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4. DEFINITION
•An isolated, usually narrow, deep probing depth of
pulpal or periodontal origin.
•Lesion with sub marginal or intrabony periradicular
bone loss of pulpal and/or periodontal origin that
communicates with the oral cavity via probing defect.
•A localized periodontal probing depth of pulpal or
periodontal origin.
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STOCK
6. WEINE
Tooth in which symptoms clinically and radiographically
simulate periodontal disease but are due to pulpal inflammation
Tooth that has both pulpal and periodontal disease
concomitantly
Tooth has no pulpal problem but require endodontic therapy
plus root amputation to gain periodontal healing
Tooth that clinically and radiographically simulated pulpal or
periapical disease but infact has periodontal disease
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7. LESIONS REQUIRING ENDODONTIC TREATMENT ONLY
necrotic pulp and apical granulomatous tissue replacing
periodontium with or without sinous tract
Chronic periapical abscess with sinus tract
Longitudinal and horizontal root fractures
Pathologic and iatrogenic root perforations
Teeth with incomplete apical root development
Endodontic implants
Teeth that require hemisection
Root submergence
GROSSMAN
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8. LESIONS REQUIRING PERIODONTAL TREATMENT ONLY
Occlusal trauma causing reversible pulpitis
Occlusal trauma plus gingival inflammation resulting in pocket
formation and reversible pulpitis
Suprabony or infrabony pocket formation treated with overzealous
root planning and curettage leading to pulpal sensitivity
Extensive infrabony pocket formation extending beyond the root
apex and sometimes coupled with lateral or apical resorption yet
with pulp that responds with in normal limits to clinical testing
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9. LESIONS REQUIRING COMBINED ENDO – PERIO TREATMENT
Any lesion in Group I That results in irreversible reactions in the
attachment apparatus and requires periodontal treatment
Any lesion in Group II that results in irreversible reactions to the
pulp tissue and also requires endodontic treatment
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15. Attachment loss asso. with
Anatomic defect on root
Nature of pathogenic flora
Necrotic & infected pulp
Host defense mechanism defect.
Aggresiveness asso with
Lateral & apical foramen
Nature of flora
Apical host defense
Periodontal probing &
radiographic examination
Radiographic examination
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16. DIFFERENTIAL DIAGNOSIS
PULPAL
PERIODONTAL
Cause
pulp infection
periodontal
Vitality
non vital
vital
Restorative
deep or extensive
not related
Plaque /calculus
not related
primary cause
Inflammation
acute
chronic
Pockets
single and narrow
multiple and wide
pH value
acidic
alkaline
Trauma
primary or secondary
contributing factor
Microbial
few
CLINICAL
coronally
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complex
17. RADIOGRAPHIC
Pattern
Bone loss
Periapical
Vertical bone loss:
localized
wider apically
radiolucent
no
generalized
wider coronally
not related
yes
HISTOPATHOLOGY
Junctional epithelium
Granulation tissues
Gingival
no apical migration
apical (minimal)
normal
present
coronal (larger)
recession
TREATMENT
Therapy
RCT
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Periodontal therapy
18. Problems in
diagnosis :
Vertical root fracture:
varied radiographic picture
Different angulations
Surgical exposure
lateral condensation excessive
Post placement
Cause
Extensive restorations
Older patients
Gingival sulcus & pocket area
Single rooted teeth
multirooted teeth
Developmental grooves
In doubt ? – Biopsy / Histological analysis
Systemic diseases mimic lesion on radiograph :
Scleroderma
Metastatic carcinoma
Osteosarcoma
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19. EFFECT OF PULP AND ITS TREATMENT ON PERIODONTIUM
Periodontal inflammation & bone loss
Sub marginal bone loss
Horizontal bone loss
Vertical intrabony pockets
Furcation involvement
Periodontal wound healing
Traumatized necrotic pulp
RC infection – compromised healing
Gingival tissue thickness
Alveolar bone level
Surgical trauma to flap
Effective flap repositioning
Root canal treatment
Doubtful pulpal status
Iatrogenic problems
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