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PULP INFLAMMATION
By / Ahmed Elgamal
DEFINITION
 Dental pulp is the soft CT of the tooth that supports
and surrounded by dentin
DENTAL PULP ZONES
The odontoblastic zone
Consists of :
 Cell body
 Odontoblastic process
which extend into predentin matrix
The cell free zone
Contain :
 Blood capillaries
 very small nerve fibers
Function ?!!
The cell rich zone
Contain :
 Blood vessels & nerves
The central zone
Contains :
 Cells embedded in collagen matrix
 Blood vessels
 Lymphatics
 Nerves
Ahmed Elgamal
FUNCTION OF DENTAL PULP
Induction
Pulp induce dentine formation which induce enamel formation
Dentin formation
Nutrition
Supplies nutrients essential for dentin via DT
Defense
 Form tertiary dentin in response to trauma
 Immunologic inflammatory reaction
Sensation
Pulp is innervated by both sensory and autonomic nerve
fibers
Causes of
pulpitis
mechanical
traumatic iatrogenic attrition abrasion
thermal
Metallic
R
Cavity
prep
chemical
erosion
Acidic
materials
bacterial
Open
cavity
Periapical
inf
CLASSIFICATION
pulpitis
involvement
involvement
partial
total
severity
acute
chronic
Communication
between pulp &
oral
environment
Open
pulpitis
Closed
pulpitis
severity
reversible irreversible Pulp
degeneration
Pulp
necrosis
REVERSIBLE PULPITIS
Early mild transient pulpitis Called pulp hyperemia
stimuli
- Dilatation of pulpal BV
- Collection of edematous fluid
- increase pressure inside pulp
pain
REVERSIBLE PULPITIS
Clinical features
 Sensitive to thermal change ( COLD )
 Sharp pain lasting for a moment
 Disappears after removal of stimuli
 No history of spontaneous pain
 No sensitivity to percussion
 teeth show :
( deep caries & large metallic R & restoration with defective margins )
MANAGEMENT
Diagnosis should be confirmed by a cold test
Remove the cause before the pulp is severely damaged
If the lesion is untreated , the duration of pain
upon stimulation can become IRREVERSIBLE
PULPITIS
IRREVERSIBLE PULPITIS
Why an inflamed pulp might be
symptomatic or asymptomatic ?
 Persistent inflammatory condition of pulp
 Caused by ( chemical , thermal , mechanical , bacterial )
 Pressure increases because of lack of escape of
inflammatory exudate
ACUTE PULPITIS
ACUTE PULPITIS
Early stage
Pain with cold and
relived by heat
Continuous &
spontaneous sever pain
even after removing
stimulus
Pain with lying down
due to change in
intrapulpal pressure
Late stage
More sever and throbbing
pain even the tooth under
constant pressure
Pt. awake at night due
to pain
pain with heat and may
relived by cold
Clinical features
 Pain may be referred
EX : lower molars referring pain to the ear
Why
Pt. has pain with cold in case of reversible and
early stage of irreversible pulpitis ?! but in case of
late stage has pain with hot ?!
Pt. has pain with sweets ?!
Referred pain with acute pulpitis and not
with reversible pulpitis ?!
CLASSIFICATION OF CHRONIC PULPITIS
Chronic closed
pulpitis
Chronic open
ulcerative
pulpitis
Chronic open
hyperplastic
pulpitis
Chronic open
pulpitis
 There are fibroblastic activity
 Newly formed capillaries
 Deposition of collagen fibers
( as a local protective barrier to wall the infection )
 Pulp abscess may present with granulation tissue
Chronic pulpitis
CHRONIC OPEN ULCERATIVE PULPITIS
 Entire or the greater part of pulp shows chronic
inflammatory changes
 The pulp is exposed to outside ( no dentine roof )
 Edema escapes through the exposure
 Minimum dull pain that become worse by thermal
changes
CHRONIC HYPERPLASTIC PULPITIS
 Also called pulp polyp
 Essentially an excessive exuberant proliferation of
chronically inflamed dental pulp tissue
 Development of granulation tissue
 Covered at time by epithelium
 The tooth is VITAL
CAUSES
 Long standing low grade infection
 extensive carious exposure of a young pulp
 Bacterial infection
CLINICAL FEATURES
 Most commonly involved are primary molars & 1st
permanent molar >> due to
 Excellent blood supply
 Large root opening
 Asymptomatic except of feeling of pressure during
mastication
 in children and young adults
 Tooth may or may not respond to thermal test
 Polypoid tissue appears
Fleshy
Reddish pulpal mass filling most of pulpal
chamber
May interferes with mouth closure
Easily bleeds ( rich blood vessels )
The cause of pain in case of
necrotic pulp ?!
Dental Pulp Inflammation Causes and Types

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Dental Pulp Inflammation Causes and Types

  • 1. PULP INFLAMMATION By / Ahmed Elgamal
  • 2. DEFINITION  Dental pulp is the soft CT of the tooth that supports and surrounded by dentin
  • 3. DENTAL PULP ZONES The odontoblastic zone Consists of :  Cell body  Odontoblastic process which extend into predentin matrix The cell free zone Contain :  Blood capillaries  very small nerve fibers Function ?!! The cell rich zone Contain :  Blood vessels & nerves The central zone Contains :  Cells embedded in collagen matrix  Blood vessels  Lymphatics  Nerves Ahmed Elgamal
  • 4. FUNCTION OF DENTAL PULP Induction Pulp induce dentine formation which induce enamel formation Dentin formation Nutrition Supplies nutrients essential for dentin via DT Defense  Form tertiary dentin in response to trauma  Immunologic inflammatory reaction Sensation Pulp is innervated by both sensory and autonomic nerve fibers
  • 5.
  • 6.
  • 7.
  • 8. Causes of pulpitis mechanical traumatic iatrogenic attrition abrasion thermal Metallic R Cavity prep chemical erosion Acidic materials bacterial Open cavity Periapical inf
  • 10. REVERSIBLE PULPITIS Early mild transient pulpitis Called pulp hyperemia stimuli - Dilatation of pulpal BV - Collection of edematous fluid - increase pressure inside pulp pain
  • 11. REVERSIBLE PULPITIS Clinical features  Sensitive to thermal change ( COLD )  Sharp pain lasting for a moment  Disappears after removal of stimuli  No history of spontaneous pain  No sensitivity to percussion  teeth show : ( deep caries & large metallic R & restoration with defective margins )
  • 12. MANAGEMENT Diagnosis should be confirmed by a cold test Remove the cause before the pulp is severely damaged If the lesion is untreated , the duration of pain upon stimulation can become IRREVERSIBLE PULPITIS
  • 13. IRREVERSIBLE PULPITIS Why an inflamed pulp might be symptomatic or asymptomatic ?
  • 14.  Persistent inflammatory condition of pulp  Caused by ( chemical , thermal , mechanical , bacterial )  Pressure increases because of lack of escape of inflammatory exudate ACUTE PULPITIS
  • 15. ACUTE PULPITIS Early stage Pain with cold and relived by heat Continuous & spontaneous sever pain even after removing stimulus Pain with lying down due to change in intrapulpal pressure Late stage More sever and throbbing pain even the tooth under constant pressure Pt. awake at night due to pain pain with heat and may relived by cold Clinical features  Pain may be referred EX : lower molars referring pain to the ear
  • 16. Why Pt. has pain with cold in case of reversible and early stage of irreversible pulpitis ?! but in case of late stage has pain with hot ?! Pt. has pain with sweets ?! Referred pain with acute pulpitis and not with reversible pulpitis ?!
  • 17.
  • 18.
  • 19.
  • 20.
  • 21. CLASSIFICATION OF CHRONIC PULPITIS Chronic closed pulpitis Chronic open ulcerative pulpitis Chronic open hyperplastic pulpitis Chronic open pulpitis
  • 22.
  • 23.  There are fibroblastic activity  Newly formed capillaries  Deposition of collagen fibers ( as a local protective barrier to wall the infection )  Pulp abscess may present with granulation tissue Chronic pulpitis
  • 24.
  • 25.
  • 26. CHRONIC OPEN ULCERATIVE PULPITIS  Entire or the greater part of pulp shows chronic inflammatory changes  The pulp is exposed to outside ( no dentine roof )  Edema escapes through the exposure  Minimum dull pain that become worse by thermal changes
  • 27. CHRONIC HYPERPLASTIC PULPITIS  Also called pulp polyp  Essentially an excessive exuberant proliferation of chronically inflamed dental pulp tissue  Development of granulation tissue  Covered at time by epithelium  The tooth is VITAL
  • 28. CAUSES  Long standing low grade infection  extensive carious exposure of a young pulp  Bacterial infection
  • 29. CLINICAL FEATURES  Most commonly involved are primary molars & 1st permanent molar >> due to  Excellent blood supply  Large root opening  Asymptomatic except of feeling of pressure during mastication  in children and young adults  Tooth may or may not respond to thermal test
  • 30.  Polypoid tissue appears Fleshy Reddish pulpal mass filling most of pulpal chamber May interferes with mouth closure Easily bleeds ( rich blood vessels )
  • 31.
  • 32.
  • 33.
  • 34.
  • 35.
  • 36.
  • 37.
  • 38.
  • 39.
  • 40.
  • 41. The cause of pain in case of necrotic pulp ?!