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HOW READ CHEST XR -10




     ANAS SAHLE ,MD
Brief review
POSITION
                             PA                                                   AP


                                                       QUALITY
                ROTATION                               PENETRATION                      INSPIRATION




                                                        LESION
OPACIT
 OPACITY
                           Homo
                       Heterogenous     Wellill defined           Zone
                                                                            Centralperipher
                                                                                               Silhouet sign
                                                                                   al
   Y                       Necrotic
  PATCHY

                                             HILUMMEDIASTINAL
  NODULE                                 Central deviasionwided

   MASS
                                            COSTO-PHRENIC ANGEL
                                                  Freeoblitern
  CAVITARY



                                                         OTHER
INFILTIRATION
                                        Bone soft tissuediaphragm
Consolidation

Infection
 causes                    Non-infection causes



                        Broncho-
                                         WEGNER              Cardiac
Pneumonia   Lymphoma    alveolar   COP             Sarcoid
                                         disease             failure
                       carcinoma
Solitary Pulmonary Nodule(SPN)
                         Appearance
 Margin                  Calcification                cavitation

             Comparison with a
                      Size
             previous x-ray to >8mm
          <8mm
             Assess growth over
             time. Location
   Upperhillar zone                     Lowerbasesup-pleural


                 Associated abnormalities
Lymph node enlargement                   Rib destruction/erosion
Cavitary lesion
 Air +
               Air-fluid level                          Air only
tissue
                                                   Wall thickness

              Straight     Wavy               Thick                        Thin
         1.   Fungal ball.
         2.   Rupture hydatid cyct                                           site
         3.   Necrotic tumor
                           ruptured
         4.   Blood glot Hydatid
              Abscess                  Irregular    Regular
                                                                Peripheral          Central
                                      inner wall   inner wall
                            cyst


                                                                Emphesemato
                                      Cavitating   Chronic          us          pneumatoc
                                      neoplasm     abscess                         ele
                                                                   bulla
LINEAR PATTERN
1) Linear (reticular) abnormality is due to
pathology involving:
     •   airways,
     •   lymphatics,
     •   veins,
     •   interstitium of the lung.
2) Volume loss is a key finding in fibrosis.
LINEAR PATTERN
                LINEAR PATTERN
LEFT VENTRICULAR FAILURE Perihilar and peripheral basal septal lines,
                         changes acutely and resolves with diuretics



Normal ageing             Coarsening of lung markings in lower zones, no
                          change on review of recent films



Lymphangitis              Coarse nodular and linear thickening of
                          markings, known malignancy, often associated
                          with pleural effusion, rapid clinical
                          deterioration of patient
LINEAR PATTERN
               LINEAR PATTERN
Atelectasis       Short thin lines, often basal, new on review of
                  previous films



Subsegmental      Longer thicker bands, often perihilar or basal,
collapse          suggest recent infection or infarction



Scarring          Any length, persist over time unchanged

Fibrosis          Volume loss is key, persists over time
Causes of fibrosis
  Mid zone lung              Lower zone lung Upper zone lung
tuberculosis                 Drug indused fibrosis        sarcoidosis
                             (most common)

Chronic extrinsic allergic   UIP
alveolitis

Radio-therapy                Asbestose-related fibrosis


Ankylosing spondylitis


Progressive massive
fibrosis

histoplasmosis
Case-1
• A 49-year-old white woman presents with progressive
  cough and dyspnea.
• She denies any history of arthritis, skin lesions, or eye
  complaints.
• On physical examination, vital signs are:
           –    pulse 90 bpm;
           –   temperature 98°F;
           –   respirations 32/min;
           –    blood pressure 119/76 mm Hg.
• General exam: patient is in moderate distress, and
  pertinent physical findings reveal clubbing of the fingers
  and bilateral “Velcro” rales on lung auscultation.
• ABGs on room air: pH 7.47; PCO2 32 mm Hg; PO2 60 mm
  Hg with further de-saturation on mild exertion
Case-1
POSITION            •PA CXR

QUALITY             •Good Technical Quality


                     •Bilateral reticular infitration
                     •At lower zone and left mid zone
LESION


                    •Central trachea and mediasteinal.
MEDIASTINALHilum

ANGELS              •Hazy left angle .

                    •No
OTHER
Case-1
1-Least likely to be associated with this condition is
a. Positive antinuclear antigen
b. Positive rheumatoid factor
c. Increased erythrocyte sedimentation rate
d. Increased IgE
2- What is the most likely diagnosis?
a. Idiopathic pulmonary fibrosis
b. Langerhans granulomatosis/histiocytosis-X disorders
c. Rheumatoid lung
d. Sarcoidosis
3- PFTS would be expected to show
a. An obstructive pattern
b. A restrictive pattern
c. A normal pattern
d. A reversible obstructive pattern
Case-2
• A 65-year-old woman from Honduras complains of arthralgias
  and difficulty getting out of a chair and doing her daily chores
  at home.
• She has muscle aches and generalized
  weakness, dyspnea, and cough.
• On physical
• examination, vital signs are:
           –   pulse 98 bpm;
           –   temperature normal;
           –   Respirations 23/min
           –   bilateral crackles on lung exam.
• Neuro exam reveals proximal muscular weakness with no
  sensory deficit.
• CPK and aldolase are increased:
• sedimentation rate is 120 mm/min.
• PFT: restrictive pattern.
Case-2
POSITION            •PA CXR

QUALITY             •Poor Technical Quality


                     •Bilateral reticular infitration
                     •Diffuse bilateral lung especially left
LESION               lower zone.




                    •Central trachea and mediasteinal.
MEDIASTINALHilum

ANGELS              •Free .

                    •Right hemi-diaphragm elevated
OTHER
Case-2
1. What is the most likely diagnosis?
a. Paraneoplastic syndrome
b. Polymyositis
c. Sjgren syndrome
d. Scleroderma
2. There is an increased association of one
of the following with this condition
a. Carcinoma of the pancreas
b. Diabetes mellitus
c. Diabetes insipidus
d. Alzheimer’s disease
Case-3
• A 48-year-old female nurse is seen with
  complaints of cough.
• She has been treated for “bronchitis”
  without much improvement.
• On exam, she is afebrile and has crackles
  in the upper zones of the lung field.
• PPD is negative and sputum for AFB is
  negative.
Case-3
POSITION            •PA CXR


QUALITY             •Good Technical Quality

                     •Bilateral reticular infitration
LESION               •Diffuse bilateral lung especially
                     middle,upper zone.


                    •Central trachea and mediasteinal.
MEDIASTINALHilum   •Bilateral hilar enlargementparathraceal



ANGELS              •Disappear.

                    •No
OTHER
Case-3
• 1. The most likely diagnosis is:
•   a. Tuberculosis
•   b. Blastomycosis
•   c. Sarcoidosis
•   d. Silicosis
• 2. All of the following findings may be seen in
  this patient except
•   a. Uveitis
•   b. Skin lesion
•   c. Bony cysts
•   d. Hypocalcemia
Case-4
•   A 56-year-old black male non-smoker is seen with a history of dyspnea
•   on walking two blocks and chronic chest congestion and cough.
•   He has been followed for progressive shortness of breath after his CABG.
•   Recently, he was ill with a flulike illness, but he denies any fever or chills
    presently.
•   Past history: reveals a GI clinic follow-up for inflammatory bowel disease for
•   which he has been on chronic steroid therapy off and on.
•   On physical examination, vital signs are:
          • pulse 110 bpm;
          • temperature normal;
          • respirations24/min;
          • blood pressure 120/78 mm Hg.
•   General exam: patient appears frail but in no distress.
•   Pertinent findings:
          • coarse rhonchi and scattered expiratory wheeze with squeaks.
          • Heart exam reveals normal S1-S2 with no gallop.
          • There is no hepatomegaly or pedal edema.
Case-4
• Laboratory data:
      • Hb 11 g; Hct 33%;
      • WBCs 15.0/μL; differential normal.
• PFTs/spirometry:
      • FVC 3.43 L (78% of predicted);
      • FEV1: 2.15 L (63% of predicted);
      • FEV1/FVC% 72%;
      • TLC 5.34 L (69% of predicted);
      • DLCO 14 cc/min/mm Hg (57% of predicted).
• Echocardiogram shows an:
      • ejection fraction of 55%.
      • no focal dyskinesia.
Case-4
POSITION            •PA CXR

QUALITY             •Poor Technical Quality


                     •Bilateral reticular infitration
                     •Diffuse bilateral lung especially
LESION               peripherial lower right zone.




                    •Central trachea and mediasteinal.
MEDIASTINALHilum

ANGELS              •Free .

                    •No
OTHER
Case-4
• 1. What is the most likely diagnosis?
•   a. Congestive heart failure
•   b. COPD
•   c. Nonspecific pneumonitis
•   d. Bronchiolitis obliterans with organizing pneumonia
    (BOOP)
• 2. There may be an increased risk of one of the
  following during therapy in this patient:
•   a. Pulmonary embolism
•   b. Staphylococcal infection
•   c. Mycobacterial infection
•   d. HIV infection
Case-5

• A 50-year-old woman is admitted with progressive
  shortness of breath.
• She was well until about 2 mo ago, when she noted
  that she was getting tired and fatigued easily.
• She gives a history of working as a domestic worker
  and “cleaning lady” for many years.
• Recently, she was working for a company that did
  maintenance work on boats in a marina area.
• She now has cough, shortness of breath, and low-grade
  fever with malaise.
• This has continued despite symptomatic treatment.
Case-5
• On exam she is found to be in:
     • mild to moderate distress
     • with harsh vesicular breath sounds,
     • Diffuse rhonchi
     • bilateral basilar crackles on lung exam, more
       on the right.
• Routine labs are normal,
• PPD is 5 mm.
• sputum is negative for fungal.
• AFB smear with cultures pending.
Case-5
POSITION            •AP CXR

QUALITY             •Poor Technical Quality


                     •Bilateral reticular (linar) infitration
                     •bilateral lower lung zone.
LESION


                    •Central trachea and mediasteinal.
MEDIASTINALHilum

ANGELS              •Bilateral hazy angels .

                    •No
OTHER
Case-5
• 1. The most likely diagnosis is
•   a. Silicosis
•   b. Asbestosis
•   c. Extrinsic allergic alveolitis
•   d. Nontuberculous mycobacterial infection
• 2. Associated with this condition is
•   a. Increased lung volumes
•   b. Decreased diffusion
•   c. Peripheral eosinophilia
•   d. Inorganic dust exposure
How  read  chest xr  10

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How read chest xr 10

  • 1. HOW READ CHEST XR -10 ANAS SAHLE ,MD
  • 3. POSITION PA AP QUALITY ROTATION PENETRATION INSPIRATION LESION OPACIT OPACITY Homo Heterogenous Wellill defined Zone Centralperipher Silhouet sign al Y Necrotic PATCHY HILUMMEDIASTINAL NODULE Central deviasionwided MASS COSTO-PHRENIC ANGEL Freeoblitern CAVITARY OTHER INFILTIRATION Bone soft tissuediaphragm
  • 4. Consolidation Infection causes Non-infection causes Broncho- WEGNER Cardiac Pneumonia Lymphoma alveolar COP Sarcoid disease failure carcinoma
  • 5. Solitary Pulmonary Nodule(SPN) Appearance Margin Calcification cavitation Comparison with a Size previous x-ray to >8mm <8mm Assess growth over time. Location Upperhillar zone Lowerbasesup-pleural Associated abnormalities Lymph node enlargement Rib destruction/erosion
  • 6. Cavitary lesion Air + Air-fluid level Air only tissue Wall thickness Straight Wavy Thick Thin 1. Fungal ball. 2. Rupture hydatid cyct site 3. Necrotic tumor ruptured 4. Blood glot Hydatid Abscess Irregular Regular Peripheral Central inner wall inner wall cyst Emphesemato Cavitating Chronic us pneumatoc neoplasm abscess ele bulla
  • 7. LINEAR PATTERN 1) Linear (reticular) abnormality is due to pathology involving: • airways, • lymphatics, • veins, • interstitium of the lung. 2) Volume loss is a key finding in fibrosis.
  • 8. LINEAR PATTERN LINEAR PATTERN LEFT VENTRICULAR FAILURE Perihilar and peripheral basal septal lines, changes acutely and resolves with diuretics Normal ageing Coarsening of lung markings in lower zones, no change on review of recent films Lymphangitis Coarse nodular and linear thickening of markings, known malignancy, often associated with pleural effusion, rapid clinical deterioration of patient
  • 9. LINEAR PATTERN LINEAR PATTERN Atelectasis Short thin lines, often basal, new on review of previous films Subsegmental Longer thicker bands, often perihilar or basal, collapse suggest recent infection or infarction Scarring Any length, persist over time unchanged Fibrosis Volume loss is key, persists over time
  • 10. Causes of fibrosis Mid zone lung Lower zone lung Upper zone lung tuberculosis Drug indused fibrosis sarcoidosis (most common) Chronic extrinsic allergic UIP alveolitis Radio-therapy Asbestose-related fibrosis Ankylosing spondylitis Progressive massive fibrosis histoplasmosis
  • 11. Case-1 • A 49-year-old white woman presents with progressive cough and dyspnea. • She denies any history of arthritis, skin lesions, or eye complaints. • On physical examination, vital signs are: – pulse 90 bpm; – temperature 98°F; – respirations 32/min; – blood pressure 119/76 mm Hg. • General exam: patient is in moderate distress, and pertinent physical findings reveal clubbing of the fingers and bilateral “Velcro” rales on lung auscultation. • ABGs on room air: pH 7.47; PCO2 32 mm Hg; PO2 60 mm Hg with further de-saturation on mild exertion
  • 13. POSITION •PA CXR QUALITY •Good Technical Quality •Bilateral reticular infitration •At lower zone and left mid zone LESION •Central trachea and mediasteinal. MEDIASTINALHilum ANGELS •Hazy left angle . •No OTHER
  • 14. Case-1 1-Least likely to be associated with this condition is a. Positive antinuclear antigen b. Positive rheumatoid factor c. Increased erythrocyte sedimentation rate d. Increased IgE 2- What is the most likely diagnosis? a. Idiopathic pulmonary fibrosis b. Langerhans granulomatosis/histiocytosis-X disorders c. Rheumatoid lung d. Sarcoidosis 3- PFTS would be expected to show a. An obstructive pattern b. A restrictive pattern c. A normal pattern d. A reversible obstructive pattern
  • 15. Case-2 • A 65-year-old woman from Honduras complains of arthralgias and difficulty getting out of a chair and doing her daily chores at home. • She has muscle aches and generalized weakness, dyspnea, and cough. • On physical • examination, vital signs are: – pulse 98 bpm; – temperature normal; – Respirations 23/min – bilateral crackles on lung exam. • Neuro exam reveals proximal muscular weakness with no sensory deficit. • CPK and aldolase are increased: • sedimentation rate is 120 mm/min. • PFT: restrictive pattern.
  • 17. POSITION •PA CXR QUALITY •Poor Technical Quality •Bilateral reticular infitration •Diffuse bilateral lung especially left LESION lower zone. •Central trachea and mediasteinal. MEDIASTINALHilum ANGELS •Free . •Right hemi-diaphragm elevated OTHER
  • 18. Case-2 1. What is the most likely diagnosis? a. Paraneoplastic syndrome b. Polymyositis c. Sjgren syndrome d. Scleroderma 2. There is an increased association of one of the following with this condition a. Carcinoma of the pancreas b. Diabetes mellitus c. Diabetes insipidus d. Alzheimer’s disease
  • 19. Case-3 • A 48-year-old female nurse is seen with complaints of cough. • She has been treated for “bronchitis” without much improvement. • On exam, she is afebrile and has crackles in the upper zones of the lung field. • PPD is negative and sputum for AFB is negative.
  • 21. POSITION •PA CXR QUALITY •Good Technical Quality •Bilateral reticular infitration LESION •Diffuse bilateral lung especially middle,upper zone. •Central trachea and mediasteinal. MEDIASTINALHilum •Bilateral hilar enlargementparathraceal ANGELS •Disappear. •No OTHER
  • 22. Case-3 • 1. The most likely diagnosis is: • a. Tuberculosis • b. Blastomycosis • c. Sarcoidosis • d. Silicosis • 2. All of the following findings may be seen in this patient except • a. Uveitis • b. Skin lesion • c. Bony cysts • d. Hypocalcemia
  • 23. Case-4 • A 56-year-old black male non-smoker is seen with a history of dyspnea • on walking two blocks and chronic chest congestion and cough. • He has been followed for progressive shortness of breath after his CABG. • Recently, he was ill with a flulike illness, but he denies any fever or chills presently. • Past history: reveals a GI clinic follow-up for inflammatory bowel disease for • which he has been on chronic steroid therapy off and on. • On physical examination, vital signs are: • pulse 110 bpm; • temperature normal; • respirations24/min; • blood pressure 120/78 mm Hg. • General exam: patient appears frail but in no distress. • Pertinent findings: • coarse rhonchi and scattered expiratory wheeze with squeaks. • Heart exam reveals normal S1-S2 with no gallop. • There is no hepatomegaly or pedal edema.
  • 24. Case-4 • Laboratory data: • Hb 11 g; Hct 33%; • WBCs 15.0/ÎĽL; differential normal. • PFTs/spirometry: • FVC 3.43 L (78% of predicted); • FEV1: 2.15 L (63% of predicted); • FEV1/FVC% 72%; • TLC 5.34 L (69% of predicted); • DLCO 14 cc/min/mm Hg (57% of predicted). • Echocardiogram shows an: • ejection fraction of 55%. • no focal dyskinesia.
  • 26. POSITION •PA CXR QUALITY •Poor Technical Quality •Bilateral reticular infitration •Diffuse bilateral lung especially LESION peripherial lower right zone. •Central trachea and mediasteinal. MEDIASTINALHilum ANGELS •Free . •No OTHER
  • 27. Case-4 • 1. What is the most likely diagnosis? • a. Congestive heart failure • b. COPD • c. Nonspecific pneumonitis • d. Bronchiolitis obliterans with organizing pneumonia (BOOP) • 2. There may be an increased risk of one of the following during therapy in this patient: • a. Pulmonary embolism • b. Staphylococcal infection • c. Mycobacterial infection • d. HIV infection
  • 28. Case-5 • A 50-year-old woman is admitted with progressive shortness of breath. • She was well until about 2 mo ago, when she noted that she was getting tired and fatigued easily. • She gives a history of working as a domestic worker and “cleaning lady” for many years. • Recently, she was working for a company that did maintenance work on boats in a marina area. • She now has cough, shortness of breath, and low-grade fever with malaise. • This has continued despite symptomatic treatment.
  • 29. Case-5 • On exam she is found to be in: • mild to moderate distress • with harsh vesicular breath sounds, • Diffuse rhonchi • bilateral basilar crackles on lung exam, more on the right. • Routine labs are normal, • PPD is 5 mm. • sputum is negative for fungal. • AFB smear with cultures pending.
  • 31. POSITION •AP CXR QUALITY •Poor Technical Quality •Bilateral reticular (linar) infitration •bilateral lower lung zone. LESION •Central trachea and mediasteinal. MEDIASTINALHilum ANGELS •Bilateral hazy angels . •No OTHER
  • 32. Case-5 • 1. The most likely diagnosis is • a. Silicosis • b. Asbestosis • c. Extrinsic allergic alveolitis • d. Nontuberculous mycobacterial infection • 2. Associated with this condition is • a. Increased lung volumes • b. Decreased diffusion • c. Peripheral eosinophilia • d. Inorganic dust exposure