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Depression (also called major depressive disorder or clinical depression) is a common but serious mood disorder. It causes severe symptoms that affect how we feel, think, and handle daily activities, such as sleeping, eating, or working. Depression is a common illness worldwide, with an estimated 3.8% of the population affected, including 5.0% among adults and 5.7% among adults older than 60 years. Approximately 280 million people in the world have depression
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CHAPTER SIX The Age of Anxiety The multiple perspectives we have been using in this book are particularly useful in understanding the impact anxiety has on U.S. society. The word “anxiety” comes from a Latin root meaning to “choke or throttle” connoting a troubled state of mind (Tone, 2009). Anxiety disorders are believed to be the most common mental health problem in the United States. Two common measures are lifetime morbid risk (the theoretical risk of getting a disorder at any point in life) and 12-month prevalence (the proportion of the population thought to suffer from the disorder in any 12-month period). Baxter et al. (2013) conducted a meta-analysis of 87 studies from 44 countries between 1980 and 2009. They found that anxiety disorders are common across the globe with an estimated current prevalence of approximately as much as 28% of the global population. The prevalence of anxiety disorders in the United States is estimated for lifetime morbid risk/12-month prevalence as follows: Specific Phobia 18.4%/12.1%, Social Anxiety Disorder 13%/7.4%, Post Traumatic Stress Disorder (PTSD) 10.1%/3.7%, Generalized Anxiety Disorder 9%/2%, Separation Anxiety Disorder 8.7% /1.2%, and Panic Disorder, 6.8%/2.4%, (Kessler, Petukhova, Sampson, Zaslovsky, & Wittchen, 2012). Although anxiety disorders are prominent, it is important to realize that their incidence has remained steady over several decades despite pharmaceutically funded efforts to make the public think there is an epidemic that needs medicated (Baxter et al., 2014). Although psychotropic medications are available for anxiety disorders, many psychological treatments also have excellent track records. Remember, from an integrative perspective it is not enough to describe anxiety symptoms, posit a biological explanation, then describe how certain drugs act biologically to (at least temporarily) decrease or eliminate these symptoms. With sentient beings, we have to look to the psychological, cultural and social variables that contribute to anxiety. We recall a client (Elijah) who lived in what could be described as a “toxic environment.” Elijah's urban residence was the regular scene of violence, and he himself had witnessed two shootings in his 23 years. He was court-ordered to receive treatment for an alcohol-related charge (drunk and disorderly conduct). Even after abstaining from all drugs for 60 days, Elijah was what could only be described as “a nervous wreck.” He showed symptoms of both Panic Disorder and PTSD (the latter related to stimuli associated with the shootings he had witnessed). In consultation with a psychiatrist, who prescribed SSRI medication, Elijah asked why he had his symptoms, and the doctor replied, “Some people have a genetic predisposition to such things.” As Charlie Brown would say, “Good grief!” In this client's case, genetic predisposition not withstanding, there were clearly psychological, cultural, and social contributors to his anxiety. His alcohol use was a .
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In this lecture on circulatory shock, Dr. Faiza, an Assistant Professor of Physiology, delves into the profound implications of inadequate blood flow throughout the body, leading to tissue damage. The session begins by defining circulatory shock and elucidating its physiological causes, including decreased cardiac output, diminished blood volume, decreased vascular tone, obstruction to blood flow, excess metabolic rate, and abnormal perfusion patterns. Dr. Faiza categorizes shock into various types, such as cardiogenic shock, hypovolemic shock, neurogenic shock, obstructive shock, anaphylactic shock, and septic shock, and explores the pathophysiological basis of each. Furthermore, the lecture examines the stages of shock, from the non-progressive phase where compensatory mechanisms aim for full recovery to the progressive phase where shock worsens steadily without intervention, potentially leading to irreversible damage. Dr. Faiza discusses therapeutic interventions for shock, including replacement therapy, blood and plasma transfusion, sympathomimetic drugs, and other adjunctive treatments like head-down positioning, oxygen therapy, and glucocorticoids.
Circulatory Shock, types and stages, compensatory mechanisms
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MedicoseAcademics
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Dr. Faiza delivers an insightful lecture on the distinguishing characteristics of skeletal, smooth, and cardiac muscles, offering a comprehensive understanding of their histology, physiological anatomy, electrophysiological properties, and contractile mechanisms. Through meticulous tabulation and comparison, she aims to equip learners with a detailed comprehension of the unique features and functionalities of each muscle type. Beginning with a detailed exploration of histological differences, Dr. Faiza outlines key distinctions such as multinucleation in skeletal muscle, mononucleation in smooth and cardiac muscles, and the presence of striations in skeletal and cardiac muscles. She meticulously elucidates the structural arrangement of each muscle type, highlighting their cellular morphology and organization within tissues. Transitioning to physiological anatomy, Dr. Faiza navigates through various aspects including innervation, level of control, initiation of contraction, and modification by hormones. By delineating the role of the somatic nervous system in skeletal muscle control, and contrasting it with the autonomic nervous system's influence on smooth and cardiac muscles, she offers a nuanced understanding of neural regulation in muscle physiology. Furthermore, Dr. Faiza delves into electrophysiological properties, elucidating the mechanisms underlying action potential generation, duration, and ionic basis in each muscle type. Through insightful analysis, she reveals how differences in action potential characteristics contribute to variations in muscle contraction speed, duration, and resistance to fatigue. Finally, Dr. Faiza meticulously examines the contractile mechanisms of skeletal, smooth, and cardiac muscles, shedding light on the sliding filament theory, mechanisms of calcium action, and speed of contraction. By comprehensively comparing these mechanisms, she provides learners with a deep understanding of the intricate processes governing muscle contraction and function. Through engaging presentation and meticulous analysis, Dr. Faiza's lecture serves as a valuable resource for students seeking a thorough understanding of the fundamental differences between skeletal, smooth, and cardiac muscles.
Difference Between Skeletal Smooth and Cardiac Muscles
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MedicoseAcademics
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Circulatory Shock, types and stages, compensatory mechanisms
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