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Senile cataract




    Presented by:- Dr Nikhil Bansal
                   J.N.M.C.,Wardha
General Concepts
 Almost universal in people over
  70 years of age.
 Occurs equally in men and
  women.
 Considerable genetic influence.
 Average age of onset is earlier
  in tropical countries.
Types of Senile cataract

1. Cortical cataract
2. Nuclear or sclerotic cataract
Cortical cataract
 Etio-pathogenesis:- Hydration followed
  by coagulation of lens proteins in the
  cortex.
  Features:- Occurs in following stages :-
1. Stage of lamellar separation
2. Stage of incipient cataract
3. Immature senile cataract
4. Mature senile cataract
5. Hypermature senile cataract
Stage of lamellar separation

 Demarcation of cortical fibres owing
  to their separation by fluid.
 Demonstrated by Slit-lamp
  examination only.
 Characteristic grey appearance of
  pupil.
 Changes are reversible.
Stage of Incipient cataract

 Wedge shaped opacities with clear
  areas in between( Lens striae).
 Most common in periphery and lower
  nasal quadrant.
 Only seen in dilated pupil.
 Irregularities in refraction, visual
  deterioration and polyopia.
Immature senile cataract

 Opacification becomes more diffuse
  and irregular.
 Lens is swollen.
 Iris shadow still visible.
 Anterior chamber becomes shallow.
Mature senile cataract

   Complete opacification.
   Whole cortex is involved .
   Lens appears pearly white in colour.
   Also known as ripe cataract.
Hyper-mature senile cataract

 Cortex is disintegrated and
  transformed into pultaceous material.

    Usually occurs in two forms:-
1. Morgagnian hyper–mature cataract
2. Sclerotic hyper–mature cataract
Morgagnian hyper–mature
cataract
 Complete cortex is liquefied and
  appears milky white in colour.
 Nucleus settles at the bottom.
 Calcium deposits may also be seen
  on the lens capsule.
Sclerotic hyper–mature cataract

 Disintegrated cortex.
 Shrunken lens.
 Wrinkled anterior capsule .
 Dense white capsular cataract in
  pupillary   area.
 Deep Anterior-Chamber.
 Tremulous Iris .
Nuclear or Sclerotic cataract
Etio-pathogenesis:- Intensification of age related
   degenerative changes associated with
   dehydration of and compaction of nucleus.
  Features:-
 Hard cataract is formed.
 Significant increase in water insoluble protein.
 Lens becomes in-elastic and looses power of
   accommodation.
 Changes begin centrally and slowly spread to
   periphery.
 Deposition of pigments gives characteristic
   colour to nucleus.
Nuclear or Sclerotic cataract
Other types of Nuclear or Sclerotic
cataract




cataracta brunescens                     cataracta nigra




                       Cataracta rubra
Complications of senile cataract
1. Phacoanaphylactic uveitis
2. Lens induced glaucoma
3. Subluxation or dislocation of lens
References




   Parson’s diseases of eye
   Comprehensive ophthalmology:-
          Khurana and Khurana
               wikipedia.org
THANK-YOU

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Senile Cataract

  • 1. Senile cataract  Presented by:- Dr Nikhil Bansal J.N.M.C.,Wardha
  • 2. General Concepts  Almost universal in people over 70 years of age.  Occurs equally in men and women.  Considerable genetic influence.  Average age of onset is earlier in tropical countries.
  • 3. Types of Senile cataract 1. Cortical cataract 2. Nuclear or sclerotic cataract
  • 4. Cortical cataract Etio-pathogenesis:- Hydration followed by coagulation of lens proteins in the cortex. Features:- Occurs in following stages :- 1. Stage of lamellar separation 2. Stage of incipient cataract 3. Immature senile cataract 4. Mature senile cataract 5. Hypermature senile cataract
  • 5. Stage of lamellar separation  Demarcation of cortical fibres owing to their separation by fluid.  Demonstrated by Slit-lamp examination only.  Characteristic grey appearance of pupil.  Changes are reversible.
  • 6. Stage of Incipient cataract  Wedge shaped opacities with clear areas in between( Lens striae).  Most common in periphery and lower nasal quadrant.  Only seen in dilated pupil.  Irregularities in refraction, visual deterioration and polyopia.
  • 7. Immature senile cataract  Opacification becomes more diffuse and irregular.  Lens is swollen.  Iris shadow still visible.  Anterior chamber becomes shallow.
  • 8. Mature senile cataract  Complete opacification.  Whole cortex is involved .  Lens appears pearly white in colour.  Also known as ripe cataract.
  • 9. Hyper-mature senile cataract  Cortex is disintegrated and transformed into pultaceous material. Usually occurs in two forms:- 1. Morgagnian hyper–mature cataract 2. Sclerotic hyper–mature cataract
  • 10. Morgagnian hyper–mature cataract  Complete cortex is liquefied and appears milky white in colour.  Nucleus settles at the bottom.  Calcium deposits may also be seen on the lens capsule.
  • 11. Sclerotic hyper–mature cataract  Disintegrated cortex.  Shrunken lens.  Wrinkled anterior capsule .  Dense white capsular cataract in pupillary area.  Deep Anterior-Chamber.  Tremulous Iris .
  • 12. Nuclear or Sclerotic cataract Etio-pathogenesis:- Intensification of age related degenerative changes associated with dehydration of and compaction of nucleus. Features:-  Hard cataract is formed.  Significant increase in water insoluble protein.  Lens becomes in-elastic and looses power of accommodation.  Changes begin centrally and slowly spread to periphery.  Deposition of pigments gives characteristic colour to nucleus.
  • 14. Other types of Nuclear or Sclerotic cataract cataracta brunescens cataracta nigra Cataracta rubra
  • 15. Complications of senile cataract 1. Phacoanaphylactic uveitis 2. Lens induced glaucoma 3. Subluxation or dislocation of lens
  • 16. References  Parson’s diseases of eye  Comprehensive ophthalmology:- Khurana and Khurana  wikipedia.org