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D E P T ; P U B LI C H E A L T H 4 T H Y R
C O U R SE T I T LE ; D E N TI ST R Y
C R E D I T H O U R S ; T H R E E ( 0 1 )
B Y ; D R . N H I A L W A N L O L ( M D)
BARO Gambella College
Module I
īļIntroduction to Dentistry
ī‚— Definition of dentistry
ī‚— Anatomy and physiology oral cavity
ī‚— Pathology of oral cavity
ī‚— Dental Caries
Introduction to Dentistry
ī‚— Definition;
Dentistry is a study of oral cavity
e.g bones, teeth and soft tissues
ī‚§ Dentistry is the science and art of preventing,
diagnosing, and treating, diseases, injuries and
malformations of the teeth, jaws and mouth and of
replacing lost or absent teeth and associated
structures.
Anatomy and physiology
ī‚— Oral cavity consist of;
ī‚— Bones..Upper–Jaw
Lower-Jaw
ī‚§ Teeth(3parts)
Crown
Neck
Root
ī‚§ Soft tissues
ī‚§ Tongue, gum, vocal mucosa, and dentine
ī‚§ Glands..salivary and lymph node
Anatomy of oral cavity
Parts of the Teeth
6
A tooth has a crown, neck, and root
īƒ˜The crown: the visible part of teeth that projects
from the gingiva
īƒ˜The neck is between the crown and the root
īƒ˜The root is the part fixed in the tooth socket, which
is variant in number
7
Layers of Teeth
8
īƒ˜Enamel: the outer most layer of teeth in the crown
īƒ˜Cementum: the outer most layer of teeth in the root
īƒ˜Dentin: the middle layer of teeth, forms the main
density of teeth
īƒ˜The pulp cavity: the central part of teeth, contains
connective tissue, blood vessels, and nerve
Sensory innervation and Blood Supply of the jaw
9
īƒ˜ Superior and inferior alveolar arteries/ veins ; branch of maxillary
artery supply tooth
īƒ˜ Upper jaw= superior alveolar nerves
īƒ˜ Lower jaw= inferior alveolar nerves; both make dental plexus
ī‚Ą Inferior alveolar nerve: all mandibular teeth, buccal soft tissues
of premolar, canine and incisors
ī‚Ą Lingual nerve: all lingual soft tissues of all teeth
ī‚Ą Long buccal nerve: buccal soft tissue of all molars
ī‚Ą Anterior superior alveolar nerve: maxillary incisors and
canine, buccal soft tissues of incisors and canines
ī‚Ą Middle superior alveolar nerve: maxillary premolar and
maxillary bone roots first molar, buccal soft tissue of premolar
Cont.â€Ļ
10
ī‚— Post. Sup. alveolar nerve: maxillary molars except
a portion of first molar, buccal soft tissue of molars
ī‚— Greater palatine nerve: lingual soft tissues of
molars and premolars
ī‚— Nasopalatine nerve: lingual soft tissue of incisors &
canines
ī‚— Mandibular premolar region, buccal soft tissue
innervated primarily by mental branch of IAN and
also by terminal branch’s of long buccal nerve
11
Tongue
12
īƒ˜Muscular organ
īƒ˜Functions in:
ī‚ĄChewing
ī‚ĄSwallowing
ī‚ĄSpeech
īƒ˜ Site of sensory reception:
īƒˇTaste
īƒˇTouch
īƒˇPain / Temperature
Salivary Glands
13
īƒ˜ Major and Minor Salivary Glands
ī‚Ą Three pairs of major Salivary Glands
īƒ˜ Parotid – below and anterior to the ear
īƒ˜ Submandibular – below the mandible
īƒ˜ Sublingual – anterior floor of the mouth
īƒ˜ Orifices / ducts:
īļ Stensen’s duct – parotid
īļ Wharton’s duct – submandibular
īļ Numerous small ducts of Sublingual
glands
īƒ˜ Over 1000 minor glands
īƒˇ Buccal, palatal, lingual
The Role of Saliva
14
īƒ˜ Physical protection provides a cleansing effect.
īƒ˜ Chemical protection contains calcium, phosphate, and fluoride. It
keeps calcium there ready to be used during remineralization.
īƒ˜ It includes buffers, bicarbonate, phosphate, and small proteins that
neutralize the acids after we ingest fermentable carbohydrates.
īƒ˜ Antibacterial substances in saliva work against the bacteria; like
lactoferrin, lysozyme, lactoperoxidase, beta-lysin, and
immunoglobulins
īƒ˜ Maintenance of homeostasis on dental surfaces:
ī‚Ą Dissolves and dilutes metabolites
ī‚Ą Maintains proper pH balance
ī‚Ą Reduces plaque
Physiology
īļFunction of oral cavity
ī‚— Digestion...mastication/chewing
ī‚— Alternate airway....breathing
ī‚— Articulation of speech...speaking
ī‚— Anatomical support
ī‚— Psychology...cosmetic
Pathology
ī‚— Pathology of oral cavity is defined as disease
affecting bones, and connective tissues of oral cavity
ī‚— Dental caries is a bacterial infection of the teeth
and soft tissues.
ī‚— Oral hygiene; caring for teeth, gum and other soft
tissues
ī‚— Connective tissue include; gum, and vocal mucosa
Dental Caries
ī‚— Bacterial infection of the teeth and connective
tissues.
ī‚— Pathophysiology of dental carier
ī‚— Food particle + bacteria= form plaque--- plaque
form dental carrier
ī‚— Dental plaque is colourless, soft, sticky material
disposed on the enamel.
Transmission of Caries Causing Bacteria
18
ī‚— Mutans streptococci are transmitted through saliva, most
frequently the mother’s, to the infant.
ī‚— When mothers have high counts of mutans streptococci in their
mouths, the babies also have high counts of the same bacteria
in their mouth.
Dental Plaque
ī‚— Dental plaque is a colorless, soft, sticky coating that adheres
to the teeth.
ī‚— Plaque remains attached to the tooth despite movements of the
tongue, water rinsing, water spray, or less than thorough
brushing.
ī‚— Formation of plaque on a tooth concentrates millions of
microorganisms on that tooth
Dental Caries: A Bacterial Infection
19
īƒ˜ There are two specific groups of bacteria found in the mouth that
are responsible for dental caries:
īƒŧ Mutans streptococci (Streptococcus mutans)
īƒŧ Lactobacilli
ī‚— They are found in relatively large numbers
in the dental plaque.
ī‚— The presence of lactobacilli in the mouth indicates a high sugar
intake.
The following mechanisms contribute to tooth decay
20
īƒ˜ Plaque bacteria that ferment dietary sucrose produce acids that
lower the pH on the tooth surface, promoting demineralization and
eventually, tooth decay.
īƒ˜ Plaque bacteria, such as S. mutans can also utilize sucrose to produce
extracellular polysaccharides, known as glucans, which are adhesive
polymers that enable mutans streptococci to avidly stick to the tooth
surface, thereby causing decay in the underlying structures.
Dental caries
21
The Caries Process
22
īƒ˜For caries to develop, three factors must occur at the
same time:
ī‚ĄA susceptible tooth
ī‚ĄDiet rich in fermentable carbohydrates
ī‚ĄSpecific bacteria (regardless of other factors,
caries cannot occur without bacteria)
Dental caries
23
The earliest sign of decay is decalcification
24
Root Caries
25
ī‚— Root caries is becoming more prevalent and is a concern
for the elderly population who often have gingival
recession exposing the root surfaces.
ī‚— Carious lesions form more quickly on root surfaces than
coronal caries because the cementum on the root
surface is softer than enamel and dentin.
ī‚— Like coronal caries, root caries has periods of
demineralization and remineralization.
Root caries
26
Erosion
27
Cause: Bruxism (Involuntary clenching
teeth; esp. during sleep)
Loss by wear of surface of tooth or
restoration caused by tooth to tooth
contact during mastication or
parafunction
Loss by wear of dental tissue caused by
abrasion by foreign substance (e.g.,
toothbrush, dentifrice)
Progressive loss of hard dental tissue by
chemical processes not involving bacterial
action Cause: GERD
Cause: Over vigorous brushing
Abrasion
Attrition
Diagnosis of Dental Caries and Non Dental Caries
28
īƒ˜Radiographs
īƒ˜Visual
īƒ˜Laser caries detector
Laser Caries Detector
īƒ˜The laser caries detector is used to
diagnose caries and reveal bacterial
activity under the enamel surface.
īƒ˜Carious tooth structure is less dense and
gives off a higher reading than non- carious
tooth structure.
Visual and radiographic appearance of seemingly intact molar
Cross section of molar showing decay
Treatments
31
īƒ˜ Elimination of risk factors
īƒ˜ Scaling, root planning & curettage
īƒ˜ Antibiotic therapy (metronidazole or tetracycline)
īƒ˜ Anti-pain (NSAIDs)
Filling
īƒ˜ Amalgam (a mixture of mercury and other metals (solid/liquid)
used for filling holes in teeth)
īƒ˜ Composite a solid material which is composed of 2/more
substances having d/t physical ch/tcs and in which each substance
retains its identity while contributing desirable properties to the
whole.
Methods of Caries Intervention
32
īƒ˜ Fluoride: A variety of types are available to strengthen the tooth against
solubility to acid.
īƒ˜ Antibacterial therapy: Products such as Chlorhexidine rinses are
effective.
īƒ˜ Salivary flow can be increased by chewing sugarless gum, for example,
those with a non-sugar sweetener such as xylitol.
Preventive measures against
caries
A. Fluoride rinse
B. Chlorhexidine rinse
C. Xylitol gum.
D. Sealants- thin, plastic material
used to cover tooth surfaces. It
protects enamel from attack by
bacterial acids.
Complications of dental carrier
I. Local
ī‚— Abscess..pus collection
ī‚— Tooth loss
ī‚— Pericorinitis
ī‚— Chronic tooth pain
ī‚— Bacterial infection of the floor of the mouth
ī‚— e.g Ludwig’s angina
Dental Carrier cont.............
II. Systemic infection
ī‚— Sepsis
ī‚— Septic arthritis
ī‚— Osteomyelitis
ī‚— Septic shock
B Y D R . N H I A L W . ( M D )
FIELDS OF DENTISTRY
Outline
ī‚— Prosthodontic
ī‚— Endodontic
ī‚— Periodontics
ī‚— Orthodontic
ī‚— Oral and maxillofacial surgery ( OMFS)
ī‚— Operative dentistry
Fields of studies
īƒŧProsthodontic
īƒŧEndodontic
īƒŧPeriodontics
īƒŧOrthodontic
īƒŧOral and maxillofacial surgery ( OMFS)
īƒŧOperative dentistry
37
1. Prosthodontics
The branch of dentistry that deals with the
replacement of missing teeth and related mouth
or jaw structures by bridges, dentures, or other
artificial devices.
38
2. Endodontic
It deals with the tooth pulp & tissues
surrounding the root of tooth.
39
39
3. Operative Dentistry (Restorative dentistry)
Concerned with restoration of parts of the teeth
that are defective as a result of disease, trauma,
or abnormal development to a state of normal
function, health, and aesthetics.
40
4.Periodontic
The specialty of dentistry that studies
supporting structures of teeth, diseases, and
conditions that affect them.
41
5. Oral and maxillofacial surgery
The dental specialty concerned with the
diagnosis and treatment of diseases affecting
the mouth, jaws and orofacial structures.
42
42
6. Orthodontics
The specialty of dentistry that is concerned with
the study and treatment of malocclusions, which
may be a result of tooth irregularity,
disproportionate jaw relationships, or both.
43
A N A T O M Y A N D N O M E N C L A T U R E O F T E E T H
Module II
Outline
ī‚— Classification
ī‚— Anatomy of tooth
ī‚— Parts of the tooth
ī‚— Layers of the tooth
ī‚— Innervation and blood supply of the teeth
ī‚— Nomenclatures system of the teeth
Objective
īļBy the this session, students will be able to;
īƒ˜Classify teeth
īƒ˜Describes normal anatomy of teeth
īƒ˜Explain parts of the tooth
īƒ˜Describes layers of the teeth
īƒ˜List nerves and blood vessels of the teeth
īƒ˜Learn different nomenclature systems of the teeth
Teeth
īƒ˜ The teeth are set in the tooth sockets and used in mastication
and speech
īļ Two class of teeth:
a) Primary/deciduous
b) Permanent
â€ĸ Children have 20 deciduous teeth; adults have 32 permanent
teeth
īƒ˜ The 20 deciduous teeth consist of
â€ĸ 2 incisor, 1 canine, and 2 molar teeth on each side of the
upper and lower jaws
â€ĸ These teeth are replaced by the incisor, canine, and premolar
teeth of the permanent teeth 47
48
Parts of the Teeth
A tooth has a crown, neck, and root
īƒ˜The crown: the visible part of teeth that
projects from the gingiva
īƒ˜The neck is between the crown and the root
īƒ˜The root is the part fixed in the tooth socket,
which is variant in number
49
50
Layers of Teeth
īƒ˜ Enamel: the outer most layer of teeth in the crown
īƒ˜ Cementum: the outer most layer of teeth in the root
īƒ˜ Dentin: the middle layer of teeth, forms the main
density of teeth
īƒ˜ The pulp cavity: the central part of teeth, contains
connective tissue, blood vessels, and nerve
51
Sensory innervation and Blood Supply of the jaw
īƒ˜ Superior and inferior alveolar arteries/ veins ; branch of maxillary
artery supply tooth
īƒ˜ Upper jaw= superior alveolar nerves
īƒ˜ Lower jaw= inferior alveolar nerves; both make dental plexus
– Inferior alveolar nerve: all mandibular teeth, buccal soft
tissues of premolar, canine and incisors
– Lingual nerve: all lingual soft tissues of all teeth
– Long buccal nerve: buccal soft tissue of all molars
– Anterior superior alveolar nerve: maxillary incisors and
canine, buccal soft tissues of incisors and canines
– Middle superior alveolar nerve: maxillary premolar and
maxillary bone roots first molar, buccal soft tissue of premolar
52
Cont.â€Ļ
â€ĸ Post. Sup. alveolar nerve: maxillary molars except
a portion of first molar, buccal soft tissue of molars
â€ĸ Greater palatine nerve: lingual soft tissues of
molars and premolars
â€ĸ Nasopalatine nerve: lingual soft tissue of incisors &
canines
â€ĸ Mandibular premolar region, buccal soft tissue
innervated primarily by mental branch of inferior
alveolar nerves and also by terminal branch of
long buccal nerve
53
54
Nomenclature of tooth
īƒ˜ Nomenclature is the first step in understanding dental
anatomy.
īƒ˜ Tooth numbering or “shorthand” system of tooth
notation is necessary in clinical practice for
recording data and communication.
īƒ˜ The various tooth notation systems are as follows:
A. Palmer notation system
B. Universal notation system
C. FDI system (Federation Dentaire Internationale)
55
A. Palmer notation system
īƒ˜ In 1861 Adolph Zsigmondy of Vienna introduced the
symbolic system for permanent dentition. He then
modified it for the primary dentition in 1874.
īƒ˜ The symbolic system is now commonly referred to as
the Palmer notation system or Zsigmondy system.
īļPrimary teeth
E D C B A │ A B C D E
E D C B A │ A B C D E
īļPermanent teeth
8 7 6 5 4 3 2 1 │ 1 2 3 4 5 6 7 8
8 7 6 5 4 3 2 1 │ 1 2 3 4 5 6 7 8
56
B. Universal notation system
īƒ˜ In this system for the permanent dentition the maxillary
teeth are numbered through 1 to 16 beginning with
upper right third molar.
īƒ˜ The mandibular teeth are numbered through 17 to 32
beginning with lower left third molar.
īƒ˜ The universal system notation for primary dentition
utilizes upper case alphabets.
īļ Primary teeth- A B C D E│F G H I J
T S R Q P│O N M L K
īļ Permanent teeth
1 2 3 4 5 6 7 8│ 9 10 11 12 13 14 15 16
32 31 30 29 28 27 26 25 │ 24 23 22 21 20 19 18 17
57
C. FDI system
īƒ˜The FDI system is a two digit system that has
been adopted by WHO.
īƒ˜In this system the first digit indicates the
quadrant and the second digit indicates the
tooth within the quadrant.
īƒ˜1 to 4 and 5 to 8 as the first digit indicates
permanent and primary dentition respectively.
īƒ˜1 to 8 and 1 to 5 as the second digit indicates
permanent and primary teeth respectively.
58
59
PERIODONTI
CS
B Y D R . N H I A L W . ( M D )
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Module III
4.Periodontic
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The specialty of dentistry that studies supporting
structures of teeth, diseases, and conditions that affect
them.
PULP AND PERIRADICULAR DISEASES
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The Dental Pulp
īƒ˜ Consists of richly vascularized and highly innervated
connective tissue
īƒ˜ It is surrounded by dentin
īƒ˜ The pulp tissue is in communication with the periodontium
and the rest of the body through the apical foramen and
accessory canals near the apex of the root
PULPITIS
īƒ˜ Is inflammation of the dental pulp resulting from untreated caries,
trauma, or multiple restorations.
īƒ˜ Pulpitis can occur when caries progresses deeply into the dentin
īƒ˜ Trauma disrupts the lymphatic and blood supply to the pulp
īƒ˜ Its principal symptom is pain
Main Cause
1.Infection: spread of dental caries to the pulp
2. Trauma
3. Physical irritation: excessive heat during cavity preparation.
4. Chemical irritation i.e. filling materials or cavity cleansers such as
alcohol, chloroform, hydrogen peroxide
5. Mixed microorganisms which are found in the oral cavity
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ī‚— This image shows how the progression of decay, if not treated,
will lead to destruction of the tooth's hard tissue and pulp.
ī‚— If treated at the beginning of the decay process (first image), a
small filling would resolve the problem.
ī‚— If allowed to continue as in the last image, root canal therapy
would be necessary.
PROGRESSION OF DECAY (CARIES)
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īƒ˜ When Dental decay reaches a depth in the dentin that
is near the pulp tissue inside of the tooth, the pulp tissue can
become inflamed (pulpitis)
Classification of dental pulp disease
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Reversible pulpitis
ī‚— Tissue is capable of
returning to normal state
of health if noxious stimuli is
removed
ī‚— Sudden mild to moderate
pain or short duration
ī‚— Sweat, sour foods or
beverages can cause pain
Irreversible pulpitis
ī‚— Higher level of inflammation
ī‚— Pulp is damaged beyond the
point of recovery
ī‚— Sharp severe pain upon
thermal stimulation
ī‚— Pain continues after the
stimulus is removed
ī‚— Heat & sweat & sour foods
elicit pain
ī‚— Pain may be spontaneous or
continuous
Diagnosis and Treatment of Pulpitis
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īƒ˜Diagnosis is based on clinical findings and is
confirmed by x-ray.
īƒ˜Treatment involves
īƒŧ Removing decay,
īƒŧ Restoring the damaged tooth, and
īƒŧ Sometimes doing root canal therapy (RCT) or extracting the
tooth.
Peri-radical Diseases
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Peri-radicular tissue
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īƒ˜ The tissues surrounding and investing the cervical, middle, and apical
regions of the root are the cementum, periodontal ligament, and
alveolar bone.
Classification of Peri-radicular lesions
Classified into four main groups:
1. Acute apical Periodontitis (AAP)
2. Chronic apical Periodontitis (CAP)
3. Acute apical abscess (AAA)
4. Chronic apical abscess (CAA)
1) Acute apical Periodontitis
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īƒ˜ Etiology
īļ Extension of pulpal inflammation into the peri-radicular tissues
īļ Irritants: bacteria toxins from necrotic pulps, chemicals
(irritants or disinfecting agents) over instrumentation &
restorations in hyper occlusion.
īƒ˜ S/symptoms
īļ moderate to severe spontaneous discomfort as well as pain on
mastication or occlusal contact.
īļ Application of pressure by fingertip can cause marked pain
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īƒ˜ Diagnosis
īļ Radiography: "Thickening”
of periodontal ligament (PDL) space
(May be not always)
īƒ˜ Treatments
īƒŧ Adjustment of occlusion (when
there is evidence of hyper
occlusion)
īƒŧ Removal of irritants or a
pathologic pulp, or
īƒŧ Release of peri radicular exudates
2) Chronic apical Periodontitis
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īƒ˜ Etiology: results from pulp necrosis and usually is a sequel to
AAP.
īƒ˜ S/symptoms:
īƒŧ Asymptomatic or slight discomfort
īƒŧ Do not respond to electrical or thermal stimuli
īƒŧ Percussion produces little or no pain there may be slight
sensitivity to palpation, indicating an alteration of the cortical
plate of bone and extension of CAP into the soft tissues
Radiography image: Well-circumscribed lesions and pear-shaped
configuration of periradicular to the canine are typical of CAP.
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3) Acute apical abscess
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īļ Etiology: microbial and nonbacterial irritants from necrotic
pulp.
īļ S/symptoms:
īƒŧ Have moderate to severe discomfort or swelling.
īƒŧ Systemic manifestations of an infective process such as a
high temperature, malaise and leukocytosis
īƒŧ Because AAA occurs only with pulp necrosis, electrical or
thermal stimulation produces no response
īƒŧ However, teeth are painful to percussion and palpation
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īƒ˜ Radiography: range from thickening of the PDL
space (infrequent) to a frank resorptive lesion (usual)
īƒ˜ Treatment: Removal of the underlying cause
(necrotic pulp), release of pressure (drainage here
possible) and root canal treatment
īƒ˜ Clinical significance
īƒŧ If untreated, a Periapical/apical abscess can develop
into osteomyelitis , septicemia, Ludwigs angina, or
cavernous sinus thrombosis.
4) Chronic apical abscess (Suppurative Apical
periodontitis)
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īƒ˜ Result of a long-standing lesion that has resulted in an
abscess that is draining to a surface
īƒ˜ Etiology
īƒŧ Pathogenesis similar to that of AAA results from pulp
necrosis and is usually associated with chronic apical
periodontitis that has formed an abscess
īƒŧ The abscess has "burrowed" through bone & soft tissue to
form a sinus on the oral mucosa or sometimes onto the skin
of the face
Contâ€Ļ
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īļ Signs and symptoms
īƒŧ Because drainage exists, CAA is usually asymptomatic
except when there is occasional closure of the sinus
pathway, which can cause pain
īƒŧ Clinical, radiographic, and histopathologic features of CAA
are similar to described for CAP.
īƒŧ An additional features is the sinus tract
A. Apical abscesses occasionally drain
extra-orally (blue arrows)
B. The tooth was retreated nonsurgical
and the chin lesion healed within a few
weeks with some scarring.
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Periapical abscess, Apical periodontitis and Cellulitis.
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Peri-coronitis
īƒ˜ Inflammatory process involving soft tissues covering
crown of partially erupted or un-erupted teeth.
īƒ˜ Develops when food debris & bacteria are present
beneath the gingival flap
īƒ˜ Usually seen associated with impacted mandibular 3rd
molars
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Clinical features
īƒ˜Severe pain
īƒ˜Swollen, red, tender gingiva
īƒ˜Discomfort in swallowing
īƒ˜Restriction of oral opening
īƒ˜Foul taste
īƒ˜Pain may radiate to ear, floor of mouth or
throat
īƒ˜Lymphadenopathy
īƒ˜Malaise & fever
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Treatment
īƒ˜ Antiseptic lavage under the gingival flap to remove gross
food debris & bacteria
īƒ˜ Gingival flap is removed surgically if the tooth to be
retained
īƒ˜ Extraction of tooth if needed
īƒ˜ Antibiotics
īƒ˜ Warm saline mouth rinse
Gingivitis
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īƒ˜ It is an inflammatory lesion confined to the tissue of the
marginal gingiva.
Cause:
īƒ˜ Accumulation of bacterial plaque at or near the gingival
margin.
īƒ˜ The bacterial component of plaque produces and releases
variety of enzymes and toxins (e.g. lipopolysacchardies )
which diffuse through the junctional epithelium and initiate
inflammatory changes in the gingival connective tissues.
Contâ€Ļ
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Clinical features
â€ĸ Redness of the gum
â€ĸ Gum bleeding
â€ĸ edema of the gum
â€ĸ Tenderness of the gum
Treatment:
â€ĸ Oral hygiene
â€ĸ Plaque control
â€ĸ Oraldine mouth wash
â€ĸ Administration of antibiotics
Acute Necrotizing Ulcerative
Gingivitis (ANUG)
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īƒ˜Definition: is an inflammatory destructive gingival
condition which exhibits characteristics clinical signs
and symptoms.
īƒ˜The other names for ANUG are "Vincent's gingivitis" or
"Vincents gingivostomatitis", 'Trench mouth'' and"
Ulcero-membraneous gingivitis“
Contâ€Ļ
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Causes
īƒ˜Fusiform bacteria
īƒ˜Treponema vincenti
īƒ˜Treponema deticola
īƒ˜Fusobacterium nucleatum
īƒ˜Prevotella intermedia
īƒ˜Porphymonas gingivalis
NB: These bacteria are found in large numbers in the
slough and necrotic tissues at the surface of the ulcer.
Contâ€Ļ..
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Clinical features
īƒ˜ Inter proximal ulcers covered with a yellowish-white or grayish
debris
īƒ˜ Easily bleed
īƒ˜ Necrosis develops rapidly
īƒ˜ Linear erythema
īƒ˜ Pain
īƒ˜ Halitosis
īƒ˜ Lymphadenitis
īƒ˜ Fever and malaise
Contâ€Ļ.
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Control of the acute phase
īƒ˜Antibacterial cleaning
īƒ˜Irrigation of the wound with 3% hydrogen peroxide
solution
īƒ˜Scaling of the affected teeth
īƒ˜Antibiotics, Metronidazole
īƒ˜2% Chloxeidine mouth wash
Modul IV
EXODONTIA
By Dr.NHIAL (MD)
Module IV Con’tâ€Ļ..
Exodontia
īƒ˜Painless removal of the tooth or root with
minimal injury to the surrounding soft tissue and
bone
īƒ˜Removal of the tooth does not require large
amount of force.
īƒ˜Pain and anxiety control is by:
īƒŧLocal anesthesia results in loss of pain,
temperature and touch but not pressure
91
Cont.â€Ļ
Duration of anesthesia
īƒ˜ local anesthesia without vasoconstrictors
īƒŧMaxillary teeth: 10 - 20 minutes
īƒŧMandibular teeth: 40 - 60 minutes
īƒŧSoft tissue: 2 – 3 hours
īƒ˜Lasts longer in mandible due to the density
of the cortical bone of the mandible.
92
Cont.â€Ļ
īƒ˜Local anesthesia with vasoconstrictor
â€ĸ Maxillary teeth: 50 – 60 minutes
â€ĸ Mandibular teeth: 90 - 100 minutes
â€ĸ Soft tissue: 3 – 4 hours
īƒ˜Long acting anesthesia with vasoconstrictor
â€ĸ Maxillary teeth: 60 - 90 minutes
â€ĸ Mandibular teeth: 3 hours
â€ĸ Soft tissue: 4- 9 hours
93
Indications for removal of teeth
īƒ˜Severe dental caries
īƒ˜Pulpal necrosis
īƒ˜Severe periodontal disease
īƒ˜Mal opposed teeth
īƒ˜Orthodontic reasons
īƒ˜Cracked teeth or fractured root
94
Indications for removal of teeth
īƒ˜Pre prosthetic extraction
īƒ˜Impacted teeth
īƒ˜Supernumerary teeth
īƒ˜Teeth associated with pathologic lesion
īƒ˜Pre radiation therapy
īƒ˜Severe attrition ,abrasion or erosion
īƒ˜Teeth involved in jaw fracture
95
Contraindications for the removal of teeth
īƒ˜ Systemic contraindications
oUn controlled diabetes
oEnd stage renal disease with severe
uremia
oUncontrolled leukemia
oUncontrolled cardiac disease
oUnstable angina pectoris
96
Contraindications for the removal of teeth
o Recent MI
o Severely uncontrolled hypertension
o Pregnancy first and last trimester
o Bleeding disorders like hemophilia
o Platelet disorders
o Pt. on anticoagulants
NB: Absolute contraindication (arterioveinous
fistula and end stage renal disease)
97
Local contraindications
īƒ˜Tooth in the area of tumor
īƒ˜Acute infection
īƒ˜Central hemangioma
98
Instrument used for extraction
1. Forceps
īƒ˜Uses of forceps
ī‚§To expand bony sockets
ī‚§To remove tooth
2. Elevator : for loosening
īƒ˜Straight elevator
īƒ˜Periosteal elevator
īƒ˜Cryer elevator
99
Techniques of extraction
1. Closed extraction
īƒ˜Adequate access and visibility needed
īƒ˜Steps for closed extraction
1. Loosening of soft tissue attachment from
tooth
2. Luxation of tooth with dental elevator
3. Adaptation of forceps to tooth
4. Luxation of tooth with forceps
5. Removal of tooth from socket 100
Post extraction care
īƒ˜ Curettage if any peri-apical pathology
īƒ˜ Curette if any debris, calculus, amalgam, tooth & bone
fragment in the socket
īƒ˜ Finger pressure is applied to buccal and lingual cortical
plates to compress the socket
īƒ˜ Moistened 2x2 inch gauze is placed
101
Contâ€Ļ
2. Open extraction
īƒ˜Indication:
īƒŧFailure to remove by closed methods
īƒŧUnfavorable root pattern
īƒŧFracture or caries extending to root
īƒŧHypercementosis
īƒŧImpacted tooth
102
Complications of exodontia
īƒŧ Fracture of crown or roots of the tooth being
extracted
īƒŧ Fracture of alveolar bone
īƒŧ Fracture of maxillary tuberosity
īƒŧ Fracture of adjacent or opposing tooth
īƒŧ Fracture of mandible
īƒŧ Dislocation of TMJ
īƒŧ Displacement of root into soft tissues ,maxillary
antrum
īƒŧ Bleeding
103
Contâ€Ļ.
īƒŧInjury to gums, lips, IAN & its branch, lingual
nerve, tongue, floor of mouth, greater palatine
artery
īƒŧ Osteomyelitis
īƒŧ Infection
īƒŧ Trismus
īƒŧ Hematoma
104
Local anesthesia
īƒ˜ Is the loss of sensation in circumscribed area of body
caused by depression of excitation in nerve endings
or inhibition of conduction process in peripheral
nerves.
īƒ˜ Produces loss of sensation with out loss of
consciousness.
105
Contâ€Ļ
Advantages of LA:
â€ĸ It can be easily mastered and can be given by
operator
â€ĸ Equipment required is limited in amount
â€ĸ Economical and transportable
â€ĸ The patency of air way is not impaired
â€ĸ Enables the patient to cooperate
Contraindications to LA:
ī‚§ Acute infection in the injection sites
ī‚§ Patients with hemorrhagic disease
106
Types of LA
1. Topical.
īƒ˜ When applied to intact mucus membrane pass through the
epidermis and anaesthetize the nerve endings e.g.. Cocaine,
lidocaine.
īƒ˜ It can be in the form of sprays, ointmentâ€Ļ
2. Infiltration:- Deposition of LA near the terminal fibers to
produce anesthesia to localized areas served by the nerve
fibers.
- Sub mucous injection
- Supra periosteal
- Sub periosteal
* Mostly used in maxilla
107
Contâ€Ļ
3. Regional anesthesia: Anesthetic solution
deposited near the nerve trunk & blocks all
impulses. It is also known as block
anesthesia.
** It is widely used in the mandible since
infiltration is unreliable due to the density of
the cortical bone of the mandible.
108
Summary of maxillary block
Posterior Superior Alveolar Nerve Block
Maxillary molars (with exception of
mesiobuccal root of maxillary 1st molar
in some cases), hard and soft tissue on
buccal aspect
Middle Superior Alveolar Nerve Block
Mesiobuccal root of maxillary 1st molar ,
premolars and
surrounding hard and soft tissue on buccal
aspect
Anterior Superior Alveolar Nerve
Block/Infraorbital Nerve Block
Maxillary central and lateral incisors and
canine, surrounding hard and soft tissue on
109
Local anesthesia in Mandible
īƒ˜ The regional or block anesthesia in the mandible is achieved
by deposition of LA around the inferior alveolar nerve at
pterygo mandibular space / Retro molar area
For mandibular block:
īƒ˜ Mandibular occlusal plane should be horizontal when
patient open his mouth
īƒ˜ Operator should stand in front of the patient for giving
anesthesia to the right side and behind for the left.
īƒ˜ The thumb of left hand is passed along the buccal surface of
the lower molar teeth until the internal oblique ridge is felt.
Then the tip is rolled in ward to lie in retro-molar fossa.
The mid point of the nail concedes the land mark for
insertion of the needle after preparation
110
Contâ€Ļ
īƒ˜ The syringe is held parallel to the mandibular
occlusal plane over 2nd premolar of the opposite side.
īƒ˜ Needle is inserted about 0.5cm to anaesthetize the
lingual nerve and advance more 1.5- 2cm to
anaesthetize the inferior alveolar nerve and long
buccal nerve
111
Difficulties and complication of LA
1. Failure to obtain Anesthesia
a) Inadequate amount LA
b) Poor operater technique
c) Missing land marks
d) PH of tissue
e) Expiration of LA
112
Contâ€Ļ
2. Pain during and after injection
Reduced by - Sharpe needle
- Tensing the tissue
- Slow injection
3. Hematoma formation: Occurs most frequently when posterior
superior alveolar block given.
4. Intra vascular injection: Frequently the patients feels faint.
Has a pale clammy skin, rapid pulse.
5. Trismus:
īƒŧ Difficulty of opening jaw due to muscle spasm
īƒŧ Injection to medial pterygoid muscle
6. Infection
113
Contâ€Ļ
7. Facial paralysis: Injection to the parotid gland
8. Prolonged impairment of sensation due to damage of the
nerves.
9. Broken Needles
-When needle broken, the tissue must be kept under continuous
pressure until the protruding end is grasped by pliers or artery
forceps.
- If the edge is embedded completely, the patient should be
informed and X-ray should be taken to confirm the position and
presence.
114
Contâ€Ļ
10. Lip trauma:
ī‚§ Children chews anaesthetized part.
ī‚§ Adults burn from hot drinks
11. Fainting
In this instance
â€ĸ Head should be lowered
â€ĸ Legs elevated
â€ĸ Tight clothes & belt loosened
â€ĸ Wet swab applied on brow
â€ĸ Stimulate respiration
115
Reading assignments
1.Maxillofacial trauma
2.Principles of preventive dentistry or Oral
health
116
117
Module V
Management of Maxillofacial injuries
Introduction
â€ĸ The facial skeleton is divided into 3 parts
â€ĸ The upper 1/3: formed by frontal bone
â€ĸ The middle 1/3: from frontal bone to the level of
upper teeth.
â€ĸ The lower 1/3: the mandible
118
The causes of maxillofacial trauma
īƒ˜Fights
īƒ˜Falls
īƒ˜RTAs
īƒ˜Occupational hazards- athletic injury,
industrial mishaps
īƒ˜Iatrogenic causes- # of tooth, alveolus,
maxillary tuberosity, #of mandible during
dental treatment.
119
â€ĸ Radiographic examination
For middle 1/3
- PA view of the skull
- Lateral view of the skull
For the mandibular fracture
- PA view of the mandible
- Right and left lateral view of the
mandible
120
Basic principle life preservation in
traumatic patient
The ABC methods
īƒŧ Maintenance of patency of Air way
īƒŧ Bleeding control
īƒŧ Maintenance of Circulation
121
Factors affecting wound healing
īąLocal
īƒ˜ Infection
īƒ˜ Foreign bodies
īƒ˜ mobility
īƒ˜ poor vascularity
īąSystemic
īƒ˜Increased age
īƒ˜Disease e.g diabetes
īƒ˜Deficiency e.g malnutrition
122
Basic principles of management of fracture
1) Reduction: the restoration of fracture
fragments to their original position.
â€ĸ It can be closed or open reduction
īļ Closed reduction:
â€ĸ Can be carried out by manipulation or
traction
â€ĸ No surgical intervention is needed
â€ĸ Occlusion of the teeth is used as a guide line
123
īƒ˜ By manipulation: by digital or hand
manipulation
- when fragments are adequately mobile
- in patients come soon after trauma
īƒ˜ By traction: fractured fragments are
subjected to gradual elastic traction
īļOpen reduction
- surgical reduction that allows visual
identification of fractured fragments
124
2) Fixation
fractured fragments are fixed to prevent
displacement and for achieving proper
approximation
īƒŧ Direct skeletal fixation: by plates or
intraosseous wiring
īƒŧ Indirect skeletal fixation: by arch bar or
intermaxillary fixation
125
3) Immobilization
- The fixation device is retained to stabilize
the reduced fragments until a bony union
takes place.
- For maxillary # 3 to 4 weeks
- For mandibular # 4 to 6 weeks
immobilization.
- In case of condylar # 2 to 3 weeks to
prevent Ankylosis.
126
Fracture of the middle 1/3 of the face
Boundaries of middle 1/3 of the face
īƒŧSuperiorly: line from zygomaticofrontal
suture, across frontonasal suture,
frontomaxillary suture.
īƒŧ Inferiorly: occlusal plane upper teeth
īƒŧPosteriorly: sphenoethmoidal junction
127
1) Le fort I
(horizontal fracture of the maxilla or Guerin's
fracture or floating fracture) separation of
dentoalveolar part of the maxilla
- Horizontal # line above the apices of the teeth at
the level of floor of the nose
- Usually bilateral
- Floating of the palate
- Hematoma within the maxillary antrum
- Bilateral hematoma of the cheek
Classification
128
129
2) Le Fort II Fractures (Low Pyramidal
Subzygomatic fracture )
- line crosses pyramidally from nasal bone
then frontal process of maxilla then lacrimal
bone and infraorbital marigin crosses the
zygomatic buttress then move backwards and
fracture above maxillary tuberosity
- Facial swelling with massive edema
- Subconjuctival ecchymosis and diplopia
- Dish faced deformity
130
- Infraorbital anesthesia both side
- Bilateral hematoma
- Retroposed upper dental arch with anterior open
bite
- Cracked pot sound on percussion of teeth
131
3) Le fort III: (High level #)
īƒ˜ Force is in lateral direction
īƒ˜ Line runs from nasofrontal region then lacrimal bone then
ethemiod bone around the optical canal involving infraorbital
fissure then greater wing of the sphenoid then
zygomaticofrontal suture, and fracture of both side of the
zygomatic arches.
Clinical features:
īƒ˜ Tenderness and separation at fronto zygomatic suture
īƒ˜ Tenderness and deformity of zygomatic arches
īƒ˜ Lengthening of face
- Depression of ocular levels
- Enophtalmos
- CSF leak via nose (csf rhinorrhea)
- Entire middle 1/3 separated from the cranial base.
- Bilateral circumorbital ecchymosis
132
Treatment of middle face fracture:
1. Manual reduction
2. Reduction by traction
3. Open reduction
133
Fracture of the mandible
Largest, strongest and heaviest bone of the face
Classification:
1)Condylar neck 35%
2)Angle 20%
3)Body 20%
4)Parasymphysis 13%
5)Symphysis 11%
6)Coronoid 1%
134
135
Management
1) Closed reduction
- dental wiring or arch bar is used to get
the occlusion
- for 6 weeks
Indication
- Non displaced #
- Lack of soft tissue over the fracture
- # of children with developing tooth bud
- Coronoid process fracture
136
2) Open reduction
Indications
- Displaced fracture
- Multiple fracture
- Associated mid face fracture
- Associated condylar fracture
Contraindicated if GA is not advisable,
sever comminuted or loss of soft tissue
and severe infection.
137
Condylar fracture
Classified as:
- Non displaced: a crack is seen on
radiograph
- Deviation: simple angulation b/n
condylar neck and ramus
- Displacement: overlap b/n condyle and
ramus
- Dislocation: condylar fragments are
pushed anteriorly and medially
138
Clinical feature:
- Pain and swelling in the region of
TMJ
- Limitation of oral opening
- Deviation towards the involved side
- Blood in external auditory canal
- Lack of condylar movement on
palpation
139
Complication of maxillofacial fracture
īƒ˜Anesthesia
īƒ˜Malunion and deformity
īƒ˜Infection
īƒ˜Derangement of occlusion
īƒ˜Ankylosis of TMJ
īƒ˜Etcâ€Ļ.
140
The Temporomandibular Joint (TMJ)
īƒŧ The Temporomandibular joint (TMJ) is a small
joint located in front of the ear where the skull
and lower jaw meet.
īƒŧ It permits the lower jaw (mandible) to move and
function.
īƒŧ Between the condyle and the fossa is a disk made
of cartilage that acts as a cushion to absorb stress
and allows the condyle to move easily when the
mouth opens and closes.
141
īƒ˜TMJ disorders are not uncommon and have a
variety of symptoms.
īļPatients may complain of :
īƒŧEaraches
īƒŧHeadaches
īƒŧLimited ability to open their mouth.
īƒŧClicking or cracking sounds in the joint
īƒŧFeel pain when opening and closing their
mouth.
142
Dislocation of TMJ
īƒ˜Excursion of condylar head beyond the
anterior limit of articular eminence
It can be of:-
- Unilateral or bilateral
- Chronic or acute
143
Causes
īƒ˜Blow on the chin while the mouth is
open
īƒ˜Excessive pressure on mandible while
extraction
īƒ˜Vomiting
īƒ˜Injudicious use of mouth gag during
GA
144
Clinical features
- Deviation of the chin
- Open bite
- Depression in front of the tragus
- Inability to close the mouth
- Drooling of the saliva
- Difficulty in speech and swallowing
145
Management
- Manipulation
- Surgical if chronic
Wishesyouthebest!!!
146

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Dentistry 2023.pptx

  • 1. D E P T ; P U B LI C H E A L T H 4 T H Y R C O U R SE T I T LE ; D E N TI ST R Y C R E D I T H O U R S ; T H R E E ( 0 1 ) B Y ; D R . N H I A L W A N L O L ( M D) BARO Gambella College
  • 2. Module I īļIntroduction to Dentistry ī‚— Definition of dentistry ī‚— Anatomy and physiology oral cavity ī‚— Pathology of oral cavity ī‚— Dental Caries
  • 3. Introduction to Dentistry ī‚— Definition; Dentistry is a study of oral cavity e.g bones, teeth and soft tissues ī‚§ Dentistry is the science and art of preventing, diagnosing, and treating, diseases, injuries and malformations of the teeth, jaws and mouth and of replacing lost or absent teeth and associated structures.
  • 4. Anatomy and physiology ī‚— Oral cavity consist of; ī‚— Bones..Upper–Jaw Lower-Jaw ī‚§ Teeth(3parts) Crown Neck Root ī‚§ Soft tissues ī‚§ Tongue, gum, vocal mucosa, and dentine ī‚§ Glands..salivary and lymph node
  • 6. Parts of the Teeth 6 A tooth has a crown, neck, and root īƒ˜The crown: the visible part of teeth that projects from the gingiva īƒ˜The neck is between the crown and the root īƒ˜The root is the part fixed in the tooth socket, which is variant in number
  • 7. 7
  • 8. Layers of Teeth 8 īƒ˜Enamel: the outer most layer of teeth in the crown īƒ˜Cementum: the outer most layer of teeth in the root īƒ˜Dentin: the middle layer of teeth, forms the main density of teeth īƒ˜The pulp cavity: the central part of teeth, contains connective tissue, blood vessels, and nerve
  • 9. Sensory innervation and Blood Supply of the jaw 9 īƒ˜ Superior and inferior alveolar arteries/ veins ; branch of maxillary artery supply tooth īƒ˜ Upper jaw= superior alveolar nerves īƒ˜ Lower jaw= inferior alveolar nerves; both make dental plexus ī‚Ą Inferior alveolar nerve: all mandibular teeth, buccal soft tissues of premolar, canine and incisors ī‚Ą Lingual nerve: all lingual soft tissues of all teeth ī‚Ą Long buccal nerve: buccal soft tissue of all molars ī‚Ą Anterior superior alveolar nerve: maxillary incisors and canine, buccal soft tissues of incisors and canines ī‚Ą Middle superior alveolar nerve: maxillary premolar and maxillary bone roots first molar, buccal soft tissue of premolar
  • 10. Cont.â€Ļ 10 ī‚— Post. Sup. alveolar nerve: maxillary molars except a portion of first molar, buccal soft tissue of molars ī‚— Greater palatine nerve: lingual soft tissues of molars and premolars ī‚— Nasopalatine nerve: lingual soft tissue of incisors & canines ī‚— Mandibular premolar region, buccal soft tissue innervated primarily by mental branch of IAN and also by terminal branch’s of long buccal nerve
  • 11. 11
  • 12. Tongue 12 īƒ˜Muscular organ īƒ˜Functions in: ī‚ĄChewing ī‚ĄSwallowing ī‚ĄSpeech īƒ˜ Site of sensory reception: īƒˇTaste īƒˇTouch īƒˇPain / Temperature
  • 13. Salivary Glands 13 īƒ˜ Major and Minor Salivary Glands ī‚Ą Three pairs of major Salivary Glands īƒ˜ Parotid – below and anterior to the ear īƒ˜ Submandibular – below the mandible īƒ˜ Sublingual – anterior floor of the mouth īƒ˜ Orifices / ducts: īļ Stensen’s duct – parotid īļ Wharton’s duct – submandibular īļ Numerous small ducts of Sublingual glands īƒ˜ Over 1000 minor glands īƒˇ Buccal, palatal, lingual
  • 14. The Role of Saliva 14 īƒ˜ Physical protection provides a cleansing effect. īƒ˜ Chemical protection contains calcium, phosphate, and fluoride. It keeps calcium there ready to be used during remineralization. īƒ˜ It includes buffers, bicarbonate, phosphate, and small proteins that neutralize the acids after we ingest fermentable carbohydrates. īƒ˜ Antibacterial substances in saliva work against the bacteria; like lactoferrin, lysozyme, lactoperoxidase, beta-lysin, and immunoglobulins īƒ˜ Maintenance of homeostasis on dental surfaces: ī‚Ą Dissolves and dilutes metabolites ī‚Ą Maintains proper pH balance ī‚Ą Reduces plaque
  • 15. Physiology īļFunction of oral cavity ī‚— Digestion...mastication/chewing ī‚— Alternate airway....breathing ī‚— Articulation of speech...speaking ī‚— Anatomical support ī‚— Psychology...cosmetic
  • 16. Pathology ī‚— Pathology of oral cavity is defined as disease affecting bones, and connective tissues of oral cavity ī‚— Dental caries is a bacterial infection of the teeth and soft tissues. ī‚— Oral hygiene; caring for teeth, gum and other soft tissues ī‚— Connective tissue include; gum, and vocal mucosa
  • 17. Dental Caries ī‚— Bacterial infection of the teeth and connective tissues. ī‚— Pathophysiology of dental carier ī‚— Food particle + bacteria= form plaque--- plaque form dental carrier ī‚— Dental plaque is colourless, soft, sticky material disposed on the enamel.
  • 18. Transmission of Caries Causing Bacteria 18 ī‚— Mutans streptococci are transmitted through saliva, most frequently the mother’s, to the infant. ī‚— When mothers have high counts of mutans streptococci in their mouths, the babies also have high counts of the same bacteria in their mouth. Dental Plaque ī‚— Dental plaque is a colorless, soft, sticky coating that adheres to the teeth. ī‚— Plaque remains attached to the tooth despite movements of the tongue, water rinsing, water spray, or less than thorough brushing. ī‚— Formation of plaque on a tooth concentrates millions of microorganisms on that tooth
  • 19. Dental Caries: A Bacterial Infection 19 īƒ˜ There are two specific groups of bacteria found in the mouth that are responsible for dental caries: īƒŧ Mutans streptococci (Streptococcus mutans) īƒŧ Lactobacilli ī‚— They are found in relatively large numbers in the dental plaque. ī‚— The presence of lactobacilli in the mouth indicates a high sugar intake.
  • 20. The following mechanisms contribute to tooth decay 20 īƒ˜ Plaque bacteria that ferment dietary sucrose produce acids that lower the pH on the tooth surface, promoting demineralization and eventually, tooth decay. īƒ˜ Plaque bacteria, such as S. mutans can also utilize sucrose to produce extracellular polysaccharides, known as glucans, which are adhesive polymers that enable mutans streptococci to avidly stick to the tooth surface, thereby causing decay in the underlying structures.
  • 22. The Caries Process 22 īƒ˜For caries to develop, three factors must occur at the same time: ī‚ĄA susceptible tooth ī‚ĄDiet rich in fermentable carbohydrates ī‚ĄSpecific bacteria (regardless of other factors, caries cannot occur without bacteria)
  • 24. The earliest sign of decay is decalcification 24
  • 25. Root Caries 25 ī‚— Root caries is becoming more prevalent and is a concern for the elderly population who often have gingival recession exposing the root surfaces. ī‚— Carious lesions form more quickly on root surfaces than coronal caries because the cementum on the root surface is softer than enamel and dentin. ī‚— Like coronal caries, root caries has periods of demineralization and remineralization.
  • 27. Erosion 27 Cause: Bruxism (Involuntary clenching teeth; esp. during sleep) Loss by wear of surface of tooth or restoration caused by tooth to tooth contact during mastication or parafunction Loss by wear of dental tissue caused by abrasion by foreign substance (e.g., toothbrush, dentifrice) Progressive loss of hard dental tissue by chemical processes not involving bacterial action Cause: GERD Cause: Over vigorous brushing Abrasion Attrition
  • 28. Diagnosis of Dental Caries and Non Dental Caries 28 īƒ˜Radiographs īƒ˜Visual īƒ˜Laser caries detector Laser Caries Detector īƒ˜The laser caries detector is used to diagnose caries and reveal bacterial activity under the enamel surface. īƒ˜Carious tooth structure is less dense and gives off a higher reading than non- carious tooth structure.
  • 29. Visual and radiographic appearance of seemingly intact molar
  • 30. Cross section of molar showing decay
  • 31. Treatments 31 īƒ˜ Elimination of risk factors īƒ˜ Scaling, root planning & curettage īƒ˜ Antibiotic therapy (metronidazole or tetracycline) īƒ˜ Anti-pain (NSAIDs) Filling īƒ˜ Amalgam (a mixture of mercury and other metals (solid/liquid) used for filling holes in teeth) īƒ˜ Composite a solid material which is composed of 2/more substances having d/t physical ch/tcs and in which each substance retains its identity while contributing desirable properties to the whole.
  • 32. Methods of Caries Intervention 32 īƒ˜ Fluoride: A variety of types are available to strengthen the tooth against solubility to acid. īƒ˜ Antibacterial therapy: Products such as Chlorhexidine rinses are effective. īƒ˜ Salivary flow can be increased by chewing sugarless gum, for example, those with a non-sugar sweetener such as xylitol. Preventive measures against caries A. Fluoride rinse B. Chlorhexidine rinse C. Xylitol gum. D. Sealants- thin, plastic material used to cover tooth surfaces. It protects enamel from attack by bacterial acids.
  • 33. Complications of dental carrier I. Local ī‚— Abscess..pus collection ī‚— Tooth loss ī‚— Pericorinitis ī‚— Chronic tooth pain ī‚— Bacterial infection of the floor of the mouth ī‚— e.g Ludwig’s angina
  • 34. Dental Carrier cont............. II. Systemic infection ī‚— Sepsis ī‚— Septic arthritis ī‚— Osteomyelitis ī‚— Septic shock
  • 35. B Y D R . N H I A L W . ( M D ) FIELDS OF DENTISTRY
  • 36. Outline ī‚— Prosthodontic ī‚— Endodontic ī‚— Periodontics ī‚— Orthodontic ī‚— Oral and maxillofacial surgery ( OMFS) ī‚— Operative dentistry
  • 38. 1. Prosthodontics The branch of dentistry that deals with the replacement of missing teeth and related mouth or jaw structures by bridges, dentures, or other artificial devices. 38
  • 39. 2. Endodontic It deals with the tooth pulp & tissues surrounding the root of tooth. 39 39
  • 40. 3. Operative Dentistry (Restorative dentistry) Concerned with restoration of parts of the teeth that are defective as a result of disease, trauma, or abnormal development to a state of normal function, health, and aesthetics. 40
  • 41. 4.Periodontic The specialty of dentistry that studies supporting structures of teeth, diseases, and conditions that affect them. 41
  • 42. 5. Oral and maxillofacial surgery The dental specialty concerned with the diagnosis and treatment of diseases affecting the mouth, jaws and orofacial structures. 42 42
  • 43. 6. Orthodontics The specialty of dentistry that is concerned with the study and treatment of malocclusions, which may be a result of tooth irregularity, disproportionate jaw relationships, or both. 43
  • 44. A N A T O M Y A N D N O M E N C L A T U R E O F T E E T H Module II
  • 45. Outline ī‚— Classification ī‚— Anatomy of tooth ī‚— Parts of the tooth ī‚— Layers of the tooth ī‚— Innervation and blood supply of the teeth ī‚— Nomenclatures system of the teeth
  • 46. Objective īļBy the this session, students will be able to; īƒ˜Classify teeth īƒ˜Describes normal anatomy of teeth īƒ˜Explain parts of the tooth īƒ˜Describes layers of the teeth īƒ˜List nerves and blood vessels of the teeth īƒ˜Learn different nomenclature systems of the teeth
  • 47. Teeth īƒ˜ The teeth are set in the tooth sockets and used in mastication and speech īļ Two class of teeth: a) Primary/deciduous b) Permanent â€ĸ Children have 20 deciduous teeth; adults have 32 permanent teeth īƒ˜ The 20 deciduous teeth consist of â€ĸ 2 incisor, 1 canine, and 2 molar teeth on each side of the upper and lower jaws â€ĸ These teeth are replaced by the incisor, canine, and premolar teeth of the permanent teeth 47
  • 48. 48
  • 49. Parts of the Teeth A tooth has a crown, neck, and root īƒ˜The crown: the visible part of teeth that projects from the gingiva īƒ˜The neck is between the crown and the root īƒ˜The root is the part fixed in the tooth socket, which is variant in number 49
  • 50. 50
  • 51. Layers of Teeth īƒ˜ Enamel: the outer most layer of teeth in the crown īƒ˜ Cementum: the outer most layer of teeth in the root īƒ˜ Dentin: the middle layer of teeth, forms the main density of teeth īƒ˜ The pulp cavity: the central part of teeth, contains connective tissue, blood vessels, and nerve 51
  • 52. Sensory innervation and Blood Supply of the jaw īƒ˜ Superior and inferior alveolar arteries/ veins ; branch of maxillary artery supply tooth īƒ˜ Upper jaw= superior alveolar nerves īƒ˜ Lower jaw= inferior alveolar nerves; both make dental plexus – Inferior alveolar nerve: all mandibular teeth, buccal soft tissues of premolar, canine and incisors – Lingual nerve: all lingual soft tissues of all teeth – Long buccal nerve: buccal soft tissue of all molars – Anterior superior alveolar nerve: maxillary incisors and canine, buccal soft tissues of incisors and canines – Middle superior alveolar nerve: maxillary premolar and maxillary bone roots first molar, buccal soft tissue of premolar 52
  • 53. Cont.â€Ļ â€ĸ Post. Sup. alveolar nerve: maxillary molars except a portion of first molar, buccal soft tissue of molars â€ĸ Greater palatine nerve: lingual soft tissues of molars and premolars â€ĸ Nasopalatine nerve: lingual soft tissue of incisors & canines â€ĸ Mandibular premolar region, buccal soft tissue innervated primarily by mental branch of inferior alveolar nerves and also by terminal branch of long buccal nerve 53
  • 54. 54
  • 55. Nomenclature of tooth īƒ˜ Nomenclature is the first step in understanding dental anatomy. īƒ˜ Tooth numbering or “shorthand” system of tooth notation is necessary in clinical practice for recording data and communication. īƒ˜ The various tooth notation systems are as follows: A. Palmer notation system B. Universal notation system C. FDI system (Federation Dentaire Internationale) 55
  • 56. A. Palmer notation system īƒ˜ In 1861 Adolph Zsigmondy of Vienna introduced the symbolic system for permanent dentition. He then modified it for the primary dentition in 1874. īƒ˜ The symbolic system is now commonly referred to as the Palmer notation system or Zsigmondy system. īļPrimary teeth E D C B A │ A B C D E E D C B A │ A B C D E īļPermanent teeth 8 7 6 5 4 3 2 1 │ 1 2 3 4 5 6 7 8 8 7 6 5 4 3 2 1 │ 1 2 3 4 5 6 7 8 56
  • 57. B. Universal notation system īƒ˜ In this system for the permanent dentition the maxillary teeth are numbered through 1 to 16 beginning with upper right third molar. īƒ˜ The mandibular teeth are numbered through 17 to 32 beginning with lower left third molar. īƒ˜ The universal system notation for primary dentition utilizes upper case alphabets. īļ Primary teeth- A B C D E│F G H I J T S R Q P│O N M L K īļ Permanent teeth 1 2 3 4 5 6 7 8│ 9 10 11 12 13 14 15 16 32 31 30 29 28 27 26 25 │ 24 23 22 21 20 19 18 17 57
  • 58. C. FDI system īƒ˜The FDI system is a two digit system that has been adopted by WHO. īƒ˜In this system the first digit indicates the quadrant and the second digit indicates the tooth within the quadrant. īƒ˜1 to 4 and 5 to 8 as the first digit indicates permanent and primary dentition respectively. īƒ˜1 to 8 and 1 to 5 as the second digit indicates permanent and primary teeth respectively. 58
  • 59. 59
  • 60. PERIODONTI CS B Y D R . N H I A L W . ( M D ) 11/19/2021 Dr Nhial 60 Module III
  • 61. 4.Periodontic 11/19/2021 Dr Nhial 61 The specialty of dentistry that studies supporting structures of teeth, diseases, and conditions that affect them.
  • 62. PULP AND PERIRADICULAR DISEASES 11/19/2021 Dr Nhial 62 The Dental Pulp īƒ˜ Consists of richly vascularized and highly innervated connective tissue īƒ˜ It is surrounded by dentin īƒ˜ The pulp tissue is in communication with the periodontium and the rest of the body through the apical foramen and accessory canals near the apex of the root
  • 63. PULPITIS īƒ˜ Is inflammation of the dental pulp resulting from untreated caries, trauma, or multiple restorations. īƒ˜ Pulpitis can occur when caries progresses deeply into the dentin īƒ˜ Trauma disrupts the lymphatic and blood supply to the pulp īƒ˜ Its principal symptom is pain Main Cause 1.Infection: spread of dental caries to the pulp 2. Trauma 3. Physical irritation: excessive heat during cavity preparation. 4. Chemical irritation i.e. filling materials or cavity cleansers such as alcohol, chloroform, hydrogen peroxide 5. Mixed microorganisms which are found in the oral cavity 11/19/2021 Dr Nhial 63
  • 64. 11/19/2021 Dr Nhial 64 ī‚— This image shows how the progression of decay, if not treated, will lead to destruction of the tooth's hard tissue and pulp. ī‚— If treated at the beginning of the decay process (first image), a small filling would resolve the problem. ī‚— If allowed to continue as in the last image, root canal therapy would be necessary. PROGRESSION OF DECAY (CARIES)
  • 65. 11/19/2021 Dr Nhial 65 īƒ˜ When Dental decay reaches a depth in the dentin that is near the pulp tissue inside of the tooth, the pulp tissue can become inflamed (pulpitis)
  • 66. Classification of dental pulp disease 11/19/2021 Dr Nhial 66 Reversible pulpitis ī‚— Tissue is capable of returning to normal state of health if noxious stimuli is removed ī‚— Sudden mild to moderate pain or short duration ī‚— Sweat, sour foods or beverages can cause pain Irreversible pulpitis ī‚— Higher level of inflammation ī‚— Pulp is damaged beyond the point of recovery ī‚— Sharp severe pain upon thermal stimulation ī‚— Pain continues after the stimulus is removed ī‚— Heat & sweat & sour foods elicit pain ī‚— Pain may be spontaneous or continuous
  • 67. Diagnosis and Treatment of Pulpitis 11/19/2021 Dr Nhial 67 īƒ˜Diagnosis is based on clinical findings and is confirmed by x-ray. īƒ˜Treatment involves īƒŧ Removing decay, īƒŧ Restoring the damaged tooth, and īƒŧ Sometimes doing root canal therapy (RCT) or extracting the tooth.
  • 69. Peri-radicular tissue 11/19/2021 Dr Nhial 69 īƒ˜ The tissues surrounding and investing the cervical, middle, and apical regions of the root are the cementum, periodontal ligament, and alveolar bone. Classification of Peri-radicular lesions Classified into four main groups: 1. Acute apical Periodontitis (AAP) 2. Chronic apical Periodontitis (CAP) 3. Acute apical abscess (AAA) 4. Chronic apical abscess (CAA)
  • 70. 1) Acute apical Periodontitis 11/19/2021 Dr Nhial 70 īƒ˜ Etiology īļ Extension of pulpal inflammation into the peri-radicular tissues īļ Irritants: bacteria toxins from necrotic pulps, chemicals (irritants or disinfecting agents) over instrumentation & restorations in hyper occlusion. īƒ˜ S/symptoms īļ moderate to severe spontaneous discomfort as well as pain on mastication or occlusal contact. īļ Application of pressure by fingertip can cause marked pain
  • 71. 11/19/2021 Dr Nhial 71 īƒ˜ Diagnosis īļ Radiography: "Thickening” of periodontal ligament (PDL) space (May be not always) īƒ˜ Treatments īƒŧ Adjustment of occlusion (when there is evidence of hyper occlusion) īƒŧ Removal of irritants or a pathologic pulp, or īƒŧ Release of peri radicular exudates
  • 72. 2) Chronic apical Periodontitis 11/19/2021 Dr Nhial 72 īƒ˜ Etiology: results from pulp necrosis and usually is a sequel to AAP. īƒ˜ S/symptoms: īƒŧ Asymptomatic or slight discomfort īƒŧ Do not respond to electrical or thermal stimuli īƒŧ Percussion produces little or no pain there may be slight sensitivity to palpation, indicating an alteration of the cortical plate of bone and extension of CAP into the soft tissues
  • 73. Radiography image: Well-circumscribed lesions and pear-shaped configuration of periradicular to the canine are typical of CAP. 11/19/2021 Dr Nhial 73
  • 74. 3) Acute apical abscess 11/19/2021 Dr Nhial 74 īļ Etiology: microbial and nonbacterial irritants from necrotic pulp. īļ S/symptoms: īƒŧ Have moderate to severe discomfort or swelling. īƒŧ Systemic manifestations of an infective process such as a high temperature, malaise and leukocytosis īƒŧ Because AAA occurs only with pulp necrosis, electrical or thermal stimulation produces no response īƒŧ However, teeth are painful to percussion and palpation
  • 75. 11/19/2021 Dr Nhial 75 īƒ˜ Radiography: range from thickening of the PDL space (infrequent) to a frank resorptive lesion (usual) īƒ˜ Treatment: Removal of the underlying cause (necrotic pulp), release of pressure (drainage here possible) and root canal treatment īƒ˜ Clinical significance īƒŧ If untreated, a Periapical/apical abscess can develop into osteomyelitis , septicemia, Ludwigs angina, or cavernous sinus thrombosis.
  • 76. 4) Chronic apical abscess (Suppurative Apical periodontitis) 11/19/2021 Dr Nhial 76 īƒ˜ Result of a long-standing lesion that has resulted in an abscess that is draining to a surface īƒ˜ Etiology īƒŧ Pathogenesis similar to that of AAA results from pulp necrosis and is usually associated with chronic apical periodontitis that has formed an abscess īƒŧ The abscess has "burrowed" through bone & soft tissue to form a sinus on the oral mucosa or sometimes onto the skin of the face
  • 77. Contâ€Ļ 11/19/2021 Dr Nhial 77 īļ Signs and symptoms īƒŧ Because drainage exists, CAA is usually asymptomatic except when there is occasional closure of the sinus pathway, which can cause pain īƒŧ Clinical, radiographic, and histopathologic features of CAA are similar to described for CAP. īƒŧ An additional features is the sinus tract
  • 78. A. Apical abscesses occasionally drain extra-orally (blue arrows) B. The tooth was retreated nonsurgical and the chin lesion healed within a few weeks with some scarring. 11/19/2021 Dr Nhial 78
  • 79. 11/19/2021 Dr Nhial 79 Periapical abscess, Apical periodontitis and Cellulitis.
  • 80. 11/19/2021 Dr Nhial 80 Peri-coronitis īƒ˜ Inflammatory process involving soft tissues covering crown of partially erupted or un-erupted teeth. īƒ˜ Develops when food debris & bacteria are present beneath the gingival flap īƒ˜ Usually seen associated with impacted mandibular 3rd molars
  • 81. 11/19/2021 Dr Nhial 81 Clinical features īƒ˜Severe pain īƒ˜Swollen, red, tender gingiva īƒ˜Discomfort in swallowing īƒ˜Restriction of oral opening īƒ˜Foul taste īƒ˜Pain may radiate to ear, floor of mouth or throat īƒ˜Lymphadenopathy īƒ˜Malaise & fever
  • 83. 11/19/2021 Dr Nhial 83 Treatment īƒ˜ Antiseptic lavage under the gingival flap to remove gross food debris & bacteria īƒ˜ Gingival flap is removed surgically if the tooth to be retained īƒ˜ Extraction of tooth if needed īƒ˜ Antibiotics īƒ˜ Warm saline mouth rinse
  • 84. Gingivitis 11/19/2021 Dr Nhial 84 īƒ˜ It is an inflammatory lesion confined to the tissue of the marginal gingiva. Cause: īƒ˜ Accumulation of bacterial plaque at or near the gingival margin. īƒ˜ The bacterial component of plaque produces and releases variety of enzymes and toxins (e.g. lipopolysacchardies ) which diffuse through the junctional epithelium and initiate inflammatory changes in the gingival connective tissues.
  • 85. Contâ€Ļ 11/19/2021 Dr Nhial 85 Clinical features â€ĸ Redness of the gum â€ĸ Gum bleeding â€ĸ edema of the gum â€ĸ Tenderness of the gum Treatment: â€ĸ Oral hygiene â€ĸ Plaque control â€ĸ Oraldine mouth wash â€ĸ Administration of antibiotics
  • 86. Acute Necrotizing Ulcerative Gingivitis (ANUG) 11/19/2021 Dr Nhial 86 īƒ˜Definition: is an inflammatory destructive gingival condition which exhibits characteristics clinical signs and symptoms. īƒ˜The other names for ANUG are "Vincent's gingivitis" or "Vincents gingivostomatitis", 'Trench mouth'' and" Ulcero-membraneous gingivitis“
  • 87. Contâ€Ļ 11/19/2021 Dr Nhial 87 Causes īƒ˜Fusiform bacteria īƒ˜Treponema vincenti īƒ˜Treponema deticola īƒ˜Fusobacterium nucleatum īƒ˜Prevotella intermedia īƒ˜Porphymonas gingivalis NB: These bacteria are found in large numbers in the slough and necrotic tissues at the surface of the ulcer.
  • 88. Contâ€Ļ.. 11/19/2021 Dr Nhial 88 Clinical features īƒ˜ Inter proximal ulcers covered with a yellowish-white or grayish debris īƒ˜ Easily bleed īƒ˜ Necrosis develops rapidly īƒ˜ Linear erythema īƒ˜ Pain īƒ˜ Halitosis īƒ˜ Lymphadenitis īƒ˜ Fever and malaise
  • 89. Contâ€Ļ. 11/19/2021 Dr Nhial 89 Control of the acute phase īƒ˜Antibacterial cleaning īƒ˜Irrigation of the wound with 3% hydrogen peroxide solution īƒ˜Scaling of the affected teeth īƒ˜Antibiotics, Metronidazole īƒ˜2% Chloxeidine mouth wash
  • 91. Module IV Con’tâ€Ļ.. Exodontia īƒ˜Painless removal of the tooth or root with minimal injury to the surrounding soft tissue and bone īƒ˜Removal of the tooth does not require large amount of force. īƒ˜Pain and anxiety control is by: īƒŧLocal anesthesia results in loss of pain, temperature and touch but not pressure 91
  • 92. Cont.â€Ļ Duration of anesthesia īƒ˜ local anesthesia without vasoconstrictors īƒŧMaxillary teeth: 10 - 20 minutes īƒŧMandibular teeth: 40 - 60 minutes īƒŧSoft tissue: 2 – 3 hours īƒ˜Lasts longer in mandible due to the density of the cortical bone of the mandible. 92
  • 93. Cont.â€Ļ īƒ˜Local anesthesia with vasoconstrictor â€ĸ Maxillary teeth: 50 – 60 minutes â€ĸ Mandibular teeth: 90 - 100 minutes â€ĸ Soft tissue: 3 – 4 hours īƒ˜Long acting anesthesia with vasoconstrictor â€ĸ Maxillary teeth: 60 - 90 minutes â€ĸ Mandibular teeth: 3 hours â€ĸ Soft tissue: 4- 9 hours 93
  • 94. Indications for removal of teeth īƒ˜Severe dental caries īƒ˜Pulpal necrosis īƒ˜Severe periodontal disease īƒ˜Mal opposed teeth īƒ˜Orthodontic reasons īƒ˜Cracked teeth or fractured root 94
  • 95. Indications for removal of teeth īƒ˜Pre prosthetic extraction īƒ˜Impacted teeth īƒ˜Supernumerary teeth īƒ˜Teeth associated with pathologic lesion īƒ˜Pre radiation therapy īƒ˜Severe attrition ,abrasion or erosion īƒ˜Teeth involved in jaw fracture 95
  • 96. Contraindications for the removal of teeth īƒ˜ Systemic contraindications oUn controlled diabetes oEnd stage renal disease with severe uremia oUncontrolled leukemia oUncontrolled cardiac disease oUnstable angina pectoris 96
  • 97. Contraindications for the removal of teeth o Recent MI o Severely uncontrolled hypertension o Pregnancy first and last trimester o Bleeding disorders like hemophilia o Platelet disorders o Pt. on anticoagulants NB: Absolute contraindication (arterioveinous fistula and end stage renal disease) 97
  • 98. Local contraindications īƒ˜Tooth in the area of tumor īƒ˜Acute infection īƒ˜Central hemangioma 98
  • 99. Instrument used for extraction 1. Forceps īƒ˜Uses of forceps ī‚§To expand bony sockets ī‚§To remove tooth 2. Elevator : for loosening īƒ˜Straight elevator īƒ˜Periosteal elevator īƒ˜Cryer elevator 99
  • 100. Techniques of extraction 1. Closed extraction īƒ˜Adequate access and visibility needed īƒ˜Steps for closed extraction 1. Loosening of soft tissue attachment from tooth 2. Luxation of tooth with dental elevator 3. Adaptation of forceps to tooth 4. Luxation of tooth with forceps 5. Removal of tooth from socket 100
  • 101. Post extraction care īƒ˜ Curettage if any peri-apical pathology īƒ˜ Curette if any debris, calculus, amalgam, tooth & bone fragment in the socket īƒ˜ Finger pressure is applied to buccal and lingual cortical plates to compress the socket īƒ˜ Moistened 2x2 inch gauze is placed 101
  • 102. Contâ€Ļ 2. Open extraction īƒ˜Indication: īƒŧFailure to remove by closed methods īƒŧUnfavorable root pattern īƒŧFracture or caries extending to root īƒŧHypercementosis īƒŧImpacted tooth 102
  • 103. Complications of exodontia īƒŧ Fracture of crown or roots of the tooth being extracted īƒŧ Fracture of alveolar bone īƒŧ Fracture of maxillary tuberosity īƒŧ Fracture of adjacent or opposing tooth īƒŧ Fracture of mandible īƒŧ Dislocation of TMJ īƒŧ Displacement of root into soft tissues ,maxillary antrum īƒŧ Bleeding 103
  • 104. Contâ€Ļ. īƒŧInjury to gums, lips, IAN & its branch, lingual nerve, tongue, floor of mouth, greater palatine artery īƒŧ Osteomyelitis īƒŧ Infection īƒŧ Trismus īƒŧ Hematoma 104
  • 105. Local anesthesia īƒ˜ Is the loss of sensation in circumscribed area of body caused by depression of excitation in nerve endings or inhibition of conduction process in peripheral nerves. īƒ˜ Produces loss of sensation with out loss of consciousness. 105
  • 106. Contâ€Ļ Advantages of LA: â€ĸ It can be easily mastered and can be given by operator â€ĸ Equipment required is limited in amount â€ĸ Economical and transportable â€ĸ The patency of air way is not impaired â€ĸ Enables the patient to cooperate Contraindications to LA: ī‚§ Acute infection in the injection sites ī‚§ Patients with hemorrhagic disease 106
  • 107. Types of LA 1. Topical. īƒ˜ When applied to intact mucus membrane pass through the epidermis and anaesthetize the nerve endings e.g.. Cocaine, lidocaine. īƒ˜ It can be in the form of sprays, ointmentâ€Ļ 2. Infiltration:- Deposition of LA near the terminal fibers to produce anesthesia to localized areas served by the nerve fibers. - Sub mucous injection - Supra periosteal - Sub periosteal * Mostly used in maxilla 107
  • 108. Contâ€Ļ 3. Regional anesthesia: Anesthetic solution deposited near the nerve trunk & blocks all impulses. It is also known as block anesthesia. ** It is widely used in the mandible since infiltration is unreliable due to the density of the cortical bone of the mandible. 108
  • 109. Summary of maxillary block Posterior Superior Alveolar Nerve Block Maxillary molars (with exception of mesiobuccal root of maxillary 1st molar in some cases), hard and soft tissue on buccal aspect Middle Superior Alveolar Nerve Block Mesiobuccal root of maxillary 1st molar , premolars and surrounding hard and soft tissue on buccal aspect Anterior Superior Alveolar Nerve Block/Infraorbital Nerve Block Maxillary central and lateral incisors and canine, surrounding hard and soft tissue on 109
  • 110. Local anesthesia in Mandible īƒ˜ The regional or block anesthesia in the mandible is achieved by deposition of LA around the inferior alveolar nerve at pterygo mandibular space / Retro molar area For mandibular block: īƒ˜ Mandibular occlusal plane should be horizontal when patient open his mouth īƒ˜ Operator should stand in front of the patient for giving anesthesia to the right side and behind for the left. īƒ˜ The thumb of left hand is passed along the buccal surface of the lower molar teeth until the internal oblique ridge is felt. Then the tip is rolled in ward to lie in retro-molar fossa. The mid point of the nail concedes the land mark for insertion of the needle after preparation 110
  • 111. Contâ€Ļ īƒ˜ The syringe is held parallel to the mandibular occlusal plane over 2nd premolar of the opposite side. īƒ˜ Needle is inserted about 0.5cm to anaesthetize the lingual nerve and advance more 1.5- 2cm to anaesthetize the inferior alveolar nerve and long buccal nerve 111
  • 112. Difficulties and complication of LA 1. Failure to obtain Anesthesia a) Inadequate amount LA b) Poor operater technique c) Missing land marks d) PH of tissue e) Expiration of LA 112
  • 113. Contâ€Ļ 2. Pain during and after injection Reduced by - Sharpe needle - Tensing the tissue - Slow injection 3. Hematoma formation: Occurs most frequently when posterior superior alveolar block given. 4. Intra vascular injection: Frequently the patients feels faint. Has a pale clammy skin, rapid pulse. 5. Trismus: īƒŧ Difficulty of opening jaw due to muscle spasm īƒŧ Injection to medial pterygoid muscle 6. Infection 113
  • 114. Contâ€Ļ 7. Facial paralysis: Injection to the parotid gland 8. Prolonged impairment of sensation due to damage of the nerves. 9. Broken Needles -When needle broken, the tissue must be kept under continuous pressure until the protruding end is grasped by pliers or artery forceps. - If the edge is embedded completely, the patient should be informed and X-ray should be taken to confirm the position and presence. 114
  • 115. Contâ€Ļ 10. Lip trauma: ī‚§ Children chews anaesthetized part. ī‚§ Adults burn from hot drinks 11. Fainting In this instance â€ĸ Head should be lowered â€ĸ Legs elevated â€ĸ Tight clothes & belt loosened â€ĸ Wet swab applied on brow â€ĸ Stimulate respiration 115
  • 116. Reading assignments 1.Maxillofacial trauma 2.Principles of preventive dentistry or Oral health 116
  • 117. 117 Module V Management of Maxillofacial injuries Introduction â€ĸ The facial skeleton is divided into 3 parts â€ĸ The upper 1/3: formed by frontal bone â€ĸ The middle 1/3: from frontal bone to the level of upper teeth. â€ĸ The lower 1/3: the mandible
  • 118. 118 The causes of maxillofacial trauma īƒ˜Fights īƒ˜Falls īƒ˜RTAs īƒ˜Occupational hazards- athletic injury, industrial mishaps īƒ˜Iatrogenic causes- # of tooth, alveolus, maxillary tuberosity, #of mandible during dental treatment.
  • 119. 119 â€ĸ Radiographic examination For middle 1/3 - PA view of the skull - Lateral view of the skull For the mandibular fracture - PA view of the mandible - Right and left lateral view of the mandible
  • 120. 120 Basic principle life preservation in traumatic patient The ABC methods īƒŧ Maintenance of patency of Air way īƒŧ Bleeding control īƒŧ Maintenance of Circulation
  • 121. 121 Factors affecting wound healing īąLocal īƒ˜ Infection īƒ˜ Foreign bodies īƒ˜ mobility īƒ˜ poor vascularity īąSystemic īƒ˜Increased age īƒ˜Disease e.g diabetes īƒ˜Deficiency e.g malnutrition
  • 122. 122 Basic principles of management of fracture 1) Reduction: the restoration of fracture fragments to their original position. â€ĸ It can be closed or open reduction īļ Closed reduction: â€ĸ Can be carried out by manipulation or traction â€ĸ No surgical intervention is needed â€ĸ Occlusion of the teeth is used as a guide line
  • 123. 123 īƒ˜ By manipulation: by digital or hand manipulation - when fragments are adequately mobile - in patients come soon after trauma īƒ˜ By traction: fractured fragments are subjected to gradual elastic traction īļOpen reduction - surgical reduction that allows visual identification of fractured fragments
  • 124. 124 2) Fixation fractured fragments are fixed to prevent displacement and for achieving proper approximation īƒŧ Direct skeletal fixation: by plates or intraosseous wiring īƒŧ Indirect skeletal fixation: by arch bar or intermaxillary fixation
  • 125. 125 3) Immobilization - The fixation device is retained to stabilize the reduced fragments until a bony union takes place. - For maxillary # 3 to 4 weeks - For mandibular # 4 to 6 weeks immobilization. - In case of condylar # 2 to 3 weeks to prevent Ankylosis.
  • 126. 126 Fracture of the middle 1/3 of the face Boundaries of middle 1/3 of the face īƒŧSuperiorly: line from zygomaticofrontal suture, across frontonasal suture, frontomaxillary suture. īƒŧ Inferiorly: occlusal plane upper teeth īƒŧPosteriorly: sphenoethmoidal junction
  • 127. 127 1) Le fort I (horizontal fracture of the maxilla or Guerin's fracture or floating fracture) separation of dentoalveolar part of the maxilla - Horizontal # line above the apices of the teeth at the level of floor of the nose - Usually bilateral - Floating of the palate - Hematoma within the maxillary antrum - Bilateral hematoma of the cheek Classification
  • 128. 128
  • 129. 129 2) Le Fort II Fractures (Low Pyramidal Subzygomatic fracture ) - line crosses pyramidally from nasal bone then frontal process of maxilla then lacrimal bone and infraorbital marigin crosses the zygomatic buttress then move backwards and fracture above maxillary tuberosity - Facial swelling with massive edema - Subconjuctival ecchymosis and diplopia - Dish faced deformity
  • 130. 130 - Infraorbital anesthesia both side - Bilateral hematoma - Retroposed upper dental arch with anterior open bite - Cracked pot sound on percussion of teeth
  • 131. 131 3) Le fort III: (High level #) īƒ˜ Force is in lateral direction īƒ˜ Line runs from nasofrontal region then lacrimal bone then ethemiod bone around the optical canal involving infraorbital fissure then greater wing of the sphenoid then zygomaticofrontal suture, and fracture of both side of the zygomatic arches. Clinical features: īƒ˜ Tenderness and separation at fronto zygomatic suture īƒ˜ Tenderness and deformity of zygomatic arches īƒ˜ Lengthening of face - Depression of ocular levels - Enophtalmos - CSF leak via nose (csf rhinorrhea) - Entire middle 1/3 separated from the cranial base. - Bilateral circumorbital ecchymosis
  • 132. 132 Treatment of middle face fracture: 1. Manual reduction 2. Reduction by traction 3. Open reduction
  • 133. 133 Fracture of the mandible Largest, strongest and heaviest bone of the face Classification: 1)Condylar neck 35% 2)Angle 20% 3)Body 20% 4)Parasymphysis 13% 5)Symphysis 11% 6)Coronoid 1%
  • 134. 134
  • 135. 135 Management 1) Closed reduction - dental wiring or arch bar is used to get the occlusion - for 6 weeks Indication - Non displaced # - Lack of soft tissue over the fracture - # of children with developing tooth bud - Coronoid process fracture
  • 136. 136 2) Open reduction Indications - Displaced fracture - Multiple fracture - Associated mid face fracture - Associated condylar fracture Contraindicated if GA is not advisable, sever comminuted or loss of soft tissue and severe infection.
  • 137. 137 Condylar fracture Classified as: - Non displaced: a crack is seen on radiograph - Deviation: simple angulation b/n condylar neck and ramus - Displacement: overlap b/n condyle and ramus - Dislocation: condylar fragments are pushed anteriorly and medially
  • 138. 138 Clinical feature: - Pain and swelling in the region of TMJ - Limitation of oral opening - Deviation towards the involved side - Blood in external auditory canal - Lack of condylar movement on palpation
  • 139. 139 Complication of maxillofacial fracture īƒ˜Anesthesia īƒ˜Malunion and deformity īƒ˜Infection īƒ˜Derangement of occlusion īƒ˜Ankylosis of TMJ īƒ˜Etcâ€Ļ.
  • 140. 140 The Temporomandibular Joint (TMJ) īƒŧ The Temporomandibular joint (TMJ) is a small joint located in front of the ear where the skull and lower jaw meet. īƒŧ It permits the lower jaw (mandible) to move and function. īƒŧ Between the condyle and the fossa is a disk made of cartilage that acts as a cushion to absorb stress and allows the condyle to move easily when the mouth opens and closes.
  • 141. 141 īƒ˜TMJ disorders are not uncommon and have a variety of symptoms. īļPatients may complain of : īƒŧEaraches īƒŧHeadaches īƒŧLimited ability to open their mouth. īƒŧClicking or cracking sounds in the joint īƒŧFeel pain when opening and closing their mouth.
  • 142. 142 Dislocation of TMJ īƒ˜Excursion of condylar head beyond the anterior limit of articular eminence It can be of:- - Unilateral or bilateral - Chronic or acute
  • 143. 143 Causes īƒ˜Blow on the chin while the mouth is open īƒ˜Excessive pressure on mandible while extraction īƒ˜Vomiting īƒ˜Injudicious use of mouth gag during GA
  • 144. 144 Clinical features - Deviation of the chin - Open bite - Depression in front of the tragus - Inability to close the mouth - Drooling of the saliva - Difficulty in speech and swallowing

Editor's Notes

  1. Most of the tooth mass composed of DENTIN
  2. Superior and inferior alveolar arteries/ veins ; branch of maxillary artery supply tooth Upper jaw= superior alveolar nn Lower jaw= inferior alveolar nn ; both make dental plexus
  3. Fluoride is safe for dental health at low concentration. NaF salt is dietary recommended; lethal dose is 5-10 g; found in tablet form for cavity prevention. Fluoride inhibits the activity of phosphatases. Used for prevention of tooth decay. Fluoride is an inorganic monatomic anion; F-, bitter taste and odorless
  4. Amalgam- is a mixture of mercury and other metals (solid/liquid) used for filling holes in teeth & for making tooth cements - From medieval Latin amalgama Composite- a solid material which is composed of 2/more substances having d/t physical ch/tcs and in which each substance retains its identity while contributing desirable properties to the whole.
  5. Most of the tooth mass composed of DENTIN
  6. Superior and inferior alveolar arteries/ veins ; branch of maxillary artery supply tooth Upper jaw= superior alveolar nn Lower jaw= inferior alveolar nn ; both make dental plexus