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MEDIASTINUM
MEDIASTINUM
   DEFINITION


   BOUNDRIES

   DIVISIONS


   NORMAL LINES & STRIPES
NORMAL ANATOMY
TECHNIQUE

 HIGH KVP TECH [120 ]--above 120
  coefficient of x ray absorption of bone &
  soft tissue approach each other at high kvp
  & so lungs are not obscured by bones.
--better penetration of mediastnum
--short exposure-less scatter radiation ,sharp
  detail outline of structure is obtained.


   WEDGE FILTER
Normal mediastinum on plain
            radiograph
* Rt mediastinum above azygous vein is formed
  by right brachiocephalic vein &s v c.
*In case of aortic or brachiocephalic artery
 ectasia or unfolding either these veins will be
 pushed laterally or mediastinal border is formed
 by aorta or bracheocephalic artery.
*Right paratrcheal stripe-between tracheal air
 column & lung . < 5 mm .
*azygous vein-outlined by air in lung at the lower
  end of paratracheal stipe.
* Oesophageal-pleural stripe -lung posterior to
  trachea contact rt wall of oesophagus.if
  oesophagus at this level contain air ,rt wall of
  oesophagus is seen as this stripe.
*Azygo-oesophageal recess—on rt side below
  azygous arch the rt lower lobe make contact
  with rt wall of esophagus & azygous vein .and
  the interface is known as azygo esophageal
  stripe.
---concave to rt side-normal
----convex to rt side—abnormal.subcarinal mass,
       left atrial enlargement
Normal mediastinum
   Paraspinal lines –lymph nodes and intercostal
    veins occupy this space between spine lung,
    Normal paraspinal stripe—1 to 2 mm wide.
   Aortico pulmonary window—pleura covering
    the angle between mid portion of aortic arch &
    main pulm artery & left pulm artery is----
    Aorticopulmonary mediastinal stripe.
   Aorticopulmonary window is sensitive place to
    look for lymph node enlargement.
The junctional lines
   Anteror junctional line –when small amount of
    fat anterior to ascending aorta,two lungs may
    be seperated by more than four layers of
    pleura. Never extend lower than where it
    envelopes rt outflow tract.
   Posterior junctional line—lungs are close
    together behind the oesophagus forms this
    line.line seperates to envelope the aortic arch
.
    When the lines are seen ,excludes mass or
     space occupying process at junctional areas
DIVISIONS OF MEDIASTINUM
COMMON CONDITIONS
APPROACH
   MEDIASTNUM WIDENING OR
    NORMAL

   PARENCHYMAL OR MEDIASTNAL

   ANTERIOR ,MIDDLE OR POSTEROR
    COMPARTMENT

   VASCULAR OR NONVASCULAR

   ORGAN OF ORIGIN.
Mediastinal /parenchymal
Mediastinal mass
    No air bronchogram
   Margins with lung obtuse
   Mediastinal lines will be disrupted.

   Can be associated spinal ,costal or sternal
    abnormalities
DIFF SIGN FOR
          LOCALISATION
   SILHOUETTE SIGN

   HILUM OVERLAY SIGN

   HILUM CONVERGENCE SIGN

   CERVICO-THORACIC SIGN

   THORACO ABDOMINAL SIGN
Silhouette sign
                              I
   If an intrathoracic opacity is situated in
    anatomic contact with a border of heart or
    aorta ,will obscure that border.
    A radioopacity causes obliteration of rt border of
    heart , is anterior in location ,Anterior
     mediastinum.
    If it overlaps but does not obliterates , it lies in
    posterior or middle mediastinum.
Silhouette sign
Hilum overlay sign
   Differentiates cardic enlargement from
    mediastinal mass
   In mediastinal mass if Hilar vessels are seen
     through the mass indicates that the mass does
     not arise from hilum
   For accuracy the film should be true frontal
    ,slight obliquity may project normal pulmonary
     artery medialy
Hilum overlay sign
Cervicothoracic sign

Well defined mass seen above the clavicle is
always situated in posterior compartment ,the
anterior compartment mass being in contact
with soft tissue rather than aerated lung is ill
defined
Cervicothoracic sign
Hilum convergence sign
Anterior mediastinal masses
   Displaced anterior junctional lines
   Obliterated cardiophrenic angle
   Obliterated retrosternal clear space
   Hilum overlay sign
ANTERIOR MEDIASTINUM
   RETROSTERNAL         PLEUROPERICARD
    GOITER                IAL CYST
   TORTUOS              MORGAGNI
    INNOMINATE            HERNIA
    ARTERY
   FAT DEPOSITION
                         STERNAL MASSES
   ENLARGED LYMPH
                         PARATHYROID
    NODES                 ADENOMA
   ASCENDING AORTA
    ANEURYSM
   THYMOMA
   GERM CELL
    TUMOUR
Ant mediastinal mass--lymphoma
Retrosternal goiter
Retrosternal goiter
ASCENDING AORTA ANEURYSM
Thymoma
Thymoma
Benign teratoma
Middle mediastinal masses
   Widened paratrcheal stripes
   Displaced azygo oesophageal recess on right
    side
   Mass on posterior trachea
   Lateral 'doughnut '
MIDDLE MEDIASTINUM
   LYMPH NODE          TRACHEAL
    ENLARGEMENT          LESIONS
   AORTIC ARCH         CARDIAC
    ANEURYSM             TUMOURS
   ENLARGED PULM
    ARTERY
   DILATATION OF
    SVC

   BRONCHOGENSTIC
Anterior and middle mediastinal
   lymph node enlargement
Tuberculosis
Pulm hypertension / sarcoidosis
Thoracic aorta aneurysm
Recurrent bronchogenic cyst
Esophageal duplication cyst
POSTERIOR MEDIASTINUM
   NEUROGENIC         PARAVETEBRAL
    TUMORS              MASSES
   PHARYNGO           NEUREENTERIC
    ESOPHAGEAL          CYSTS
    POUCH              BOCHDALEK
   HIATUS HERNIA       HERNIA
   DESCENDING         PANCREATIC
    AORTA               PSEUDOCYST
    ANEURYSM           ABCESSES ,
   OESOPHAGEAL         FIBROSIS
    DIALATION
Neuroenteric cyst
Neuroenteric cyst
Bochdalek's hernia
More than one compartment
   Since no tissue plane in diff compartments in
    some conditions multiple comp are involved
   Enlarged lymph nodes.
   Mediastinitis
   Hematomas
   Vascular entities
   Bronchogenic cancer
   Metastates
   lymphangiomas
Characterization of mass on C T
     Does it contain fat
     Does it contain fluid?
     Does it enhance following the
      administration of i v contrast
Thank you
Cystic hygroma
             TECHNIQUE
                           Transverse images of thin
                           slices of lung (1 to 1.5 mm
                           thick) are obtained at non-
                           contiguous intervals,
 In routine CT, slices 3 to 10 mm thickcm apart,
                           usually 1 to 2 are obtained
                           throughout.
       contiguously, imaging 100% of the lung
HRCT
   Mediastinal window: C/W = 35/350
   Lung window: C/W = -450/1350
   Bone window: C/W = 500/1800
NORMAL ANATOMY
The junctional lines
Cystic hygroma
Germ cell tumour
Thymic cyst
NORMAL VARIANT
   Azygous Lobe:-
      Area of lung medial to the azygous fissure
BronchoPulmonarySegments
                     Right Lung 10 Segments
   Upper Lobe :- apical
                    anterior
                  posterior
   Middle Lobe :- medial
                    lateral
   Lower Lobe :- superior
                    anterior basal
                    medial basal
                    lateral basal
                    posterior basal
BronchoPulmonarySegments




                        Lef superior
                             inferior
   Lower lobe :- superior
                  anterior basal
                  lateral basal
VASCULAR ANATOMY
   The bronchial arteries provide systemic blood
    to the lung tissue
   Arising from the aorta; supply nearly all lung
    tissue
   The alveoli are supplied by the pulmonary
    circulation
Secondary Pulmonary Lobule
INDICATIONS OF HRCT
Descending aorta aneurysm
Consolidation
Posterior mediastinum
BRONCHIECTASIS
EMPHYSEMA
   Abnormal permanent enlargement of air
    spaces distal to terminal bronchioles with
    destruction of alveolar walls without obvious
    fibrosis
EMPHYSEMA
   Types

   Centilobular
   Panacinar
   Paraseptal or subpleural
EMPHYSEMA
EMPHYSEMA
INTERLOBULAR SEPTAL
            THICKENING
   Seen in patients with interstitial lung disease.

   Types :- smooth
              nodular / irregular
INTERLOBULAR SEPTAL
     THICKENING
SEPTAL THICKENING
             (SMOOTH) 
   Pulmonary edema
   Lymphangitic spread of carcinoma
   Amyloidosis
SEPTAL THICKENING
       (NODULAR/IRREGULAR)
   Lymphangitic spread of carcinoma
   Pulmonary Fibrosis
   Rare: amyloidosis & asbestosis
   Sarcoidosis
NODULES

   Smallest diameter detected 1-2 mm

   Classification

    Appearance - well-defined (likely interstitial)
                  ill-defined (likely air-space)
          Distribution - centrilobular perilymphatic, or
    random
CENTRILOBAR
               MICRONODULES
   Centrilobular nodules can be identified in close
    association to pulmonary artery branches

   Centrilobular nodules are often centered 5-10 mm
    from the pleural surface

   Centrilobular nodules are usually of similar size and
    spaced at regular distances from each other.
CENTRILOBAR
            MICRONODULES
   Endobronchial tuberculosis
   Any bronchopneumonia
   Endobronchial spread of timor
   Silicosis or Coal workers’ pneumoconiosis
PERILYMPHATIC NODULES

   Perilymphatic nodules are usually well-defined
    and occur in relation to the lymphatics. They
    often affect the pleural surfaces and the
    peribronchovascular, interlobular septa, and
    centrilobular interstitial components.
PERILYMPHATIC
            MICRONODULES
   Sarcoidosis
   Lymphangitic spread of carcinoma
   Silicosis & coal workers’ pneumoconiosis
   Lymphoid interstitial pneumonitis (rare)
   Amyloidosis (rare)
    
RANDOMLY DISTRIBUTED
            NODULES
   Random nodules are usually well-defined and
    appear diffuse, but uniform in distribution.
RANDOMLY DISTRIBUTED
         MICRONODULES
   Miliary tuberculosis
   Fungal infections
   Hematogenous metastasis
   Sarcoidosis (when diffuse)
TREE-IN-BUD
   "Tree-in-bud" appearance represents dilated and
    fluid-filled (i.e. pus, mucus, or inflammatory
    exudate) centrilobular bronchioles.

   Abnormal "tree-in-bud" bronchioles appear more
    irregular , lack of tapering or knobby/bulbous
    appearance at the tip of their branches.
TREE-IN-BUD
   Infection: Tuberculosis, mycobacterium avium
    intracellulare complex (MAC), bacterial,
    fungal
   Airway disease (i.e. cystic fibrosis or
    bronchiectasis)
   Allergic bronchopulmonary aspergilosis (rare)
Ground-glass Opacity
   Ground-glass , increased hazy opacity within
    the lungs that is not associated with obscured
    underlying vessels .

    Minimal thickening of the septal or alveolar
    interstitium, thickening of alveolar walls, or
    the presense of cells or fluid filling the alveolar
    spaces.
GROUND GLASS OPACITY
            (ACUTE)
   Pulmonary edema
   Hemorrhage
   Pneumocystic pneumonia (PCP)
   Acute interstitial pneumonia
   Hypersensitivity pneumonia
   Early idiopathic pulmonary fibrosis (IPF)
GROUND GLASS OPACITY
           (CHRONIC)
   Desquamative interstitial pneumonitis
   Idiopathic pulmonary fibrosis
   Alveolar proteinosis
   Hypersensitivity pneumonitis - chronic
   Sarcoidosis
   Lipoid pneumonia
   Bronchoalveolar carcinoma
MOSAIC ATTENUATION

   Decreased attenuation which results from regional
    differences in lung perfusion secondary to airway
    disease or pulmonary vascular disease.
   Distribution is often patch, hence the designation
    "mosaic."
   Pulmonary arteries will be reduced in size in the
    lucent lung fields thus allowing mosaic perfusion to
    be distinguished from ground-glass opacities.
MOSAIC ATTENUATION
   Bronchiolitis Obliterans
   Cystic fibrosis
   Chronic PE
AIR TRAPPING

   Abnormal retention of gas within the lung
    following expiration.
   On HRCT, the lung parencyhma remains
    lucent on expiration, while normal lung areas
    show increased attenuation.
   Inspiration scans can be completely normal in
    air trapping.
AIR TRAPPING
   Obliterative bronchiolitis
   Asthma
   Hypersensitivity pneumonitis
   Normal variant (seen in superior segement of
    left lobe, middle lobe or lingula)
HONEYCOMBING

   Honeycombing
        extensive lung fibrosis
        alveolar destruction
        cystic appearance on gross pathology.
    Honeycombing
    presence of thich-walled, air-filled cysts,
    usually between the size of 3mm to 1cm in
    diameter.
HONEYCOMBING
   Interstitial fibrosis (IPF, RA, scleroderma,
    drug reaction, asbestosis, end stage
    hypersensitivity pneumonitis)
   End stage sarcoidosis
APPROACH TO DIAGNOSIS
An acute appearance suggests pulmonary edema
 or pneumonia
APPROACH TO DIAGNOSIS
Reticulonodular lower lung predominant
 distribution with decreased lung volumes
 suggests: (APC)
1. Asbestosis
2. Aspiration (chronic)
3. Pulmonary fibrosis (idiopathic)
4. Collagen vascular disease
APPROACH TO DIAGNOSIS
  A middle or upper lung predominant
   distribution suggests:
1. Mycobacterial or fungal disease
2. Silicosis
3. Sarcoidosis
4. Langerhans Cell Histiocytosis
APPROACH TO DIAGNOSIS
Associated lymphadenopathy suggests :
1. Sarcoidosis
2. Neoplasm (lymphangitic carcinomatosis,
   lymphoma, metastases)
3. Infection (viral, mycobacterial, or fungal)
4. Silicosis
APPROACH TO DIAGNOSIS
   Associated pleural thickening and/or
    calcification suggest asbestosis.
APPROACH TO DIAGNOSIS
Associated pleural effusion suggests :
1. Pulmonary edema
2. Lymphangitic carcinomatosis
3. Lymphoma
4. Collagen vascular disease


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Mediastnum ppt

  • 2. MEDIASTINUM  DEFINITION  BOUNDRIES  DIVISIONS  NORMAL LINES & STRIPES
  • 4. TECHNIQUE  HIGH KVP TECH [120 ]--above 120 coefficient of x ray absorption of bone & soft tissue approach each other at high kvp & so lungs are not obscured by bones. --better penetration of mediastnum --short exposure-less scatter radiation ,sharp detail outline of structure is obtained.  WEDGE FILTER
  • 5. Normal mediastinum on plain radiograph * Rt mediastinum above azygous vein is formed by right brachiocephalic vein &s v c. *In case of aortic or brachiocephalic artery ectasia or unfolding either these veins will be pushed laterally or mediastinal border is formed by aorta or bracheocephalic artery. *Right paratrcheal stripe-between tracheal air column & lung . < 5 mm . *azygous vein-outlined by air in lung at the lower end of paratracheal stipe.
  • 6. * Oesophageal-pleural stripe -lung posterior to trachea contact rt wall of oesophagus.if oesophagus at this level contain air ,rt wall of oesophagus is seen as this stripe. *Azygo-oesophageal recess—on rt side below azygous arch the rt lower lobe make contact with rt wall of esophagus & azygous vein .and the interface is known as azygo esophageal stripe. ---concave to rt side-normal ----convex to rt side—abnormal.subcarinal mass, left atrial enlargement
  • 7. Normal mediastinum  Paraspinal lines –lymph nodes and intercostal veins occupy this space between spine lung,  Normal paraspinal stripe—1 to 2 mm wide.  Aortico pulmonary window—pleura covering the angle between mid portion of aortic arch & main pulm artery & left pulm artery is---- Aorticopulmonary mediastinal stripe.  Aorticopulmonary window is sensitive place to look for lymph node enlargement.
  • 8. The junctional lines  Anteror junctional line –when small amount of fat anterior to ascending aorta,two lungs may be seperated by more than four layers of pleura. Never extend lower than where it envelopes rt outflow tract.  Posterior junctional line—lungs are close together behind the oesophagus forms this line.line seperates to envelope the aortic arch .  When the lines are seen ,excludes mass or space occupying process at junctional areas
  • 11. APPROACH  MEDIASTNUM WIDENING OR NORMAL  PARENCHYMAL OR MEDIASTNAL  ANTERIOR ,MIDDLE OR POSTEROR COMPARTMENT  VASCULAR OR NONVASCULAR  ORGAN OF ORIGIN.
  • 13. Mediastinal mass  No air bronchogram  Margins with lung obtuse  Mediastinal lines will be disrupted.  Can be associated spinal ,costal or sternal abnormalities
  • 14. DIFF SIGN FOR LOCALISATION  SILHOUETTE SIGN  HILUM OVERLAY SIGN  HILUM CONVERGENCE SIGN  CERVICO-THORACIC SIGN  THORACO ABDOMINAL SIGN
  • 15. Silhouette sign I  If an intrathoracic opacity is situated in anatomic contact with a border of heart or aorta ,will obscure that border. A radioopacity causes obliteration of rt border of heart , is anterior in location ,Anterior mediastinum. If it overlaps but does not obliterates , it lies in posterior or middle mediastinum.
  • 17. Hilum overlay sign  Differentiates cardic enlargement from mediastinal mass  In mediastinal mass if Hilar vessels are seen through the mass indicates that the mass does not arise from hilum  For accuracy the film should be true frontal ,slight obliquity may project normal pulmonary artery medialy
  • 19. Cervicothoracic sign Well defined mass seen above the clavicle is always situated in posterior compartment ,the anterior compartment mass being in contact with soft tissue rather than aerated lung is ill defined
  • 22. Anterior mediastinal masses  Displaced anterior junctional lines  Obliterated cardiophrenic angle  Obliterated retrosternal clear space  Hilum overlay sign
  • 23. ANTERIOR MEDIASTINUM  RETROSTERNAL  PLEUROPERICARD GOITER IAL CYST  TORTUOS  MORGAGNI INNOMINATE HERNIA ARTERY  FAT DEPOSITION  STERNAL MASSES  ENLARGED LYMPH  PARATHYROID NODES ADENOMA  ASCENDING AORTA ANEURYSM  THYMOMA  GERM CELL TUMOUR
  • 31. Middle mediastinal masses  Widened paratrcheal stripes  Displaced azygo oesophageal recess on right side  Mass on posterior trachea  Lateral 'doughnut '
  • 32. MIDDLE MEDIASTINUM  LYMPH NODE  TRACHEAL ENLARGEMENT LESIONS  AORTIC ARCH  CARDIAC ANEURYSM TUMOURS  ENLARGED PULM ARTERY  DILATATION OF SVC  BRONCHOGENSTIC
  • 33. Anterior and middle mediastinal lymph node enlargement
  • 35. Pulm hypertension / sarcoidosis
  • 39. POSTERIOR MEDIASTINUM  NEUROGENIC  PARAVETEBRAL TUMORS MASSES  PHARYNGO  NEUREENTERIC ESOPHAGEAL CYSTS POUCH  BOCHDALEK  HIATUS HERNIA HERNIA  DESCENDING  PANCREATIC AORTA PSEUDOCYST ANEURYSM  ABCESSES ,  OESOPHAGEAL FIBROSIS DIALATION
  • 43. More than one compartment  Since no tissue plane in diff compartments in some conditions multiple comp are involved  Enlarged lymph nodes.  Mediastinitis  Hematomas  Vascular entities  Bronchogenic cancer  Metastates  lymphangiomas
  • 44. Characterization of mass on C T  Does it contain fat  Does it contain fluid?  Does it enhance following the administration of i v contrast
  • 46. Cystic hygroma TECHNIQUE  Transverse images of thin slices of lung (1 to 1.5 mm thick) are obtained at non- contiguous intervals,  In routine CT, slices 3 to 10 mm thickcm apart, usually 1 to 2 are obtained throughout. contiguously, imaging 100% of the lung
  • 47. HRCT  Mediastinal window: C/W = 35/350  Lung window: C/W = -450/1350  Bone window: C/W = 500/1800
  • 53. NORMAL VARIANT  Azygous Lobe:- Area of lung medial to the azygous fissure
  • 54. BronchoPulmonarySegments Right Lung 10 Segments  Upper Lobe :- apical anterior  posterior  Middle Lobe :- medial lateral  Lower Lobe :- superior anterior basal medial basal lateral basal posterior basal
  • 55. BronchoPulmonarySegments Lef superior inferior  Lower lobe :- superior anterior basal lateral basal
  • 57. The bronchial arteries provide systemic blood to the lung tissue  Arising from the aorta; supply nearly all lung tissue  The alveoli are supplied by the pulmonary circulation
  • 63.
  • 65. EMPHYSEMA  Abnormal permanent enlargement of air spaces distal to terminal bronchioles with destruction of alveolar walls without obvious fibrosis
  • 66. EMPHYSEMA  Types  Centilobular  Panacinar  Paraseptal or subpleural
  • 69. INTERLOBULAR SEPTAL THICKENING  Seen in patients with interstitial lung disease.  Types :- smooth nodular / irregular
  • 70. INTERLOBULAR SEPTAL THICKENING
  • 71. SEPTAL THICKENING (SMOOTH)   Pulmonary edema  Lymphangitic spread of carcinoma  Amyloidosis
  • 72. SEPTAL THICKENING (NODULAR/IRREGULAR)  Lymphangitic spread of carcinoma  Pulmonary Fibrosis  Rare: amyloidosis & asbestosis  Sarcoidosis
  • 73. NODULES  Smallest diameter detected 1-2 mm  Classification Appearance - well-defined (likely interstitial) ill-defined (likely air-space) Distribution - centrilobular perilymphatic, or random
  • 74. CENTRILOBAR MICRONODULES  Centrilobular nodules can be identified in close association to pulmonary artery branches  Centrilobular nodules are often centered 5-10 mm from the pleural surface  Centrilobular nodules are usually of similar size and spaced at regular distances from each other.
  • 75.
  • 76. CENTRILOBAR MICRONODULES  Endobronchial tuberculosis  Any bronchopneumonia  Endobronchial spread of timor  Silicosis or Coal workers’ pneumoconiosis
  • 77. PERILYMPHATIC NODULES  Perilymphatic nodules are usually well-defined and occur in relation to the lymphatics. They often affect the pleural surfaces and the peribronchovascular, interlobular septa, and centrilobular interstitial components.
  • 78.
  • 79. PERILYMPHATIC MICRONODULES  Sarcoidosis  Lymphangitic spread of carcinoma  Silicosis & coal workers’ pneumoconiosis  Lymphoid interstitial pneumonitis (rare)  Amyloidosis (rare)   
  • 80. RANDOMLY DISTRIBUTED NODULES  Random nodules are usually well-defined and appear diffuse, but uniform in distribution.
  • 81.
  • 82. RANDOMLY DISTRIBUTED MICRONODULES  Miliary tuberculosis  Fungal infections  Hematogenous metastasis  Sarcoidosis (when diffuse)
  • 83. TREE-IN-BUD  "Tree-in-bud" appearance represents dilated and fluid-filled (i.e. pus, mucus, or inflammatory exudate) centrilobular bronchioles.  Abnormal "tree-in-bud" bronchioles appear more irregular , lack of tapering or knobby/bulbous appearance at the tip of their branches.
  • 84.
  • 85. TREE-IN-BUD  Infection: Tuberculosis, mycobacterium avium intracellulare complex (MAC), bacterial, fungal  Airway disease (i.e. cystic fibrosis or bronchiectasis)  Allergic bronchopulmonary aspergilosis (rare)
  • 86. Ground-glass Opacity  Ground-glass , increased hazy opacity within the lungs that is not associated with obscured underlying vessels .  Minimal thickening of the septal or alveolar interstitium, thickening of alveolar walls, or the presense of cells or fluid filling the alveolar spaces.
  • 87.
  • 88. GROUND GLASS OPACITY (ACUTE)  Pulmonary edema  Hemorrhage  Pneumocystic pneumonia (PCP)  Acute interstitial pneumonia  Hypersensitivity pneumonia  Early idiopathic pulmonary fibrosis (IPF)
  • 89. GROUND GLASS OPACITY (CHRONIC)  Desquamative interstitial pneumonitis  Idiopathic pulmonary fibrosis  Alveolar proteinosis  Hypersensitivity pneumonitis - chronic  Sarcoidosis  Lipoid pneumonia  Bronchoalveolar carcinoma
  • 90. MOSAIC ATTENUATION  Decreased attenuation which results from regional differences in lung perfusion secondary to airway disease or pulmonary vascular disease.  Distribution is often patch, hence the designation "mosaic."  Pulmonary arteries will be reduced in size in the lucent lung fields thus allowing mosaic perfusion to be distinguished from ground-glass opacities.
  • 91.
  • 92. MOSAIC ATTENUATION  Bronchiolitis Obliterans  Cystic fibrosis  Chronic PE
  • 93. AIR TRAPPING  Abnormal retention of gas within the lung following expiration.  On HRCT, the lung parencyhma remains lucent on expiration, while normal lung areas show increased attenuation.  Inspiration scans can be completely normal in air trapping.
  • 94.
  • 95.
  • 96. AIR TRAPPING  Obliterative bronchiolitis  Asthma  Hypersensitivity pneumonitis  Normal variant (seen in superior segement of left lobe, middle lobe or lingula)
  • 97. HONEYCOMBING  Honeycombing extensive lung fibrosis alveolar destruction cystic appearance on gross pathology. Honeycombing presence of thich-walled, air-filled cysts, usually between the size of 3mm to 1cm in diameter.
  • 98.
  • 99. HONEYCOMBING  Interstitial fibrosis (IPF, RA, scleroderma, drug reaction, asbestosis, end stage hypersensitivity pneumonitis)  End stage sarcoidosis
  • 100. APPROACH TO DIAGNOSIS An acute appearance suggests pulmonary edema or pneumonia
  • 101. APPROACH TO DIAGNOSIS Reticulonodular lower lung predominant distribution with decreased lung volumes suggests: (APC) 1. Asbestosis 2. Aspiration (chronic) 3. Pulmonary fibrosis (idiopathic) 4. Collagen vascular disease
  • 102. APPROACH TO DIAGNOSIS  A middle or upper lung predominant distribution suggests: 1. Mycobacterial or fungal disease 2. Silicosis 3. Sarcoidosis 4. Langerhans Cell Histiocytosis
  • 103. APPROACH TO DIAGNOSIS Associated lymphadenopathy suggests : 1. Sarcoidosis 2. Neoplasm (lymphangitic carcinomatosis, lymphoma, metastases) 3. Infection (viral, mycobacterial, or fungal) 4. Silicosis
  • 104. APPROACH TO DIAGNOSIS  Associated pleural thickening and/or calcification suggest asbestosis.
  • 105. APPROACH TO DIAGNOSIS Associated pleural effusion suggests : 1. Pulmonary edema 2. Lymphangitic carcinomatosis 3. Lymphoma 4. Collagen vascular disease
  • 106.

Hinweis der Redaktion

  1. Images are usually displayed using &quot;lung windows,&quot; in which air appears black, aerated lung dark grey, and other structures white
  2. This image of normal left lung shows central, branching pulmonary arteries and bronchi. The bronchovascular bundles are made up of these paired structures and their surrounding interstitium (connective tissue). In cross section, the bronchus is a thin-walled, white circle with central air (black), and the adjacent artery appears as a solid, white circle. More peripherally, numerous small &quot;dots&quot; and a few branching lines represent small pulmonary arteries and veins. Throughout, arteries branch at acute angles, and veins branch at 90° angles. The pleura of the major interlobar fissure is a thin, horizontal line traversing the lung. The peripheral pleural surface, which cannot be seen, is smooth.
  3. This image shows complete opacification of most of the left upper lobe. Vessels are not visible in this area. When the bronchi remain aerated, they are seen as branching lucencies called air-bronchograms, which are present in this image. This image represents infectious pneumonia, which is limited by the major fissure, resulting in a sharp border. The advancing anteromedial margin shows ground-glass opacity
  4. This image shows patchy ground-glass opacities throughout both lungs. Note that in the regions of ground-glass, one can see the vessels.