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BRAIN INJURIES

The brain may be injured:
• By direct intrusion, either by a foreign object
  such as a penetrating weapon, bullet
                )
• By deformation of the brain in closed head
  injuries                   )
                  (direct&indirect)
Mechanism of indirect brain damage




 • The mechanism is mainly:
 1- acceleration deceleration
 2- rotational movement shearing strain)
Coup & contrecoup injury
3-The pressure gradient theory.
TYPES OF BRAIN INJURIES

A- concussion:
B- brain contusion
C- brain laceration
D- brain edema
E- difuse axonal injury
A-Brain concussion

   It is a state of sudden transient loss
    of consciousness following head
    injuries. It is characterised by
    momentary physiological disruption of
    the function of the brain with little or
    no noticeable anatomical
    * Fate of concussion
    changes.(shaking of the brain)
    1- Complete uncomplicated
    recovery
    2- compression
    3- death
B- brain contusion
C- brain laceration
Both contusion and laceration can occur in
cases of coup or contre-coup lesion.
Occure in the outer layers .in the frontal &temporal lobes
D- cerebral edema

 - Increased brain size&weight
 - flattining of gyri&disappearance of sulci
 - pallor of the brain
  -Signs of herniation &shifting
 - difficulty in the differentiation between
  grey&white matter
 - slicing the brain show wet cut surface&fluid
  oozing
Brain edema
Practical notes


   There may be no coup damage at all, only
    contrecoup.
   May be no fracture of skull, even in the
    presence of severe coup and contrecoup
    lesions.
   The most common site for contrecoup injury is
    in the frontal lobes .It is often at the tips of the
    frontal poles and may be symmetrical, if a fall
    on the occiput has occurred.
Notes…..cont.

   In a temporal impact, the contrecoup damage
    may be not be on the contralateral
    hemisphere, but on the opposite side of the
    ipsilateral hemisphere from impact against the
    falx cerebri.
   The degree of contrecoup damage may be
    severe,more than coup , especially in the frontal
    lobes and tips of the temporal lobes.
Sequelae of Head Injuries:
   1- Retrograde amnesia:
       Loss of memory concerning the accident itself and for
    sometimes before the accident (partial amnesia).
   2- Post-traumatic automatism:
       The patient may do voluntary acts after the accident or
    even commits a crime. But he forgets every thing about what
    he did.
   3- Post traumatic neurosis:
       The patient may complain of some vague symptoms as
    headache, anxiety and insomnia. Sometimes all of these
    symptoms disappear after the victim receives the financial
    compensation.
   4- Pos traumatic epilepsy:
       Due to adhesion between the site of the trauma and the
    meninges.
   5- Sepsis: meningitis, brain abscess and sinus thrombosis.
Causes of death in head injuries:

1.   Rapid death: concussion, compression and
     brain laceration
2.   Delayed death:
     Infections: e.g. meningitis, brain abscess and
     sinus thrombosis.
    Indirectly from epilepsy
--          -
         1-           hyperflexion
         2-          hyperextension
            (whiplash injury)
         3-        compression
-                                -
-
     -
    -
    -


-
-
-
-
Cut throut


   -                -


    -                -

   -            -
-

    Haemothorax
    Pneumothorax
      Empyema
traumatic
     (Whiplash injuiry)
spontaneous
             Ruptured Acute MI -
     Ruptured cardiac aneurysme-
               Fatty degenaration
-
-
-
-
(Du,typhoid,TB   -   -
                 -   -
                 -   -

                 -   -
                 -   -
-
-
-
-
-
-

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Brain ,vert.,neck,chest&abdominal inj.

  • 1. BRAIN INJURIES The brain may be injured: • By direct intrusion, either by a foreign object such as a penetrating weapon, bullet ) • By deformation of the brain in closed head injuries ) (direct&indirect)
  • 2. Mechanism of indirect brain damage • The mechanism is mainly: 1- acceleration deceleration 2- rotational movement shearing strain)
  • 4.
  • 5.
  • 7.
  • 8.
  • 9.
  • 10. TYPES OF BRAIN INJURIES A- concussion: B- brain contusion C- brain laceration D- brain edema E- difuse axonal injury
  • 11. A-Brain concussion  It is a state of sudden transient loss of consciousness following head injuries. It is characterised by momentary physiological disruption of the function of the brain with little or no noticeable anatomical * Fate of concussion changes.(shaking of the brain) 1- Complete uncomplicated recovery 2- compression 3- death
  • 12. B- brain contusion C- brain laceration Both contusion and laceration can occur in cases of coup or contre-coup lesion. Occure in the outer layers .in the frontal &temporal lobes
  • 13. D- cerebral edema  - Increased brain size&weight  - flattining of gyri&disappearance of sulci  - pallor of the brain  -Signs of herniation &shifting  - difficulty in the differentiation between grey&white matter  - slicing the brain show wet cut surface&fluid oozing
  • 15. Practical notes  There may be no coup damage at all, only contrecoup.  May be no fracture of skull, even in the presence of severe coup and contrecoup lesions.  The most common site for contrecoup injury is in the frontal lobes .It is often at the tips of the frontal poles and may be symmetrical, if a fall on the occiput has occurred.
  • 16. Notes…..cont.  In a temporal impact, the contrecoup damage may be not be on the contralateral hemisphere, but on the opposite side of the ipsilateral hemisphere from impact against the falx cerebri.  The degree of contrecoup damage may be severe,more than coup , especially in the frontal lobes and tips of the temporal lobes.
  • 17. Sequelae of Head Injuries:  1- Retrograde amnesia:  Loss of memory concerning the accident itself and for sometimes before the accident (partial amnesia).  2- Post-traumatic automatism:  The patient may do voluntary acts after the accident or even commits a crime. But he forgets every thing about what he did.  3- Post traumatic neurosis:  The patient may complain of some vague symptoms as headache, anxiety and insomnia. Sometimes all of these symptoms disappear after the victim receives the financial compensation.  4- Pos traumatic epilepsy:  Due to adhesion between the site of the trauma and the meninges.  5- Sepsis: meningitis, brain abscess and sinus thrombosis.
  • 18. Causes of death in head injuries: 1. Rapid death: concussion, compression and brain laceration 2. Delayed death: Infections: e.g. meningitis, brain abscess and sinus thrombosis.  Indirectly from epilepsy
  • 19. -- - 1- hyperflexion 2- hyperextension (whiplash injury) 3- compression - -
  • 20. - - - - - - - -
  • 21.
  • 22. Cut throut  - - - -  - -
  • 23.
  • 24.
  • 25. - Haemothorax Pneumothorax Empyema
  • 26. traumatic (Whiplash injuiry) spontaneous Ruptured Acute MI - Ruptured cardiac aneurysme- Fatty degenaration
  • 28. (Du,typhoid,TB - - - - - - - - - -