5. Standardizati
on
ï Usual 1 mV = 10 mm
ï In special cases ECG may be intentionally recorded at one-half
standardization (1 mV =5mm) or two times normal standardization
(1 mV = 20 mm). However, overlooking this change in gain may lead
to the mistaken diagnosis of low or high voltage.
6. Rhythm
ï Sinus rhythm
ï bradycardia or tachycardia
ï SR with APBs orVPBs
ï SR with AV block
ï Nonsinus:PSVT),Afib or flutter,VT and AV junctional escape
7. Sinus rhythm ï Discrete P waves that are always positive (upright) in lead II (and
negative in aVR
8. Heart Rate
ï Normally, the ventricular (QRS) rate and atrial (P) rates are the
same (1:1 AV conduction)
ï Tachycardia >100
ï Bradycardia <60
ï Irregular
ï Regularly irregular :Wenchebachâs
ï Irregularly irregular :Fib
9. PR Interval
ï The normal PR interval (measured from the beginning of the P
wave to the beginning of the QRS complex) is 0.12 to 0.2 sec
ï First-degree AV block
ï A short PR interval with sinus rhythm and with a wide QRS
complex and a delta wave is seen in theWolff-Parkinson-White
(WPW) pattern
ï A short PR interval with retrograde P waves (negative in lead II)
generally indicates an ectopic (atrial or AV junctional) pacemaker.
10. P wave
ï Normal not exceed 2.5 mm in amplitude and is less than 3 mm
(120 ms) wide in all leads
ï Tall, peaked P waves may be a sign of right atrial overload (P
pulmonale)
ï Wide (and sometimes notched P) waves are seen with left atrial
abnormality.
11. QRS Interval
ï 0.1 sec (100 ms) or less, measured by eye
ï 110 ms if measured by computer
12. QT/QTc
Interval
ï Shortened :hyperkalaemia and digitalis effect
ï Prolonged:hypocalcemia or hypokalemia, drug effects (quinidine,
procainamide, amiodarone, or sotalol), or myocardial ischemia
15. R wave
progression
ï Inspect leadsV1 toV6
ï Normal increase in R/S ratio occurs as you move across the chest
ï Poor: (small or absent R waves in leadsV1 toV3)
ï AWMI
ï The term reversed R wave progression
ï Tall R waves in leadV1 that progressively decrease in
amplitude:RVH, posterior (or posterolateral) infarction, and
dextrocardia
17. UWave
ï U Waves Look for prominent U waves.These waves, usually most
apparent in chest leadsV2-V4, may be a sign of hypokalemia or
drug effect or toxicity (e.g., ami-odarone ami-odarone, dofetilide,
quinidine, or sotalol).
20. (1) standardizationâ10 mm/mV; 25 mm/sec
(2) rhythmânormal sinus
(3) heart rateâ75 beats/min
(4) PR intervalâ0.16 sec
(5) P wavesânormal size
(6) QRS widthâ0.08 sec (normal)
(7) QT intervalâ0.4 sec (slightly prolonged for rate)
(8) QRS voltageânormal
(9) QRS axisâabout 30° (biphasic QRS complex in lead II with positive QRS complex
in lead I)
(10) R wave progression:early precordial transition with relatively tall R wave in lead
V2
(11) abnormal Q wavesâleads II, III, and aVF
(12) ST segments: elevated in leads II, III, aVF,V4,V5, andV6 slightly depressed in
leads V1 and V2
(13)T wavesâinverted in leads II, III, aVF, andV3 throughV6
(14) U wavesânot prominent. Impression:This ECG is consistent with an
inferolateral (or infero-posterolateral) wall myocardial infarction of indeterminate
age, possibly recent or evolving. Comment:The relatively tall R wave in lead V2 could
reflect loss of lateral potentials or actual posterior wall involvement
EXAMPLE
22. What ECG
findings may
be present in
pulmonary
embolus?
ï Sinus tachycardia (the most common ECG finding)
ï Right atrial enlargement (P pulmonale)âtall P waves in the
inferior leads
ï Right axis deviation
ï T wave inversions in leadsV1-V2
ï Incomplete right bundle branch block (IRBBB)
ï S1Q3T3 patternâan S wave in lead I, a Q wave in lead III, and an
invertedT wave in lead III.Although this is only occasionally seen
with pulmonary embolus, it is quite suggestive that a pulmonary
embolus has occurred.