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PEDIATRIC ACCIDENTS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
OVERVIEW OF COMMON ACCIDENTAL INGESTION -Counseling if suicide attempt - Liver assessment ,[object Object],[object Object],Liver necrosis in 2-5 days; nausea; vomiting; pain in R upper quadrant; jaundice; coagulation; abnormalities, hepatoxic Acetaminophen (Tylenol) Anticipatory guidance Bleeding precautions Counseling if suicide attempt   Emesis Hydration Vitamin K Activated charcoal   Tinnitus Hyperpyrexia Seizures Bleeding Hyperventilation   Salicylate (Aspirin)   INTERVENTIONS  NURSING TREATMENT  CLINICAL MANIFESTATION  INGESTION
Keep warm and inactive ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],Corrosives (drain or oven cleanser, chlorine bleach, battery acid) If vomiting, reduce aspiration ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],Hydrocarbons (kerosene, turpentine, gasoline) ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],-Developmental regression -Impaired growth (encelophalopathy) -Irritability -Increased clumsiness Lead (paint, also in soil near heavily trafficked roadways, household dust)
BURNS ,[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
FRACTURES ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
MUSCULOSKELETAL DISORDERS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
ONCOLOGY DISORDER ,[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
NEPHROBLASTOMA (Wilms Tumor) ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
NEUROBLASTOMA ,[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
HODKIN’S LYMPHOMA ,[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
RENAL DISORDERS 2/3 of cases in children under 4-7 years; more common in boys Average age of onset about 2-1/2 years most common in boys Incidence Antigen- antibody reaction secondary to infection elsewhere in the body, usually a Group A beta hemolytic streptococcal infection of the upper respiratory tract Cause unknown; may follow the toxic effects of mercury or Tridione exposure or bee sting Etiology Post-streptococcal glomerulonephritis Childhood nephrosis Other names ACUTE GLOMERULONEPHRITIS NEPHROTIC SYNDROME
Urine – contains RBC and increase specific gravity Urine – shows heavy albuminuria Laboratory findings Varying degrees of HPN maybe present, when BP is increased, cerebral manifestations may occur such as, headache, drowsiness, diplopia, vomiting and convulsions Usually normal, transient elevation may occur early Blood pressure Periorbital edema appears insidiously; tea-colored urine from hematuria, hypertension oliguria Edema appears insidiously; usually first noticed about the eyes and can advance to the legs, arms, back, pentoneal cavity and scrotum; massive protenuria, anorexia; pallor Clinical manifestations Inflammation of the kidneys; damage to the glomeruli allows excretion of red blood cells Increased permeability of the glomerular membrane to protein Pathology
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],Nursing interventions ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],Treatments

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Pediatric Accidents

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  • 22. RENAL DISORDERS 2/3 of cases in children under 4-7 years; more common in boys Average age of onset about 2-1/2 years most common in boys Incidence Antigen- antibody reaction secondary to infection elsewhere in the body, usually a Group A beta hemolytic streptococcal infection of the upper respiratory tract Cause unknown; may follow the toxic effects of mercury or Tridione exposure or bee sting Etiology Post-streptococcal glomerulonephritis Childhood nephrosis Other names ACUTE GLOMERULONEPHRITIS NEPHROTIC SYNDROME
  • 23. Urine – contains RBC and increase specific gravity Urine – shows heavy albuminuria Laboratory findings Varying degrees of HPN maybe present, when BP is increased, cerebral manifestations may occur such as, headache, drowsiness, diplopia, vomiting and convulsions Usually normal, transient elevation may occur early Blood pressure Periorbital edema appears insidiously; tea-colored urine from hematuria, hypertension oliguria Edema appears insidiously; usually first noticed about the eyes and can advance to the legs, arms, back, pentoneal cavity and scrotum; massive protenuria, anorexia; pallor Clinical manifestations Inflammation of the kidneys; damage to the glomeruli allows excretion of red blood cells Increased permeability of the glomerular membrane to protein Pathology
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