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Prof Dr Hussein Abdeldayem
Prof and Chief of Ped Neurology Unit,
alex university
NEURO-MOTOR SYSTEM
CP
AFP= GBS
FLOPPY BABY: SMA
Neuromotor disorder general view: make it easy
ACUTE FLACCID PARALYSIS
AFP
Dr Hussein Abdeldayem, MD
Professor of Pediatric Neurology
Brain (
pyramidal
cells and
tracts)
AHC PNs
NM
junction
Muscles
UMNL L M N L
Anatomy
1- UML
 Control Circuits
 1-cerebellum : motor and premotor areas
 2-basal ganglia
control and modify motor activity- prevent invol movements
 3- cerebellum
LMNL
 Motor unit – a lower motor neuron (ventral horn cell/CN
nucleus) + muscle fibers under its control
 alpha motor neuron AHC
 Root (anterior)
 axon (nerve)
 MN synapse
 muscle fibers
AHC
CEREBRAL MOTOR DISORDER
• CEREBRAL PALSY
C/P SUGGESTIONS
• Cognitive dysfunctions as GDD/MR
• Language delay
• Seizures
• TONE:
PYRAMIDAL:SPASTIC
EXTRPYRAMIDAL: LEADPIPE/cogwheel
CEREBELLUM: HYPOTONIA
INVESTIGATION
BRAIN MRI
LMNL CAUSES
• AHC:
1- GENETIC: SMA
2- ACQUIRED : polio
• Roots and Peripheral Nerves:
genetic: myelocele, HSMN
acquired: GBS (post-infectious), PNitis
• N-M junction:
genetic: Myasthenia Gavis
Acquired: botulinum toxin
• Muscular:
congenital: Myopathy, DMD
acquired: myositis
C/P SUGGESTIONS
• Normal Brain Function with only Motor
Dysfunction ( Motor Delay)
• Atonia: Floppy/ hypotonia
• Areflexia
Neuromotor disorder general view: make it easy

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Neuromotor disorder general view: make it easy

Hinweis der Redaktion

  1. Major neurotransmitters: glutamate (excitatory) and GABA (inhibitory) Damage results in weakness, with loss of voluntary movements especially fine, skilled movements, but preservation of other forms of movements including segments reflexes They both (1-2) receive input from several motor and sensory cortical areas and project back to the cerebral cortex via the thalamus. They integrate and modulate motor activity primarily through the cerebral cortex and direct activation pathways.
  2. Final common pathway – many motor units through which all activities in the motor system must act
  3. central causes : Cerebral palsy as cerebellar or atonic diplegiaCP, , syndromes as Down syndrome or Prader Willi syndrome, neurodegenerative or neurometabolic disorders