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The pericardium
and
the pericardial sinuses
Pericardium:

           (Peri-around, Cardium-heart)


 It is a double-walled, fluid filled sac.

 It contains the heart and the juxtacardiac parts of its
  great vessels(the aorta, the vena cava and the
  pulmonary artery).
Functions of the pericardium:

 Keeps the heart contained in the thoracic
  cavity(cardiac seat belt).

 Prevents over-expanding of heart when blood
  volume increases.

 Limits the heart‟s movements.

    Acts as a shock absorber with the help of the
    fluid filled sac.
Inner
                                visceral
              Inner Serous
                  layer
                                 Outer
Pericardium                     Parietal

                  Outer
                              Single Layer
              Fibrous layer
FIBROUS PERICARDIUM:
 The fibrous pericardium is a sac made of
  tough connective tissue
 It is roughly conical and clothes the heart.

Attachments:
 Superiorly, it is continuous with the adventitia
  of the great vessels and also the pre-tracheal
  fascia.

    Inferiorly, it is attached the the central tendon
    of the diaphragm and a small muscular part of
    its left side.
     Anteriorly, it is attached to the posterior
    surface of the sternum by superior and inferior
    sternopericardial ligaments. The extents of
    these ligaments are extremely variable and the
    suerior one is often undetectable.

     The pericardium is securely anchored by
    these attachments and maintains the general
    thoracic position of the heart, serving as the
    cardiac seat belt‟.
Relations:

Anteriorly,
   Seperated from the thoracic wall by the lungs
  and the pleural coverings.
    But,in a small area behind the lower left
  halfof the body of the sternum and the sternal
  ends of the left 4th and 5th costal cartilages , the
  pericardium is in direct contact with the thoracic
  wall.
    Until it regresses, the lower end of the
  thymus is anterior to the upper part.
Posteriorly,
   The principal bronchi, the esophagus, the
  esophageal plexus, the descending thoracic
  aorta, and the posterior parts of the mediastinal
  surface of both lungs.

Laterally,
     Pleural coverings of the mediastinal surface
   of the lungs.
     The phrenic nerve with its accompaning
   vessels, descends between the mediastinal
   pleura and the fibrous pericardium on either
   side.
Inferiorly,
 the pericardium is seperated from the liver and
   the fundus of stomach by the diaphragm.


The aorta, the superior vena cava, the pulmonary
arteries and veins receive extensions of the fibrous
pericardium except the inferior vena cava which
traverses the central tendon.
SEROSAL PERICARDIUM:
   It is closed sac within the fibrous
  pericardium and has a visceral and a parietal
  layer.
    The visceral layer or the epicardium covers
  the heart and the great vessels and is
  reflected into the parietal layer which lines the
  inner surface of the fibrous pericardium.
    The reflections of the serosal layer are
  arranged as two complex tubes : the aorta
  and the pulmonary trunk are enclosed min
  one and the superior and inferior vena cavae
  and the pulmonary veins in the other.
   The tube surrounding the veins has an
  inverted J shape.
   The cul-de-sac within its curve is behind the
  left atrium and is termed the „OBLIQUE SINUS‟.
   The passage between the two pericardial
  tubes is termed the „TRANSVERSE SINUS‟.
Vascular supply and lymphatic
drainage:
• The arteries are derived from the internal
  thoracic, the musculophrenic arteries and the
  descending thoracic aorta.

• The veins are tributaries of the azygous
  system.

Innervation:
• The pericarduium is innervated by the
  vagus, together with the phrenic nerves and
  the sympathetic trunks
Applied aspects:

Pericardial effusion:
•          Accumulation of excess fluid in the
  pericardial space.
•          When this obstructs the beating of
  heart, it is termed cardiac tamponade.
•          Symptoms are severe edema, low
  BP, shortness of breath, dizziness, chest
  pain, cough, rapid pulse.
•          Causes are inflammation, rheumatoid
  arthritis, surgery, cancer, infection, kidney
  failure, hemorrhage, trauma or idiopathic.
•   Treatment: Giving NSAIDS, excess fluid drained
    using a needle or in severe cases, surgery.

PERICARDITIS:
•   Inflammation of the pericardium.

•       Infections that can cause pericarditis include
    viral infections, bacterial
    infections, tuberculosis, and fungal infections.
    Patients with AIDS frequently develop infections
    that produce pericarditis.
      Autoimmune disorders that can cause
    pericarditis include rheumatoid
    arthritis, lupus, and scleroderma.

 Pericarditis occurs in up to 15% of patients who
  have acute myocardial infarctions (heart
  attacks). There is also a late form of post-heart-
  attack pericarditis, called Dressler‟s
  syndrome, that occurs weeks to months after
  the heart attack.

• Some of the drugs that can produce pericarditis
  include
  procainamide, hydralazine, phenytoin, and
•     Many forms of cancer can metastasize (spread) to
  the pericardial sac, and produce pericarditis.In many
  cases, no definite cause for pericarditis can be
  identified - this is called “idiopathic" pericarditis.”
•     The most common symptom caused by pericarditis
  is chest pain. The pain can severe, and is often made
  worse by changing position or with deep breathing.
  Patients can also have shortness of breath, or fever.
• Pericarditis can produce complications, namely
  tamponade, chronic pericarditis, and constriction.
  These complications - which are discussed below –
  can produce reduced cardiac pumping, lung
  congestion, and organ failure.
•     Acute pericarditis is treated by a) identifying the
    underlying cause, b) treating the underlying cause,
    c) giving anti-inflammatory drugs (to reduce
    inflammation and help prevent chronic problems),
    and d) giving analgesics to control the pain. Most
    cases of acute pericarditis resolve within a few
    weeks, and leave no permanent cardiac problems.

•     Tamponade is treated by draining the fluid from
     the pericardial sac, usually via a tiny catheter.
     Removing the fluid relieves the pressure on the
     heart, and restores normal cardiac function almost
     immediately.
•        Chronic pericarditis is treated by identifying and
    treating the underlying cause, if possible. If
    recurrent pericardial effusions become a
    problem, surgery can be done to create a
    permanent opening that allows the fluid to drain
    from the pericardial sac, thus preventing
    tamponade.

•        Constrictive pericarditis is a very difficult
    therapeutic problem. Symptoms can be treated with
    bed rest, diuretics, and digitalis, but definitive
    treatment requires surgery to strip the thickened
    pericardial lining from the heart. This surgery is
    usually quite difficult
The pericardium and the pericardial sinuses

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The pericardium and the pericardial sinuses

  • 2. Pericardium: (Peri-around, Cardium-heart)  It is a double-walled, fluid filled sac.  It contains the heart and the juxtacardiac parts of its great vessels(the aorta, the vena cava and the pulmonary artery).
  • 3. Functions of the pericardium:  Keeps the heart contained in the thoracic cavity(cardiac seat belt).  Prevents over-expanding of heart when blood volume increases.  Limits the heart‟s movements.  Acts as a shock absorber with the help of the fluid filled sac.
  • 4. Inner visceral Inner Serous layer Outer Pericardium Parietal Outer Single Layer Fibrous layer
  • 5.
  • 6. FIBROUS PERICARDIUM:  The fibrous pericardium is a sac made of tough connective tissue  It is roughly conical and clothes the heart. Attachments:  Superiorly, it is continuous with the adventitia of the great vessels and also the pre-tracheal fascia.  Inferiorly, it is attached the the central tendon of the diaphragm and a small muscular part of its left side.
  • 7. Anteriorly, it is attached to the posterior surface of the sternum by superior and inferior sternopericardial ligaments. The extents of these ligaments are extremely variable and the suerior one is often undetectable.  The pericardium is securely anchored by these attachments and maintains the general thoracic position of the heart, serving as the cardiac seat belt‟.
  • 8. Relations: Anteriorly,  Seperated from the thoracic wall by the lungs and the pleural coverings.  But,in a small area behind the lower left halfof the body of the sternum and the sternal ends of the left 4th and 5th costal cartilages , the pericardium is in direct contact with the thoracic wall.  Until it regresses, the lower end of the thymus is anterior to the upper part.
  • 9. Posteriorly,  The principal bronchi, the esophagus, the esophageal plexus, the descending thoracic aorta, and the posterior parts of the mediastinal surface of both lungs. Laterally,  Pleural coverings of the mediastinal surface of the lungs.  The phrenic nerve with its accompaning vessels, descends between the mediastinal pleura and the fibrous pericardium on either side.
  • 10. Inferiorly,  the pericardium is seperated from the liver and the fundus of stomach by the diaphragm. The aorta, the superior vena cava, the pulmonary arteries and veins receive extensions of the fibrous pericardium except the inferior vena cava which traverses the central tendon.
  • 11.
  • 12. SEROSAL PERICARDIUM:  It is closed sac within the fibrous pericardium and has a visceral and a parietal layer.  The visceral layer or the epicardium covers the heart and the great vessels and is reflected into the parietal layer which lines the inner surface of the fibrous pericardium.  The reflections of the serosal layer are arranged as two complex tubes : the aorta and the pulmonary trunk are enclosed min one and the superior and inferior vena cavae and the pulmonary veins in the other.
  • 13. The tube surrounding the veins has an inverted J shape.  The cul-de-sac within its curve is behind the left atrium and is termed the „OBLIQUE SINUS‟.  The passage between the two pericardial tubes is termed the „TRANSVERSE SINUS‟.
  • 14.
  • 15.
  • 16. Vascular supply and lymphatic drainage: • The arteries are derived from the internal thoracic, the musculophrenic arteries and the descending thoracic aorta. • The veins are tributaries of the azygous system. Innervation: • The pericarduium is innervated by the vagus, together with the phrenic nerves and the sympathetic trunks
  • 17. Applied aspects: Pericardial effusion: • Accumulation of excess fluid in the pericardial space. • When this obstructs the beating of heart, it is termed cardiac tamponade. • Symptoms are severe edema, low BP, shortness of breath, dizziness, chest pain, cough, rapid pulse. • Causes are inflammation, rheumatoid arthritis, surgery, cancer, infection, kidney failure, hemorrhage, trauma or idiopathic.
  • 18.
  • 19. Treatment: Giving NSAIDS, excess fluid drained using a needle or in severe cases, surgery. PERICARDITIS: • Inflammation of the pericardium. • Infections that can cause pericarditis include viral infections, bacterial infections, tuberculosis, and fungal infections. Patients with AIDS frequently develop infections that produce pericarditis.
  • 20.
  • 21. Autoimmune disorders that can cause pericarditis include rheumatoid arthritis, lupus, and scleroderma.  Pericarditis occurs in up to 15% of patients who have acute myocardial infarctions (heart attacks). There is also a late form of post-heart- attack pericarditis, called Dressler‟s syndrome, that occurs weeks to months after the heart attack. • Some of the drugs that can produce pericarditis include procainamide, hydralazine, phenytoin, and
  • 22. Many forms of cancer can metastasize (spread) to the pericardial sac, and produce pericarditis.In many cases, no definite cause for pericarditis can be identified - this is called “idiopathic" pericarditis.” • The most common symptom caused by pericarditis is chest pain. The pain can severe, and is often made worse by changing position or with deep breathing. Patients can also have shortness of breath, or fever. • Pericarditis can produce complications, namely tamponade, chronic pericarditis, and constriction. These complications - which are discussed below – can produce reduced cardiac pumping, lung congestion, and organ failure.
  • 23. Acute pericarditis is treated by a) identifying the underlying cause, b) treating the underlying cause, c) giving anti-inflammatory drugs (to reduce inflammation and help prevent chronic problems), and d) giving analgesics to control the pain. Most cases of acute pericarditis resolve within a few weeks, and leave no permanent cardiac problems. • Tamponade is treated by draining the fluid from the pericardial sac, usually via a tiny catheter. Removing the fluid relieves the pressure on the heart, and restores normal cardiac function almost immediately.
  • 24. Chronic pericarditis is treated by identifying and treating the underlying cause, if possible. If recurrent pericardial effusions become a problem, surgery can be done to create a permanent opening that allows the fluid to drain from the pericardial sac, thus preventing tamponade. • Constrictive pericarditis is a very difficult therapeutic problem. Symptoms can be treated with bed rest, diuretics, and digitalis, but definitive treatment requires surgery to strip the thickened pericardial lining from the heart. This surgery is usually quite difficult