SlideShare ist ein Scribd-Unternehmen logo
1 von 12
Downloaden Sie, um offline zu lesen
Emergency Management of Pediatric Burns
Jennifer L. Reed, MD and Wendy J. Pomerantz, MD, MS, FAAP
Key Words: burns, emergency care, management
Burn-related injuries are a leading cause of morbidity and
mortality in children. Burn injuries rank third among
injury-related deaths in children aged 1 to 9 years.1
In 2001,
there were more than 181,000 fire- and burn-related injuries,
more than 4200 hospitalizations, and 672 deaths in children
aged 0 to 19 years in the United States.2
Pediatric patients
and the elderly have the highest morbidity and mortality
associated with burn injuries.3
A majority of these children
are initially seen in emergency departments (EDs) around
the country; therefore, it is necessary that all emergency
professionals are proficient in burn management in the
pediatric population. Many burns are the result of uninten-
tional events, and others are the result of nonaccidental
trauma. However, most burns are preventable. As a primary
care provider in the ED, it is necessary to have the knowl-
edge and skills not only to treat burns, but to also counsel
families regarding burn prevention and to identify burns
resulting from child abuse.
THERMAL BURNS
Thermal burns are the most common type of burn in
childhood. They can be a result of flames, scalds, contact,
cold, or radiation. In the toddler age group, scald burns from
hot liquid or hot grease are seen commonly and account
for 80% of all thermal injuries.4
This type of burn accounts
for a majority of childhood burn hospitalizations in this
age group. Toddlers also have the highest rate of contact
burns such as those that occur when touching hot metal
from a stove, grill, or home space heater. Young school-age
children have an innate curiosity and tend to play with
dangerous equipment such as matches and cigarette lighters
resulting in thermal burns. Older school-age children and
teenage populations are more commonly burned from risk-
taking activities, fireworks, and careless use of flammable
substances such as gasoline, lighter fluid, or hairspray. In
addition, household fires commonly caused by unattended
cigarettes or candles are a major contributor to pediatric
burn injuries and death in all age groups. Cigarettes are
responsible for 35% of fatal house fires in the United States,
many of which kill children.3
More than half of all fire-
related deaths occur in homes that do not have smoke
alarms, and fire-related injuries are the most common cause
of fire- and burn-related deaths in children.5
Native Amer-
ican children, African-American children, and children of
low-income families are at increased risk for death and
injury related to fire.5,6
Burn injuries are more common in
boys than in girls, and an increase in fire-related deaths is
seen during the winter months.2,5
Pathophysiology
The skin is an important organ system that functions
to protect the body from infectious agents, to regulate the
body temperature by preventing heat loss, and to serve as
a barrier to prevent body fluid loss. When the skin is dam-
aged by a burn, devastating sequela may follow. The skin
consists of 2 layers, the epidermis and the dermis. The epi-
dermis includes 4 layers: stratum corneum, stratum lucidum,
stratum granulosum, and stratum germinativum. The stratum
corneum is the most important layer that protects the
body from water loss and infection. Beneath the epidermis,
the dermis consists of hair follicles, sweat glands, nerve
fibers, and connective tissue. This layer is vital in regulating
heat loss.
Classification
Burns are classified as first, second, or third degree.
More commonly, they are referred to as superficial, partial
thickness, or full thickness, respectively. Oftentimes it is
difficult to correctly identify the depth of a burn, and it is
common to have several depths exhibited in one injury, with
the center usually demonstrating a higher degree of burn
than the periphery.7
The thickness of the burn is directly
related to the source of the burn and the time the skin is
in contact with the source. Areas of thin skin such as the
118 Pediatric Emergency Care  Volume 21, Number 2, February 2005
Review Article
Division of Emergency Medicine, Cincinnati Children’s Hospital Medical
Center, Cincinnati, OH.
Drs Reed and Pomerantz have no relationship to or financial interest in any
of the products referred to in this manuscript.
Address correspondence and reprint requests to Jennifer L. Reed, MD,
Cincinnati Children’s Hospital Medical Center, Division of Emergency
Medicine, 3333 Burnet Avenue ML2008, Cincinnati, OH 45229. E-mail:
Jennifer.Reed@cchmc.org.
Copyright n 2005 by Lippincott Williams  Wilkins
ISSN: 0749-5161/05/2102-0118
Copyright © Lippincott Williams  Wilkins. Unauthorized reproduction of this article is prohibited.
ears, volar surface of the arm, dorsum of the hand, and peri-
neum may often incur deeper burns.8
Superficial or first-degree burns are classically ery-
thematous and painful. The most common example is a
sunburn from ultraviolet exposure. These burns involve the
intact epidermis without blistering (Fig. 1). Because these
burns only involve the outer layer of the epidermis, fluid loss
is not a problem. Oftentimes, several days after the initial
burn, peeling of the skin will begin because of superficial
cell death. They heal without scarring in 4 to 5 days.7
Partial-thickness or second-degree burns can be classi-
fied as superficial partial-thickness or deep partial-thickness
burns. Superficial partial-thickness burns involve partial
destruction of the dermis and appear red and painful with
blister formation (Figs. 2 and 3). They have a weeping or
moist appearance, and healing usually occurs with mini-
mal scarring in 7 to 10 days.7
Dark-skinned individuals
may lose melanin and develop hypopigmentation upon
healing. Deep partial-thickness burns involve greater than
50% of the dermis, destroying the nerve fibers and,
consequently, are usually less painful. They have a white
pale appearance and usually take 2 to 3 weeks or more to
heal. These burns can be difficult to distinguish from third-
degree burns. Often, severe scarring will develop, and pa-
tients are at risk for contractures. Patients with significant
partial-thickness burns are at risk for fluid loss. Surgical
consultation is necessary for deep partial-thickness burns
because skin grafting is usually necessary for long-term
treatment. A good rule of thumb is if a burn does not
completely heal in 7 to 10 days, referral to a surgeon for
further evaluation is necessary.7
Full-thickness or third-degree burns are the most
severe burns (Fig. 4). They appear white, waxy, or leathery
and do not bleed or demonstrate any capillary refill (Fig. 5).
FIGURE 1. A superficial (first degree) burn.
n 2005 Lippincott Williams  Wilkins 119
Pediatric Emergency Care  Volume 21, Number 2, February 2005 Emergency Management of Pediatric Burns
Copyright © Lippincott Williams  Wilkins. Unauthorized reproduction of this article is prohibited.
Typically, these burns are painless because they have
completely destroyed the nerve fibers in the dermis. Patients
with these burns are at high risk for infection and severe
fluid loss. These burns usually take several weeks to heal,
and patients should be referred to a surgeon immediately
because of risk of significant scarring upon healing. These
burns often require skin grafting.
Although the terminology is not commonly used,
fourth-degree burns may also be seen. These involve destruc-
tion of the underlying structures such as tendons, nerves,
muscles, bone, and deep fascia. Most commonly, these are
seen with severe electrical injuries, and they require imme-
diate surgical consultation and transfer to a burn center.
Besides classifying burns as superficial, partial thick-
ness, or full thickness, it is also necessary to determine
the extent of the burn. This is calculated based on total
body surface area (TBSA). The ‘‘rule of 9s’’ is a method to
determine TBSA of adult burn victims. The head and each
arm from the shoulder to the fingertips are each estimated
to be 9% of the TBSA. The anterior trunk, posterior trunk,
and each leg from the groin to the toes are each estimated
to be 18% of TBSA. The neck and groin are each estimated
to be 1% of TBSA. This method is very accurate for adult
and teenage burn patients, but it is not accurate in the
assessment of pediatric burns because of the difference in
body proportions. Alternatively, the Lund and Browder
Chart9
may be used to correctly determine percent surface
area burns for children of various ages10
(Fig. 6). It has been
slightly modified to account for the varying body propor-
tions in different ages of children. Another simple method
is to measure the percent burn by using the child’s palm
which is equal to 1% TBSA burn.9,10
Although 1% TBSA
is the accepted measurement, a study demonstrated that the
surface of the palm in any age group more closely approx-
imates 0.5% TBSA, and a second study determined that
the entire hand more accurately represents 0.8% TBSA in
any age group.11,12
Clearly, controversy exists as to the
most accurate measurement when using a patient’s hand
to calculate percent TBSA. Superficial burns should not
be included in the surface area burn calculation because
they only involve the epidermis and do not put a patient
at risk for fluid loss. Whichever method is chosen, one
should quickly determine the best estimate of TBSA burn
involvement.
Initial Assessment
The initial approach to burn treatment is similar
regardless of the etiology of the burn. Initial assessment
should first include airway, breathing, and circulation
followed by determination of the depth of burn, TBSA of
the burn, and the involved body parts. Burns that are circum-
ferential should be noted and carefully observed because
they have the potential to require escharotomy. Circumferential
burns to the chest may interfere with the patient’s ability
to ventilate and need to be addressed if problems arise.
In addition, one should obtain historical information in-
cluding date, time, cause of burn, general medical history,
and immunization status. Significant burns associated with
trauma should be evaluated in the standard Advanced
Trauma Life Support protocol with a primary survey con-
sisting of airway, breathing, and circulation and a secondary
survey evaluating for other injuries.
One should immediately remove all clothing that is
hot, burned, or exposed to chemicals to prevent continued
damage to the skin. To decrease burn pain, cool saline-
soaked gauze should be applied to the burned skin, but
caution needs to be taken with large burns to prevent the
complication of hypothermia. Applying ice to the wound
or submersing the wound in ice water not only produces
hypothermia, but also worsens damage to the skin and
should be avoided.13
For large burns, a clean sheet may be
used to cover the burns during the initial assessment in
the ED. This will decrease pain and provide a superficial
covering until each burn can be assessed in detail. Tar prod-
uct burns can be challenging. It is necessary to immediately
cool the hot tar with cool water. To remove the tar, use a
petroleum-based product such as bacitracin which will dis-
solve the tar and allow for easier removal.14
In the ED, laboratory data should be obtained for
patients with severe burns. This includes a complete blood
count, electrolytes with blood urea nitrogen and creatinine,
and possibly liver function tests. These laboratory values
will serve as a baseline because the patient may experience
major fluid shifts and changes in metabolic status. Urinaly-
sis is also important to assess the presence of myoglobin
that can result from muscle breakdown. Significant myo-
globin can lead to renal function impairment. In addition
FIGURE 2. A partial-thickness (second degree) burn with
significant blister formation.
120 n 2005 Lippincott Williams  Wilkins
Reed and Pomerantz Pediatric Emergency Care  Volume 21, Number 2, February 2005
Copyright © Lippincott Williams  Wilkins. Unauthorized reproduction of this article is prohibited.
for fire-related burns, a baseline chest radiograph along
with carbon monoxide levels should be obtained.
House fires, indoor fires, chemical fires, and chemical
ingestions may involve respiratory tract burns resulting in
inflammation and edema. If a patient’s physical examina-
tion findings demonstrate stridor, hoarseness, carbonaceous
sputum, perioral or perinasal burns, or singed nose hair,
eyebrows, or eyelashes, one should anticipate airway com-
promise and promptly secure the patient’s airway with
intubation. It is important not to rely on chest radiograph
because it is often normal at initial presentation.15
Many
advocate the use of bronchoscopy to directly visualize the
airway. In the ED setting, it can be just as useful to directly
visualize the upper airway with a laryngoscope. If blisters
are seen in the oropharynx or oral mucosa, or the mucosa
appears dry and erythematous, one should anticipate airway
compromise.16
Edema of the airway may not be apparent
until 48 hours after a burn. If the physician waits until ob-
vious compromise, results may be catastrophic, and intu-
bation can be extremely difficult. During intubation, it is
important to have various-size endotracheal tubes available
and anticipate a narrowed airway. Supraglottic airway injury
is usually the result of direct thermal injury, whereas lower
airway edema and irritation are a result of chemicals or tox-
ins such as smoke inhalation ultimately leading to chemical
pneumonitis.17,18
All patients who are victims of house or
indoor fires should also be evaluated for carbon monoxide
poisoning, and 100% oxygen should be administered.
Intravenous access should be obtained immediately
in intact skin. If necessary though, catheters may be placed
through burned skin, and intraosseous access can be used
in young children. Any significant burn injury results in
major fluid loss for patients. A burn that is 15% to 20%
TBSA will produce hypovolemic shock unless appropri-
ately managed with crystalloid fluid replacement. Isotonic
saline, most commonly lactated Ringer solution, is recom-
mended for resuscitation in the first 24 hours after a sig-
nificant burn.14
Large volumes of fluid are needed for proper
resuscitation because only 20% to 30% of the isotonic fluid
remains in the intravascular space.19
Thus, it is necessary to
FIGURE 3. A partial-thickness (second degree) burn. One blister is intact, but most of the blister formation has been debrided.
n 2005 Lippincott Williams  Wilkins 121
Pediatric Emergency Care  Volume 21, Number 2, February 2005 Emergency Management of Pediatric Burns
Copyright © Lippincott Williams  Wilkins. Unauthorized reproduction of this article is prohibited.
appropriately calculate the extent of the burn and fluid-
resuscitate each patient individually.
The patient should receive fluid resuscitation in the
first 24 hours based on the modified Parkland formula:
4 mL Â TBSA Â weight in kilograms. Half of the total
fluid should be given in the first 8 hours and the second
half over the next 16 hours.4
In children younger than
5 years, calculated maintenance requirements should be
added to this total volume to more accurately predict fluid
needs.4
Children with burns to less than 15% TBSA usually
do not have significant capillary leak and can be managed
with isotonic fluid at 1.5 times the maintenance rate while
closely following the patient’s urine output and hydration
status.14
Children less than 20 kg may develop hypogly-
cemia and should have a 5% dextrose–containing solution
administered at a maintenance rate along with the calcu-
lated volume of fluid necessary for resuscitation.14
Be-
cause pediatric burn victims are also prone to hypothermia
caused by the loss of integument, initial fluid should be
warmed.
Treatment
Pain control is extremely important in burn manage-
ment. Most minor burns can be controlled with nonsteroidal
anti-inflammatory medications or acetaminophen. Partial-
thickness burns can be extremely painful because of the
damage of the epidermis and consequent exposure of the
underlying pain fibers. Simply covering the patient with
a sheet can dramatically reduce pain by decreasing the
environmental exposure. Narcotic pain control though is
often necessary in burns of partial thickness or deeper. In
children, morphine intravenously is most commonly used.
One should be very careful to monitor the patient’s hemo-
dynamic stability when administering any narcotic.
Each burn should be cleaned with mild soap and water
while avoiding extremely cold water or ice. Any skin that
is loose at the initial presentation needs to be debrided.
Controversy exists as to whether to unroof blisters or keep
them intact. One author suggests leaving small blisters
intact as long as they do not interfere with wound care or
movement over a joint. This author recommends all large
FIGURE 4. A full-thickness (third degree) burn in a Caucasian patient. Note the leathery appearance.
122 n 2005 Lippincott Williams  Wilkins
Reed and Pomerantz Pediatric Emergency Care  Volume 21, Number 2, February 2005
Copyright © Lippincott Williams  Wilkins. Unauthorized reproduction of this article is prohibited.
blisters be unroofed; when in doubt, unroof the blister.7
A second author believes that removing blisters makes care
of the burn easier and recommends that only blisters on
the soles of the feet or those less than 1 cm should be left
intact.20
There is scientific data that show burn fluid may
have a negative effect on the burn including suppressing
neutrophil and lymphocyte response as well as interfering
with fibrinolysis. Burn blister fluid also contains substances
that increase inflammatory response which may increase
the risk of infection.21
Needle aspiration of a blister should
never be performed; bacteria may be introduced into the
space and incite infection.22
All partial-thickness and deeper burns are at increased
risk for infection. Consequently, attention to chemophylaxis
is extremely important. Tetanus status should be evaluated
for all significant burns. If it has been greater than 5 years
since the last tetanus immunization, the patient needs a
booster injection. For those 7 years of age or older, Td
(adult diptheria and tetanus toxoid) is indicated. In a child
younger than 7 years, DTaP (acellular pertussis vaccine and
tetanus toxoid) should be administered unless there is a
contraindication to pertussis vaccine; if this is the case, DT
(diptheria and tetanus toxoid) should be given. If a patient
has not been immunized against tetanus in the past, TIG
(tetanus immunoglobulin) in addition to the appropriate
tetanus vaccine should be administered intramuscularly at
2 different sites.23
For most burns, there is no role for initial intravenous
antibiotic therapy. Using antibiotics initially may increase
the risk of colonization of more virulent organisms. There-
fore, intravenous antibiotics should be reserved only for
obvious secondary wound infections.20,24
Due to an intact epidermis that retains all protec-
tive function, superficial burns only require application of
moisturizer for treatment. For partial-thickness burns,
most authors recommend topical antibiotic ointment such
as bacitracin and polymyxin (Polysporin; Warner-Lambert
Consumer Healthcare, Freiburg, Germany), or mupirocin
topical (Bactroban; GlaxoSmithKline, Research Triangle
Park, NC) along with a nonadhesive dressing such as
petroleum gauze (Adaptic; Johnson and Johnson, New
Brunswick, NJ) or bismuth-impregnated gauze (Xeroform;
FIGURE 5. A full-thickness (third degree) burn in an African-American patient.
n 2005 Lippincott Williams  Wilkins 123
Pediatric Emergency Care  Volume 21, Number 2, February 2005 Emergency Management of Pediatric Burns
Copyright © Lippincott Williams  Wilkins. Unauthorized reproduction of this article is prohibited.
Kendall Healthcare, Mansfield, MA) followed by a gauze
dressing.7,24
One author recommends that dressings be
changed twice a day because of the shorter half-life of
ointments.24
This has been the standard in most institutions.
Recently, however, some authors have suggested that once-
daily dressing changes are adequate for smaller burns and
have decreased cost, pain with dressing changes, and need
for nursing time. It is still recommended for infection-prone
wounds or large extensive burns that dressings be changed
twice daily.20
Any exudates or previously applied antibi-
otic ointment needs to be completely removed, and new
antibiotic ointment needs to be reapplied with each dressing
change.
For full-thickness burns, creams are most often used.
Silver sulfadiazine 1% cream (Silvadene; Aventis Pharma-
ceuticals, Inc, Bridgewater, NJ) is commonly used. Appli-
cation is painless, and it is bactericidal. Disadvantages
include cutaneous hypersensitivity reactions, thrombocyto-
penia, and leukopenia.17,24
Silver sulfadiazine (Silvadene;
Aventis Pharmaceuticals, Inc) may result in eschar forma-
tion, therefore interfering with the assessment of burn depth
and healing. It cannot be used in those with sulfa allergies
and should be avoided in premature infants and newborns
younger than 1 month because of the complication of
kernicterus.17,24
Silver nitrate 0.5% solution can also be
used for full-thickness burns. Application is painless, and it
is effective against a large spectrum of bacteria as well as
fungi.14
Disadvantages include that it will readily stain,
has poor eschar penetration, and leaches electrolytes from
wounds oftentimes leading to hyponatremia in significant
burns.14,19
Mafenide acetate 0.5% cream (Sulfamylon;
Mylan Laboratories, Inc, Canonsburg, PA) is another widely
used topical burn preparation. It is bacteriostatic and has
good activity against Pseudomonas aeruginosa. One major
advantage is its ability to effectively penetrate eschar; it is
widely used on burns of the external ear to prevent infec-
tious chondritis.14,20
Disadvantages include the possibility
of a cutaneous rash and painful application. Because it is a
carbonic anhydrase inhibitor, it may also lead to metabolic
acidosis.
Instead of using topical creams and ointments, burn
wounds may be managed with synthetic occlusive dressings
such as Biobrane (Bertek Pharmaceuticals, Inc, Morgantown,
WV), human allograft, or pigskin.8
These can be applied to
clean partial-thickness wounds that are less than 24 hours
old.20
The dressing adheres to the wound until epithelization
occurs. The disadvantage is that the dressing can only cover
1% to 2% TBSA burn and is very expensive, prohibiting
routine use. For outpatient treatment, one should trim the
edges of the dressing as it begins to fall off and continue
to change an overlying gauze dressing once daily.6
Infection in burns is a major complication and may
often be difficult to recognize. A typical burn exhibits
inflammation along with erythema, tenderness, and edema.
If these symptoms worsen or are accompanied by fever,
malaise, or lymphangitis, infection should be suspected.
Infected burns should be treated with intravenous antibiotics
and hospital admission. Infection can not only cause sepsis,
but it can also lead to deeper burn damage.
Most pediatric burns can be managed in an outpatient
setting. Superficial burns can be treated with moisturizing
lotions, pain control, and reassurance. Partial-thickness burns
that are less than 10% of TBSA or full-thickness burns that
are less than 2% of TBSA can be managed in the out-
patient setting as long as they do not involve the face, hands,
feet, or genital region.7
Children with small superficial
hand burns may be treated as outpatients as long as there is
appropriate follow-up, and the caregiver can adequately
care for the child. The complications that arise from hand
burns such as infection and contractures necessitate that all
other hand wounds be treated on an inpatient basis.25
Most
suggest twice-daily dressing changes with antibiotic oint-
ment for the first week followed by once-daily dressing
FIGURE 6. The modified Lund and Browder Chart used
to estimate the TBSA burns of children. Adapted and pub-
lished with permission from Shriners Hospitals for Children,
Cincinnati, OH.
124 n 2005 Lippincott Williams  Wilkins
Reed and Pomerantz Pediatric Emergency Care  Volume 21, Number 2, February 2005
Copyright © Lippincott Williams  Wilkins. Unauthorized reproduction of this article is prohibited.
changes for the remainder of the healing process. Initially,
frequent visits to the primary care physician must be initi-
ated to monitor wound healing and infection. Admission to
a general or pediatric hospital is recommended for partial-
thickness burns 10% to 20% TBSA and full-thickness burns
up to 5% TBSA.17
Transfer to a burn center is recommended
for any partial-thickness burn greater than 20% TBSA in any
age patient or greater than 10% TBSA in children younger
than 10 years. Full-thickness burns greater than 5%; burns to
the face, hands, feet, major joints, or genital region; inha-
lation injury; significant burns with multiple trauma;
electrical burns and lightning injury; chemical burns; cir-
cumferential burns; burns caused by child abuse or other
cases requiring long-term emotional or rehabilitation sup-
port; and burns in patients with preexisting comorbidities
should also be treated at a burn hospital.14,26
If the home
environment is unsafe or the caregiver is unable to provide
appropriate care, the child should be admitted to the hospital
regardless of the size of the burn.
Prevention
Scald burns are the most common etiology for burn
injuries resulting in hospitalization in infants, toddlers, and
preschoolers, and more than 80% are caused by tap-water
scalds.3,27
It has been shown that if the temperature of water
is reduced from 558C to 498C (1318F to 1208F), it will take
10 minutes to produce the same burn that at the higher
temperature took 30 seconds.28
Therefore, it is recommended
that all hot-water heaters be set at less than 498C (1208F).
Hot liquid that is heated to 608C (1408F) can cause a burn in
5 seconds, and liquid heated to 718C (1608F) will cause a
burn in 1 second. Hot grease can be as hot as 2048C (4008F)
and cause immediate severe burns. In children younger than
5 years, the time that it takes to cause these burns may be
half of what it would take to cause the same burn in older
children and adults.29
Other ways to prevent burn-related
injuries include turning pot handles on the stove inward and
never leaving hot bowls or cups of liquid unattended. In
addition, avoid using a tablecloth when children are present
because a child can easily pull the tablecloth and spill hot
liquid onto himself. As with any preventable injury, super-
vising the child at all times can prevent some of the most
serious injuries.
Over the past 20 years though, there has been a drop
in fire-related deaths. Much of this is because of the com-
mitment to educate children and adults about fire preven-
tion. Programs such as ‘‘Stop, Drop, and Roll’’ to address
burning clothing, the development of flame-retardant
sleepwear, and the use of smoke detectors have prevented
numerous fire-related injuries and deaths. It is important
to have a smoke detector on each level of a house as well
as in each bedroom. Parents should have a specific escape
plan communicated to their children to be used in the event
of a house fire. Matches should be kept out of reach
of children and only used with parental presence. Lighters
should also be kept away from children. All lighters should
be child-resistant, and parents should not allow children
to use lighters as a ‘‘toy.’’29
Unfortunately, fireworks are
easily accessible to teenagers and older children. Parents
should not permit their children to ignite or play with fire-
works. Firework displays should be left to professionals.
ELECTRICAL BURNS
Electrical burns result in over 1500 deaths per year
and more than 4000 ED visits.4,30
Electrical burns also
account for 2% to 3% of all admissions to hospital burn
centers, and that number is slowly increasing.4
Up to one
third of electrical burns are household burns, seen mostly
among children.
Electrical burns result from thermal energy that is
produced as an electrical current passes through the body.
The amount of thermal energy produced is directly pro-
portional to the degree of electric current. The extent of
injury is dependent on 6 factors including the resistance
of skin, mucosa and internal structures, type of current,
frequency of the current, duration of contact, intensity of the
current, and the pathway taken by the current.31
Generally,
high-voltage injury is more severe than low-voltage injury.
The longer the victim is in contact with the electrical source,
the more severe the damage.
Resistance is inversely proportional to tissue injury.
Nerves, muscles, and blood vessels have low electrical resis-
tance; therefore, electricity will preferentially flow through
these structures and cause severe damage. Conductivity de-
creases as one moves from these structures to skin, ten-
dons, bone, and fat, in that order.17
Water will additionally
decrease resistance, thus resulting in increased internal
damage. Consequently, the areas of the body with increased
moisture, such as the axilla and antecubital fossa, will sus-
tain greater injury. Thick calloused adult skin may exhibit
greater damage at the site of entry than more internally as
this type of skin impedes current flow. On the other hand,
thin wet skin or mucous membranes may exhibit minor
thermal injury at the site of contact but more significant
internal injury because of lower resistance to flow.
The type of current also plays a major role in the
extent of tissue injury. Alternating current is more dangerous
than direct current because it produces muscle tetany
caused by the continual contraction and relaxation of the
muscle with each cycle. Typically, alternating current is
found in household electricity. A 60-Hz alternating current
will change direction 120 times per second.31
This frequency
is so rapid that it prevents muscle relaxation and keeps
the muscle in a continual refractory state leading to muscle
tetany. If tetany occurs in the muscles of the chest wall,
suffocation may result. In addition, if a patient is holding
n 2005 Lippincott Williams  Wilkins 125
Pediatric Emergency Care  Volume 21, Number 2, February 2005 Emergency Management of Pediatric Burns
Copyright © Lippincott Williams  Wilkins. Unauthorized reproduction of this article is prohibited.
onto the electrical source, the patient may be unable to let
go because of muscle tetany, therefore increasing the dura-
tion of contact and extent of the injury. On the other hand,
direct current produces muscle contraction only at the
beginning and end of the current flow.30
This type of cur-
rent is used in the medical setting and is found in lightning
strikes. If this current passes through the heart, the patient
is at risk for ventricular fibrillation or asystole.
Electrical burns can be the result of low-, medium-, or
high-voltage current. They may result in burns anywhere
from partial-thickness to deep burns involving underlying
structures. Low-current injuries (200 V) are often seen in
young children from household electrical sources such as
putting an electrical cord or plug in their mouth or placing
objects into electrical outlets. Medium-current (200–1000 V)
and high-current (1000 V) injuries are commonly seen in
older children and teenagers with risk-taking behavior,
utility workers, and people caught in electrical storms.
These would include lightning strikes and high-tension elec-
trical injuries that occur when children or teens climb trees,
electrical poles, or other utility constructions and contact
large electrical wires.
The direction the current flows through the body can
also predict the severity of injury. Electric current will flow
from the point of contact to the ground or another area of
the body that completes the electrical circuit. Most com-
monly, low current follows a path of least resistance, whereas
high current will follow a path directly to the ground.
Flow through the body usually takes 1 of 3 pathways: hand
to hand, hand to foot, or foot to foot. Hand-to-hand flow is
the most dangerous and has a 60% mortality rate because
of risk of spinal cord transection at C4 to C8, suffocation
caused by tetany of the chest wall muscles, and myocar-
dial muscle damage.31
The hand-to-foot pathway carries a
20% mortality rate largely secondary to associated cardiac
arrythmias. The foot-to-foot pathway is associated with less
than 5% mortality.31
Lightning may injure a child in 4 different ways
including direct strike, stride potential, side flash, and flash-
over phenomenon. Direct strike is the most dangerous form
of a lightning strike; the electrical current passes directly
through the child. Alternatively, lightning can strike as a
side flash. This occurs when the lightning has already
passed through a primary source such as a tree or a person
and then travels through the air to a second victim. Stride
potential is defined as lightning hitting the ground and
entering through one leg of a person while exiting through
the other leg. When lightning flows outside of the body, it
is referred to as flashover phenomenon. This usually occurs
when a patient has on wet clothing and often results in
thermal skin burns caused by the ignition of clothing.
Initial assessment of the electrical burn victim
includes airway, breathing, and circulation. History should be
taken to determine the type of current and voltage involved.
Placing a patient on cardiac monitors immediately while
initiating appropriate fluid resuscitation and pain control is
important. A patient who is a victim of a high-voltage
injury may be unresponsive, pulseless, and apneic. In addition,
these patients often sustain additional injuries after being
thrown off of electrical poles or other structures, thereby
resulting in traumatic injuries to the abdomen, head,
neck, and extremities. Immobilizing the cervical spine
and following the basic Advanced Trauma Life Support
protocol with primary and secondary survey in this
situation is necessary. Oftentimes, a patient may appear to
have minor superficial injury when actually there is
significant underlying tissue damage. It is important to
anticipate severe internal damage until proven otherwise.
Clinically, electrical injuries can affect multiple organ
systems; thus, it is important to perform a thorough eval-
uation. The cutaneous manifestations include flame burns,
flash burns, and arc burns as well as mottled cyanotic skin.
Flame burns are usually full thickness secondary to the
effects of burning clothes. Flash burns are usually partial
thickness resulting from lightning injuries. Current can easily
jump across flexed joints producing arc burns. In addition,
skin of a victim may appear mottled and cyanotic because
of autonomic instability, although this is generally transient.
The cardiac effects are the most life-threatening and
include cardiac dysrhythmias or myocardial damage. Most
commonly, ventricular fibrillation is seen in low-voltage
alternating-current injuries. Lightning strikes and high-
voltage injuries most commonly produce asystole. Tachy-
cardia and hypertension may be seen secondary to excessive
catecholamine production.
Musculoskeletal injury may be extensive in high-
voltage injuries. Oftentimes, tissue that initially appears
well perfused, later becomes edematous and necrotic. Initial
strong pulses do not always predict that blood vessels
are intact, and compartment syndrome can develop. Renal
failure can occur by either the direct effect of the current on
the kidney tissue, hypoxic damage to the kidney, or renal
tubular damage secondary to excess myoglobin deposition
from extensive muscle damage.31
Electrical injuries produce
damage to the central nervous system including the brain,
spinal cord, peripheral motor and sensory nerves, and sym-
pathetic fibers. Manifestations are classified as immediate,
secondary (within 5 days), or delayed (after 5 days). The
most common immediate manifestations that may be seen
on presentation to the ED include painful sensation, loss
of consciousness, respiratory center paralysis, confusion,
motor paralysis, visual disturbances, deafness, sensory defi-
cits, hemiplegia, quadriparesis, seizures, amnesia, disorien-
tation, and intracranial bleeds.31
Although not as common,
one should also be aware that pulmonary, abdominal, and
ocular manifestations may occur as well.
126 n 2005 Lippincott Williams  Wilkins
Reed and Pomerantz Pediatric Emergency Care  Volume 21, Number 2, February 2005
Copyright © Lippincott Williams  Wilkins. Unauthorized reproduction of this article is prohibited.
Low-voltage household current can produce injury of
the oral cavity and the lips. This most common occurs
when a toddler places a household plug or cord in his/her
mouth.30
The result is coagulation necrosis and a deep burn
to the corner of the mouth. These burns appear as a cen-
tral black area surrounded by a gray-white ring surrounded
by an erythematous circle. This type of injury is often
painless because of the destruction of nerves. Because the
commissure of the lip is involved, children need close
follow-up to prevent excessive scarring and contractures.
The most serious complication is significant bleeding from
the labial artery 1 to 2 weeks after the injury when the
eschar separates.14,32
These children do not necessarily
need to be admitted to the hospital at the time of the injury.
Instead, the physician needs to arrange follow-up the next
day and educate the parents as to the complications that
would necessitate a return visit to the hospital.30
All patients who present with significant electrical
current injuries should undergo laboratory testing as would
a patient with severe thermal burns. These laboratory tests
include a complete blood count, electrolytes, blood urea
nitrogen, creatinine, and a urinalysis with urine myoglobin.
Patients should also have an electrocardiograph and chest
radiograph performed and be placed on cardiac monitors.
A physician should consider obtaining creatine phosphoki-
nase with cardiac enzymes. With significant trauma asso-
ciated with electrical burns, consider head, spine, and
abdominal imaging.
All patients with high-voltage injuries should be
admitted to the hospital. Low-voltage injuries less commonly
produce complications. One author found that none of
the patients in his study exposed to low-voltage current
developed cardiac arrhythmias despite laboratory test–
confirmed muscle damage (elevated creatine phosphokinase)
in a few patients.30
Thus, the recommendation is to observe
patients in the ED for 4 hours on cardiac monitors. If
the patient does not have cardiac arrhythmias, did not lose
consciousness, and has only minor wounds, outpatient man-
agement can be safely arranged.30
Management of patients with medium- and high-
voltage injuries (200 V) includes observing children on a
cardiac monitor for 24 to 72 hours because of the increased
risk of cardiac arrythmias.14
In patients with significant
muscle damage, pigmented urine is often present and puts
a patient at risk for the development of renal failure.
Foley catheters should be placed in these at-risk patients.
Increasing the patient’s urine output to a goal of 2 mL/kg/h and
alkalinizing the urine with bicarbonate will help prevent
this complication.14
Patients may develop hyperkalemia
caused by extensive muscle tissue damage; consequently,
potassium levels need to monitored closely.
Household electrical cords are responsible for the
majority of electrocutions in children younger than 12 years,
yet there are no federal safety standards in effect to pre-
vent household electrical injuries. Thus, it is important for
parents to keep all electrical outlets covered with plastic
plugs and keep children away from all electrical cords. This
will prevent children from placing objects into the electri-
cal outlets or electrical cords into their mouths resulting
in electrocution. Older children and teenagers should be
counseled on the dangers of climbing electrical structures
and be taught to avoid other risk-taking behaviors. To
prevent lightning strikes, children should be taught to stay
indoors during storms, exit the water immediately, and avoid
contact with metal objects.
CHEMICAL BURNS
There are between 25,000 and 100,000 chemical burns
reported in the United States each year, and collectively, they
have a morbidity and mortality rate of less than 1%. Children
and adults are reported to have similar rates of exposure.33
Over 25,000 different chemicals can produce either acid
or alkali burns.4
The burn is usually a result of a direct
chemical injury, but there also may be components of a
thermal burn caused by an exothermic reaction.17
Acid burns result in coagulation necrosis, which
usually limits the depth and penetration of the burn.
Common household products that contain acids include
drain cleaners (sulfuric acid or hydrochloric acid), toilet
cleaners (hydrochloric acid or phosphoric acid), and car
batteries (sulfuric acid).34
Ingestion of acids will lead to
gastric injury and often leaves patients with strictures of the
pylori. In severe cases, involvement of the esophagus,
stomach, and small intestine can be seen.
Alkalis produce liquefactive necrosis, thus causing
deeper penetration and a more significant burn. Alkalis
include lye (sodium hydroxide), cement (calcium, potas-
sium, and sodium hydroxide), fertilizers (anhydrous am-
monia), oven and drain cleaners (sodium or potassium
hydroxide), paint strippers (sodium hydroxide), and various
detergents.17,34
Ingesting alkalis can lead to significant
gastrointestinal injury and perforation and, most commonly,
esophageal strictures.
Regardless of the type of chemical burn, it is neces-
sary initially to remove all clothing and perform copious
irrigation for at least 30 minutes to prevent further injury
to the tissue. Do not attempt to neutralize chemical burns;
this can lead to further thermal burn injury because of
the exothermic reaction. Measuring the resultant pH will
determine whether the tissue has been adequately irrigated.
The pH should be measured approximately 15 minutes after
irrigation to allow chemicals from deeper tissues to diffuse
to the surface. Testing the pH too early may lead to false-
negative results.34
Because alkali substances are less water-
soluble, these burns often take longer to neutralize. Any
chemical injury to the eye should be treated in the same
n 2005 Lippincott Williams  Wilkins 127
Pediatric Emergency Care  Volume 21, Number 2, February 2005 Emergency Management of Pediatric Burns
Copyright © Lippincott Williams  Wilkins. Unauthorized reproduction of this article is prohibited.
way followed by consultation with an ophthalmologist. In
cases of ingestion, do not induce emesis. Instead, treatment
with ingestion of milk or water is recommended followed
by diagnostic endoscopy. Activated charcoal is contrain-
dicated; it does not bind to acid or alkali and will complicate
endoscopy.34
Hydrofluoric acid burn treatment is an exception to
the typical burn treatment regimen. It is extremely corrosive
and causes liquefactive necrosis and bone corrosion. Cal-
cium gluconate gel should be massaged into the affected
area for at least 30 minutes.34
If the pain is persistent or the
hydrofluoric acid strength is greater than 20%, 10% calcium
gluconate should be injected into the tissues. In severe
hydrofluoric acid burns, calcium ions may be trapped as
calcium fluoride, and hypocalcemia will occur which may
lead to ventricular arrythmias. Patients with hydrofluoric
acid burns should be placed on a cardiac monitor, and cal-
cium levels should be monitored closely with intravenous
calcium replacement as indicated.14
To prevent chemical injuries, all caustic substances
most commonly used to clean bathrooms or kitchens should
be kept out of reach of children. The cabinets where these
products are stored should have child safety locks. Chem-
icals stored in a garage or shed should also be kept out of
reach of children. All chemicals should all be kept in their
original containers or bottles and should be used appropri-
ately with the proper ventilation and protective equipment. In
addition, care should be taken while using these products to
keep children away.
CHILD ABUSE OR NONACCIDENTAL BURNS
Between 10% and 20% of burns in children are
inflicted.4
It is estimated that between 16% and 20% of
patients with burns resulting in hospital admission are
victims of abuse, and most of these types of burns are more
extensive and severe.35
Most inflicted burns are scald or contact burns and
oftentimes have recognizable patterns such as a triangular
shape from the tip of an iron or linear parallel lines from
a radiator. Toddlers who are submersed in hot water will
present with scald burns to the buttocks, thighs, and feet.
The most common areas involved in scald burns of abuse
include the buttocks and perineum. These areas are rarely
involved in unintentional scald burns particularly in the
nonambulatory child. Severe burns to the hands and feet in
a glove or stockinglike pattern, often symmetric and with
a distinct line of demarcation, are classic for nonaccidental
immersion.36
Alternatively, a child may be submersed more
than once and may not have specific lines of demarcation.
Oftentimes the palms, soles, or buttocks are spared because
of contact with the cool floor of the water-containing object
such as the floor of a bathtub or sink. When the buttocks are
spared, it is described as a ‘‘doughnut hole’’ appearance.27,37
The flexion creases in the anterior hip, popliteal fossa,
antecubital space, and lower abdomen may be spared when
the trunk is in flexion.27,37
This is seen when a child is in
a defensive position with arms, legs, and trunk flexed
while being forced into hot liquid against his/her will. Splash
burns can be seen when a hot liquid is thrown at the
child. This is less common and will leave an ‘‘arrowhead’’
appearance as the hot liquid runs down the patient’s body
because of gravitational force.37
Cigarette burns appear as deep, small, circular burns.
Another less common form of child abuse is burning through
microwave ovens. A small child is placed in a microwave
oven, and significant burns are seen on the area of the
skin closest to the area that emits microwaves. In addition
stun guns burns may be seen in abuse cases. Because stun
guns are easily obtainable by the public, partial-thickness
electrical burns can result.
When a child presents with these types of burns,
physicians need to have a high suspicion of abuse, espe-
cially when the history of the injury does not match the
pattern of the burn or there is no witness to the injury.
Historically, one should be suspicious of abuse if there is
significant delay in the time to presentation of an injury, if
the parents seem uninterested or do not want to manage
the child’s postburn care, if the child is brought to the
hospital by someone other than the parent, or if there is
documentation of previous unexplained injuries or neglect.36
On physical examination, one should be concerned about
the potential of abuse if the mechanism of injury does not
match the child’s developmental age, burns are older than
the history suggests, other injuries are seen such as fractures
and bruising, burns are symmetric or localized to the buttock
and perineum, or the child is passive to invasive painful
procedures.36
The burn itself should be treated appropri-
ately, and social services should be consulted. Reporting
suspected abuse to the appropriate child protective authority
is mandated by law for all physicians suspecting abuse.
Regardless of the size or severity of the burn, if there is
concern regarding the safety of the child, the child should
be admitted to the hospital until the case has been properly
investigated.
Prevention of child abuse can be difficult. It is im-
portant for the physician to report any suspicion of abuse
at the first sign to prevent further fatal abuse of the child.
Reporting at the initial suspicion may also help to prevent
abuse in siblings by having them removed from the abusive
home and placed in appropriate foster care.
CONCLUSION
Pediatric burns result in significant morbidity and
mortality. Emergency department physicians are usually one
of the first healthcare providers to encounter these injuries.
Thus, it is imperative that physicians receive education on
128 n 2005 Lippincott Williams  Wilkins
Reed and Pomerantz Pediatric Emergency Care  Volume 21, Number 2, February 2005
Copyright © Lippincott Williams  Wilkins. Unauthorized reproduction of this article is prohibited.
the proper diagnosis and management of all burn injuries
to prevent further complications in patients. In addition,
one should be proficient in recognizing signs of burn abuse.
Incorporating burn prevention strategies into physician’s
daily routine is necessary to limit future burn-related injuries
and deaths.
ACKNOWLEDGMENT
The authors thank Donna Mertens, RN, for her
assistance in obtaining the photographs included in this
manuscript.
REFERENCES
1. Center for Disease Control and Prevention. Ten leading causes of
injury death by age group—2001 highlighting unintentional injury
deaths. Available at: ftp://ftp.cdc.gov/pub/ncipc/10LC-2001/PDF/101c-
unintentional.pdf. Accessed March 7, 2004.
2. Center for Disease Control and Prevention, National Center for Injury
Prevention and Control, WISQUARS. Overall fire/burn nonfatal injuries
and rates per 100,000 and fire/burn deaths and rates per 100,000.
Available at: http://webapp.cdc.gov/cgi-bin/broker.exe. Accessed June
14, 2004.
3. McLoughlin E, McGuire A. The causes, cost and prevention of child-
hood burn injuries. Am J Dis Child. June 1990;144(6):677–683.
4. Joffe MD. Burns. In: Fleisher GR, Ludwig S, eds. Textbook of Pediatric
Emergency Medicine. 4th ed. Philadelphia: Lippincott Williams 
Wilkins; 2000:1427–1434.
5. Centers for Disease Control and Prevention, National Center for Injury
Prevention and Control. Fire and burn injuries fact sheet. Available
at: http://www.cdc.gov/ncipc/factsheets/fire.htm. Accessed February 3,
2004.
6. Stewart C. Emergency care of pediatric burns. Emerg Med Rep. October
2000;5(10):101–111.
7. Passaretti D, Billmire DA. Management of pediatric burns. J Craniofac
Surg. September 2003;14(5):713–718.
8. Schonfeld N. Outpatient management of burns in children. Pediatr
Emerg Care. September 1990;6(3):249–253.
9. Lund C, Browder N. The estimation of areas of burns. Surg Gynecol
Obstet. 1944;79:352–358.
10. Merten DM, Jenkins ME, Warden GD. Outpatient burn management.
Nurs Clin North Am. June 1997;32(2):343–364.
11. Sheridan RL, Retras L, Basha G, et al. Planimetry study of the percent
of body surface represented by the hand and palm: sizing irregular
burns is more accurately done with the palm. J Burn Care Rehabil.
November–December 1995;16(6):605–606.
12. Perry RJ, Moore CA, Morgan BD, et al. Determining the approximate
area of a burn: an inconsistency investigated and re-evaluated. Br J
Med. May 25, 1996;312(7042):1338.
13. Purdue GF, Layton TR, Copeland CE. Cold injury complicating burn
therapy. J Trauma. February 1985;25(2):167–168.
14. Sheridan RL. Burns. Crit Care Med. November 2002;30(11 suppl):
S500–S514.
15. Sheridan RL. Airway management and respiratory care of the burn
patient. Int Anesthesiol Clin. Summer 2000;38(3):129–145.
16. Heimbach DM, Waeckerle JF. Inhalation injuries. Ann Emerg Med.
December 1988;17(12):1316–1320.
17. Smith ML. Pediatric burns: management of thermal, electrical, and
chemical burns and burn-like dermatologic conditions. Pediatr Ann.
June 2000;29(6):367–378.
18. Cortiella J, Marvin JA. Management of the pediatric burn patient.
Nurs Clin North Am. June 1997;32(2):311–329.
19. Monafo WW. Initial management of burns. N Engl J Med. November
21, 1996;335(21):1581–1586.
20. Kao CC, Garner WL. Acute burns. Plast Reconstr Surg. June 2000;
101(7):2482–2493.
21. Rockwell WB, Ehrlich HP. Should burn blister fluid be evacuated?
J Burn Care Rehabil. January to February 1990;11(1):93–95.
22. Clayton MC, Solem LD. No ice, no butter. Advice on management
of burns for primary care physicians. Postgrad Med. May 1995;97(5):
151–155, 159–160, 165.
23. American Academy of Pediatrics. Tetanus. In: Pickering LD, eds.
Red book: 2003 Report of the Committee on Infectious Diseases.
26th ed.Elk Grove Village, IL: American Academy of Pediatrics; 2003:
611–616.
24. Palmieri T, Greenhalgh D. Topical treatment of pediatric patients with
burns: a practical guide. Am J Clin Dermatol. 2002;3(8):529–534.
25. Sheridan RL, Baryza MJ, Pessina MA, et al. Acute hand burns in
children: management and long-term outcome based on a 10-year
experience with 698 injured hands. Ann Surg. April 1999;229(4):
558–564.
26. Smith S, Duncan M, Mobley J, et al. Emergency room management
of minor burn injuries: a quality management evaluation. J Burn Care
Rehabil. January–February 1977;18(1 pt 1):76–80.
27. Stratman E, Melski J. Scald abuse. Arch Dermatol. March 2002;
138(3):318–320.
28. Moritz AR, Henriques FC. Studies of thermal injury: II. The relative
importance of time and surface temperature in the causation of cuta-
neous burns. Am J Pathol. 1947;23:695–720.
29. Shriners Hospitals for Children. Pamphlet: Burn Prevention Tips.
Tampa, FL: Shriners Hospitals for Children; September 1999:1–24.
30. Zubair M, Besner GE. Pediatric electrical burns: management strategies.
Burns. August 1997;23(5):413–420.
31. Baum CR. Environmental injuries. In: Fleisher GR, Ludwig S, eds.
Textbook of Pediatric Emergency Medicine. 4th ed. Philadelphia:
Lippincott Williams  Wilkins; 2000:959–963.
32. Jain S, Bandi V. Electrical and lightning injuries. Crit Care Clin. April
1999;15(2):319–331.
33. Hostetler MA. Burns, Chemical [last updated January 20, 2004]. Avail-
able at: http://www.emedicine.com/ped/topic2735.htm. Accessed June
14, 2004.
34. Bates N. Acid and alkali injury. Emerg Nurse. December 1999–January
2000;7(8):21–26.
35. Deitch E, Staats M. Child abuse through burning. J Burn Care Rehabil.
1982;3:89–94.
36. Andronicus M, Oates RK, Peat J, et al. Non-accidental burns in children.
Burns. September 1998;24(6):552–558.
37. Peck MD, Priolo-Kapel D. Child abuse by burning: a review of
the literature and an algorithm for medical investigations. J Trauma.
November 2002;53(5):1013–1022.
n 2005 Lippincott Williams  Wilkins 129
Pediatric Emergency Care  Volume 21, Number 2, February 2005 Emergency Management of Pediatric Burns
Copyright © Lippincott Williams  Wilkins. Unauthorized reproduction of this article is prohibited.

Weitere ähnliche Inhalte

Ähnlich wie 9 emergency mgmt of burns -peds emerg care 2005

Ähnlich wie 9 emergency mgmt of burns -peds emerg care 2005 (20)

Manejo ambulatorio de quemaduras
Manejo ambulatorio de quemadurasManejo ambulatorio de quemaduras
Manejo ambulatorio de quemaduras
 
Burns.pptx
Burns.pptxBurns.pptx
Burns.pptx
 
Burns
BurnsBurns
Burns
 
Abc of burns
Abc of burnsAbc of burns
Abc of burns
 
The thermal injury
The thermal injuryThe thermal injury
The thermal injury
 
Copy2-BURNS slides.pptx
Copy2-BURNS slides.pptxCopy2-BURNS slides.pptx
Copy2-BURNS slides.pptx
 
Burns
BurnsBurns
Burns
 
Major burn management
Major burn managementMajor burn management
Major burn management
 
Burn in children
Burn in childrenBurn in children
Burn in children
 
burn.pptx
burn.pptxburn.pptx
burn.pptx
 
3 Burn Management
3 Burn Management3 Burn Management
3 Burn Management
 
Burn(2)
Burn(2)Burn(2)
Burn(2)
 
51316 ch08 188_217
51316 ch08 188_21751316 ch08 188_217
51316 ch08 188_217
 
Physiotherapy in burns
Physiotherapy in burnsPhysiotherapy in burns
Physiotherapy in burns
 
Quemaduras
QuemadurasQuemaduras
Quemaduras
 
BURNS.pptx
BURNS.pptxBURNS.pptx
BURNS.pptx
 
diseasepaper
diseasepaperdiseasepaper
diseasepaper
 
Burns -RBXY1.ppt
Burns -RBXY1.pptBurns -RBXY1.ppt
Burns -RBXY1.ppt
 
Burns in pediatrics presentation
Burns in pediatrics presentationBurns in pediatrics presentation
Burns in pediatrics presentation
 
Classification, Principles, assessment and management of burn
Classification, Principles, assessment and  management of burnClassification, Principles, assessment and  management of burn
Classification, Principles, assessment and management of burn
 

Kürzlich hochgeladen

Statistical modeling in pharmaceutical research and development.
Statistical modeling in pharmaceutical research and development.Statistical modeling in pharmaceutical research and development.
Statistical modeling in pharmaceutical research and development.ANJALI
 
Biomechanics- Shoulder Joint!!!!!!!!!!!!
Biomechanics- Shoulder Joint!!!!!!!!!!!!Biomechanics- Shoulder Joint!!!!!!!!!!!!
Biomechanics- Shoulder Joint!!!!!!!!!!!!ibtesaam huma
 
Tans femoral Amputee : Prosthetics Knee Joints.pptx
Tans femoral Amputee : Prosthetics Knee Joints.pptxTans femoral Amputee : Prosthetics Knee Joints.pptx
Tans femoral Amputee : Prosthetics Knee Joints.pptxKezaiah S
 
Presentació "Real-Life VR Integration for Mild Cognitive Impairment Rehabilit...
Presentació "Real-Life VR Integration for Mild Cognitive Impairment Rehabilit...Presentació "Real-Life VR Integration for Mild Cognitive Impairment Rehabilit...
Presentació "Real-Life VR Integration for Mild Cognitive Impairment Rehabilit...Badalona Serveis Assistencials
 
systemic bacteriology (7)............pptx
systemic bacteriology (7)............pptxsystemic bacteriology (7)............pptx
systemic bacteriology (7)............pptxEyobAlemu11
 
Basic principles involved in the traditional systems of medicine PDF.pdf
Basic principles involved in the traditional systems of medicine PDF.pdfBasic principles involved in the traditional systems of medicine PDF.pdf
Basic principles involved in the traditional systems of medicine PDF.pdfDivya Kanojiya
 
Measurement of Radiation and Dosimetric Procedure.pptx
Measurement of Radiation and Dosimetric Procedure.pptxMeasurement of Radiation and Dosimetric Procedure.pptx
Measurement of Radiation and Dosimetric Procedure.pptxDr. Dheeraj Kumar
 
Wessex Health Partners Wessex Integrated Care, Population Health, Research & ...
Wessex Health Partners Wessex Integrated Care, Population Health, Research & ...Wessex Health Partners Wessex Integrated Care, Population Health, Research & ...
Wessex Health Partners Wessex Integrated Care, Population Health, Research & ...Wessex Health Partners
 
Presentation on Parasympathetic Nervous System
Presentation on Parasympathetic Nervous SystemPresentation on Parasympathetic Nervous System
Presentation on Parasympathetic Nervous SystemPrerana Jadhav
 
SYNDESMOTIC INJURY- ANATOMICAL REPAIR.pptx
SYNDESMOTIC INJURY- ANATOMICAL REPAIR.pptxSYNDESMOTIC INJURY- ANATOMICAL REPAIR.pptx
SYNDESMOTIC INJURY- ANATOMICAL REPAIR.pptxdrashraf369
 
low cost antibiotic cement nail for infected non union.pptx
low cost antibiotic cement nail for infected non union.pptxlow cost antibiotic cement nail for infected non union.pptx
low cost antibiotic cement nail for infected non union.pptxdrashraf369
 
SWD (Short wave diathermy)- Physiotherapy.ppt
SWD (Short wave diathermy)- Physiotherapy.pptSWD (Short wave diathermy)- Physiotherapy.ppt
SWD (Short wave diathermy)- Physiotherapy.pptMumux Mirani
 
Case Report Peripartum Cardiomyopathy.pptx
Case Report Peripartum Cardiomyopathy.pptxCase Report Peripartum Cardiomyopathy.pptx
Case Report Peripartum Cardiomyopathy.pptxNiranjan Chavan
 
VarSeq 2.6.0: Advancing Pharmacogenomics and Genomic Analysis
VarSeq 2.6.0: Advancing Pharmacogenomics and Genomic AnalysisVarSeq 2.6.0: Advancing Pharmacogenomics and Genomic Analysis
VarSeq 2.6.0: Advancing Pharmacogenomics and Genomic AnalysisGolden Helix
 
Presentation for Bella Mahl 2024-03-28-24-MW-Overview-Bella.pptx
Presentation for Bella Mahl 2024-03-28-24-MW-Overview-Bella.pptxPresentation for Bella Mahl 2024-03-28-24-MW-Overview-Bella.pptx
Presentation for Bella Mahl 2024-03-28-24-MW-Overview-Bella.pptxpdamico1
 
Lippincott Microcards_ Microbiology Flash Cards-LWW (2015).pdf
Lippincott Microcards_ Microbiology Flash Cards-LWW (2015).pdfLippincott Microcards_ Microbiology Flash Cards-LWW (2015).pdf
Lippincott Microcards_ Microbiology Flash Cards-LWW (2015).pdfSreeja Cherukuru
 
Culture and Health Disorders Social change.pptx
Culture and Health Disorders Social change.pptxCulture and Health Disorders Social change.pptx
Culture and Health Disorders Social change.pptxDr. Dheeraj Kumar
 
Glomerular Filtration and determinants of glomerular filtration .pptx
Glomerular Filtration and  determinants of glomerular filtration .pptxGlomerular Filtration and  determinants of glomerular filtration .pptx
Glomerular Filtration and determinants of glomerular filtration .pptxDr.Nusrat Tariq
 
The next social challenge to public health: the information environment.pptx
The next social challenge to public health:  the information environment.pptxThe next social challenge to public health:  the information environment.pptx
The next social challenge to public health: the information environment.pptxTina Purnat
 

Kürzlich hochgeladen (20)

Statistical modeling in pharmaceutical research and development.
Statistical modeling in pharmaceutical research and development.Statistical modeling in pharmaceutical research and development.
Statistical modeling in pharmaceutical research and development.
 
Biomechanics- Shoulder Joint!!!!!!!!!!!!
Biomechanics- Shoulder Joint!!!!!!!!!!!!Biomechanics- Shoulder Joint!!!!!!!!!!!!
Biomechanics- Shoulder Joint!!!!!!!!!!!!
 
Tans femoral Amputee : Prosthetics Knee Joints.pptx
Tans femoral Amputee : Prosthetics Knee Joints.pptxTans femoral Amputee : Prosthetics Knee Joints.pptx
Tans femoral Amputee : Prosthetics Knee Joints.pptx
 
Presentació "Real-Life VR Integration for Mild Cognitive Impairment Rehabilit...
Presentació "Real-Life VR Integration for Mild Cognitive Impairment Rehabilit...Presentació "Real-Life VR Integration for Mild Cognitive Impairment Rehabilit...
Presentació "Real-Life VR Integration for Mild Cognitive Impairment Rehabilit...
 
systemic bacteriology (7)............pptx
systemic bacteriology (7)............pptxsystemic bacteriology (7)............pptx
systemic bacteriology (7)............pptx
 
Basic principles involved in the traditional systems of medicine PDF.pdf
Basic principles involved in the traditional systems of medicine PDF.pdfBasic principles involved in the traditional systems of medicine PDF.pdf
Basic principles involved in the traditional systems of medicine PDF.pdf
 
Measurement of Radiation and Dosimetric Procedure.pptx
Measurement of Radiation and Dosimetric Procedure.pptxMeasurement of Radiation and Dosimetric Procedure.pptx
Measurement of Radiation and Dosimetric Procedure.pptx
 
Wessex Health Partners Wessex Integrated Care, Population Health, Research & ...
Wessex Health Partners Wessex Integrated Care, Population Health, Research & ...Wessex Health Partners Wessex Integrated Care, Population Health, Research & ...
Wessex Health Partners Wessex Integrated Care, Population Health, Research & ...
 
Presentation on Parasympathetic Nervous System
Presentation on Parasympathetic Nervous SystemPresentation on Parasympathetic Nervous System
Presentation on Parasympathetic Nervous System
 
SYNDESMOTIC INJURY- ANATOMICAL REPAIR.pptx
SYNDESMOTIC INJURY- ANATOMICAL REPAIR.pptxSYNDESMOTIC INJURY- ANATOMICAL REPAIR.pptx
SYNDESMOTIC INJURY- ANATOMICAL REPAIR.pptx
 
low cost antibiotic cement nail for infected non union.pptx
low cost antibiotic cement nail for infected non union.pptxlow cost antibiotic cement nail for infected non union.pptx
low cost antibiotic cement nail for infected non union.pptx
 
SWD (Short wave diathermy)- Physiotherapy.ppt
SWD (Short wave diathermy)- Physiotherapy.pptSWD (Short wave diathermy)- Physiotherapy.ppt
SWD (Short wave diathermy)- Physiotherapy.ppt
 
Case Report Peripartum Cardiomyopathy.pptx
Case Report Peripartum Cardiomyopathy.pptxCase Report Peripartum Cardiomyopathy.pptx
Case Report Peripartum Cardiomyopathy.pptx
 
VarSeq 2.6.0: Advancing Pharmacogenomics and Genomic Analysis
VarSeq 2.6.0: Advancing Pharmacogenomics and Genomic AnalysisVarSeq 2.6.0: Advancing Pharmacogenomics and Genomic Analysis
VarSeq 2.6.0: Advancing Pharmacogenomics and Genomic Analysis
 
Presentation for Bella Mahl 2024-03-28-24-MW-Overview-Bella.pptx
Presentation for Bella Mahl 2024-03-28-24-MW-Overview-Bella.pptxPresentation for Bella Mahl 2024-03-28-24-MW-Overview-Bella.pptx
Presentation for Bella Mahl 2024-03-28-24-MW-Overview-Bella.pptx
 
Lippincott Microcards_ Microbiology Flash Cards-LWW (2015).pdf
Lippincott Microcards_ Microbiology Flash Cards-LWW (2015).pdfLippincott Microcards_ Microbiology Flash Cards-LWW (2015).pdf
Lippincott Microcards_ Microbiology Flash Cards-LWW (2015).pdf
 
Culture and Health Disorders Social change.pptx
Culture and Health Disorders Social change.pptxCulture and Health Disorders Social change.pptx
Culture and Health Disorders Social change.pptx
 
Glomerular Filtration and determinants of glomerular filtration .pptx
Glomerular Filtration and  determinants of glomerular filtration .pptxGlomerular Filtration and  determinants of glomerular filtration .pptx
Glomerular Filtration and determinants of glomerular filtration .pptx
 
The next social challenge to public health: the information environment.pptx
The next social challenge to public health:  the information environment.pptxThe next social challenge to public health:  the information environment.pptx
The next social challenge to public health: the information environment.pptx
 
Epilepsy
EpilepsyEpilepsy
Epilepsy
 

9 emergency mgmt of burns -peds emerg care 2005

  • 1. Emergency Management of Pediatric Burns Jennifer L. Reed, MD and Wendy J. Pomerantz, MD, MS, FAAP Key Words: burns, emergency care, management Burn-related injuries are a leading cause of morbidity and mortality in children. Burn injuries rank third among injury-related deaths in children aged 1 to 9 years.1 In 2001, there were more than 181,000 fire- and burn-related injuries, more than 4200 hospitalizations, and 672 deaths in children aged 0 to 19 years in the United States.2 Pediatric patients and the elderly have the highest morbidity and mortality associated with burn injuries.3 A majority of these children are initially seen in emergency departments (EDs) around the country; therefore, it is necessary that all emergency professionals are proficient in burn management in the pediatric population. Many burns are the result of uninten- tional events, and others are the result of nonaccidental trauma. However, most burns are preventable. As a primary care provider in the ED, it is necessary to have the knowl- edge and skills not only to treat burns, but to also counsel families regarding burn prevention and to identify burns resulting from child abuse. THERMAL BURNS Thermal burns are the most common type of burn in childhood. They can be a result of flames, scalds, contact, cold, or radiation. In the toddler age group, scald burns from hot liquid or hot grease are seen commonly and account for 80% of all thermal injuries.4 This type of burn accounts for a majority of childhood burn hospitalizations in this age group. Toddlers also have the highest rate of contact burns such as those that occur when touching hot metal from a stove, grill, or home space heater. Young school-age children have an innate curiosity and tend to play with dangerous equipment such as matches and cigarette lighters resulting in thermal burns. Older school-age children and teenage populations are more commonly burned from risk- taking activities, fireworks, and careless use of flammable substances such as gasoline, lighter fluid, or hairspray. In addition, household fires commonly caused by unattended cigarettes or candles are a major contributor to pediatric burn injuries and death in all age groups. Cigarettes are responsible for 35% of fatal house fires in the United States, many of which kill children.3 More than half of all fire- related deaths occur in homes that do not have smoke alarms, and fire-related injuries are the most common cause of fire- and burn-related deaths in children.5 Native Amer- ican children, African-American children, and children of low-income families are at increased risk for death and injury related to fire.5,6 Burn injuries are more common in boys than in girls, and an increase in fire-related deaths is seen during the winter months.2,5 Pathophysiology The skin is an important organ system that functions to protect the body from infectious agents, to regulate the body temperature by preventing heat loss, and to serve as a barrier to prevent body fluid loss. When the skin is dam- aged by a burn, devastating sequela may follow. The skin consists of 2 layers, the epidermis and the dermis. The epi- dermis includes 4 layers: stratum corneum, stratum lucidum, stratum granulosum, and stratum germinativum. The stratum corneum is the most important layer that protects the body from water loss and infection. Beneath the epidermis, the dermis consists of hair follicles, sweat glands, nerve fibers, and connective tissue. This layer is vital in regulating heat loss. Classification Burns are classified as first, second, or third degree. More commonly, they are referred to as superficial, partial thickness, or full thickness, respectively. Oftentimes it is difficult to correctly identify the depth of a burn, and it is common to have several depths exhibited in one injury, with the center usually demonstrating a higher degree of burn than the periphery.7 The thickness of the burn is directly related to the source of the burn and the time the skin is in contact with the source. Areas of thin skin such as the 118 Pediatric Emergency Care Volume 21, Number 2, February 2005 Review Article Division of Emergency Medicine, Cincinnati Children’s Hospital Medical Center, Cincinnati, OH. Drs Reed and Pomerantz have no relationship to or financial interest in any of the products referred to in this manuscript. Address correspondence and reprint requests to Jennifer L. Reed, MD, Cincinnati Children’s Hospital Medical Center, Division of Emergency Medicine, 3333 Burnet Avenue ML2008, Cincinnati, OH 45229. E-mail: Jennifer.Reed@cchmc.org. Copyright n 2005 by Lippincott Williams Wilkins ISSN: 0749-5161/05/2102-0118 Copyright © Lippincott Williams Wilkins. Unauthorized reproduction of this article is prohibited.
  • 2. ears, volar surface of the arm, dorsum of the hand, and peri- neum may often incur deeper burns.8 Superficial or first-degree burns are classically ery- thematous and painful. The most common example is a sunburn from ultraviolet exposure. These burns involve the intact epidermis without blistering (Fig. 1). Because these burns only involve the outer layer of the epidermis, fluid loss is not a problem. Oftentimes, several days after the initial burn, peeling of the skin will begin because of superficial cell death. They heal without scarring in 4 to 5 days.7 Partial-thickness or second-degree burns can be classi- fied as superficial partial-thickness or deep partial-thickness burns. Superficial partial-thickness burns involve partial destruction of the dermis and appear red and painful with blister formation (Figs. 2 and 3). They have a weeping or moist appearance, and healing usually occurs with mini- mal scarring in 7 to 10 days.7 Dark-skinned individuals may lose melanin and develop hypopigmentation upon healing. Deep partial-thickness burns involve greater than 50% of the dermis, destroying the nerve fibers and, consequently, are usually less painful. They have a white pale appearance and usually take 2 to 3 weeks or more to heal. These burns can be difficult to distinguish from third- degree burns. Often, severe scarring will develop, and pa- tients are at risk for contractures. Patients with significant partial-thickness burns are at risk for fluid loss. Surgical consultation is necessary for deep partial-thickness burns because skin grafting is usually necessary for long-term treatment. A good rule of thumb is if a burn does not completely heal in 7 to 10 days, referral to a surgeon for further evaluation is necessary.7 Full-thickness or third-degree burns are the most severe burns (Fig. 4). They appear white, waxy, or leathery and do not bleed or demonstrate any capillary refill (Fig. 5). FIGURE 1. A superficial (first degree) burn. n 2005 Lippincott Williams Wilkins 119 Pediatric Emergency Care Volume 21, Number 2, February 2005 Emergency Management of Pediatric Burns Copyright © Lippincott Williams Wilkins. Unauthorized reproduction of this article is prohibited.
  • 3. Typically, these burns are painless because they have completely destroyed the nerve fibers in the dermis. Patients with these burns are at high risk for infection and severe fluid loss. These burns usually take several weeks to heal, and patients should be referred to a surgeon immediately because of risk of significant scarring upon healing. These burns often require skin grafting. Although the terminology is not commonly used, fourth-degree burns may also be seen. These involve destruc- tion of the underlying structures such as tendons, nerves, muscles, bone, and deep fascia. Most commonly, these are seen with severe electrical injuries, and they require imme- diate surgical consultation and transfer to a burn center. Besides classifying burns as superficial, partial thick- ness, or full thickness, it is also necessary to determine the extent of the burn. This is calculated based on total body surface area (TBSA). The ‘‘rule of 9s’’ is a method to determine TBSA of adult burn victims. The head and each arm from the shoulder to the fingertips are each estimated to be 9% of the TBSA. The anterior trunk, posterior trunk, and each leg from the groin to the toes are each estimated to be 18% of TBSA. The neck and groin are each estimated to be 1% of TBSA. This method is very accurate for adult and teenage burn patients, but it is not accurate in the assessment of pediatric burns because of the difference in body proportions. Alternatively, the Lund and Browder Chart9 may be used to correctly determine percent surface area burns for children of various ages10 (Fig. 6). It has been slightly modified to account for the varying body propor- tions in different ages of children. Another simple method is to measure the percent burn by using the child’s palm which is equal to 1% TBSA burn.9,10 Although 1% TBSA is the accepted measurement, a study demonstrated that the surface of the palm in any age group more closely approx- imates 0.5% TBSA, and a second study determined that the entire hand more accurately represents 0.8% TBSA in any age group.11,12 Clearly, controversy exists as to the most accurate measurement when using a patient’s hand to calculate percent TBSA. Superficial burns should not be included in the surface area burn calculation because they only involve the epidermis and do not put a patient at risk for fluid loss. Whichever method is chosen, one should quickly determine the best estimate of TBSA burn involvement. Initial Assessment The initial approach to burn treatment is similar regardless of the etiology of the burn. Initial assessment should first include airway, breathing, and circulation followed by determination of the depth of burn, TBSA of the burn, and the involved body parts. Burns that are circum- ferential should be noted and carefully observed because they have the potential to require escharotomy. Circumferential burns to the chest may interfere with the patient’s ability to ventilate and need to be addressed if problems arise. In addition, one should obtain historical information in- cluding date, time, cause of burn, general medical history, and immunization status. Significant burns associated with trauma should be evaluated in the standard Advanced Trauma Life Support protocol with a primary survey con- sisting of airway, breathing, and circulation and a secondary survey evaluating for other injuries. One should immediately remove all clothing that is hot, burned, or exposed to chemicals to prevent continued damage to the skin. To decrease burn pain, cool saline- soaked gauze should be applied to the burned skin, but caution needs to be taken with large burns to prevent the complication of hypothermia. Applying ice to the wound or submersing the wound in ice water not only produces hypothermia, but also worsens damage to the skin and should be avoided.13 For large burns, a clean sheet may be used to cover the burns during the initial assessment in the ED. This will decrease pain and provide a superficial covering until each burn can be assessed in detail. Tar prod- uct burns can be challenging. It is necessary to immediately cool the hot tar with cool water. To remove the tar, use a petroleum-based product such as bacitracin which will dis- solve the tar and allow for easier removal.14 In the ED, laboratory data should be obtained for patients with severe burns. This includes a complete blood count, electrolytes with blood urea nitrogen and creatinine, and possibly liver function tests. These laboratory values will serve as a baseline because the patient may experience major fluid shifts and changes in metabolic status. Urinaly- sis is also important to assess the presence of myoglobin that can result from muscle breakdown. Significant myo- globin can lead to renal function impairment. In addition FIGURE 2. A partial-thickness (second degree) burn with significant blister formation. 120 n 2005 Lippincott Williams Wilkins Reed and Pomerantz Pediatric Emergency Care Volume 21, Number 2, February 2005 Copyright © Lippincott Williams Wilkins. Unauthorized reproduction of this article is prohibited.
  • 4. for fire-related burns, a baseline chest radiograph along with carbon monoxide levels should be obtained. House fires, indoor fires, chemical fires, and chemical ingestions may involve respiratory tract burns resulting in inflammation and edema. If a patient’s physical examina- tion findings demonstrate stridor, hoarseness, carbonaceous sputum, perioral or perinasal burns, or singed nose hair, eyebrows, or eyelashes, one should anticipate airway com- promise and promptly secure the patient’s airway with intubation. It is important not to rely on chest radiograph because it is often normal at initial presentation.15 Many advocate the use of bronchoscopy to directly visualize the airway. In the ED setting, it can be just as useful to directly visualize the upper airway with a laryngoscope. If blisters are seen in the oropharynx or oral mucosa, or the mucosa appears dry and erythematous, one should anticipate airway compromise.16 Edema of the airway may not be apparent until 48 hours after a burn. If the physician waits until ob- vious compromise, results may be catastrophic, and intu- bation can be extremely difficult. During intubation, it is important to have various-size endotracheal tubes available and anticipate a narrowed airway. Supraglottic airway injury is usually the result of direct thermal injury, whereas lower airway edema and irritation are a result of chemicals or tox- ins such as smoke inhalation ultimately leading to chemical pneumonitis.17,18 All patients who are victims of house or indoor fires should also be evaluated for carbon monoxide poisoning, and 100% oxygen should be administered. Intravenous access should be obtained immediately in intact skin. If necessary though, catheters may be placed through burned skin, and intraosseous access can be used in young children. Any significant burn injury results in major fluid loss for patients. A burn that is 15% to 20% TBSA will produce hypovolemic shock unless appropri- ately managed with crystalloid fluid replacement. Isotonic saline, most commonly lactated Ringer solution, is recom- mended for resuscitation in the first 24 hours after a sig- nificant burn.14 Large volumes of fluid are needed for proper resuscitation because only 20% to 30% of the isotonic fluid remains in the intravascular space.19 Thus, it is necessary to FIGURE 3. A partial-thickness (second degree) burn. One blister is intact, but most of the blister formation has been debrided. n 2005 Lippincott Williams Wilkins 121 Pediatric Emergency Care Volume 21, Number 2, February 2005 Emergency Management of Pediatric Burns Copyright © Lippincott Williams Wilkins. Unauthorized reproduction of this article is prohibited.
  • 5. appropriately calculate the extent of the burn and fluid- resuscitate each patient individually. The patient should receive fluid resuscitation in the first 24 hours based on the modified Parkland formula: 4 mL Â TBSA Â weight in kilograms. Half of the total fluid should be given in the first 8 hours and the second half over the next 16 hours.4 In children younger than 5 years, calculated maintenance requirements should be added to this total volume to more accurately predict fluid needs.4 Children with burns to less than 15% TBSA usually do not have significant capillary leak and can be managed with isotonic fluid at 1.5 times the maintenance rate while closely following the patient’s urine output and hydration status.14 Children less than 20 kg may develop hypogly- cemia and should have a 5% dextrose–containing solution administered at a maintenance rate along with the calcu- lated volume of fluid necessary for resuscitation.14 Be- cause pediatric burn victims are also prone to hypothermia caused by the loss of integument, initial fluid should be warmed. Treatment Pain control is extremely important in burn manage- ment. Most minor burns can be controlled with nonsteroidal anti-inflammatory medications or acetaminophen. Partial- thickness burns can be extremely painful because of the damage of the epidermis and consequent exposure of the underlying pain fibers. Simply covering the patient with a sheet can dramatically reduce pain by decreasing the environmental exposure. Narcotic pain control though is often necessary in burns of partial thickness or deeper. In children, morphine intravenously is most commonly used. One should be very careful to monitor the patient’s hemo- dynamic stability when administering any narcotic. Each burn should be cleaned with mild soap and water while avoiding extremely cold water or ice. Any skin that is loose at the initial presentation needs to be debrided. Controversy exists as to whether to unroof blisters or keep them intact. One author suggests leaving small blisters intact as long as they do not interfere with wound care or movement over a joint. This author recommends all large FIGURE 4. A full-thickness (third degree) burn in a Caucasian patient. Note the leathery appearance. 122 n 2005 Lippincott Williams Wilkins Reed and Pomerantz Pediatric Emergency Care Volume 21, Number 2, February 2005 Copyright © Lippincott Williams Wilkins. Unauthorized reproduction of this article is prohibited.
  • 6. blisters be unroofed; when in doubt, unroof the blister.7 A second author believes that removing blisters makes care of the burn easier and recommends that only blisters on the soles of the feet or those less than 1 cm should be left intact.20 There is scientific data that show burn fluid may have a negative effect on the burn including suppressing neutrophil and lymphocyte response as well as interfering with fibrinolysis. Burn blister fluid also contains substances that increase inflammatory response which may increase the risk of infection.21 Needle aspiration of a blister should never be performed; bacteria may be introduced into the space and incite infection.22 All partial-thickness and deeper burns are at increased risk for infection. Consequently, attention to chemophylaxis is extremely important. Tetanus status should be evaluated for all significant burns. If it has been greater than 5 years since the last tetanus immunization, the patient needs a booster injection. For those 7 years of age or older, Td (adult diptheria and tetanus toxoid) is indicated. In a child younger than 7 years, DTaP (acellular pertussis vaccine and tetanus toxoid) should be administered unless there is a contraindication to pertussis vaccine; if this is the case, DT (diptheria and tetanus toxoid) should be given. If a patient has not been immunized against tetanus in the past, TIG (tetanus immunoglobulin) in addition to the appropriate tetanus vaccine should be administered intramuscularly at 2 different sites.23 For most burns, there is no role for initial intravenous antibiotic therapy. Using antibiotics initially may increase the risk of colonization of more virulent organisms. There- fore, intravenous antibiotics should be reserved only for obvious secondary wound infections.20,24 Due to an intact epidermis that retains all protec- tive function, superficial burns only require application of moisturizer for treatment. For partial-thickness burns, most authors recommend topical antibiotic ointment such as bacitracin and polymyxin (Polysporin; Warner-Lambert Consumer Healthcare, Freiburg, Germany), or mupirocin topical (Bactroban; GlaxoSmithKline, Research Triangle Park, NC) along with a nonadhesive dressing such as petroleum gauze (Adaptic; Johnson and Johnson, New Brunswick, NJ) or bismuth-impregnated gauze (Xeroform; FIGURE 5. A full-thickness (third degree) burn in an African-American patient. n 2005 Lippincott Williams Wilkins 123 Pediatric Emergency Care Volume 21, Number 2, February 2005 Emergency Management of Pediatric Burns Copyright © Lippincott Williams Wilkins. Unauthorized reproduction of this article is prohibited.
  • 7. Kendall Healthcare, Mansfield, MA) followed by a gauze dressing.7,24 One author recommends that dressings be changed twice a day because of the shorter half-life of ointments.24 This has been the standard in most institutions. Recently, however, some authors have suggested that once- daily dressing changes are adequate for smaller burns and have decreased cost, pain with dressing changes, and need for nursing time. It is still recommended for infection-prone wounds or large extensive burns that dressings be changed twice daily.20 Any exudates or previously applied antibi- otic ointment needs to be completely removed, and new antibiotic ointment needs to be reapplied with each dressing change. For full-thickness burns, creams are most often used. Silver sulfadiazine 1% cream (Silvadene; Aventis Pharma- ceuticals, Inc, Bridgewater, NJ) is commonly used. Appli- cation is painless, and it is bactericidal. Disadvantages include cutaneous hypersensitivity reactions, thrombocyto- penia, and leukopenia.17,24 Silver sulfadiazine (Silvadene; Aventis Pharmaceuticals, Inc) may result in eschar forma- tion, therefore interfering with the assessment of burn depth and healing. It cannot be used in those with sulfa allergies and should be avoided in premature infants and newborns younger than 1 month because of the complication of kernicterus.17,24 Silver nitrate 0.5% solution can also be used for full-thickness burns. Application is painless, and it is effective against a large spectrum of bacteria as well as fungi.14 Disadvantages include that it will readily stain, has poor eschar penetration, and leaches electrolytes from wounds oftentimes leading to hyponatremia in significant burns.14,19 Mafenide acetate 0.5% cream (Sulfamylon; Mylan Laboratories, Inc, Canonsburg, PA) is another widely used topical burn preparation. It is bacteriostatic and has good activity against Pseudomonas aeruginosa. One major advantage is its ability to effectively penetrate eschar; it is widely used on burns of the external ear to prevent infec- tious chondritis.14,20 Disadvantages include the possibility of a cutaneous rash and painful application. Because it is a carbonic anhydrase inhibitor, it may also lead to metabolic acidosis. Instead of using topical creams and ointments, burn wounds may be managed with synthetic occlusive dressings such as Biobrane (Bertek Pharmaceuticals, Inc, Morgantown, WV), human allograft, or pigskin.8 These can be applied to clean partial-thickness wounds that are less than 24 hours old.20 The dressing adheres to the wound until epithelization occurs. The disadvantage is that the dressing can only cover 1% to 2% TBSA burn and is very expensive, prohibiting routine use. For outpatient treatment, one should trim the edges of the dressing as it begins to fall off and continue to change an overlying gauze dressing once daily.6 Infection in burns is a major complication and may often be difficult to recognize. A typical burn exhibits inflammation along with erythema, tenderness, and edema. If these symptoms worsen or are accompanied by fever, malaise, or lymphangitis, infection should be suspected. Infected burns should be treated with intravenous antibiotics and hospital admission. Infection can not only cause sepsis, but it can also lead to deeper burn damage. Most pediatric burns can be managed in an outpatient setting. Superficial burns can be treated with moisturizing lotions, pain control, and reassurance. Partial-thickness burns that are less than 10% of TBSA or full-thickness burns that are less than 2% of TBSA can be managed in the out- patient setting as long as they do not involve the face, hands, feet, or genital region.7 Children with small superficial hand burns may be treated as outpatients as long as there is appropriate follow-up, and the caregiver can adequately care for the child. The complications that arise from hand burns such as infection and contractures necessitate that all other hand wounds be treated on an inpatient basis.25 Most suggest twice-daily dressing changes with antibiotic oint- ment for the first week followed by once-daily dressing FIGURE 6. The modified Lund and Browder Chart used to estimate the TBSA burns of children. Adapted and pub- lished with permission from Shriners Hospitals for Children, Cincinnati, OH. 124 n 2005 Lippincott Williams Wilkins Reed and Pomerantz Pediatric Emergency Care Volume 21, Number 2, February 2005 Copyright © Lippincott Williams Wilkins. Unauthorized reproduction of this article is prohibited.
  • 8. changes for the remainder of the healing process. Initially, frequent visits to the primary care physician must be initi- ated to monitor wound healing and infection. Admission to a general or pediatric hospital is recommended for partial- thickness burns 10% to 20% TBSA and full-thickness burns up to 5% TBSA.17 Transfer to a burn center is recommended for any partial-thickness burn greater than 20% TBSA in any age patient or greater than 10% TBSA in children younger than 10 years. Full-thickness burns greater than 5%; burns to the face, hands, feet, major joints, or genital region; inha- lation injury; significant burns with multiple trauma; electrical burns and lightning injury; chemical burns; cir- cumferential burns; burns caused by child abuse or other cases requiring long-term emotional or rehabilitation sup- port; and burns in patients with preexisting comorbidities should also be treated at a burn hospital.14,26 If the home environment is unsafe or the caregiver is unable to provide appropriate care, the child should be admitted to the hospital regardless of the size of the burn. Prevention Scald burns are the most common etiology for burn injuries resulting in hospitalization in infants, toddlers, and preschoolers, and more than 80% are caused by tap-water scalds.3,27 It has been shown that if the temperature of water is reduced from 558C to 498C (1318F to 1208F), it will take 10 minutes to produce the same burn that at the higher temperature took 30 seconds.28 Therefore, it is recommended that all hot-water heaters be set at less than 498C (1208F). Hot liquid that is heated to 608C (1408F) can cause a burn in 5 seconds, and liquid heated to 718C (1608F) will cause a burn in 1 second. Hot grease can be as hot as 2048C (4008F) and cause immediate severe burns. In children younger than 5 years, the time that it takes to cause these burns may be half of what it would take to cause the same burn in older children and adults.29 Other ways to prevent burn-related injuries include turning pot handles on the stove inward and never leaving hot bowls or cups of liquid unattended. In addition, avoid using a tablecloth when children are present because a child can easily pull the tablecloth and spill hot liquid onto himself. As with any preventable injury, super- vising the child at all times can prevent some of the most serious injuries. Over the past 20 years though, there has been a drop in fire-related deaths. Much of this is because of the com- mitment to educate children and adults about fire preven- tion. Programs such as ‘‘Stop, Drop, and Roll’’ to address burning clothing, the development of flame-retardant sleepwear, and the use of smoke detectors have prevented numerous fire-related injuries and deaths. It is important to have a smoke detector on each level of a house as well as in each bedroom. Parents should have a specific escape plan communicated to their children to be used in the event of a house fire. Matches should be kept out of reach of children and only used with parental presence. Lighters should also be kept away from children. All lighters should be child-resistant, and parents should not allow children to use lighters as a ‘‘toy.’’29 Unfortunately, fireworks are easily accessible to teenagers and older children. Parents should not permit their children to ignite or play with fire- works. Firework displays should be left to professionals. ELECTRICAL BURNS Electrical burns result in over 1500 deaths per year and more than 4000 ED visits.4,30 Electrical burns also account for 2% to 3% of all admissions to hospital burn centers, and that number is slowly increasing.4 Up to one third of electrical burns are household burns, seen mostly among children. Electrical burns result from thermal energy that is produced as an electrical current passes through the body. The amount of thermal energy produced is directly pro- portional to the degree of electric current. The extent of injury is dependent on 6 factors including the resistance of skin, mucosa and internal structures, type of current, frequency of the current, duration of contact, intensity of the current, and the pathway taken by the current.31 Generally, high-voltage injury is more severe than low-voltage injury. The longer the victim is in contact with the electrical source, the more severe the damage. Resistance is inversely proportional to tissue injury. Nerves, muscles, and blood vessels have low electrical resis- tance; therefore, electricity will preferentially flow through these structures and cause severe damage. Conductivity de- creases as one moves from these structures to skin, ten- dons, bone, and fat, in that order.17 Water will additionally decrease resistance, thus resulting in increased internal damage. Consequently, the areas of the body with increased moisture, such as the axilla and antecubital fossa, will sus- tain greater injury. Thick calloused adult skin may exhibit greater damage at the site of entry than more internally as this type of skin impedes current flow. On the other hand, thin wet skin or mucous membranes may exhibit minor thermal injury at the site of contact but more significant internal injury because of lower resistance to flow. The type of current also plays a major role in the extent of tissue injury. Alternating current is more dangerous than direct current because it produces muscle tetany caused by the continual contraction and relaxation of the muscle with each cycle. Typically, alternating current is found in household electricity. A 60-Hz alternating current will change direction 120 times per second.31 This frequency is so rapid that it prevents muscle relaxation and keeps the muscle in a continual refractory state leading to muscle tetany. If tetany occurs in the muscles of the chest wall, suffocation may result. In addition, if a patient is holding n 2005 Lippincott Williams Wilkins 125 Pediatric Emergency Care Volume 21, Number 2, February 2005 Emergency Management of Pediatric Burns Copyright © Lippincott Williams Wilkins. Unauthorized reproduction of this article is prohibited.
  • 9. onto the electrical source, the patient may be unable to let go because of muscle tetany, therefore increasing the dura- tion of contact and extent of the injury. On the other hand, direct current produces muscle contraction only at the beginning and end of the current flow.30 This type of cur- rent is used in the medical setting and is found in lightning strikes. If this current passes through the heart, the patient is at risk for ventricular fibrillation or asystole. Electrical burns can be the result of low-, medium-, or high-voltage current. They may result in burns anywhere from partial-thickness to deep burns involving underlying structures. Low-current injuries (200 V) are often seen in young children from household electrical sources such as putting an electrical cord or plug in their mouth or placing objects into electrical outlets. Medium-current (200–1000 V) and high-current (1000 V) injuries are commonly seen in older children and teenagers with risk-taking behavior, utility workers, and people caught in electrical storms. These would include lightning strikes and high-tension elec- trical injuries that occur when children or teens climb trees, electrical poles, or other utility constructions and contact large electrical wires. The direction the current flows through the body can also predict the severity of injury. Electric current will flow from the point of contact to the ground or another area of the body that completes the electrical circuit. Most com- monly, low current follows a path of least resistance, whereas high current will follow a path directly to the ground. Flow through the body usually takes 1 of 3 pathways: hand to hand, hand to foot, or foot to foot. Hand-to-hand flow is the most dangerous and has a 60% mortality rate because of risk of spinal cord transection at C4 to C8, suffocation caused by tetany of the chest wall muscles, and myocar- dial muscle damage.31 The hand-to-foot pathway carries a 20% mortality rate largely secondary to associated cardiac arrythmias. The foot-to-foot pathway is associated with less than 5% mortality.31 Lightning may injure a child in 4 different ways including direct strike, stride potential, side flash, and flash- over phenomenon. Direct strike is the most dangerous form of a lightning strike; the electrical current passes directly through the child. Alternatively, lightning can strike as a side flash. This occurs when the lightning has already passed through a primary source such as a tree or a person and then travels through the air to a second victim. Stride potential is defined as lightning hitting the ground and entering through one leg of a person while exiting through the other leg. When lightning flows outside of the body, it is referred to as flashover phenomenon. This usually occurs when a patient has on wet clothing and often results in thermal skin burns caused by the ignition of clothing. Initial assessment of the electrical burn victim includes airway, breathing, and circulation. History should be taken to determine the type of current and voltage involved. Placing a patient on cardiac monitors immediately while initiating appropriate fluid resuscitation and pain control is important. A patient who is a victim of a high-voltage injury may be unresponsive, pulseless, and apneic. In addition, these patients often sustain additional injuries after being thrown off of electrical poles or other structures, thereby resulting in traumatic injuries to the abdomen, head, neck, and extremities. Immobilizing the cervical spine and following the basic Advanced Trauma Life Support protocol with primary and secondary survey in this situation is necessary. Oftentimes, a patient may appear to have minor superficial injury when actually there is significant underlying tissue damage. It is important to anticipate severe internal damage until proven otherwise. Clinically, electrical injuries can affect multiple organ systems; thus, it is important to perform a thorough eval- uation. The cutaneous manifestations include flame burns, flash burns, and arc burns as well as mottled cyanotic skin. Flame burns are usually full thickness secondary to the effects of burning clothes. Flash burns are usually partial thickness resulting from lightning injuries. Current can easily jump across flexed joints producing arc burns. In addition, skin of a victim may appear mottled and cyanotic because of autonomic instability, although this is generally transient. The cardiac effects are the most life-threatening and include cardiac dysrhythmias or myocardial damage. Most commonly, ventricular fibrillation is seen in low-voltage alternating-current injuries. Lightning strikes and high- voltage injuries most commonly produce asystole. Tachy- cardia and hypertension may be seen secondary to excessive catecholamine production. Musculoskeletal injury may be extensive in high- voltage injuries. Oftentimes, tissue that initially appears well perfused, later becomes edematous and necrotic. Initial strong pulses do not always predict that blood vessels are intact, and compartment syndrome can develop. Renal failure can occur by either the direct effect of the current on the kidney tissue, hypoxic damage to the kidney, or renal tubular damage secondary to excess myoglobin deposition from extensive muscle damage.31 Electrical injuries produce damage to the central nervous system including the brain, spinal cord, peripheral motor and sensory nerves, and sym- pathetic fibers. Manifestations are classified as immediate, secondary (within 5 days), or delayed (after 5 days). The most common immediate manifestations that may be seen on presentation to the ED include painful sensation, loss of consciousness, respiratory center paralysis, confusion, motor paralysis, visual disturbances, deafness, sensory defi- cits, hemiplegia, quadriparesis, seizures, amnesia, disorien- tation, and intracranial bleeds.31 Although not as common, one should also be aware that pulmonary, abdominal, and ocular manifestations may occur as well. 126 n 2005 Lippincott Williams Wilkins Reed and Pomerantz Pediatric Emergency Care Volume 21, Number 2, February 2005 Copyright © Lippincott Williams Wilkins. Unauthorized reproduction of this article is prohibited.
  • 10. Low-voltage household current can produce injury of the oral cavity and the lips. This most common occurs when a toddler places a household plug or cord in his/her mouth.30 The result is coagulation necrosis and a deep burn to the corner of the mouth. These burns appear as a cen- tral black area surrounded by a gray-white ring surrounded by an erythematous circle. This type of injury is often painless because of the destruction of nerves. Because the commissure of the lip is involved, children need close follow-up to prevent excessive scarring and contractures. The most serious complication is significant bleeding from the labial artery 1 to 2 weeks after the injury when the eschar separates.14,32 These children do not necessarily need to be admitted to the hospital at the time of the injury. Instead, the physician needs to arrange follow-up the next day and educate the parents as to the complications that would necessitate a return visit to the hospital.30 All patients who present with significant electrical current injuries should undergo laboratory testing as would a patient with severe thermal burns. These laboratory tests include a complete blood count, electrolytes, blood urea nitrogen, creatinine, and a urinalysis with urine myoglobin. Patients should also have an electrocardiograph and chest radiograph performed and be placed on cardiac monitors. A physician should consider obtaining creatine phosphoki- nase with cardiac enzymes. With significant trauma asso- ciated with electrical burns, consider head, spine, and abdominal imaging. All patients with high-voltage injuries should be admitted to the hospital. Low-voltage injuries less commonly produce complications. One author found that none of the patients in his study exposed to low-voltage current developed cardiac arrhythmias despite laboratory test– confirmed muscle damage (elevated creatine phosphokinase) in a few patients.30 Thus, the recommendation is to observe patients in the ED for 4 hours on cardiac monitors. If the patient does not have cardiac arrhythmias, did not lose consciousness, and has only minor wounds, outpatient man- agement can be safely arranged.30 Management of patients with medium- and high- voltage injuries (200 V) includes observing children on a cardiac monitor for 24 to 72 hours because of the increased risk of cardiac arrythmias.14 In patients with significant muscle damage, pigmented urine is often present and puts a patient at risk for the development of renal failure. Foley catheters should be placed in these at-risk patients. Increasing the patient’s urine output to a goal of 2 mL/kg/h and alkalinizing the urine with bicarbonate will help prevent this complication.14 Patients may develop hyperkalemia caused by extensive muscle tissue damage; consequently, potassium levels need to monitored closely. Household electrical cords are responsible for the majority of electrocutions in children younger than 12 years, yet there are no federal safety standards in effect to pre- vent household electrical injuries. Thus, it is important for parents to keep all electrical outlets covered with plastic plugs and keep children away from all electrical cords. This will prevent children from placing objects into the electri- cal outlets or electrical cords into their mouths resulting in electrocution. Older children and teenagers should be counseled on the dangers of climbing electrical structures and be taught to avoid other risk-taking behaviors. To prevent lightning strikes, children should be taught to stay indoors during storms, exit the water immediately, and avoid contact with metal objects. CHEMICAL BURNS There are between 25,000 and 100,000 chemical burns reported in the United States each year, and collectively, they have a morbidity and mortality rate of less than 1%. Children and adults are reported to have similar rates of exposure.33 Over 25,000 different chemicals can produce either acid or alkali burns.4 The burn is usually a result of a direct chemical injury, but there also may be components of a thermal burn caused by an exothermic reaction.17 Acid burns result in coagulation necrosis, which usually limits the depth and penetration of the burn. Common household products that contain acids include drain cleaners (sulfuric acid or hydrochloric acid), toilet cleaners (hydrochloric acid or phosphoric acid), and car batteries (sulfuric acid).34 Ingestion of acids will lead to gastric injury and often leaves patients with strictures of the pylori. In severe cases, involvement of the esophagus, stomach, and small intestine can be seen. Alkalis produce liquefactive necrosis, thus causing deeper penetration and a more significant burn. Alkalis include lye (sodium hydroxide), cement (calcium, potas- sium, and sodium hydroxide), fertilizers (anhydrous am- monia), oven and drain cleaners (sodium or potassium hydroxide), paint strippers (sodium hydroxide), and various detergents.17,34 Ingesting alkalis can lead to significant gastrointestinal injury and perforation and, most commonly, esophageal strictures. Regardless of the type of chemical burn, it is neces- sary initially to remove all clothing and perform copious irrigation for at least 30 minutes to prevent further injury to the tissue. Do not attempt to neutralize chemical burns; this can lead to further thermal burn injury because of the exothermic reaction. Measuring the resultant pH will determine whether the tissue has been adequately irrigated. The pH should be measured approximately 15 minutes after irrigation to allow chemicals from deeper tissues to diffuse to the surface. Testing the pH too early may lead to false- negative results.34 Because alkali substances are less water- soluble, these burns often take longer to neutralize. Any chemical injury to the eye should be treated in the same n 2005 Lippincott Williams Wilkins 127 Pediatric Emergency Care Volume 21, Number 2, February 2005 Emergency Management of Pediatric Burns Copyright © Lippincott Williams Wilkins. Unauthorized reproduction of this article is prohibited.
  • 11. way followed by consultation with an ophthalmologist. In cases of ingestion, do not induce emesis. Instead, treatment with ingestion of milk or water is recommended followed by diagnostic endoscopy. Activated charcoal is contrain- dicated; it does not bind to acid or alkali and will complicate endoscopy.34 Hydrofluoric acid burn treatment is an exception to the typical burn treatment regimen. It is extremely corrosive and causes liquefactive necrosis and bone corrosion. Cal- cium gluconate gel should be massaged into the affected area for at least 30 minutes.34 If the pain is persistent or the hydrofluoric acid strength is greater than 20%, 10% calcium gluconate should be injected into the tissues. In severe hydrofluoric acid burns, calcium ions may be trapped as calcium fluoride, and hypocalcemia will occur which may lead to ventricular arrythmias. Patients with hydrofluoric acid burns should be placed on a cardiac monitor, and cal- cium levels should be monitored closely with intravenous calcium replacement as indicated.14 To prevent chemical injuries, all caustic substances most commonly used to clean bathrooms or kitchens should be kept out of reach of children. The cabinets where these products are stored should have child safety locks. Chem- icals stored in a garage or shed should also be kept out of reach of children. All chemicals should all be kept in their original containers or bottles and should be used appropri- ately with the proper ventilation and protective equipment. In addition, care should be taken while using these products to keep children away. CHILD ABUSE OR NONACCIDENTAL BURNS Between 10% and 20% of burns in children are inflicted.4 It is estimated that between 16% and 20% of patients with burns resulting in hospital admission are victims of abuse, and most of these types of burns are more extensive and severe.35 Most inflicted burns are scald or contact burns and oftentimes have recognizable patterns such as a triangular shape from the tip of an iron or linear parallel lines from a radiator. Toddlers who are submersed in hot water will present with scald burns to the buttocks, thighs, and feet. The most common areas involved in scald burns of abuse include the buttocks and perineum. These areas are rarely involved in unintentional scald burns particularly in the nonambulatory child. Severe burns to the hands and feet in a glove or stockinglike pattern, often symmetric and with a distinct line of demarcation, are classic for nonaccidental immersion.36 Alternatively, a child may be submersed more than once and may not have specific lines of demarcation. Oftentimes the palms, soles, or buttocks are spared because of contact with the cool floor of the water-containing object such as the floor of a bathtub or sink. When the buttocks are spared, it is described as a ‘‘doughnut hole’’ appearance.27,37 The flexion creases in the anterior hip, popliteal fossa, antecubital space, and lower abdomen may be spared when the trunk is in flexion.27,37 This is seen when a child is in a defensive position with arms, legs, and trunk flexed while being forced into hot liquid against his/her will. Splash burns can be seen when a hot liquid is thrown at the child. This is less common and will leave an ‘‘arrowhead’’ appearance as the hot liquid runs down the patient’s body because of gravitational force.37 Cigarette burns appear as deep, small, circular burns. Another less common form of child abuse is burning through microwave ovens. A small child is placed in a microwave oven, and significant burns are seen on the area of the skin closest to the area that emits microwaves. In addition stun guns burns may be seen in abuse cases. Because stun guns are easily obtainable by the public, partial-thickness electrical burns can result. When a child presents with these types of burns, physicians need to have a high suspicion of abuse, espe- cially when the history of the injury does not match the pattern of the burn or there is no witness to the injury. Historically, one should be suspicious of abuse if there is significant delay in the time to presentation of an injury, if the parents seem uninterested or do not want to manage the child’s postburn care, if the child is brought to the hospital by someone other than the parent, or if there is documentation of previous unexplained injuries or neglect.36 On physical examination, one should be concerned about the potential of abuse if the mechanism of injury does not match the child’s developmental age, burns are older than the history suggests, other injuries are seen such as fractures and bruising, burns are symmetric or localized to the buttock and perineum, or the child is passive to invasive painful procedures.36 The burn itself should be treated appropri- ately, and social services should be consulted. Reporting suspected abuse to the appropriate child protective authority is mandated by law for all physicians suspecting abuse. Regardless of the size or severity of the burn, if there is concern regarding the safety of the child, the child should be admitted to the hospital until the case has been properly investigated. Prevention of child abuse can be difficult. It is im- portant for the physician to report any suspicion of abuse at the first sign to prevent further fatal abuse of the child. Reporting at the initial suspicion may also help to prevent abuse in siblings by having them removed from the abusive home and placed in appropriate foster care. CONCLUSION Pediatric burns result in significant morbidity and mortality. Emergency department physicians are usually one of the first healthcare providers to encounter these injuries. Thus, it is imperative that physicians receive education on 128 n 2005 Lippincott Williams Wilkins Reed and Pomerantz Pediatric Emergency Care Volume 21, Number 2, February 2005 Copyright © Lippincott Williams Wilkins. Unauthorized reproduction of this article is prohibited.
  • 12. the proper diagnosis and management of all burn injuries to prevent further complications in patients. In addition, one should be proficient in recognizing signs of burn abuse. Incorporating burn prevention strategies into physician’s daily routine is necessary to limit future burn-related injuries and deaths. ACKNOWLEDGMENT The authors thank Donna Mertens, RN, for her assistance in obtaining the photographs included in this manuscript. REFERENCES 1. Center for Disease Control and Prevention. Ten leading causes of injury death by age group—2001 highlighting unintentional injury deaths. Available at: ftp://ftp.cdc.gov/pub/ncipc/10LC-2001/PDF/101c- unintentional.pdf. Accessed March 7, 2004. 2. Center for Disease Control and Prevention, National Center for Injury Prevention and Control, WISQUARS. Overall fire/burn nonfatal injuries and rates per 100,000 and fire/burn deaths and rates per 100,000. Available at: http://webapp.cdc.gov/cgi-bin/broker.exe. Accessed June 14, 2004. 3. McLoughlin E, McGuire A. The causes, cost and prevention of child- hood burn injuries. Am J Dis Child. June 1990;144(6):677–683. 4. Joffe MD. Burns. In: Fleisher GR, Ludwig S, eds. Textbook of Pediatric Emergency Medicine. 4th ed. Philadelphia: Lippincott Williams Wilkins; 2000:1427–1434. 5. Centers for Disease Control and Prevention, National Center for Injury Prevention and Control. Fire and burn injuries fact sheet. Available at: http://www.cdc.gov/ncipc/factsheets/fire.htm. Accessed February 3, 2004. 6. Stewart C. Emergency care of pediatric burns. Emerg Med Rep. October 2000;5(10):101–111. 7. Passaretti D, Billmire DA. Management of pediatric burns. J Craniofac Surg. September 2003;14(5):713–718. 8. Schonfeld N. Outpatient management of burns in children. Pediatr Emerg Care. September 1990;6(3):249–253. 9. Lund C, Browder N. The estimation of areas of burns. Surg Gynecol Obstet. 1944;79:352–358. 10. Merten DM, Jenkins ME, Warden GD. Outpatient burn management. Nurs Clin North Am. June 1997;32(2):343–364. 11. Sheridan RL, Retras L, Basha G, et al. Planimetry study of the percent of body surface represented by the hand and palm: sizing irregular burns is more accurately done with the palm. J Burn Care Rehabil. November–December 1995;16(6):605–606. 12. Perry RJ, Moore CA, Morgan BD, et al. Determining the approximate area of a burn: an inconsistency investigated and re-evaluated. Br J Med. May 25, 1996;312(7042):1338. 13. Purdue GF, Layton TR, Copeland CE. Cold injury complicating burn therapy. J Trauma. February 1985;25(2):167–168. 14. Sheridan RL. Burns. Crit Care Med. November 2002;30(11 suppl): S500–S514. 15. Sheridan RL. Airway management and respiratory care of the burn patient. Int Anesthesiol Clin. Summer 2000;38(3):129–145. 16. Heimbach DM, Waeckerle JF. Inhalation injuries. Ann Emerg Med. December 1988;17(12):1316–1320. 17. Smith ML. Pediatric burns: management of thermal, electrical, and chemical burns and burn-like dermatologic conditions. Pediatr Ann. June 2000;29(6):367–378. 18. Cortiella J, Marvin JA. Management of the pediatric burn patient. Nurs Clin North Am. June 1997;32(2):311–329. 19. Monafo WW. Initial management of burns. N Engl J Med. November 21, 1996;335(21):1581–1586. 20. Kao CC, Garner WL. Acute burns. Plast Reconstr Surg. June 2000; 101(7):2482–2493. 21. Rockwell WB, Ehrlich HP. Should burn blister fluid be evacuated? J Burn Care Rehabil. January to February 1990;11(1):93–95. 22. Clayton MC, Solem LD. No ice, no butter. Advice on management of burns for primary care physicians. Postgrad Med. May 1995;97(5): 151–155, 159–160, 165. 23. American Academy of Pediatrics. Tetanus. In: Pickering LD, eds. Red book: 2003 Report of the Committee on Infectious Diseases. 26th ed.Elk Grove Village, IL: American Academy of Pediatrics; 2003: 611–616. 24. Palmieri T, Greenhalgh D. Topical treatment of pediatric patients with burns: a practical guide. Am J Clin Dermatol. 2002;3(8):529–534. 25. Sheridan RL, Baryza MJ, Pessina MA, et al. Acute hand burns in children: management and long-term outcome based on a 10-year experience with 698 injured hands. Ann Surg. April 1999;229(4): 558–564. 26. Smith S, Duncan M, Mobley J, et al. Emergency room management of minor burn injuries: a quality management evaluation. J Burn Care Rehabil. January–February 1977;18(1 pt 1):76–80. 27. Stratman E, Melski J. Scald abuse. Arch Dermatol. March 2002; 138(3):318–320. 28. Moritz AR, Henriques FC. Studies of thermal injury: II. The relative importance of time and surface temperature in the causation of cuta- neous burns. Am J Pathol. 1947;23:695–720. 29. Shriners Hospitals for Children. Pamphlet: Burn Prevention Tips. Tampa, FL: Shriners Hospitals for Children; September 1999:1–24. 30. Zubair M, Besner GE. Pediatric electrical burns: management strategies. Burns. August 1997;23(5):413–420. 31. Baum CR. Environmental injuries. In: Fleisher GR, Ludwig S, eds. Textbook of Pediatric Emergency Medicine. 4th ed. Philadelphia: Lippincott Williams Wilkins; 2000:959–963. 32. Jain S, Bandi V. Electrical and lightning injuries. Crit Care Clin. April 1999;15(2):319–331. 33. Hostetler MA. Burns, Chemical [last updated January 20, 2004]. Avail- able at: http://www.emedicine.com/ped/topic2735.htm. Accessed June 14, 2004. 34. Bates N. Acid and alkali injury. Emerg Nurse. December 1999–January 2000;7(8):21–26. 35. Deitch E, Staats M. Child abuse through burning. J Burn Care Rehabil. 1982;3:89–94. 36. Andronicus M, Oates RK, Peat J, et al. Non-accidental burns in children. Burns. September 1998;24(6):552–558. 37. Peck MD, Priolo-Kapel D. Child abuse by burning: a review of the literature and an algorithm for medical investigations. J Trauma. November 2002;53(5):1013–1022. n 2005 Lippincott Williams Wilkins 129 Pediatric Emergency Care Volume 21, Number 2, February 2005 Emergency Management of Pediatric Burns Copyright © Lippincott Williams Wilkins. Unauthorized reproduction of this article is prohibited.