is released into the
atmosphere.
 We add more and more Co2 and destroy more
and more of the plants that clean the air for
us.
 Stop destroying and promote regrowth of
natural areas — especially forests.
 Burn less (fossil fuels, forest fires, etc.)
 Oxygen insufficiency means “ deficient in
oxygen”.
 The normal range of oxygen in the external
blood should be 80-100 mm of Hg.
 For treating Oxygen insufficiency effectively,
early diagnosis and correct cause should be
ruled out.
 The only management for Oxygen
insufficiency is Oxygen administration.
 Oxygenation means the delivery of
oxygen to the body’s tissues and cell
it is necessary to maintain life and
health
 Oxygenation results from the co-
operative function of 3 major system
1.Pulmonary
2.Hematological
3.Cardiovascular system
 Pulmonary ventilation:-
A movement of air into and out
of lungs. It’s main purpose is to
supply fresh air. composed of…..
1.Inspiration: air flows into the lungs.
2.Expiration: air moves out of lungs.
 Oxygen insufficiency is a condition in
which the body as a whole or a region is
deprived of adequate oxygen supply.
 Oxygen insufficiency is a failure to
provide adequate oxygen to cells of the
body and to remove excess carbon
dioxide from them.
 Transference of
1.Oxygen from the atmosphere to the
tissues.
2.And carbon dioxide from the tissues
to the atmosphere.
 External respiration:-
It takes place in the lungs. The O2
is absorbed from air and into the
blood and Co2 is excreted from the
blood into the air.
 Internal respiration:
or tissue respiration O2 is
transferred from blood to the tissue
which gives up Co2
 Ventilation
 Pulmonary diffusion or gaseous
exchange at the alveolar level
 Transport of gases between lungs
and tissues through blood
 Gases exchange at tissue level
 Utilization of oxygen and production
of Co2 by the tissues(cellular
respiration)
 It is the process of movement of air
from the atmosphere to the lungs
and vice versa.
 Inspiration is an active process
brought about by the contraction of
inspiratory muscles, where as
Expiration is a passive process due
to elastic recoil of lungs.
 The space between parietal pleura
and visceral pleura is filled with the
pleural fluid, which lubricates the
lungs movements which is called as
intrapleural pressure.
 IPP is the pressure in the lungs.
 Easily determined by a water filled
spirometer attached to a
kymograph.
1.TIDAL VOLUME(TV):
Is the volume of air that moves into the
lunges with each inspiration or that amount
that moves out with each expiration. It is
about 0.5 liters In an adult.
2.INSPIRATORY RESERVE VOLUME(IRV)
It is the air inspired with a maximal
inspiratory effort in excess of the tidal
volume.
It is about 3.3l in male and 1.9l in females
 DURING INSPIRATION:
 DURING EXPIRATION:
 PULMONARY VENTILATION:-
Is the amount of air inspired per minute.
i.e. 500ml/breath/minute which is about
6lit/minute.
 ALVEOLAR VENTILATION:-
It is the volume of gas that participates in
the exchange of O2 and Co2 out of the
500ml of air entering the lungs, during
inspiration only 350 reaches the alveoli.
 Therefore alveolar ventilation
=350mlx12 breaths/minute
=4.5lit/minute.
3.EXPIRATORY RESERVE
VOLUME(ERV):
It is the volume of expelled by a
forceful expiration and after the end
of the normal tidal expiration.
1lit in males,0.7 in females.
4.RESIDUAL VOLUME(RV)
Air left in the lungs after the
maximal expiratory efforts.
1.2lit in males,1.1lit in females.
 VITAL CAPACITY(VT):
4.8lit in male,3.1lit in female
 TOTAL LUNG CAPACITY(TLC):
 INSPIRATORY CAPACITY(IC):
IC=IVR+TV
 FUNCTIONAL RESIDUAL CAPACITY:
FRC=RV+ERV
 TRANSPORT OF OXYGEN (O2):
It depends primarily on the amount of
Hb in the RBCs.
METHODS BY WHICH O2 IS CARRIED BY
THE BLOOD
About 97% of the O2 transported from
the lungs to the tissues is by chemical
combination with Hb of RBCs.
 The rest 3% is carried in the
dissolved state in plasma.
 DEVELOPMENTAL FACTORS:
At birth, fluid filled lungs drains first
and PCO2 rises.
This causes the neonate to take first
breath.
Lungs are gradually expanded till 2weeks
of age.
Change in aging that effect respiratory
system of elders, due to infection physical
or emotional stress.
 PHYSIOLOGICAL FACTORES:
 BEHAVIROL FACTORE:
 LIFE STYLE FACTORE:
 ENVIORNMENTAL FACTORE:
 MEDICATION:
3. Inability of the tissue to
extract oxygen from the
blood
4. Decreased diffusion
of oxygen from the
alveoli to the blood
5. Poor tissue perfusion with
oxygenated blood
6. Impaired ventilation
TYPES
OF
HYPOXIA
Due to reduced oxygen tension in
arterial blood (supply problem)
Causes:-
Low o2 tension in the inhaled air.
Leaking mask, inadequate o2 regulator function
Impaired gas exchange in the lungs e.g. CHRONIC
BRONCHITIS & EMPHYSEMA
Gross ventilation/perfusion mismatch, as occur in
high G forces
 DUE TO DECREASED OXYGEN O2 CARRYING CAPACITY OF THE
BLOOD (Transport Problem)
CO poisoning chemicals/ drugs
hemorrhage/ hemolysis Anemia
OCCURS WHEN BLOOD CIRCULATION THROUGH
TISSUE IS REDUCED (Distribution Problem)
Causes:-
 High G forces
 Syncope (fainting)
 Heart failure
 Shock
DUE TO INABILITY OF THE TISSUES TO MAKE
USE OF THE OXYGEN SUPPLIED TO THEM
(Utilization Problem)
EXAMPLE:
 CYANIDE POISONING
 ALCOHOL & BARBITURATE
 OXYGEN TOXICITY
PATHOPHYSIOLOGY
ANXIETYAND
TIRED
HEADACHE,
DIZZINESS,
IRRITABILITY AND
MEMORY LOSS
NAUSEA
VOMITTING
OLIGURIA /
ANURIA
VISUAL
IMPAIREMENT
 Clubbing of finger
 Impairment in
judgement
 Shortness of breath
1. History Collection 2. Physical
Examination
3. Pulmonary
function test
4. Arterial blood
gas analysis
5. SPUTUM STUDIES 6. CHEST X- RAY
AND CT- SCAN
7. BRONCHOSCOPY
8. Thoracentesis 9. Spirometry
10. Pulse Oxymetry 11. Pulmonary
Angiography
1. POSITIONING
2. BREATHING EXERCISES
3. NEBULIZATION
4. CHEST PHYSIOTHERAPY
5. SUCTIONING
6. OXYGEN THERAPY
 Nasal canula
 Face mask
 Non breather mask
 Venture mask
 Face tent
 Transtracheal oxygenation
 Check the identification data of
the patient.
 Confirm diagnosis and the need
of oxygenation.
 Assess the patient for any sign
of clinical anoxia.
 Monitor for result of ABG.
 Oxygen should be monitored
for toxicity.
 Check that oxygen is properly
humidified.
 Precaution to be taken to
prevent infection.
 Discontinue oxygen therapy
gradually.
 Place a calling bell near patient
for emergency.
 Since oxygen supports
combustion, fire precautions to
be taken during oxygen therapy.
 Do proper documentation
including rate of flow of oxygen.
 Infection
 Combustion
 Drying of mucous membrane
of respiratory tract.
 Oxygen toxicity
 Atelectasis
 Oxygen induced apnea
 Ulceration, edema and
visual impairment
 Asphyxia
 It is a positive pressure or
negative pressure breathing
device that can maintain
ventilation and oxygen
delivery for prolonged period.
 Positive pressure ventilation
 Negative pressure
ventilation
MODES OF
VENTILATOR
 Continuous mandatory volume (CMV)
 Assist control ventilation (ACV)
 Pressure support ventilation (PSV)
 Intermittent mandatory ventilation (IMV)
 Continuous positive airway pressure
(CPAP)
 Synchronized intermittent mandatory
ventilation (SIMV)
 Positive end expiratory pressure (PEEP)
MODES OF VENTILATOR
Continuous mandatory volume is a mode
of mechanical ventilation where breaths
are delivered based on set variables and
makes no effort to sense patient effort
When the patient triggers the ventilator,
he/she receives a breaths of identical
duration and magnitude as the
mandatory breath.
The ventilator only provides support of
each breath to a preset amount of pressure,
thus the volume breathed can differ from
breath to breath taken.
It refers to any mode where a regular series of
breaths are scheduled but the ventilator
senses patient effort and reschedules
mandatory breaths based on the calculated
need of the patient.
 The ventilator adjunct is used with only
spontaneous ventilation; the patient breaths
spontaneously through the ventilator at an
elevated baseline pressure through the
breathing cycle.
 It facilitate the liberation from mechanical
ventilation.
 A demand valve is placed in it, so that
patient could take spontaneous breaths
without taking breath through apparatus of
the ventilator.
 PEEP is the alveolar pressure above
atmospheric pressure that exist at the end of
expiration
These are of two types
 Extrinsic PEEP
 Intrinsic PEEP
1) Impaired gas exchange related
to broncho- construction and
inflammation of airways.
2) Ineffective airway
clearance related to
increased mucous production
due to upper respiratory
infection and asthma.
3) Activity intolerance
related to dyspnoea and
hypoxia manifested by
fatigue.
4) Anxiety related to
difficulty in breathing as
manifested by asking more
doubts.
Abstract
It was recently established that supplemental oxygen
administration significantly enhances memory formation in healthy
young adults. In the present study, a double-blind, placebo-
controlled design was employed to assess the cognitive and
physiological effects of subjects' inspiration of oxygen or air
(control) prior to undergoing simple memory and reaction-time
tasks. Arterial blood oxygen saturation and heart rate were
monitored during each of six phases of the experiment,
corresponding to baseline, gas inhalation, word presentation,
reaction time, distractor and word recall, respectively.
The results confirm that oxygen administration significantly
enhances cognitive performance above that seen in the air
inhalation condition. Subjects who received oxygen recalled more
words and had faster reaction times.
 Text book of anatomy and physiology
Author-Ashalata
2nd
edition
page no 368-770
 Text book of advance nursing
practice
Author-Shabeer Basheer
1ST
edition
page no 198-208
 Text book of medical surgical nursing
Author-Suzanne c and Brenda Bare
11th
edition
page no 723-745
 Book of medical surgical nursing
Author-Usha Nair
page no 965-966
 Medical surgical nursing
Shafer’s
7th
edition
page no 358-359
Oxygen insufficency

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Oxygen insufficency

  • 2.  At the end of the seminar the group will be able to get the knowledge about the oxygen insufficiency, develop a positive attitude towards it and practice this knowledge in teaching and clinical areas.
  • 3. At the end of the class the student will be able to :  Define oxygen insufficiency.  Enlist the etiology of oxygen insufficiency.  Enumerate factors affecting oxygenation  Know the types of oxygen insufficiency.  Discuss the pathophysiology of oxygen insufficiency.  Enlist sign and symptoms of oxygen insufficiency.
  • 4.  Explain different diagnostic evaluation of oxygen insufficiency.  Describe the management of oxygen insufficiency.  Identify the nurses role in the management of oxygen insufficiency.  Explain about oxygen therapy.  Discuss about nurses responsibility while administering oxygen.
  • 5.
  • 6.  All living things use oxygen or depend on organisms that use oxygen in some way.
  • 7.  We use oxygen to break down simple sugar and release energy.  This can be done through respiration or fermentation.  Animals mainly use respiration.
  • 8.  The process that breaks apart simple food molecules to release energy.  It occurs inside cells.  What YOU do with the oxygen you take in.
  • 9. The molecule most living things use for energy — including us!  We break down food into smaller molecules during digestion. One of the small molecules is glucose.  Glucose leaves your intestines, goes into your blood and is taken to every cell in your body.
  • 10.  In your cells, oxygen is used to split glucose apart — releasing energy, water and carbon dioxide.
  • 11.  Plants take in carbon dioxide and water and use them to make food. Their food is simple sugar — glucose.
  • 12.  Plants pull the carbon off Co2 and use the carbon in glucose. (They do not need the oxygen for this. They get that from water, H2O.)  Plants release the oxygen (O2) back into the atmosphere.  Other organisms use the free oxygen for respiration.
  • 13.  We keep destroying natural areas, especially forested areas with many plants and replacing them with buildings, parking lots, lawns, etc.  Fewer plants mean less oxygen and more carbon dioxide.  This disturbs the balance of the natural cycle.
  • 14.  Every time something burns (combustion), more carbon dioxide is released into the atmosphere.  We add more and more Co2 and destroy more and more of the plants that clean the air for us.
  • 15.  Stop destroying and promote regrowth of natural areas — especially forests.  Burn less (fossil fuels, forest fires, etc.)
  • 16.  Oxygen insufficiency means “ deficient in oxygen”.  The normal range of oxygen in the external blood should be 80-100 mm of Hg.  For treating Oxygen insufficiency effectively, early diagnosis and correct cause should be ruled out.  The only management for Oxygen insufficiency is Oxygen administration.
  • 17.  Oxygenation means the delivery of oxygen to the body’s tissues and cell it is necessary to maintain life and health
  • 18.  Oxygenation results from the co- operative function of 3 major system 1.Pulmonary 2.Hematological 3.Cardiovascular system
  • 19.  Pulmonary ventilation:- A movement of air into and out of lungs. It’s main purpose is to supply fresh air. composed of….. 1.Inspiration: air flows into the lungs. 2.Expiration: air moves out of lungs.
  • 20.  Oxygen insufficiency is a condition in which the body as a whole or a region is deprived of adequate oxygen supply.  Oxygen insufficiency is a failure to provide adequate oxygen to cells of the body and to remove excess carbon dioxide from them.
  • 21.  Transference of 1.Oxygen from the atmosphere to the tissues. 2.And carbon dioxide from the tissues to the atmosphere.
  • 22.  External respiration:- It takes place in the lungs. The O2 is absorbed from air and into the blood and Co2 is excreted from the blood into the air.  Internal respiration: or tissue respiration O2 is transferred from blood to the tissue which gives up Co2
  • 23.  Ventilation  Pulmonary diffusion or gaseous exchange at the alveolar level  Transport of gases between lungs and tissues through blood  Gases exchange at tissue level  Utilization of oxygen and production of Co2 by the tissues(cellular respiration)
  • 24.  It is the process of movement of air from the atmosphere to the lungs and vice versa.  Inspiration is an active process brought about by the contraction of inspiratory muscles, where as Expiration is a passive process due to elastic recoil of lungs.
  • 25.  The space between parietal pleura and visceral pleura is filled with the pleural fluid, which lubricates the lungs movements which is called as intrapleural pressure.  IPP is the pressure in the lungs.
  • 26.  Easily determined by a water filled spirometer attached to a kymograph.
  • 27. 1.TIDAL VOLUME(TV): Is the volume of air that moves into the lunges with each inspiration or that amount that moves out with each expiration. It is about 0.5 liters In an adult. 2.INSPIRATORY RESERVE VOLUME(IRV) It is the air inspired with a maximal inspiratory effort in excess of the tidal volume. It is about 3.3l in male and 1.9l in females
  • 28.  DURING INSPIRATION:  DURING EXPIRATION:
  • 29.  PULMONARY VENTILATION:- Is the amount of air inspired per minute. i.e. 500ml/breath/minute which is about 6lit/minute.  ALVEOLAR VENTILATION:- It is the volume of gas that participates in the exchange of O2 and Co2 out of the 500ml of air entering the lungs, during inspiration only 350 reaches the alveoli.
  • 30.  Therefore alveolar ventilation =350mlx12 breaths/minute =4.5lit/minute.
  • 31. 3.EXPIRATORY RESERVE VOLUME(ERV): It is the volume of expelled by a forceful expiration and after the end of the normal tidal expiration. 1lit in males,0.7 in females.
  • 32. 4.RESIDUAL VOLUME(RV) Air left in the lungs after the maximal expiratory efforts. 1.2lit in males,1.1lit in females.
  • 33.  VITAL CAPACITY(VT): 4.8lit in male,3.1lit in female  TOTAL LUNG CAPACITY(TLC):  INSPIRATORY CAPACITY(IC): IC=IVR+TV  FUNCTIONAL RESIDUAL CAPACITY: FRC=RV+ERV
  • 34.  TRANSPORT OF OXYGEN (O2): It depends primarily on the amount of Hb in the RBCs. METHODS BY WHICH O2 IS CARRIED BY THE BLOOD About 97% of the O2 transported from the lungs to the tissues is by chemical combination with Hb of RBCs.
  • 35.  The rest 3% is carried in the dissolved state in plasma.
  • 36.  DEVELOPMENTAL FACTORS: At birth, fluid filled lungs drains first and PCO2 rises. This causes the neonate to take first breath. Lungs are gradually expanded till 2weeks of age. Change in aging that effect respiratory system of elders, due to infection physical or emotional stress.
  • 37.  PHYSIOLOGICAL FACTORES:  BEHAVIROL FACTORE:  LIFE STYLE FACTORE:  ENVIORNMENTAL FACTORE:  MEDICATION:
  • 38. 3. Inability of the tissue to extract oxygen from the blood 4. Decreased diffusion of oxygen from the alveoli to the blood
  • 39. 5. Poor tissue perfusion with oxygenated blood 6. Impaired ventilation
  • 41. Due to reduced oxygen tension in arterial blood (supply problem) Causes:- Low o2 tension in the inhaled air. Leaking mask, inadequate o2 regulator function Impaired gas exchange in the lungs e.g. CHRONIC BRONCHITIS & EMPHYSEMA Gross ventilation/perfusion mismatch, as occur in high G forces
  • 42.  DUE TO DECREASED OXYGEN O2 CARRYING CAPACITY OF THE BLOOD (Transport Problem) CO poisoning chemicals/ drugs hemorrhage/ hemolysis Anemia
  • 43. OCCURS WHEN BLOOD CIRCULATION THROUGH TISSUE IS REDUCED (Distribution Problem) Causes:-  High G forces  Syncope (fainting)  Heart failure  Shock
  • 44. DUE TO INABILITY OF THE TISSUES TO MAKE USE OF THE OXYGEN SUPPLIED TO THEM (Utilization Problem) EXAMPLE:  CYANIDE POISONING  ALCOHOL & BARBITURATE  OXYGEN TOXICITY
  • 48.  Clubbing of finger  Impairment in judgement  Shortness of breath
  • 49. 1. History Collection 2. Physical Examination
  • 50. 3. Pulmonary function test 4. Arterial blood gas analysis
  • 51. 5. SPUTUM STUDIES 6. CHEST X- RAY AND CT- SCAN 7. BRONCHOSCOPY
  • 52. 8. Thoracentesis 9. Spirometry 10. Pulse Oxymetry 11. Pulmonary Angiography
  • 54. 3. NEBULIZATION 4. CHEST PHYSIOTHERAPY
  • 56.  Nasal canula  Face mask  Non breather mask  Venture mask  Face tent  Transtracheal oxygenation
  • 57.  Check the identification data of the patient.  Confirm diagnosis and the need of oxygenation.  Assess the patient for any sign of clinical anoxia.
  • 58.  Monitor for result of ABG.  Oxygen should be monitored for toxicity.  Check that oxygen is properly humidified.  Precaution to be taken to prevent infection.
  • 59.  Discontinue oxygen therapy gradually.  Place a calling bell near patient for emergency.  Since oxygen supports combustion, fire precautions to be taken during oxygen therapy.  Do proper documentation including rate of flow of oxygen.
  • 60.  Infection  Combustion  Drying of mucous membrane of respiratory tract.  Oxygen toxicity
  • 61.  Atelectasis  Oxygen induced apnea  Ulceration, edema and visual impairment  Asphyxia
  • 62.  It is a positive pressure or negative pressure breathing device that can maintain ventilation and oxygen delivery for prolonged period.
  • 63.  Positive pressure ventilation  Negative pressure ventilation
  • 65.  Continuous mandatory volume (CMV)  Assist control ventilation (ACV)  Pressure support ventilation (PSV)  Intermittent mandatory ventilation (IMV)  Continuous positive airway pressure (CPAP)  Synchronized intermittent mandatory ventilation (SIMV)  Positive end expiratory pressure (PEEP) MODES OF VENTILATOR
  • 66. Continuous mandatory volume is a mode of mechanical ventilation where breaths are delivered based on set variables and makes no effort to sense patient effort
  • 67. When the patient triggers the ventilator, he/she receives a breaths of identical duration and magnitude as the mandatory breath.
  • 68. The ventilator only provides support of each breath to a preset amount of pressure, thus the volume breathed can differ from breath to breath taken.
  • 69. It refers to any mode where a regular series of breaths are scheduled but the ventilator senses patient effort and reschedules mandatory breaths based on the calculated need of the patient.
  • 70.  The ventilator adjunct is used with only spontaneous ventilation; the patient breaths spontaneously through the ventilator at an elevated baseline pressure through the breathing cycle.
  • 71.  It facilitate the liberation from mechanical ventilation.  A demand valve is placed in it, so that patient could take spontaneous breaths without taking breath through apparatus of the ventilator.
  • 72.  PEEP is the alveolar pressure above atmospheric pressure that exist at the end of expiration These are of two types  Extrinsic PEEP  Intrinsic PEEP
  • 73.
  • 74. 1) Impaired gas exchange related to broncho- construction and inflammation of airways.
  • 75. 2) Ineffective airway clearance related to increased mucous production due to upper respiratory infection and asthma.
  • 76. 3) Activity intolerance related to dyspnoea and hypoxia manifested by fatigue.
  • 77. 4) Anxiety related to difficulty in breathing as manifested by asking more doubts.
  • 78. Abstract It was recently established that supplemental oxygen administration significantly enhances memory formation in healthy young adults. In the present study, a double-blind, placebo- controlled design was employed to assess the cognitive and physiological effects of subjects' inspiration of oxygen or air (control) prior to undergoing simple memory and reaction-time tasks. Arterial blood oxygen saturation and heart rate were monitored during each of six phases of the experiment, corresponding to baseline, gas inhalation, word presentation, reaction time, distractor and word recall, respectively. The results confirm that oxygen administration significantly enhances cognitive performance above that seen in the air inhalation condition. Subjects who received oxygen recalled more words and had faster reaction times.
  • 79.  Text book of anatomy and physiology Author-Ashalata 2nd edition page no 368-770  Text book of advance nursing practice Author-Shabeer Basheer 1ST edition page no 198-208
  • 80.  Text book of medical surgical nursing Author-Suzanne c and Brenda Bare 11th edition page no 723-745  Book of medical surgical nursing Author-Usha Nair page no 965-966  Medical surgical nursing Shafer’s 7th edition page no 358-359