1. OCCUPATIONAL HEALTH
Dr.Siva.P.M
Junior Resident
Community medicine
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2. INTRODUCTION
⢠Occupational health :
â promotion and maintenance of highest degree of physical, mental and
social well being of workers
â All three levels of prevention
â Adaptation of work to man and vice versa
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3. Ergonomics â ergon â work ; nomos â law
⢠Fitting job to the worker
⢠Objective â achieve mutual adjustment of man and his work for
improvement of human efficiency and well being
⢠To reduce industrial accidents and increase efficiency of workers
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4. ⢠Occupational environment
⢠Sum of external conditions and influences âprevail at the place of
work âbearing on health of workers
⢠3 interactions
â Man and agents(Physical, Chemical, Biological)
â Man and machine
â Man and man
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9. ⢠Noise
â Auditory effects â hearing loss
â Non auditory â nervousness, fatigue, interfere with communication
â Degree of injury â intensity & frequency range, duration of
exposure, susceptibility
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11. ⢠Ionizing radiation
â X rays and radio active isotopes(cobalt 60, phosphorous 32)
â Bone marrow â more sensitive
â Genetic
changes, malformation, cancer, leukemia, depilation, ulceration, sterilit
y, death
â Maximum permissible â 5 rem/year to whole body
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12. CHEMICAL HAZARDS
⢠Acts in three ways â local action, inhalation, ingestion
⢠Local action
â Dermatitis, eczema, ulcer, cancer (irritant action)
â Aromatic amino and nitro compounds â systemic
⢠Inhalation
â Respirable dust - <5 microns
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15. ⢠Ingestion
â Lead, antimony, cadmium, mercury, phosphorous, chromium, zinc etc
â through â contaminated hand or food or cigarettes
⢠Biological hazards
â Brucellosis, leptospirosis, anthrax, hydatidosis, psittacosis, tetanus, enc
ephalitis, fungal infections, schistosomiasis
â Working among animal products
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16. ⢠Mechanical hazards
â Machinery, protruding and moving parts
â 10% of accidents
⢠Psychosocial hazards
â Failure to adapt to an alien psychosocial environment
â Frustration, lack of job satisfaction, insecurity, poor human
relationship, emotional tension â affect physical and mental health
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17. OCCUPATIONAL DISEASES
⢠Pneumoconiosis
⢠Dust â 0.5 to 3 microns â health hazard- variable period of exposure â lung
disease â pneumoconiosis
⢠Hazardous effect depends on
â Chemical composition
â Fineness
â Concentration
â Health status of the person
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18. ⢠Important dust diseases
â silicosis
â anthracosis
â byssinosis
â bagassosis
â asbestosis
â farmerâs lung
⢠No cure â only prevention essential
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19. SILICOSIS (NOTIFIABLE- FACTORIES ACT
1948 AND MINES ACT 1952)
⢠Major cause of disability and mortality
⢠Inhalation of dust with â silica or silicon dioxide
⢠Higher concentration â higher hazard
⢠Incubation period â few months to 6 years
⢠Particles â phagocytosis â accumulation â block lymph channels
⢠Dense nodular fibrosis â 3-4mm
⢠c/f â insidious onset â irritant cough, dyspnoea, chest pain
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20. ⢠Decreased TLC â advanced disease
⢠X ray â snow-storm appearance
⢠Prone to PTB â silico-tuberculosis
⢠No effective treatment
⢠Fibrotic â cant reversed
⢠Rigorous dust control
⢠Regular physical examination by doctors
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21. ANTHRACOSIS(NOTIFIABLE-INDIAN MINES ACT
1952, COMPENSATABLE-WORKMENâS
COMPENSATION ACT 1959
⢠First phase â simple pneumoconiosis â ventilatory impairment â after
12year exposure â anthracite coal dust
⢠Second phase â
â progressive massive fibrosis
â severe respiratory disability
â premature death
⢠Once simple pneumoconiosis
â progress even without further exposure
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22. BYSSINOSIS
⢠Inhalation of cotton fiber
⢠Chronic cough â progressive dyspnoea â chronic bronchitis â emphysema
⢠India has large textile industry â 35% of factory workers
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23. BAGASSOSIS
⢠Inhalation of bagasse or sugar-cane dust
⢠Thermophilic actinomycete â thermoactinomyces sacchari
⢠Breathlessness, cough, haemoptysis, slight fever
⢠Acute diffuse bronchiolitis â initial
⢠Skiagram â mottling in lungs or shadow
⢠Resolution of inflammationâ if treated early
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25. â Personal protection
⢠Masks
⢠Mechanical filters
â Medical control
⢠Initial and periodical medical examinations
â Bagasse control
⢠Keeping moisture >20%
⢠Spraying bagasse with 2% propionic acid- fungicide
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26. ASBESTOSIS
⢠Fibrous material â commercial name
⢠Silicates of varying composition of bases (Mg,Fe,Ca,Na,Al)
⢠Two types â
â Serpentine or chrysolite (90% production)
â Amphibole
⢠Crocidolite (blue)
⢠Amosite (brown)
⢠Anthrophyllite(white)
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27. ⢠Inhalation â ROE
⢠Insoluble fibers â pulmonary fibrosis â insufficiency â death
⢠Carcinoma of bronchus (high-if add with smoking)
⢠Mesothelioma of pleura or peritoneum(crocidolite)
⢠Disease doesnât appear â until 5-10 yrs of exposure
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28. ⢠Irritation â fibrosis â peribronchial â diffuse âbasal in location
⢠Dyspnoea â out of proportion to clinical signs
⢠Clubbing â cardiac distress â cyanosis
⢠Sputum â Asbestos Bodies(fibers coated with fibrin)
⢠X ray â ground glass appearance â lower third of lung fields
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29. ⢠Preventive measures
â Use of safer type asbestos
â Substitution of other insulants
â Rigorous dust control
â Periodic examination of workers
â Continuing research
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30. FARMERâS LUNG
⢠Inhalation of mouldy hay grain dust
⢠>30% moisture
â favors bacteria, fungi growth
â rise temperature 40-50 deg C
â favors Thermophilic actinomycete
⢠Micropolyspora faeni â main cause
⢠Repeated attacks â pulmonary fibrosis â damage - corpulmonale
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31. CONCLUSION
⢠Pneumoconiosis â one of the occupational disease â due to dust inhalation
⢠No specific treatment
⢠Prevention is important
⢠Periodical examination and preplacement examination of workers also
important
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