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Vascular Intervention Radiology
Varicose Vein & Treatment
DR. NARENDRA SINGH PATEL
MBBS, MD - RADIODIAGNOSIS, FSVIR
WHAT IS VARICOSE VEIN ?
 Varicose veins are dilated tortuous superficial venous channels.
Perforating
veins
Indirect veins-
These consist of small
superficial veins which
penetrate the deep fascia
to connect with vessel in
muscle and in turn end in
Deep vein.
CAUSES – Pathology - RISK FACTORS
 1) Incompetent / malfunction/ diseased venous
valves
 2) dilation of vessels
 3) increase back pressure
 4) disorder - DVT
WHAT HAPPENS
 hemodynamic stress or valve agenesis.
 Decreased compliance and contractility and reduced smooth muscle content.
 Due to weakening of the vein wall, the vein dilates, stretching the valve cusp commissure and
separating the leaflets.
 Dilated saphenous veins undergo valvular remodelling with increased collagen and reduced elastin
content
 Consequently discolouration of skin , dryness, skin cracks then ulceration.
Who should be screened?
 Aching
 •Fatigue, heaviness in legs
 •Pain: throbbing, burning, stabbing
 •Cramping
 •Swelling (peripheral edema)
 •Itching
 •Restless legs
 •Numbness
 •Leg ulcerations –usually on the lower leg could be medial or lateral with typically
acceptable arterial circulation
CEAP classification stands for Clinical (C), Etiological (E),
Anatomical (A), and Pathophysiological (P).
Clinical classification
S: Symptomatic
A: Asymptomatic
C0 No visible or palpable signs of venous disease
C1 - Telangiectasies or reticular veins
C2 - Varicose veins; distinguished from reticular veins by a diameter of 3mm or more
C3 - Edema
C4 - Changes in skin and subcutaneous tissue secondary to CVD
C4a - Pigmentation or eczema
C4b - Lipodermatosclerosis or atrophie blanche
C5 - Healed venous ulcer.
C6 - Active venous ulcer.
The Etiological classification divides into:
Ec: - Congenital
Ep: - Primary
Es: - Secondary
En: - No venous cause identified
Anatomical classification divides into four categories:
As: superficial veins
Ap: perforating veins
Ad: deep veins
An: no venous location identified
pathophysiology classification, divided into four categories:
Pr: Reflux
Po: obstruction
Pr,o: reflux and obstruction
Pn: no venous pathophysiology identifiable
How can we make diagnose ?
 Mapping venous anatomy,
 Identifying anatomic variants ,
 Finding the sources of venous insufficiency.
What are the points to be proven ?
Find out cause of venous insufficiency .
 Points to be role out
 1) DEEP VEIN DISORDERS -
 a) external compression
 b) TOTUOUS ANATOMY
 c) Deep Vein reflux
 d) D V T
 2) superficial vein disorders
 A) dilation
 B) reflux
 C) disorder
Valves distribution
1) infrapopliteal segment
2) femoro-popliteal segment,
3) common femoral vein (CFV) near the inguinal ligament,
4) superficial femoral vein (SFV) just distal to the deep femoral vein (DFV) tributary,
5) popliteal vein (PV) near the adductor hiatus.
Technique
Patients are evaluated in the standing position to ensure maximum venous distention. The patient will need to be able to support
their weight on the opposite leg to participate in maneuvers to elicit reflux.
Slight limb flexion and outward rotation provides optimal visualisation of the great saphenous vein. The entire length of the GSV
is first examined using axial grayscale technique, noting the maximal vein diameter (normally <4 mm). Any varicose tributaries
are then identified and traced distally. Next, the SFJ is assessed for reflux. Color or power doppler imaging are used in
combination with sudden compression and release of distal venous segments (35) to identify sites of reflux. Since color Doppler
imaging often underestimates the degree of venous reflux, pulsed-wave doppler imaging is preferred while performing
compression and release (36).
For assessment of the short saphenous vein, the knee is slightly flexed and the muscles of the thigh are relaxed. Using axial
grayscale technique, the SSV is serially examined from the calf upwards until its termination at the SPJ, again noting the
maximal diameter, and assessing for venous competence of the SPJ. A thigh extension of the SSV is also assessed for reflux if
present. Comprehensive deep venous evaluation must also be performed for detection of DVT and reflux. Chronic DVT findings
may be subtle and manifest as webs, focal wall thickening, or calcification. Persistent or repeated venous obstructions can
contribute to venous hypertension. Perforating veins in the thigh and the leg are lastly examined in transverse and oblique planes
to identify the longitudinal axis of perforator.
GRADE I - incompetence found only during Valsalva’s maneuver on standing position;
GRADE II - incompetence found during Valsalva’s maneuver on standing and supine
positions;
GRADE III - incompetence found spontaneously on standing position;
GRADE IV - incompetence found spontaneously on standing and supine positions.
Choi, J, Hong, S, Park, S, Park, C, Seol, H, Cha, I,
TREATMENT OPTIONS
 VENASEAL CLOSER DEVICE
 LASER ABALATIONS ( BEST in category )
 SURGICAL REMOVAL OF SUPERFICIAL VEIN
 LIGATION OF THE VEIN AND PERFORATORS
 SCLEROTHERAPY
 RADIOFREQUNCY ABALATION
LASER ABALATIONS……….BEST WHY ?
 SHORTEST DURATION PROCEDURE - 2 HOURS WALKIN to WALKOUT
 DAY CARE PROCEDURE – No night emergency .
 NO ADMISSION / IPD -
 LESS COMPLICATED / POST PROCEDURE INFECTION
 LESS ASSOCIATED PERIPROCEDURAL MORBIDITY AND FASTER RECOVERY
 TREAT THE PERFORATORS
HOW DO WE DO IT ???
 HISTORY OF ILLNESS
 PRE- INTERVENTIONAL PREPARATION
 INVESTIGATIONS
 INTRA PROCEDURE CARE
 POST PROCEDURE CARE
HISTORY OF ILLNESS
 DURATION,
 PAST ILNESS,
 TREATMENT DONE BEFORE
 ANY OTHER ASSOCIATED DISEASE eg. DM
 TYPE OF DAILY ROUTINE
PRE - INTERVENTIONAL PREPARATION
 INVESTIGATIONS FOR DIAGNOSIS --
COLOR DOPPLER { REFLUX IN SFJ AND PERFORATORS, MAY BE SPJ,
DILATED GSV WITH SSV }
 COUNSELLING, { I DO NOT USE THE WORD OPERATION }.
 PT, INR, LA SENSITIVITY, OTHER ROUTINE PREPARATIONS, PAC
INTRA - PROCEDURE CARE
 TALK TO PATIENT DURING PROCEDURE
 SPINAL OR LOCAL BLOCK ANAESTHE SIA.
 CAN USE GA in SOS only
 MAKE SURE LUMEN OF VEIN COLLAPSE BY PERI VENULAR
ANAESTHESIA
 SPEED OF PULLING LASER FIBRE IS AS ABALATION
 DO NOT BE IN HURRY …………… TAKE YOUR TIME
 REPEAT USG IF ANY THING LEFT………… AND
POST PROCEDURE CARE
 ADEQUATE COMPRESSION DESSING 2 – 3 DAYS
 WOUND DRESSING ………..
 ADVICES TO THE PATIENT FOR EXERCISE, STOCKINGS
 FOLLOW UP AFTER 3 - 5 DAYS
 IF NEEDED MULTIPLE SITTINGS CAN BE DONE IN CHRONIC CASES.
RESULTS
MORE THAN 100 PATIENTS TREATED
WHEN PATIENT CAME WITH MULTIPLE
ULCERS,,,,,,,
CASE - 1
CASE - 2
WHEN PATIENT CAME WITH MULTIPLE ULCERS,,,,,,,
AFTER TREATMENT
WHEN PATIENT CAME WITH MULTIPLE
ULCERS,,,,,,,
CASE - 3
AFTER TREATMENT
WHEN PATIENT CAME WITH MULTIPLE ULCERS,,,,,,,
CASE - 4
CASE - 5
Lacture  varicose
Lacture  varicose
Lacture  varicose
Lacture  varicose
Lacture  varicose
Lacture  varicose
Lacture  varicose
Lacture  varicose
Lacture  varicose
Lacture  varicose
Lacture  varicose
Lacture  varicose
Lacture  varicose
Lacture  varicose
Lacture  varicose

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Lacture varicose

  • 1. Vascular Intervention Radiology Varicose Vein & Treatment DR. NARENDRA SINGH PATEL MBBS, MD - RADIODIAGNOSIS, FSVIR
  • 2. WHAT IS VARICOSE VEIN ?  Varicose veins are dilated tortuous superficial venous channels.
  • 3.
  • 4. Perforating veins Indirect veins- These consist of small superficial veins which penetrate the deep fascia to connect with vessel in muscle and in turn end in Deep vein.
  • 5. CAUSES – Pathology - RISK FACTORS  1) Incompetent / malfunction/ diseased venous valves  2) dilation of vessels  3) increase back pressure  4) disorder - DVT
  • 6. WHAT HAPPENS  hemodynamic stress or valve agenesis.  Decreased compliance and contractility and reduced smooth muscle content.  Due to weakening of the vein wall, the vein dilates, stretching the valve cusp commissure and separating the leaflets.  Dilated saphenous veins undergo valvular remodelling with increased collagen and reduced elastin content  Consequently discolouration of skin , dryness, skin cracks then ulceration.
  • 7. Who should be screened?  Aching  •Fatigue, heaviness in legs  •Pain: throbbing, burning, stabbing  •Cramping  •Swelling (peripheral edema)  •Itching  •Restless legs  •Numbness  •Leg ulcerations –usually on the lower leg could be medial or lateral with typically acceptable arterial circulation
  • 8.
  • 9. CEAP classification stands for Clinical (C), Etiological (E), Anatomical (A), and Pathophysiological (P). Clinical classification S: Symptomatic A: Asymptomatic C0 No visible or palpable signs of venous disease C1 - Telangiectasies or reticular veins C2 - Varicose veins; distinguished from reticular veins by a diameter of 3mm or more C3 - Edema C4 - Changes in skin and subcutaneous tissue secondary to CVD C4a - Pigmentation or eczema C4b - Lipodermatosclerosis or atrophie blanche C5 - Healed venous ulcer. C6 - Active venous ulcer.
  • 10. The Etiological classification divides into: Ec: - Congenital Ep: - Primary Es: - Secondary En: - No venous cause identified Anatomical classification divides into four categories: As: superficial veins Ap: perforating veins Ad: deep veins An: no venous location identified pathophysiology classification, divided into four categories: Pr: Reflux Po: obstruction Pr,o: reflux and obstruction Pn: no venous pathophysiology identifiable
  • 11.
  • 12. How can we make diagnose ?  Mapping venous anatomy,  Identifying anatomic variants ,  Finding the sources of venous insufficiency.
  • 13. What are the points to be proven ? Find out cause of venous insufficiency .  Points to be role out  1) DEEP VEIN DISORDERS -  a) external compression  b) TOTUOUS ANATOMY  c) Deep Vein reflux  d) D V T  2) superficial vein disorders  A) dilation  B) reflux  C) disorder
  • 14. Valves distribution 1) infrapopliteal segment 2) femoro-popliteal segment, 3) common femoral vein (CFV) near the inguinal ligament, 4) superficial femoral vein (SFV) just distal to the deep femoral vein (DFV) tributary, 5) popliteal vein (PV) near the adductor hiatus.
  • 15. Technique Patients are evaluated in the standing position to ensure maximum venous distention. The patient will need to be able to support their weight on the opposite leg to participate in maneuvers to elicit reflux. Slight limb flexion and outward rotation provides optimal visualisation of the great saphenous vein. The entire length of the GSV is first examined using axial grayscale technique, noting the maximal vein diameter (normally <4 mm). Any varicose tributaries are then identified and traced distally. Next, the SFJ is assessed for reflux. Color or power doppler imaging are used in combination with sudden compression and release of distal venous segments (35) to identify sites of reflux. Since color Doppler imaging often underestimates the degree of venous reflux, pulsed-wave doppler imaging is preferred while performing compression and release (36). For assessment of the short saphenous vein, the knee is slightly flexed and the muscles of the thigh are relaxed. Using axial grayscale technique, the SSV is serially examined from the calf upwards until its termination at the SPJ, again noting the maximal diameter, and assessing for venous competence of the SPJ. A thigh extension of the SSV is also assessed for reflux if present. Comprehensive deep venous evaluation must also be performed for detection of DVT and reflux. Chronic DVT findings may be subtle and manifest as webs, focal wall thickening, or calcification. Persistent or repeated venous obstructions can contribute to venous hypertension. Perforating veins in the thigh and the leg are lastly examined in transverse and oblique planes to identify the longitudinal axis of perforator.
  • 16.
  • 17. GRADE I - incompetence found only during Valsalva’s maneuver on standing position; GRADE II - incompetence found during Valsalva’s maneuver on standing and supine positions; GRADE III - incompetence found spontaneously on standing position; GRADE IV - incompetence found spontaneously on standing and supine positions. Choi, J, Hong, S, Park, S, Park, C, Seol, H, Cha, I,
  • 18. TREATMENT OPTIONS  VENASEAL CLOSER DEVICE  LASER ABALATIONS ( BEST in category )  SURGICAL REMOVAL OF SUPERFICIAL VEIN  LIGATION OF THE VEIN AND PERFORATORS  SCLEROTHERAPY  RADIOFREQUNCY ABALATION
  • 19. LASER ABALATIONS……….BEST WHY ?  SHORTEST DURATION PROCEDURE - 2 HOURS WALKIN to WALKOUT  DAY CARE PROCEDURE – No night emergency .  NO ADMISSION / IPD -  LESS COMPLICATED / POST PROCEDURE INFECTION  LESS ASSOCIATED PERIPROCEDURAL MORBIDITY AND FASTER RECOVERY  TREAT THE PERFORATORS
  • 20. HOW DO WE DO IT ???  HISTORY OF ILLNESS  PRE- INTERVENTIONAL PREPARATION  INVESTIGATIONS  INTRA PROCEDURE CARE  POST PROCEDURE CARE
  • 21. HISTORY OF ILLNESS  DURATION,  PAST ILNESS,  TREATMENT DONE BEFORE  ANY OTHER ASSOCIATED DISEASE eg. DM  TYPE OF DAILY ROUTINE
  • 22. PRE - INTERVENTIONAL PREPARATION  INVESTIGATIONS FOR DIAGNOSIS -- COLOR DOPPLER { REFLUX IN SFJ AND PERFORATORS, MAY BE SPJ, DILATED GSV WITH SSV }  COUNSELLING, { I DO NOT USE THE WORD OPERATION }.  PT, INR, LA SENSITIVITY, OTHER ROUTINE PREPARATIONS, PAC
  • 23. INTRA - PROCEDURE CARE  TALK TO PATIENT DURING PROCEDURE  SPINAL OR LOCAL BLOCK ANAESTHE SIA.  CAN USE GA in SOS only  MAKE SURE LUMEN OF VEIN COLLAPSE BY PERI VENULAR ANAESTHESIA  SPEED OF PULLING LASER FIBRE IS AS ABALATION  DO NOT BE IN HURRY …………… TAKE YOUR TIME  REPEAT USG IF ANY THING LEFT………… AND
  • 24.
  • 25.
  • 26. POST PROCEDURE CARE  ADEQUATE COMPRESSION DESSING 2 – 3 DAYS  WOUND DRESSING ………..  ADVICES TO THE PATIENT FOR EXERCISE, STOCKINGS  FOLLOW UP AFTER 3 - 5 DAYS  IF NEEDED MULTIPLE SITTINGS CAN BE DONE IN CHRONIC CASES.
  • 27. RESULTS MORE THAN 100 PATIENTS TREATED WHEN PATIENT CAME WITH MULTIPLE ULCERS,,,,,,, CASE - 1
  • 28. CASE - 2 WHEN PATIENT CAME WITH MULTIPLE ULCERS,,,,,,,
  • 30. WHEN PATIENT CAME WITH MULTIPLE ULCERS,,,,,,, CASE - 3
  • 32. WHEN PATIENT CAME WITH MULTIPLE ULCERS,,,,,,, CASE - 4
  • 33.

Editor's Notes

  1. The most common of these syndromes is left iliac vein compression by the right iliac artery, known as May-Thurner syndrome (MTS). 
  2. These factors are exacerbated by muscle pump dysfunction, most notably of the calf muscles. These mechanisms serve to produce global or regional venous hypertension, particularly with standing or ambulation. Contributing to the macrocirculatory hemodynamic disturbances are alterations within the microcirculation.24,25 Unabated venous hypertension may result in dermal changes with hyperpigmentation; subcutaneous tissue fibrosis, termed “lipodermatosclerosis”; and eventual ulceration.
  3. Calf muscle contraction elevates the pressure of the deep venous compartment of leg to approximately 140 mmHg, propelling venous blood into the popliteal and femoral veins (10,12). During muscle relaxation, the pressure gradient is reversed and causes physiological reflux, lasting approximately 200 to 300 milliseconds in veins with competent valves.